little to no movement, connected by fibrous tissue or cartilage
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4 signs suggesting acute inflammation in a joint
redness, joint swelling, pain, stiffness
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dislocation
complete separation of 2 bones where they meet at a joint
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subluxation
a partial dislocation of one of the bones in a joint
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contracture
shortening and hardening of muscles, tendons, or other tissue, leading to deformed and rigidity of joints
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ankyloses
abnormal stiffening and immobility of a joint due to fusion of the boxes
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6 things to inspect on a joint
size, swelling, color, contour, masses, deformity
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what signals joint irritation?
swelling
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what should be specifically noted about palpation of joints and overlying skin?
heat, tenderness, swelling, or masses
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how to test active ROM
ask patient to model your own movements with you as the control
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how to test passive ROM
tested when a limitation or injury is noticed in active ROM
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what is the MOST sensitive sign of joint disease
limitation in ROM
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scoliosis
lateral curvature of thoracic and lumbar segments of the spine, usually with some rotation of the involved vertebral bodies.
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functional scoliosis findings
flexible, appears with standing and disappears with forward bending, may compensate for other abnormalities like leg length discrepancy
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structural scoliosis findings
fixed, curve shows when standing and bending forward. Rib hump with forward flexion, unequal shoulder elevation, unequal scapulae, obvious curvature, and unequal hip level
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2 techniques for examining carpal tunnel
phalen test and tinel sign
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phalen test
hold both hands back to back while flexing the wrists 90 degrees
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tinel sign
direct percussion of the location of the median nerve at the wrist
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rheumatoid arthritis
chronic inflammatory pain condition started by an autoimmune response, inflammaroty events, or infection. Inflammation of synovial joints, hyperplasia, and swelling lead to fibrosis, cartilage and bone destruction limiting motion and show deformity
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swan neck
flexion contracture curving like a swans neck-often associated w/ chronic rheumatoid arthritis
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boutonniere deformities
knuckle appears as if pushed through a buttonhole- associated w/ chronic rheumatoid arthritis
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osteoarthritis
noninflammatory, localized, progressive disorder involving deterioration of articular cartilages, subchondral bone remodeling, synovial inflammation and formation of new bone at joint surfaces
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bouchard's nodes
bony overgrowths of the PIP joints (less common) often associated with osteoarthritis
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herberden's nodes
bony overgrowths (osteophytes) of the DIP joints. commonly associated with osteoarthritis
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pallor
red pink tones from oxygenated hemoglobin in blood are lost skin takes on color of connective tissues- commonly pale/white
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erythema
intense redness of skin from excess blood in the dilated superficial capillaries
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cyanosis
bluish mottled color from decreased perfusion; tissues having high levesl of deoxygenated blood
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jaundice
yellowish skin color due to increased bilirubin in blood
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causes of pallor
acute high stress states (anxiety/fear), cold exposure, edema, cigarette smoking
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causes of erythema
fever, local inflammation, emotional reactions (blush)
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cause of cyanosis
decreased oxygenation, changes in LOC, signs of respiratory distress
Asymmetry: not regularly round or oval, 2 halves of lesion don't look the same Border Irregularity: notching, scalloping, ragged edges, poorly defined margins Color variation: areas of brown, tan, black, blue, red, white or combo Diameter: greater than 6mm (size of pencil eraser) Elevation/Evolution: changes to the lesion (size)
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Pressure Ulcer stage 1
non-blanchable erythema; intact skin, red but unbroken. Localized redness, doesn't blanch. Changes in temp or firmness
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Pressure Ulcer stage 2
partial thickness, skin loss, loss of epidermis, and exposed dermis. Superficial ulcer looks shallow like an abrasion or blister. Red pink wound bed, no visible fat/deeper tissue
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Pressure Ulcer Stage 3
full thickness skin loss; injury extends to sub-q tissue, resembles crater. Visible sub-q fat, granulation tissues, rolled edges. No visible muscle, bone, tendon
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Pressure Ulcer Stage 4
Full thickness skin/tissue loss; injury to all skin layers, extends to supporting tissue, exposes muscle, bone, tendon and may show slough and eschar, rolled edges, and tunneling
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tinea capitus
rounded, patchy hair loss, broken-off hairs, pustules, scales on skin
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pediculosis capitus (head lice)
