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Specific / Medical Causes of Neck Pain
Fractures
Infections
Inflammatory disorders
Malignancy
Vascular pathology
Neurological pathology
Mechanical (MSK related) Neck Pain Causes
Disc
Facet
Instability/Ligament
Muscle
Postural
Wry Neck (Adult) Presentation
Acute neck pain / stiffness / muscle spasm
Often awake from sleep with it
Sudden onset
Restricted ROM
Abnormal head posture: Usually head tilted to one side and chin turned to the other
May experience headaches, dizziness, nausea
Wry Neck (Adult) Causes
No clear cause
Could be muscle spasm, facet sprain, synovial fold etc.
Wry Neck (Adult) Treatment
Rest
Heat
Medications
Manual Therapy
NO HARM
Wry Neck (Children) Causes
Early weeks of life- often SCM tumor or fibrosis
Abnormal development of the cervical vertebrae
Cervical spine articular disruption
Intracranial pathology
Inflammation (juvenile idiopathic arthritis) or infection in the neck, head, spine, CNS, upper lobe chest
Trauma (fracture/dislocation, spinal haematoma)
Wry Neck (Children) Treatment
Heat pack, massage and basic analgesia should be appropriate
Cervical Instability Presentation
Neurovascular compromise including 5D’s and 3N’s
Headaches
Dizziness
Widespread neurological changes
Lump in throat
Fatigue with prolonged postures
Cervical Instability Causes
Lax/damage to the ligaments from:
Trauma
Congenital abnormalities
Inflammatory disease
Generalized hypermobility syndromes
Cervical Instability Treatment
Education
Manual therapy (lower cervical and thoracic)
Motor control and strength training
Whiplash Causes
Sudden acceleration and deceleration (forced forward and whipped backward)
Car accidents
Contact sports
Falls
Whiplash Treatment
Accurate advice and education
Relative rest (not too much, not enough)
Simple medications
Exercise as appropriate to patient condition
Manual therapy
Specific dizziness assessment and management
Referral as required (imaging, medical, psychological)
Whiplash Presentation
Neck pain
Stiffness
Difficulty moving head, reduced ROM
Headaches
Muscle spasms
Case Study- C6 Radicular pain
Onset- gradual L lower neck pain - now with L arm, forearm, and thumb ache
Symptoms worsened with increased workload
Arm symptoms suggest C6 nerve root irritation, but no neurological deficits reported
Radiculopathy not confirmed due to absence of sensory / motor changes
C5-C6 segment- common site of degeneration and nerve root compression
C6 nerve root- refers pain to forearm and thumb
Hairdressing posture- repetitive arm elevation, neck flexion / rotation → ↑ cervical load
Possible foraminal narrowing- aggravated by extension and rotation
Muscle fatigue and joint stress- from sustained postures and repetitive use
Case Study- Cervicogenic Headache
Onset- prolonged neck extension and sustained computer posture
Pain- localized to upper cervical region, movement-provoked
No migrane history or red flags
Considerations: Joint Compression: C1–C3 under load in extension; C1–C2 rotation restricted Muscle Tone: ↑ Suboccipital tone; postural strain from upper traps, levator scap, SCM Posture: Forward Head Posture → ↓ Deep Neck Flexor activation, ↑ passive structure strain
Cervicogenic Headache Presentation
Unilateral pain in head (starting from one side of the posterior head and nek, migrating to the front)
Neck pain and stiffness
Pain aggravated by neck movements or sustained postures
Cervicogenic Headache Treatment
Manual therapy
Exercise
Education
Postural control
Pain relivers (NSAIDs)
Thoracic Outlet Syndrome Pathophysiology/Etiology
Nerve or blood vessels that gets compressed above or below the clavicle (infraclavicular fossa and supraclavicular fossa)
Can be just nerve or nerve and vein/artery
This condition comes on a lot when people are recovering from something traumatic like a first rib fracture or whiplash
Thoracic Outlet Syndrome Presentation (nTOS)
Neck, scap, trapezius, and/or arm pain potentially difficult to localize
Pins and needles, numbness
Thoracic Outlet Syndrome Presentation (vTOS)
Arm pain
Swelling
Cyanosis
Thoracic Outlet Syndrome Presentation (aTOS)
Intermittent ischaemia
Pain
Pallor
Claudication
Paraesthesias
Coldness
Thoracic Outlet Syndrome Treatment (Conservative)
Education:
Positions of ease and compression
Avoid provoking postures
Reduce training load overhead (if appropriate)
Muscle/Motor Retraining:
Deep neck flexors
Scapulothoracic strength / proprioception
Neural Glides:
Gliding / flossing of ulna, radius, median nerves
Taping:
Proprioception facilitation of scapular posture
Thoracic Outlet Syndrome Treatment (Surgical)
Main aim of surgery is to decompress the brachial plexus / subclavian artery or vein as it passes through the main site of compression.
