personality characteristic that are inflexible and maladpative causes functional impairment and/or subjective distress
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inflexible and maladpative- personality disorders
enduring pattern on behavior orgininate in childhood
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functional impairment- personality disorders
required crosses areas of life
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subjective distress- personality disorders
not required often family and friends are distressed
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personality disorders- DSM 5
an enduring pattern of inner experience and behavior that deviates markedly from the expectations of the individuals culture pattern is manifested in 2+ areas (cognitive, affectivity, interpersonal functioning, impulse control)
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prevalence of personality disorders
.5%-2.5% of population
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gender differences of personality disorders
men: antisocial PD women: histrionic and borderline PD
fearful or anxious avoidant, dependent, obsessive-compulsive
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paranoid personality disorders
A pervasive distrust and suspiciousness of others, such that their motives are interpreted as malevolent
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causes of paranoid personality disorders
possible role of early experiences cultural factors (prisoners, refugees, people with hearing impairments, older adults) possible relationship to schizphrenia
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treatment for paranoid personality disorder
unlikely to seek on their own focus on developing trust cognitive therapy no empirically-supported treatments (poor improvement rates)
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schizoid personality disorder
a pervasive pattern of detachment from social relationships and a restricted range of expression of emotions
neither desires nor enjoys relationships no unusual thought process like paranoid
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causes of schizoid PD
limited research precursor: childhood shyness connection with ASD? lower density of DA receptors social skills therapy
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schizotypal PD
a pervasive pattern of social and interpersonal deficits marked by acute discomfort with and reduced capacity for close relationships as well as cognitive or perceptual distortions and eccentricities of behavior
ideas of reference, odd beliefs, unusual perceptual experiences, odd behavior
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magical thinking
attribution of casual relationships between actions and events which cannot be justified by reason and observation
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schizotypal PD causes
schizphrenia phenotype genetic and environmental
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schizotypal PD treatment
multidimensional social skills training, antipsychotic meds, community treatment treatment for comorbid depression: 30-50%
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antisocial personality disorder
pervasive pattern of disregard and violation of the rights of others and evidence of conduct disorder before age 15
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ASPD and legal consequences
80-90% of inmates fulfill criteria for ASPD while only 15-25% score for psychopathy
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ASPD video
tommy lynn sells killer who lost count of how many people he killed
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ASPD causes- under arousal hypothesis
patients have abnormally low cortical arousal seek stimulation to boost arousal physiological correlates: dec, skin conductance, lower resting HR, more slow-frequency brain-wave activity
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ASPD causes- fearlessness hypothesis
higher threshold for experiencing fear leads to all other major features of aspd
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acquired psychopathy
phineas gage previous to brain injury: most efficient/capable forman after injury: disrepectful, profanity, impatient, stubborn
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aspd treatment
rarely identify themselves as needing treatment incarceration: prevents further antisocial acts parent child interaction training
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borderline personality disorder
a pervasive pattern of instability of interpersonal relationships, self-image, affect and control over impulses
fear of abandonment self-destructive behaviors impulsivity
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splitting- BPD
defense mechanism all or none thinking
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BPD video
ever changing and extreme views/reactions difficulty with relationships
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BPD prevalence
most common personality disorder 1-2% of population 15% of psychiatric settings
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BPD comorbidities
depression- 20% suicide- 6% bipolar- 40% subtance abuse- 67% eating disorders- 25% of people with bulimia have BPD
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BPD causes
genetic/biological vulnerability (high sensitivity, high reactivity, slow return to baseline) invalidating social environment- high rate of abuse chronic emotional dysregulation
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treatment for BPD
DBT structured skills training prioritize treatment based on behavior severity
1 yr follow up less suicidal, less angry, better socially adjusted
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narcisstic personality disorder
a pervasive pattern of grandiosity, need for admiration, and lack of empathy
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causes and treatment of narcissistic personality disorder
deficits in early childhood learning -lack of empathy and altruism development -remain self-centered sociological view -increased indidvidual focus comorbid depression -experience of what the deserve doesnt match with reality -treatment attempts to replace fantasies with focus on attainable day-to-day pleasures -focus on others feelongs
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histrionic personality disorder
a pervasive pattern of excessive emotionality and attention seeking
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causes of histrionic PD
little research links with ASPD sex-typed alternative expression
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treatment of histrionic PD
problematic interpersonal relationships -attention seeking -long-term consequences -little empirical support
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avoidant personality disorder
disorder characterized by social discomfort and avoidance of interpersonal contact
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causes of avoidant personality disorder
child or infant with difficult temperament--parental rejection/lack of uncritcal love report childhood isolation, rejection, conflict
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treatment for avoidant personality disorder
well-studied behavioral interventions focus on anxiety and social skills issues treatment similar to social phobia therapeutic alliance is key to success- collaborative connection
