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Heart failure (HF) (4)
Impaired cardiac pumping or filling
When the heart cannot pump enough blood to the body
Abrupt onset or slow, progressive
abrupt onset usually associated with MI
Heart failure is associated with: (3)
CAD
HTN
MI
Heart failure risk factors: (4)
DM
Smoking
Obesity
Increased cholesterol
Heart failure cause (5)
Caused by interference with normal regulation of cardiac output (CO)
Preload (volume of blood in ventricles before contraction)
Afterload (resistance in which the ventricle pumps against)
Myocardial contractility (ability of heart to contract)
Heart rate
Ejection fraction (2)
Heart’s ability to pump out blood to your body
amount of blood pumped out over amount of blood in the chamber = percentage pumped out is the ejection fraction
amount of blood pumped out/amount of blood in the chamber = %
Types of heart failure
Left-sided and right-sided
Most common type of heart failure
Left-sided initially, then as the left side becomes dysfunctional then bilateral HF
Left-sided heart failure
Left-ventricular dysfunction
Blood backs up through left atrium
Pulmonary congestion and edema
Right-sided heart failure
Backward blood flow to right atrium and venous circulation
Heart failure clinical manifestations (10)
Pulmonary edema
Fatigue
Dyspnea
Tachycardia
Edema
Nocturia
Skin changes
Behavioral changes
Chest pain (decreased perfusion)
Weight changes
Dyspnea in HF
Paroxysmal nocturnal dyspnea
When patient asleep
Reabsorption of fluid when patient is flat (want to elevate HOB)
Feelings of suffocation – wake in a panic
Edema in HF
Sudden weight gain = exacerbated HF
Nocturia in HF
Renal blood flow increases when laying down
Skin changes in HF (2)
Dusky color
Lower extremities swollen & shiny
Behavioral changes in HF
Decreased oxygen and perfusion
Restlessness, confusion
Weight changes in HF
Fluid retention, so looks larger but
Muscles are wasting so decreased muscle mass
Pulmonary edema (4)
Life-threatening medical emergency
Fluid in alveoli & interstitial spaces of lungs
Interferes with gas exchange
Most common cause – left-sided HF
Pulmonary edema S+S
↑RR, ↓PaO2, ↑HR
SOB (accessory muscle use)
Anxious, pale, cyanotic
Orthopnea
Wheezing, coughing
Crackles, wheezes, rhonchi throughout lungs
Blood-tinged sputum
Orthopnea
Shortness of breath when laying down, relieved by sitting or standing up
Rhonchi
Low pitched, snoring, gurgling sounds heard in the large airways during breathing, caused by mucus, secretions, fluid.
If you see blood-tinged sputum automatically think:
Pulmonary edema
How to treat pulmonary edema
Treat underlying cause
oxygen, diuretics, BP meds
Complications of heart failure (5)
Pleural effusion
Dysrhythmias
Left ventricular thrombus
Hepatomegaly
Renal failure
Pleural effusion (6)
Fluid in pleural sac around lungs, restricts lungs from expanding all the way
SOB, decreased breath sounds
Trachea can deviate to opposite side of fluid
Tx:
Thoracentesis (needle inserted and fluid is withdrawn)
Antibiotics if infection
Dysrhythmias and HF
Heart not working properly, working harder, getting bigger and thicker. Can disrupt electrical function
Left ventricular thrombus and HF
Decreased cardiac output
Increased risk of pooling
Hepatomegaly and HF
Fluid backs up into systemic circulation causing other organs (of a lower pressure) to enlarge, especially the liver
Renal failure and HF
Decreased cardiac output
Decreased perfusion to kidneys
Causing kidneys to not work properly, can ultimately fail
Heart failure classification
Class 1: no limitation of physical activity (early)
Class 2: slight limitation of physical activity
Class 3: marked limitation of physical activity
usually comfortable at rest
Class 4: inability to carry on any physical activity without discomfort (advanced)
S+S at rest
Heart failure diagnostic studies (4)
Echocardiography (ultrasound)
BNP
Chest X-ray (see enlargement of heart)
ECG (can support dx)
BNP
B-type natriuretic peptide
When there’s increased volume in the heart, there’s increased pressure, BNP is released
If pt is SOB should draw for BNP
If increased we look at the heart
Nursing management: Acute decompensated heart failure (12)
↓ Intravascular volume
Diuretics (IV then transitioned to oral)
↓ Venous return
High Fowler’s position (legs down low or sitting over side of bed)
Diuretics (both help with lung expansion)
↓ Afterload
Managing BP (so heart doesn’t have to pump against such increased resistance)
↑ Gas exchange
IV morphine (relax pt, decreases anxiety)
Oxygen (pulse oximetry)
↑Cardiac function
↓ Anxiety (morphine, Ativan)
Collaborative care: chronic heart failure (10)
Multidisciplinary clinic
O2 if <90%
Self management: recognize S+S
Exercise & activity
30-45 mins, 3-5x week
cardiac rehab program
Devices
Cardiac transplantation
Medication therapy
Nutritional therapy
Chronic heart failure devices
Pacemaker
Implantable cardioverter-defibrillator (ICD)
Ventricular assist device (VAD)
Pacemaker (3)
Help regulate the heart beat with small evenly timed shocks
Involves implanting electrodes into one or more of the hearts chambers
Implanted generator
Implantable cardioverter-defibrillator (ICD) (2)
Implanted defibrillator
Capable of sensing a stopped heart and delivering an electric shock powerful enough to restart it
Chronic heart failure: medication therapy (6)
Diuretics (Lasix)
ACE Inhibitors (“prils”)
Combination medication
ARB (Valsartan) + Neprilysin (Sacubitril)
Beta blockers (“olol”)
Mineralocorticoid receptor antagonists (Spironolactone)
Inotropic drugs (dobutamine, milrinone)
Chronic heart failure: nutritional therapy (3)
Sodium restriction (DASH diet)
1.5 - 2 L fluid/day
Daily weight
Want weight to stay the same or decrease, an increase means something isn’t working
Nursing management: chronic heart failure (11)
History & Physical
Health Promotion (Treat & control underlying heart disease)
O2 (nasal prongs when eating)
Positioning (high fowlers)
Intake/Output (s+s peripheral edema)
Education
ECG
Administer medications & monitor lab values
Nutrition
Encourage rest/activity
Support