Usually this would involve one or a combination of the following:
Anterior scalene release
Subclavius muscle release
First rib resection
Removal of cervical rib (if present)
Example TOS Treatment
Education- reducing compressive load via repeated activity
Posture- review sitting and standing / prolonged postures
Depending on site of compression -
positions of ease + exercise
taping (scapular proprioception using rocktape / kinesiotape)
soft tissue modalities as indicated
manual therapy over cervicothoracic junction if indicated
Exercise- 4pt kneel periscapular awareness/cervical extension training, serratus anterior wall push-ups, lat dorsi strengthening (and so on…)
Plan- consider imaging if not done already, refer onward if concerned regarding atrial compromise or possible DVT, review responsiveness to load management education and exercise program
What is Cervicogenic dizziness?
A condition that can be defined as a dysfunction/damage to the mechanoreceptors or sympathetic nerves in the upper cervical region causing symptoms that include dizziness, light-headedness, disorientation, postural imbalance, and/or headaches; all associated with neck movement
Cervicogenic Headaches- Aetiology
There is no clear aetiology that defines the onset of cervicogenic dizziness at present
Associated with many neck-related pathologies, such as:
OA of the cervical facet joints
WAD whiplash associated disorder
Cervical artery insufficiency
Any pathology that can affect the typical function of cervical proprioception could be considered a cause for cervicogenic dizziness
Cervicogenic Headaches- Pathophysiology
Deep neck musculature has high muscle spindle density
Cervical joints also contribute to proprioceptive feedback
Dysfunction or damage to the cervical proprioceptors impairs the information provided to the occular motor, cervical motor, and lower limb motor systems
Pain may also contribute via maladaptive strategies, and recruitment patterns
Pain can also adversely modulate cervical afferent input
Cervicogenic Headaches Presentation
Dizziness
Imbalance
Light-headedness
Concurrent neck pain- aggravated by neck movement NOT head position
Symptoms present daily
Cervicogenic Headaches Assessment
To assess for cervicogenic dizziness, a musculoskeletal assessment of the cervical spine is performed:
Active cervical ROM
Palpation
MLT, MMT
PAVIMS, PPIVMS
Joint position error testing
Cervical Torsion test
Head neck differentiation test
Smooth pursuit torsion test
Idiopathic Thoracic Conditions
Scoliosis
Scheuermann disease
be aware of
juvenile kyphosis, juvenile idiopathic arthritis
hypermobility syndromes (Marfans, Loeys-Dietz, Elhers Danlos Syndrome)
neuromuscular conditions: cerebral palsy, muscular dystrophy
Thoracic Conditions MSK
Intervertebral joint strain
Costovertebral strain
Rib fracture
Muscle or ligament sprain/strain
Nerve injury or irritation
be aware of
SCJ injury
DISH (Diffuse Idiopathic Skeletal Hyperostosis
Thoracic Conditions Non MSK
Osteoporotic vertebral fracture
Metastatic disease
Shingles (herpes zoster virus)
Structural Scoliosis Presentation
Shoulders uneven
Prominent shoulder blade
Visible curve in erect standing
Iliac crest heigh uneven
“Rib Hump” in forward flexion
Functional Scoliosis
A functional scoliosis will appear curved in standing, however will have no curvature/ rib hump in forward flexion
No bony changes on imaging
Scheuermann’s Disease
Etiology: Unsure
Vertebral end plate irregularities during growth affecting 4 or more vertebral bodies
Variance causes some of the vertebrae to become wedge-shaped
Results in hyper kyphosis of thoracic spine and a compensatory hyper lordosis of lumbar spine
Scheuermann’s Disease Presentation
Rigid hyperkyphotic curve, accentuated in flexion
Curve does not reduce in extension/supine/prone
Cervical lumbar hyper lordosis
