Results for "incomplete dominance"

Let’s break this down! I’ll give you a study guide covering asexual reproduction, sexual reproduction (in humans and plants), and Mendelian genetics — then I’ll create a test for you. ⸻ Study Guide for Asexual Reproduction, Sexual Reproduction, and Mendelian Genetics ⸻ I. Asexual Reproduction • Definition: Involves a single parent, producing offspring genetically identical to the parent (clones). • Types of Asexual Reproduction: • Binary Fission: Simple cell division (e.g., bacteria). • Budding: New organism grows from a bud on the parent (e.g., yeast, hydra). • Fragmentation/Regeneration: Organism splits, and each part grows into a new organism (e.g., starfish). • Vegetative Propagation: New plants grow from parts of the parent plant (e.g., runners in strawberries). • Parthenogenesis: Offspring develop from unfertilized eggs (e.g., some lizards, insects). • Pros and Cons: • Pros: Fast, no need for a mate, energy-efficient. • Cons: No genetic variation, susceptible to environmental changes. ⸻ II. Sexual Reproduction (Humans & Plants) Humans: • Process: • Gametes (sperm and egg) produced by meiosis. • Fertilization forms a zygote (diploid cell). • Male Reproductive System: Testes, epididymis, vas deferens, prostate, urethra, penis. • Female Reproductive System: Ovaries, fallopian tubes, uterus, cervix, vagina. • Hormonal Regulation: • Male: Testosterone (sperm production, secondary characteristics). • Female: Estrogen & progesterone (menstrual cycle, pregnancy). Plants: • Process: Alternation of generations (sporophyte & gametophyte stages). • Pollination: Transfer of pollen to stigma. • Fertilization: Sperm (from pollen) fuses with egg in ovule. • Structures: Stamen (male), carpel/pistil (female). • Seed & Fruit Development: Fertilized ovules become seeds; ovary becomes fruit. ⸻ III. Mendelian Genetics • Gregor Mendel’s Experiments: Pea plants, discovered inheritance patterns. • Key Concepts: • Genes & Alleles: Genes determine traits, alleles are gene variants. • Dominant vs. Recessive Alleles: Dominant alleles mask recessive ones. • Homozygous vs. Heterozygous: Same alleles (AA or aa) vs. different alleles (Aa). • Laws of Inheritance: • Law of Segregation: Allele pairs separate during gamete formation. • Law of Independent Assortment: Genes for different traits sort independently. • Genetic Crosses: Punnett squares, monohybrid/dihybrid crosses. • Probability & Ratios: Phenotypic/genotypic ratios. • Non-Mendelian Inheritance: Incomplete dominance, codominance, multiple alleles, polygenic traits, sex-linked traits. ⸻ AP Biology Practice Test Total Questions: 30 (Multiple Choice) Section 1: Asexual Reproduction (6 questions) 1. Which form of asexual reproduction involves an organism splitting into two identical cells? a) Budding b) Fragmentation c) Binary fission d) Parthenogenesis 2. Which organism commonly reproduces through budding? a) Bacteria b) Starfish c) Hydra d) Fern 3. A disadvantage of asexual reproduction is: a) Slow reproduction rate b) High genetic diversity c) Vulnerability to environmental changes d) Requirement of a mate 4. Which plant structure is involved in vegetative propagation? a) Petal b) Stigma c) Runner d) Anther 5. Parthenogenesis involves: a) Fertilized eggs developing into offspring b) Unfertilized eggs developing into offspring c) Fusion of gametes d) Regeneration of lost body parts 6. What is the primary benefit of asexual reproduction in stable environments? a) Genetic variation b) Rapid population growth c) Evolutionary adaptability d) Reduced mutation rates ⸻ Section 2: Sexual Reproduction (8 questions) 7. In humans, fertilization typically occurs in the: a) Uterus b) Vagina c) Ovary d) Fallopian tube 8. The male gamete in plants is contained in the: a) Ovule b) Anther c) Pollen grain d) Stigma 9. Which hormone triggers ovulation? a) Testosterone b) Progesterone c) Luteinizing hormone (LH) d) Estrogen 10. The female gametophyte in flowering plants is the: a) Ovary b) Pollen tube c) Embryo sac d) Sepal 11. Which part of the male reproductive system produces sperm? a) Epididymis b) Vas deferens c) Testes d) Prostate gland 12. The process where pollen is transferred from anther to stigma is: a) Germination b) Pollination c) Fertilization d) Sporulation 13. What structure develops into a seed after fertilization in plants? a) Ovule b) Ovary c) Stamen d) Pistil 14. Which term describes the fusion of egg and sperm to form a zygote? a) Gametogenesis b) Meiosis c) Fertilization d) Pollination ⸻ Section 3: Mendelian Genetics (16 questions) 15. Who is considered the “Father of Genetics”? a) Charles Darwin b) Gregor Mendel c) Rosalind Franklin d) James Watson 16. The physical expression of a trait is called: a) Genotype b) Phenotype c) Allele d) Chromosome 17. An organism with the genotype Aa is: a) Homozygous dominant b) Homozygous recessive c) Heterozygous d) Diploid 18. A Punnett square shows: a) The process of DNA replication b) Possible genetic combinations of offspring c) Chromosome number in gametes d) Evolutionary relationships 19. The expected phenotypic ratio for a monohybrid cross is: a) 1:2:1 b) 9:3:3:1 c) 3:1 d) 4:0 20. Which of Mendel’s laws states that allele pairs separate during gamete formation? a) Law of Independent Assortment b) Law of Segregation c) Law of Dominance d) Law of Inheritance 21. Incomplete dominance results in: a) Blended traits b) Both traits expressed equally c) One trait completely masking another d) A 9:3:3:1 ratio 22. A cross between two heterozygous individuals (Aa x Aa) produces what genotypic ratio? a) 3:1 b) 1:2:1 c) 9:3:3:1 d) 2:2 23-30

