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what is the pregnant pt at risk of and why
caries and erosion risk
due to sugary diet caused by cravings = caries susceptibility
acid reflux/ monring sickness = erosion
perio risk
tooth mobility due to pdl disturbances
what is teratogenic damage and when does it happen
harmful effects of drugs on the foetus most prone in 1sr trimester
what di we nit do in third trimester
treat pt supine due to prevention of hypotensive syndrome
correct positioning avoids maternal vena cava and arota compression
what is the placental barrier
acts as an interface between mother and foetus
gas exchnage and transfer of nutrients and waste products
secretion of hormones for foetal growth and development
transfers immunity from mum to foetus
what is greatest risk to foetus
organogenesis in 1st trimester
what are the placental barrier mechanisms of transfer
simple diffusion
molecular weight
lipid solubility
degree of ionisation
protien binding
when do we not use amalgam
children under 15
pregnant or breastfeeding women
except when strictly necessary based on medical needs of pt
what are the concerns of resin composites
elution of bisphenol a (bpa);
is an endocrine distruptor
mimics oestrogen
new resin comps associated with short term bpa in saliva and urine
bpa can cross placental barrier
no restrictions tho
what do we know about amalgam
surpisingly little
chemical form of mercur determines the celluler uptake;
mercury vapour - passive diffusion
methyl mercury - active transport by amino acid carriers