“Barrel chest’ shape of thorax
Scheuermann’s Disease Treatment
Kyphosis relatively fixed in adulthood, aims during growth period for maintain thoracic ROM, increase hamstring length and abdominal strength, encourage general activity
Most Common Thoracic Injuries
Intervertebral facet joint sprain
Costovertebral/costotransverse sprain
Sternoclavicular joint sprain/dislocation
Rib fracture
Rib cartilage fracture
Intervertebral (facet joint / thoracic disc) Presentation
Pain with gross thoracic movement (flexion / extension / LF / rotation)
Pain with prolonged upright postures
Pain with unilateral / bilateral overhead arm movement
Moreso central / paraspinal thoracic pain
Pain with central PA of level above and below the irritated structure
Generally: pain with unilateral rotation and LF (facet), extension or flexion (facet/disc)
Bilateral or unilateral thoracic spine pain
Morning stiffness common (inflammatory)
Costovertebral Presentation
Pain associated with breathing and increased exertion
Pain with increased intra-abdominal pressure (cough/sneeze/laugh/abdominal contraction)
Pain with deep inspiration or forced expiration
Pinpoint pain over rib/transverse process
Moreso unilateral thoracic pain
Pain reproduced with deep inspiration with deep inspiration or expiration
Thoracic flexion/extension OK, painful with rotation
Pain reduced with rib support during inspiration/cough/sneeze
Rib springs provocative on irritated rib
Osteoporotic Fracture Summary
Complaint:
Mid-low central thoracic pain
Etiology:
Often sudden onset of pain with minimal or no trauma (step off curb, fall onto buttocks)
Associated Symptoms:
Previous fractures with minimal trauma
Pattern Recognition:
Women > Men
Post-menopause
Family history of OP
Corticosteroid use hx
Excess alcohol or smoking
Poor diet
Costal Cartilage Fractures Treatment
Supported cough/sneeze, symptomatic management
Protective padding recommended for RTS >3
Treatments for Mobility Deficits
PPVIMs
Unilateral PAVIMS
Central PAVIMS
MWM (transverse glide with rotation, ceph/caud glide with extension)
Always combined with exercise, which may target
mobility (ie. thoracic ext with foam roller, exercise ball)
endurance and strength
Treatments for Hypermobility
Exercise training working on motor control during functional movements
endurance of paraspinal muscles (postural training)
flexion/extension control in 4pt or 2pt kneel
side flexion control over exercise ball (side lie)
whole body coordination - lunge + upper thoracic rotation
Costovertebral Joint Summary
Complaint:
Unilateral pain. Pain located over transverse process/rib articulation
Etiology:
Generally related to clear MOI (hyper rotation, impact to chest wall)
Associated Symptoms:
Pain with deep inspiration or forced expiration
Pattern Recognition:
Pain reduced with support and provocation action (ie. cough, deep breath)
Gross movements of trunk generally OK (can be painful in rotation)
Risk Factors for Spinal Osteoporotic Fractures
Age >64
Prolonged corticosteroid use
Trauma
Combination of: older age, female, trauma and steroid use hx
Subcutaneous emphysema
Air escape from lung into subcutaneous tissue
Feels like ‘bubble wrap’ under skin
Pneumothorax
Air escape from lung into pleural cavity
Area of lung collapse around it
Increasing shortness of breath / work of breathing
Sternoclavicular Dislocations
Anterior (more obvious, most common and least dangerous)
Posterior (subtle, with mediastinal structures at risk)
Dislocations generally reduced under general anesthesia ± thoracic surgery
Post reduction will be managed in sling, AROM will start around 3 weeks post reduction
Costal Cartilage Fractures- Conservative Management
Supported cough/sneeze, symptomatic management
Protective padding recommended for RTS >3
Costal Cartilage Fracture- Prognosis
Pain settled relatively quick and good RTS rates
