Label a diagram of a neuron
Dendrite- Allows transfer of info
Cell body- Provides structure
Nucleus- Cells control center
Axon- Main component of nerve signal transmission
Motor end plate- Where the neuron is unmyelinated and joins with a muscle
Synapse- The gap between the muscle and the MEP
Muscle- Receives the message
Acetylcholines role in neuromuscular junctions
A small molecule that acts a chemical messenger that triggers the firing of motor neurons and affecting voluntary movements (contracts)
Cholinesterase role in neuromuscular junctions
Breaks down the acetylcholine (relaxes)
SFT steps
action potential arrives at the neuromuscular junction
cell membrane is depolarized, acetylcholine is released, sodium goes into cell
calcium is released from sarcoplasmic reticulum into muscle
calcium binds to troponin (on tropomyosin) causes movement and revealing myosin binding sites on actin
ATP is hydrolyzed to form ADP + phosphate
myosin head binds to actin and forms cross bridges (stays there until ATP molecule releases it) - if calcium is still there cross bridge is still there
ADP is released causing myosin heads to activate and move towards center of sarcomere (Power stroke)
power stroke is continued till z lines are pulled toward H zone
calcium is transported back causing termination of cross bridge.
myosin binding sites covered by tropomyosin and troponin (returns to original state)
Myofibril
Elongated contractile threads found in striated muscle cells
Myofilament
The structure that makes up the myofibril, examples include Actin and Myosin
Sarcomere
Structural unit of a myofibril consisting of a dark band and near a pale band
Actin (I band)
thin filaments only, The protein that forms the contractile filaments of muscle cells
H zones
Contains only thick filaments and shortens during contraction
Myosin (A band)
The fibrous protein that forms the contractile filaments of muscle cells, Thick bands
Z line
end of sarcomere
Tropomyosin
Found in actin, blocks muscle contraction
Sarcoplasmic reticulum
Contains and releases calcium
Calcium ions
Bonds to troponin and reveals the binding sites on the actin
ATP
Fuel for muscle contraction, splits into ADP and P
Type I
Activity- Light activity
Glycogen- Low
Type IIa
Activity- Long duration low intensity
Glycogen- High
Type IIb
Activity- Short duration high intensity
Glycogen- High
Flexion
Decrease the angle of a joint
Extension
Increase the angle of a joint
Abduction
movement away from the midline of the body
Adduction
Movement toward the midline of the body
Pronation
Rotation of Forearm where the palm of the hand is facing down
Supination
Rotation of Forearm where the palm of the hand is facing up
Elevation
Raising of the shoulder girdle
Depression
Lowering of the shoulder girdle
Rotation
Movement around a pivotal point
Circumduction
A circular movement/direction resembling a cone
Dorsiflexion
Raising of the foot/toes towards the body \n - Decreasing angle between tibia and foot
Plantar flexion
Pointing of the toes/ foot towards the body \n - increasing angle between tibia and foot
Eversion
Turning of the sole of the foot outwards
Inversion
Turning of the sole of the foot inwards
Isotonic
when the muscles either shortens or lengthens during contraction
Isometric
When a muscle contracts with no resulting movement
Isokinetic
A muscle contraction with constant speed
Concentric vs Eccentric
Muscle shortens, muscle lengthens
Agonist
Prime mover - contracts to cause movement
Antagonist
Relaxes to allow movement
DOMS
delayed onset muscle soreness (24-72 hours after eccentric muscle contraction) and is associated with structural muscle damage, inflammatory reactions in the muscle, overstretching and overtraining. \n \n DOMS is prevented/minimized by reducing the eccentric component of muscle actions during early training, starting training at a low intensity and gradually increasing the intensity, and warming up before exercise, cooling down after exercise.
Fixator
Stabilizes the origin of a prime mover
Synergist
Neutralizer, whom contract isometrically to prevent unwanted actions against the agonist/ antagonist