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consciousness
awareness of yourself & your environment
encompasses thoughts, feelings, & perceptions, allowing for subjective experiences
RAS filters out diff stuff for diff ppl
altered state of consciousness
your focus is split, or your quality of cognition declines
preconscious
smth I’m not currently aware of, but could be if I wanted to
ex. walking into biopsych test, not specifically thinking abt medulla, but if I get Q on respiration, then ik medulla
nonconscious
automatic ns (things we do w/o thinking)
subconscious
I’m not fully aware of my motives, but those feelings still influence my behaviors
unconscious
inaccessible to us
no way to prove/disprove this; the one stage of consciousness that not everyone agrees w/
biological rhythm
physiological functions
ex. mensuration, puberty
can’t be adjusted
circadian rhythm
24-hr cycle
ex. sleep
can be adjusted (in 14 days)
why do we sleep?
health of body
restores us, ex. immune system, cellular damage, etc
replenishes chemicals used throughout day
integrates today’s experiences into memory
throws out unnecessary memories
helps us deal w/ stress, anxiety, & emotions
regulates eating
less sleep = obesity
stream of consciousness waves (from most active to least active)
gamma
beta
alpha
theta
delta
hypnogogic sensation
when you feel like you’re falling so body jerks
happens during NREM 1
how long is sleep cycle?
90 mins
should be going thru cycle 5 times every night
order of sleep cycle
NREM1 → NREM2 → NREM3 → NREM2 → NREM1 → REM = one 90 min sleep cycle
cycle begins more heavily in NREM, but as you cont., more time spent in REM
NREM1
lightest stage of sleep
starting to emit theta waves
NREM2
sleep spindles occur
respiration slows down a little more
sleep talking/walking occurs
NREM3
slow wave/restorative sleep
deep sleep
taking out trash in brain
deciding which memories to keep/discard
REM
rapid eye mvmt
indicates you’re dreaming
body enters sleep paralysis, but brain is highly active, esp in cortex
“paradoxical sleep”
respiration rates inc
REM rebound
inc amt of time spent in REM if I’m habitually depriving it of its normal amt
activation synthesis
dream theory from biological POV
synthesizing active neurons
dreams = brain trying to make sense of random neural activity
information processing
dream theory from biological & psychological POV
dreams help you get info into memory system & process emotions of day
Freud’s dream theory
1st person who says dreams can mean smth
dreams = “royal road to unconscious”
manifest (storyline you can account for) v.s. latent (hidden symbols, interpreted version of dreams) content
somnambulism
genetic
sleep walking disorder
affects NREM sleep
more common in men than women
starts in childhood, sometimes fixes itself w/ puberty
sleep apnea
as you’re laying down, throat closes → stop breathing → brain forces you to rise out of deep sleep to snort up air
narcolepsy
bout of uncontrollable sleep (short amt of time)
cataplexy = sudden loss of muscle tone so body goes limp
almost immediately fall into REM
treatment = scheduled naps
genetic
night terrors
affects NREM sleep
indiv looks disturbed; can get up, clench teeth, make noises, etc
more common in boys than girls
starts in childhood, sometimes fixes itself w/ puberty
insomnia
most common sleep disorder
two types:
problem falling sleep (more common)
problem staying asleep
stimulants
stimulates ns (CNS & sympathetic NS)
dilates pupils, inc HR, etc
ex. cocaine, nicotine, caffeine
can cause paranoia & insomnia
depressants
reduce/slow neural activity & body functions
ex. alc (most common)
hallucinogens
distort perception & evoke sensory images w/o actual sensory input
ex. LSD, shrooms
No. 1 hallucination = seeing smth
but for schizo, it’s hearing smth
opiates
NS slowed AND indiv feels sense of pleasure
ex. opium, morphine, heroin
psychoactive drugs
chemicals that change perceptions & moods
agonist
substance that initiatives physiological response when combined w/ receptor
tolerance
diminished response to drug
antagonist
drug that blocks certain neurotransmitter from activating its receptors
withdrawal
symptoms that occur when indiv ends use of addictive substance
sensation
detecting smth from environment
biological process = same for everybody
perception
subjective to everyone
diff due to…
prev experiences
what you’re paying attention to/interpreting
transduction
us converting sensed energy into neural impulse
bottom-up processing
going from senses to brain
top-down processing
from brain to senses
when brain is trying to interpret smth in absence of stimuli
e.g. trying to recall someone’s voice when they’re not there
absolute threshold
smallest amt of stimuli you can detect reliably (50% of the time)
declines over time
sensory adaptation
occurs when you have constant unchanging stimuli, so you get used to it
happening at 2 diff lvls
sensory neurons fire less frequently
habituation
signal detection theory
psychological theory
trying to make sense of why absolute threshold isn’t constant (why only 50% reliable?)
reason = psychological state isn’t constant; can’t give full attention to everything @ same time
difference threshold/just noticeable difference (JND)
smallest amt of change in ongoing stimuli that you can detect reliably (50%)
selective attention
pay attention to some info, & exclude irrelevant stuff
diff types
change blindness
inattentional blindness
cocktail party phenomenon
change blindness
when we fail to notice change in visual stimuli bc attention focused elsewhere
inattentional blindness
when we fail to notice smth that’s fully visible b/c brain is focused elsewhere
cocktail party phenomenon
when you weren’t paying attention to someone’s convo until they say your name
JND & Weber’s Law
size of JND = proportional to strength of OG stimuli
e.g. changing temp of jacuzzi > changing temp og lukewarm water
if you start w/ smth strong → takes more change to notice diff
if you start w/ smth not as strong → takes less change to notice diff
can we sense smth below our absolute threshold?