Clicking can persist well beyond acute healing times
General Rib Injury Treatments
aims of txt
analgesia
restore normal breathing mechanics (pump handle/bucket handle)
teach supported cough/sneeze
maintain cervical, shoulder and thoracic
can consider:
taping (dynamic generally preferable)
compression
positions of comfort
Intervertebral discs are made of:
Nucleus
Annulus
Cartilaginous endplate
Anterior longitudinal ligament
Posterior longitudinal ligament
Typical Aging Disc
dehydration
reduced height
NP and anulus less distinct
calcification of endplate
genetic and loading causes
Typical Young Disc
more fluid nucleus
more mobile nucleus
acute injury and rapid recovery
disc compression of nerve root
classic derangement syndrome
Stages of Disc Herniation
bulging - disc protrudes outward but remains intact
protrusion - nucleus pushes against outer layer but is still contained
extrusion - nucleus tears through the outer ring and extends beyond the discs boundary
sequestration - a fragment of the disc breaks off and separates from the disc
Discogenic Pain Presentation (Typical)
often caused by flexion activities
can be sudden onset or gradual onset
central back pain BUT can radiate
nerve root (radicular / radiculopathy) can happen
pain with increased intra-abdominal pressure
often displays direction preference
Dysfunction Syndrome
Pain directly related to mechanical loading
Presence of load = pain
Absence of load = no pain
Pain is intermittent depending on load
Pain often at end- ROM due to stretching or compression
Conceptual model in the spine is articular dysfunction
Classified in respect to movement loss
ie. extension dysfunction = loss extension
Derangement Syndrome
Conceptual model for derangement related to an internal derangement of the IVD
Mechanical presentation
direction preference
repeated movements and sustained postures can aggravate or or alleviate symptoms
centralization or peripheralization
May have postural abnormality
flexion and/or side shift
Facet Joint Pain Presentation
innervation = medial branches dorsal of the same level
richly innervated with nociceptors
often caused by repetitive loading in extension or rotation
increased risk with increased lordotic posture
often unilateral
tender on palpation
local muscle tension / spasm
age-related change like any synovial joint
Spondylolisthesis Stages
Grade 1- <25% slip
Grade 2- <50% slip
Grade 3- <75% slip
Grade 4- >75% slip
Grade 5- completely off (paralysis)
Pars Stress Fracture
Can be congenital or traumatic
Sports with repeated extension (ie. dance, gymnastics)
Disc Pain Presentation
Central or unilateral back pain
Radicular pain common
Often flexion related coughing/sneeze
Disc Pain Treatment
Repeated movements (derangement)
Facet Joint Pain Presentation
Unilateral
Normally extension related
Facet Joint Pain Treatment
(normally) mobilization
Instability Presentation
Central pain
Normally extension related
Instability Treatment
Stabilization exercises
SIJ Presentation
Unilateral buttock pain
SIJ loading, SL weight bearing aggravating
SIJ Treatment
Mobilize or Stabilize
Inflammatory Back Pain
Night Pain
Morning Stiffness
Autoimmune disease
Systemic
3 Causes of Discogenic Change
Trauma
Degenerative change
Prolonged postures
Posterior Chest Wall Injury
Posterior chest wall injury
Subclavian artery/vein
Trachea
What is Spondylolysis
A stress fracture or defect in the pars interarticularis
Commonly affects children and teens involved in sports like gymnastics, weight-lifting and football- repetitive trauma is a significant cause
Spondylolisthesis
A condition where a vertebra in the spine slips forward out of its normal position
Symptoms may include LBP, pain down glutes or thigh, and hamstring tightness
Causes can be degenerative, isthmic or due to birth defects or trauma
Spondylosis
Age related wear-and-tear of the spinal discs