intense itching in occiput. Nits seen in occipital area and around ears 2-3 mm oval translucent bodies adherent to hair shafts
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loss of skin turgor
less elasticity due to loss of collagen as aging increases
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clubbing of nails
clubbing can occur due to chronic lung inflammation, lung cancers, heart defects with right-to-left causing release of growth factors and promotion of growth of vessels
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vesicle
elevated cavity w/ free fluid; up to 1 cm a "blister: clear serum flows if wall ruptured ex. herpes simplex
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wheal
superficial, raised, transient and erythematous; slightly irregular shape from edema ex. mosquito bite
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annular
begins in center and spreads to periphery ex. ringworm
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discrete
distinct individual lesion that remain separate ex. skin tags
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grouped
clusters of lesions ex. contact dermatitis
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macule
solely a color change, flat and circumscribed of less than 1 cm ex. freckles
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cerebral cortex
center for governing thought, memory, reasoning, sensation and voluntary movement
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frontal lobe
personality, behavior emotions, and intellectual function. Precentral gyrus initiates voluntary movement
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parietal lobe
postcentral gyrus in the parietal lobe is the primary center for sensation
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temporal lobe
primary auditory reception center. Functions of hearing, taste, and smell
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occipital lobe
primary visual receptor center
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wernicke's area
in temporal lobe- language comprehension. If damaged the person hears speaking/sound but has attached (receptive aphasia)
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broca's area
located in frontal lobe- mediates motor speech. When injured, the person can't speak. They understand want to say but only garbled speech results
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cerebellum
motor coordination of voluntary movements, equilibrium (body balance), and muscle tone. Not in charge of movement itself, but the coordination and smoothness of it. Operates below the conscious level
coordinates movement, maintains posture and equilibrium
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corticospinal crossed/uncrossed
mediates voluntary movement, purposeful movements
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extrapyramidal tracts
all motor nerve fibers originating in motor cortex, basal ganglia, brain stem, and spinal cord outside the pyramidal tract. maintains muscle tone, and body movements (gross automatic movements)
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upper motor neurons
descending motor fibers, can influence or modify lower motor neurons. Located in CNS. Convey impulses from motor areas of cerebral cortex to lower motor neurons in anterior horn cells of spinal cord
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lower motor neurons
located mainly in PNS. Final common pathway, funnels neural signals and provides final direct contact with muscles. Any movement is translated to action by lower motor neuron (LMN) fibers
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upper motor neuron disease examples
stroke, cerebral palsy, multiple sclerosis
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lower motor neuron disease example
bell palsy in face, spinal cord lesions, polio myelitis
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Cerebellar function tests that assess coordination
rapid alternating movements (RAM), heel-to-shin test, finger-nose-finger test
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Rapid alternating movements (RAM)
ask person to pat knees with both hands, lift up, turn hands over, and pat knees with the backs of hangs. Then ask client to repeat faster. OR ask patient to touch thumb to each finger and then reverse the motion
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Heel-to-Shin test
test lower-extremity coordination by asking the person, who is in a supine position, place heel to the opposite knee and run it down shin from knee to ankle. Normally heel moves in a straight line down skin
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Finger-Nose-Finger test
ask patient to touch index finger to touch finger to nose. After a few times move finger to a different spot. Movement should be smooth and accurate
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3 cerebellar function tests that assess balance
- gait: observe as the person walks 10-20 feet, turns and returns to starting point. The patient can also be asked to walk in a straight line heel-to-toe - the Romberg test: ask person to stand with feet together and arms at sides. Then ask to close their eyes and hold the position for 20 seconds - Ask person to perform shallow knee bend on one leg and then the other. May use one hand supported on table
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Method of testing for pain
tested by ability to feel a pinprick. Apply a broken tongue blade lightly to skin alternating "sharp" and "dull" and ask the patient to identify the sensation
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method for testing touch
apply a wisp of cotton to skin and brush over skin in random order of sites and at irregular intervals. Have the patient identify when the cotton contracts the skin
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method for testing vibration
place a tuning fork over bony prominences. Use a low pitch fork due to the vibrations slower decay. Ask patient to identify when vibration starts and stops
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method for testing position
test ability to perceive passive movements by moving a finger or big toe up and down and have patient identify which way it moved. The test is performed with patient's eyes closed. Vary the movement of up/down. Digit should be held by the sides
