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what % and what disorder is most prevalent in children
anxiety disorders are most prevalent
Disorder of uncontrolled behaviour
also known as externalizing problems
DSM-5 classification - disruptive, impulse-control and conduct disorder
oppositional defiant disorder
conduct disorder
attention deficit hyperactivity disorder
Disruptive mood dysregulation disorder
Referred to as ‘temper tantrum disorder’
persistent irritability
Episodes of temper outbursts, three or more times per week
In DSM-IV-R called childhood bipolar disorder
Attention - Deficit/Hyperactivity Disorder
Not recognized until research by Virginia Douglas
Difficulty concentrating on tasks
Difficulty sustaining attention over time
Trouble following through on instructions
Forgetful in daily activities
Boys with ADHD are more likely than girls with ADHD to be aggressive. In girls, hyperactivity is more likely to be manifested in talkativeness.
Types of ADHD
ADHD predominantly attention deficit – problems with executive functions
ADHD predominantly hyperactive
ADHD combined
Hyperactivity
constantly in motion
trouble sitting still
unable to stop moving or talking when asked to be quiet
activities and movements may seem haphazard
Thomas Brown’s model of executive functions impaired in ADHD
1. Activation: Organizing, prioritizing, and activating to work
2. Focus: focusing, sustaining focus, and shifting focus to tasks
3. Effort: Regulating alertness, sustaining effort, and processing speed
4. Emotion: Managing frustration & modulating emotions
5. Memory: Utilizing working memory & accessing recall
6. Action: Monitoring & self-regulating action
children with ADHD have
Peer-relations difficulty since their behaviour can be annoying to others
Learning disabilities in 15- 30%
Comorbidity with many disorders
Genetic predisposition - biological theories and ADHD
ADHD is considered to be one of the most heritable phenotypes
Estimates of heritability: 75%
Differences in brain structure and function - biological theories and ADHD
reductions in volume in the cerebrum and cerebellum
delays in cortical maturation
smaller basal ganglia volumes
dysfunction in dopaminergic and noradrenergic systems
Possible environmental risk factors in ADHD
pre and perinatal factors
maternal smoking, alcohol and substance abuse
maternal stress
low birth weight and prematurity
environmental toxins
dietary factors and nutritional deficiencies
psychosocial adversity
family adversity and low income
conflict parents and children hostility
early deprivation
Psychological Theories of ADHD
Diathesis -stress theory of ADHD
Hyperactivity develops when predisposition to disorder is coupled with an authoritarian upbringing (Bettelheim)
Attention -seeking and hyperactivity
Treatment of ADHD
Stimulant Drugs
Drugs such as Methylphenidate (Ritalin) used to reduce attention deficit
Side effects: sleep problems, loss of appetite
higher prescriptions in recent years
Psychological Treatment of ADHD
Parent training and changes in classroom management based on operant conditioning principles
Reinforcement for behaving appropriately
Oppositional Defiant Disorder
Three main themes:
Pattern of disobedient, hostile, and defiant behaviour towards authority figures
angry irritable mood
vindictiveness
Children with ODD do NOT demonstrate serious violations of societal norms
Conduct Disorder
More severe than ODD
Marked by callousness, viciousness, lack of remorse \
Repetitive pattern of behaviour that includes: 3 or more
Aggression to people and animals
bullying, threatening, cruel
Destruction of property
Deceitfulness or theft
Serious violations of rules, rights of others
CD is a criteria for antisocial personality disorder
comorbidity with ODC and ADHD
Etiology of CD - Biological Factors in Conduct Disorder
Genetic influences
Aggressive behaviour clearly heritable
Delinquent behaviour seems not to be heritable
Neuropsychological deficits
Poor verbal skills, difficulty w/ executive functioning, problems w/ memory
Neurological correlates (brain imaging studies)
Possible amygdala dysfunction
Etiology of CD - Psychological, learning and cognitive Factors in Conduct Disorder
Hostile/ineffective parenting practices
inconsistent parental discipline and parental adjustment difficulties
Learning theories
Modelling and operant conditioning