yes, subliminally (below your threshold)
characteristics of waves
wave length
amplitude
complexity
vision & waves
stimulus = electromagnetic energy
wavelengths determine hue we see
amplitude determines how bright hue is
complexity = range of wavelengths, how saturated color is
sound & waves
stimulus = pressure waves (created when smth vibrates)
frequency = rate of vibration
high freq = small wavelength
freq = pitch of sound
measure pitch in Hertz
humans can only hear 20-20k Hertz
amplitude = amt of pressure of waves
high amp → high pressure → high volume (loud)
measured in decibels
tambre = tone saturation (combo of diff sounds)
dark adaptation
happens when we go from light environment to dark
light adaptation
happens when we go from dark environment to light
takes shorter time than dark adaptation
lens
responsible for inverting the world
can change its shape thru accommodation
pupil
opening to eye
iris
adjusts size of pupil
cornea
protective outer layer
curvature helps focus light rays inward towards eye so we can see things clearly
blind spot
black dot in vision that brain fills in for you
fovea
where we see greatest detail b/c highest density of cones
retina
where transduction takes place
covered w/ neurons known as photoreceptors
2 types
rods → black, white, grey
cones → color vision
trichromatic (Young/Helmholtz) theory
eye has 3-color sensitive cones: R, G, B
activating them in diff ways results in seeing other colors
color blindness
sex-linked/x-linked trait → more common in men
deficiency in cones
red-green
yellow-blue
black-white (rare)
opponent process theory
when one color is excited, the other is inhibited
ex. if I stare at smth blue, yellow is inhibited, & vice versa
pairs:
black white
red green
yellow blue
pina
funnels sound into ear
inner ear
where transduction takes place (in cochlear on vascular membrane)
includes cochlear & semicircular canals
cochlear
responsible for audition & transduction
semicircular canals
responsible for balance
middle ear
job = conduct vibrations
includes ear drum & 3 tiny bones (stapes, incus, malleus)
outer ear
pina & ear canal
conduction hearing loss
middle ear porblem, 2 possible scenarios
smth happens to eardrum (e.g. tear)
more rare; bones in mid ear start to degenerate → can’t conduct vibrations
sensory neural hearing loss
hair cells die
auditory nerve doesn’t work
place theory
brain perceives pitch based on where hair cells are moving
frequency theory
brain perceives pitch based on how often auditory nerve is firing
volley principle
grps of hair cells take turns sending action potentials
sensory interaction
experience of one sense influences experience of another sense (ex. smell & taste)
gustation (taste)
sense receptors = taste buds
universal love for sweet & distaste for bitter
bitter = back of tongue by throat
sour = side of tongue
salty = front of tongue
umami = whole tongue
sweet = front of tongue
olfaction (smell)
sense receptors = hair cells @ top of nasal cavities
only sense that bypasses thalamus & goes to olfactory bulb
how are gustation and olfaction similar?
both sense chemical molecules
only sense where sensor cells die & are regenerated
need smell for full taste experience
vestibular sense
sense of balance
transduction happens in canals
canals like cochlear filled w/ liquid + hair cells
reliant on info your brain gets abt body position & ground
ex. where head is compared to ground/how head is moving
ex. tilt head, hair cells move
kinesthesis
provides info to brain abt what body parts are doing
receptors in muscles, joints, etc in body
this is where transduction occurs
will send info to brain
also helps w/ posture
touch
stimulus = what experience we’re having
no one particular
diff layers of skin respond diff to sensations
pain is body’s way of letting us know smth is wrong
somatic pain = muscle/tendons in body; quick/sharp pain
visceral pain = internal organs; more consistent (constant ache)
gate control theory
can’t prove it
“gate” in spinal cord that can be opened allowing msgs to go up to bran/remain closed and msg doesn’t go up
“disease”/amputation makes door remain open, causing msg to go up
mirrors used to treat phantom limb
see that it’s fine, gates will close
monocular cues
there are way more of these than binocular cues
illusions of depth
interposition
when one object partially blocks view of another, the one that’s blocked is perceived as being further away
figure-ground relationship
figure is separate from bckgrd
relative size
assuming two objs are abt same size, the one that casts the smaller retinal image is perceived as being further away
proximity (Gestalt grouping principle)
b/c things are close to one another, they’re percieved as unit/having a relationship
retinal clarity/atmospheric perspective
hazy/blurry objects are perceived as being further away from us
similarity (Gestalt grouping principle
we grp things tg that are similar
common fate (Gestalt grouping principle)
when ppl/animals travelling in same direction, we percieve them as grp
closure (Gestalt grouping principle)
we see things as complete objects, even when they aren’t; filling in gaps, seeing things the way we think they should be even if it’s not actually like that
continuity
we see uninterrupted patterns over smth broken up
law of pragnanz
when brain sees ambiguous visual stimuli, we see it in simplest way possible
texture gradient
objects closer to us, we can see in greater detail + we can also see the spacing in btwn the objects
depth perception isn’t developed until…
the time we learn to crawl
binocular cues
helps w/ depth perception
retinal disparity: brain perceives depth thru 2 diff images it receives (one from left eye, one from right eye); the greater the disparity, the closer the object
convergence: kinesthesis for your eyes; part of how brain judges depth is based on mvmt of eye muscles more inward; the more eye converges, the closer the object
light & shadow
nearby objects are going to be brighter
linear perspective
when parallel lines are converging, appears more in depth