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stereognosis
place a familiar object (key, cotton ball) in patients hand and ask them to identify it
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graphesthesia
have the patient "read" a number when it is traced on skin. Trace a single digit on palm of hand with blunt instrument have them identify it
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extinction
simultaneously touch both sides of the body at the same point, ask person to identify how many sensation are felt and where
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point location
touch skin and withdraw stimulus promptly. Ask patient to put finger where you touched
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Components of the neurologic recheck exam performed routinely on hospitalized persons monitored for neurologic deficit
level of consciousness, motor functions, pupillary response, vital signs
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3 areas of assessment on the Glasgow Coma Scale
eye opening response, motor response, verbal response
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spastic hemiparesis
arm immobile against body, flexion of the shoulder, elbow, wrist, and fingers and adduction does not swing freely. Leg stiff and extended and circumducts with each step. Causes: UMN lesion of corticospinal tract. ex. stroke, trauma
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cerebellar ataxia
staggering, wide-based gait; difficult w/ turns; uncoordinated movement w/ and Romberg sign. Causes: alcohol or barbiturate effect on cerebellum; cerebellar tumor; multiple sclerosis
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parkinsonian (festinating)
stooped posture: trunk is pitched forward; elbows, hips, and knees flexed. steps short and shuffling. Hesitation to begin walking and difficult and stop suddenly. Person holds body rigid. Walks and turns body as one fixed unit. Difficult w/ any change in direction. Causes: parkinsonism
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steppage
slapping quality-looks like walking up stairs and finding no stair there. Lifts knee and foot high and slaps down hard and flat to compensate for foot drop. Causes: weakness of peroneal and anterior tibial muscles; caused LMN lesion at spinal cord. ex. poliomyelitis
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Direct light reflex with a consensual light reflex
when one eye is exposed to bright light, a direct light reflex (constriction of that pupil) consensual light reflex (simultaneous constrictions of other pupil) occurs
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why do you advance a light in from the side when testing the pupillary light relex
if you advance from front, the pupils constrict to accommodate for near vision, thus you don't know what the pure response to the light would have been
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cataracts
clouding of the crystalline lens partly due to ultraviolet radiation
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age related macular degeneration
loss of central vision caused by yellow deposits (drusen) and neovascularity in the macula
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glaucoma
optic nerve neuropathy characterized by loss of peripheral vision caused by increased intraocular pressure
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visual impairment
not being able to see letters on the eye chart at the line 20/50 or below.
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what kind of impact does VI and blindness have on physical and mental health
increases the risk for lost productivity, chronic health conditions, accidents and injuries, social isolation, depression, and mortality
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how could VI be improved or corrected
improved through glasses, contact lenses, and refractive surgery
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how to test for accommodation
ask person to focus on a distant object, to allow pupils to dilate. Have person shift gaze to a near object (finger) 7-8 cm from patient's nose. Normal response will show pupillary constriction and convergence of axes of the eyes
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how do you perform the cover test on children, Normal response? Abnormal response?
have child stare straight ahead at your nose or at familiar puppet. Cover one eye w/ opaque card. Look at uncovered eye: normal response should be a steady, fixed gaze. If muscle weakness exists, the covered eye drifts into a relaxed position. Then uncover other eye and observe. Should stare straight ahead. If it jumps to re-establish fixation, eye msucle weakness exists. Repeat w/ other eye
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Phoria
mild weakness noted only when fusion is blocker
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Tropia
more severe than phoria. constant malalignment of the eyes
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conjunctivitis
"pink eye" red, beefy-looking vessels at periphery but usually clearer around iris, commonly from viral/bacterial infection, allergy, or chemical irritation. Purulent discharge accompanies bacterial infection. Symptoms include itching, burning, foreign body sensation and eyelids stuck together on awakening
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subconjunctival hemorrhage
red patch on sclera, subconjunctival hemorrhage- usually not serious. Red patch sharp edges, occurs from increased intraocular pressure from coughing, vomiting, weight lifting, labor during childbirth, straining at stool/trauma
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hyphema
blood in the anterior chamber is a serious result of herpes zoster infection. Also occurs w/ blunt trauma or spontaneous hemorrhage. Suspect school rupture or major intraocular trauma. Gravity settles blood in front of iris.
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blepharitis (inflammation of the eyelids)
red, scaly, greasy flakes and thickened, crusted lid margin occur with staphylococcal infection or seborrheic dermatitis of the lid edge. Sx: burning, itching, tearing, foreign body sensation and some pain