Cognitive Biases
Social-information processing theory
Mistaken views of neutral peer behaviour
Etiology of CD -Chaotic social environment
Noise levels, crowding, unpredictability in home and neighbourhood
higher in communities with more chaos and low-socio economic places
Course of treatment of CD
Some improvements seen when issues addressed at younger age
Severe cases typically persist and develop into APD in adulthood
Family Interventions - Parental Management Training
Multi-systemic Treatment
Cognitive Approaches - Anger management - Moral reasoning skills training
Prevention of CD
Beginning treatment before age 3
Identifying families and mothers at risk
Prenatal and postnatal risks in mother:
Maternal antisocial behaviour
Young age of pregnancy
Smoking during pregnancy
Maternal depression soon after birth
Partner cruelty
Harsh parenting
Neurodevelopmental Disorders DSM-5
Attention-Deficit/Hyperactivity Disorder (ADHD)
Specific Learning Disorders
Communication Disorders
Motor Disorders
Intellectual Disability Disorder
Autism Spectrum Disorders (ASD)
Specific Learning Disorders
Inadequate development in specific area of academic, language, speech, or motor skills
Not due to intellectual disability autism, a demonstrable physical disorder, or deficient educational opportunities
Usually have average or aboveaverage intellect
Specific Learning Disorders - Reading disorder
• Difficulty with word recognition and reading comprehension
Also known as dyslexia
Specific Learning Disorders - Mathematics disorder -Dyscalculia
Difficulties rapidly and accurately recalling arithmetic facts, counting objects correctly and quickly, or aligning numbers in columns
Specific Learning Disorders - Dysgraphia - disorder of written expression
difficulties in composing written work
spelling errors, errors in grammar or very poor handwriting
Etiology of Learning Disorders - Biological Factors
Heritable component
Chromosome 13 is implicated in dyslexia
Brain Structure Differences
Left temporo-parietal cortex ↓ activation in LD
Brain area responsible for ‘phonological awareness’
Treatment of Learning Disorders
most often occurs within special-education programs in the public schools
Individualized programs should be implemented
Duration of treatment should match the severity of the LD
parental involvement
Communication disorder - Language Disorder, Communication disorder
Language disorder
Child sees a car but has trouble communicating the word for it
Communication disorder - Speech Sound Disorder (Phonology Disorder)
says Wabbit not rabbit
Childhood Onset Fluency Disorder (Stuttering)
most recover
Motor disorders
Developmental Coordination Disorder
Marked impairment in motor coordination, such as troubles tying shoelaces, buttoning shirts
Diagnosis only made if significant impairment
Tourette’s disorder
multiple motor tics and one or more vocal tics
Tics
involuntary, repetitive movements or vocalizations
Intellectual Disability Disorder
Previously termed Mental Retardation
Significant limitations in intellectual functioning and adaptive behaviour Diagnostic Criteria
Intelligence-Test Scores (IQ = 70 or lower)
Adaptive Functioning (deficits in conceptual skills, social skills and practical skills
Classification Intellectual Disability Disorder
Four levels of intellectual disability disorder (DSM-5):
Mild (most common), moderate, severe, profound •
Use both IQ scores and adaptive functioning to determine severity levels
Previous DSM-IV-TR classifications (IQ score based):
Mild • 50–55 to 70 IQ; 85% of people with Intellectual Disability
Moderate • 35-40 to 50-55 IQ; 10%
Severe • 20-25 to 35-40 IQ; 3 to 4%
Profound • below 20 to 25 IQ; 1-2%
Etiology Intellectual Disability Disorder
No Identifiable Etiology
Accidents and physical abuse. Illnesses such as measles, whooping cough, chicken pox, Hib disease, Reye’s syndrome, exposure to the Zika virus
Etiology Intellectual Disability Disorder - Heredity Disorders (5%)
Genetic or Chromosomal Anomalies
Phenylketonuria (PKU); Fragile X syndrome
what is intellectual disability disorder
Down syndrome, or trisomy 21; maternal alcohol consumption
Late pregnancy and perinatal problems (10%) - etiology of intellectual disability disorder
Fetal malnutrition, placental insufficiency, prematurity, low birth weight, viral and other infections (e.g., HIV infection)