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dependent personality disorder
pervasive and excessive need to be taken care of leads to submissve and clinging behavior and fears of seperation
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causes of dependent PD
little research early experience (death of a parent, rejection, attachment, genetics)
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treatment for dependent PD
limited empirical support caution: dependence on therapist gradual increases in independence, personal responsibility, confidence
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obsessive-compulsive personality disorder
pervasive pattern of preoccupation with orderliness, perfectionism, and mental and interpersoanl control, at the expense of flexibility, openness, and efficiency
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causes of obsessive compulsive personality disorder
limited research weak genetic contributions
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treatment for obsessive compulsive personality disorder
similar to OCD address fears related to need for orderliness limited efficacy data
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old age
65+ 12% of population (36 mill)
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old age by 2030
20% of population
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old age gender ratio
women: men 3:2
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jeanne calment
122 yrs old- longest confirmed living lifespan smoked for 96 yrs (2 cigarettes a day) not a necessarily healthy lifestyle
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cognitive changes in normal aging
mild slower processing speed novel tasks are more difficult recalling names are particularly difficult does not impact everyday functioning
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benign senescent forgetfulness
age-related memory decline that is distinct from memory impairment due to known neurological damage or disease
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age associated memory impairment
normal part of aging executive functionng actually declines faster than memory
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paradox of aging
well-being increases with age
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hartshorne and germine
wexler test results how cognitive abilities change with age plenty abilities start decline at age 30 some do increase until 50
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socioemotional selectivity theory (carstensen and colleagues)
-motivational theory -suggests that secondary to understanding of constraints on life longevity, olrder adults alter their strategies for emotional regulation -older adults focus on and demonstrate a bias towards positively valenced material -reduced time horizons prompt older adults to prioritize achieving emotional gratification and thus exhibit increased positivity in attention and recall
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positivity bias
emotional memory older adults remember positive images more than younger adults
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barrier task
director and matcher goal is to verbally match tangrams as quickly as possible
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normal aging of the brain
aging does not equal dementia increased age is primary risk factor for dementia every 10 years prevalence of dementia doubles normal aging is associated with variable degrees of cognitive weakness, cortical atrophy, accumulation of alzheimer-type pathology, and reduced cerebral blood flow
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cognitive reserve
individuals with higher _____ make more efficient use of the brains resources in the presence of pathology use/challenge of the brain can lead to higher resilience toward aging
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mild cogniyive impairment
older adult with weakness in one cognitive domain subjective and objective memory problems, but otherwise intact cognition and ADLs progresses to dementia at a rate of 15-20% per year
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amnesic MCI
transitional stage of cognitive impairment preceding alzheimers dementia
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dementia
acquired impairments in mulitple aspects of cognition severe enough to impair normal activties not a specific disease consequences of several diseases
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warning signs of dementia
memory loss that affects normal activties excessive word finding problems difficulty performing familiar tasks disorientatiom to time or place changes in mood, behavior, personality poor judgement and decision making
age-related, irreversible brain disorder that develops gradually and results in memory loss, decline in thinking ability and changes in behavior and personality these deficits and changes are due to the breakdown and dealth of brain cells
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neurofibrillary tangles
twisted protein fibers (tau) found within cells causes abnormal neuronal functioning leaves behind a "tombstone" cell
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neuritic (senile) plaques
deposits of beta-amyloid protein that form in spaces between cells interfere with communication between cells causes neurons to breakdown and die
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treating alzheimers (or any other dementia)
slow decline but do not stop the degeneration of the brain no treatment has been proven to arrest, reverse, or prevent AD
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declarative long-term memory
memory for facts and events medial temporal lobe diencephalon
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anterograde declarative long-term memory
learning and memory formation of new material
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retrograde declarative long-term memory
recall of previously acquired memories
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procedural long-term memory
memory for a behavioral response not conscious recall learned through repeated exposure or practice preserved in amnesia and AD based on different brain structures than declarative memory striatum, cerebellum, motor cortex
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alzheimers patient video
socially graced (conversation) and knows her husband could not remember that she talked to the doctor earlier, her last name, her birthday, doesnt remember words he gave, poor word finding (confrontational)
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bi-temporal-parietal hypometabolism
bilaterally (r and l hemisphere) there is low metabolism in both temporal and parietal *key feature of AD on PET scan*
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inducing a mood in patients with severe anterograde amnesia
each mood induction elicited appropriate modd for over 15 min in patients with severe amnesia the mood endured beyond their ability to recall the induction
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prevalence of intellectual disability
1% both in US and worldwide
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intellectual disability
significanly subaverage general intellectual functioning existing corrently with deficits in adapative behavior and manifested during the developmental period