Environmental Influences (15-20%) - etiology of intellectual disability disorder
Deprivation, lack of nurturance, reduced stimulation
Effects of ↓ socio-economic conditions
Prevention and Treatment Intellectual Disability
Environmental Interventions and Enrichment Programs
Behavioural Interventions Based on Operant Conditioning
Applied Behaviour Analysis
Cognitive Interventions
Self-instructional training
Autism Spectrum Disorder (ASD)
Consisted of several subcategories, including autistic disorder (autism), Asperger’s Disorder, Rett’s disorder, childhood disintegrative disorder , pervasive developmental disorder – not otherwise specified
DSM-5 eliminated subcategories since distinctions found to be ‘inconsistent’, more related to:
symptom severity levels, language levels, intellectual levels • Prevalence is increasing
Onset: infancy and early developmental period
Comorbidity: depression, anxiety, ADHD
Autism Spectrum Disorder (ASD) Characteristics
Deficits in social communication and social interaction
Troubles adjusting behaviour in changing social contexts
Limited imaginative play
Repetitive and rigid behaviour (insistence on sameness)
Unusual motor movements
Rett’s Disorder
Very rare; found only in girls
Development normal until 1st-2nd year of life
Head growth decelerates
Loses ability to use hands purposefully
Stereotyped movements such as handwringing or handwashing
Walks in an uncoordinated manner
Poor speech
Childhood Disintegrative Disorder
very rare
Normal development in the first 2 years of life then significant loss of:
Social, play, language, and motor skills
Extreme Autistic Aloneness, Communication Deficits, • Obsessive-Compulsive and Ritualistic Acts
Extreme Autistic Aloneness
Rarely engage others in play
Fail to offer spontaneous greetings
Communication Deficits
Echolalia echo speech
Pronoun reversal
Obsessive-Compulsive and Ritualistic Acts
Upset easily over changes
Prone to stereotypic behaviour
Etiology of ASD - Psychological bases
Psychoanalytic and behavioural perspectives
It was previously thought that parents play a crucial role in ASD
This is not credible and it is cruel
Etiology of ASD - genetic factors
risk of autism in siblings of people with the disorder is about 75% higher
fragile X syndrome - chromosome abornmalities
linked genetically to broader spectrum of deficits in communications and social areas
autism reflects exceeding complex variation, more than 1000 genes
Etiology of ASD - neurological factors and environmental risks
epileptic seizures
abnormal brainwave patterns
Possible brain regions implicated include:
Cerebellum
Amygdala and corpus callosum
Medial frontal cortex and medial temporal cortex
possible role of: maternal infections, drugs, and toxicants as well as metabolic and nutritional factors
Treatment of ASD
early intervention is critical to providing a better chance of success in school and in living independently
treatments that are most effective using modelling and operant conditioning
early intensive behavioural intervention
most effective if delivered early
children with higher initial cognitive levels and fewer early social interaction deficits show best response
Disorders of Overcontrolled Behaviour
Separation Anxiety (now included among Anxiety Disorders)
Social Phobia (also known as Social Anxiety)
Selective Mutism
Specific Phobia
Generalized Anxiety Disorder
Obsessive-Compulsive Disorder
Post-Traumatic Stress
Panic Disorder Depression
Childhood Fears and Anxiety Disorders
most common disorder in childhood
some anxious children report helicopter parents style
Separation Anxiety Disorder
Unrealistic concern about separation from major attachment figures
Unrealistic and persistent worries about harm to major attachment figures
Fears of abandonment
Refusal to attend school
Avoidance of being alone
Experience of nightmares involving separation themes
Experience of physical complaints in anticipation of being separated from attachment figures
Social Phobia
Extremely quiet, shy, avoid strangers
May include selective mutism
Theories of social phobia in children
individual differences in behavioural inhibition
Higher risk when parent has social phobia
Treatment of Fears and Phobias in Children
Similar to that employed with adults
Exposure to feared object while performing some action to inhibit their anxiety
CBT shows great promise in treating childhood anxiety