Lecture 11: Dihybrid cross, incomplete dominance, codominance

Incomplete dominance and Co-Dominance

incomplete dominance quiz thing

Bio Quiz 1 Unit 3: complete dominance, incomplete dominance, codominance, sex linked diseases, blood types, gel interpretation, and pedigrees

Incomplete Dominance, Codominance, Polygenic Traits, and Epistasis!

Incomplete Dominance, Codominance, Polygenic Traits, and Epistasis

Incomplete Dominance and Codominance

Co-Dominance and Incomplete Dominance in Genetics

s2 - incomplete dominance + co-dominance (check rest of notes)

Incomplete dominance ( 画到F1) (copy)

Genetic - Extensions to Mendel: Incomplete dominance and multiple alleles

Incomplete Dominance

1.3 Macro Intro Breaking a bond = hydrolysis Build/make a bond = remove water, dehydration synthesis 1.4 Macros Nucleic Acids DNA and RNA Made from nucleotides A, T, C, G, U Proteins Amino acids Polypeptide To make it into a protein you need to fold and modify Carbs Monosaccharides Ex. glucose Polysaccharides Ex. starch, cellulose, glycogen, chitin Lipids nonpolar Ex. phospholipids Saturated (butter) vs unsaturated (oil) 1.5 Macros structure + function Uses covalent bonds between nucleotides Main structure want it to be covalent bond so its strong Bases use hydrogen bonds DNA is antiparallel, equally spaced read in opposite directions Protein Primary - Amino acids Secondary - Pleats and coils (hydrogen bonding) Tertiary - Interactions between the R-groups (unique shapes) Quaternary - 2 or more chains (any bond) Carbs Chains of sugars using covalent bonds 1.6 Nucleic Acids DNA Deoxyribose sugar T Double stranded RNA Ribose sugar U Single stranded Common Both use nucleotides A, G, C U2 Cells Organelles Ribosomes = protein synthesis Found on rough ER or free Show common ancestry Endoplasmic Reticulum Rough = ribosomes Smooth = makes lipids, detox Golgi complex Protein trafficking Packaging and transport of proteins mitochondria Site of cellular respiration, ATP production Double membrane Own DNA circular DNA Chloroplast Site of photosynthesis Own circular DNA Lysosome Hydrolytic enzymes Apoptosis Vacuole Large in plants Small in animal cells 2.3 Cell Size Small cells Inc surface area to volume ratio More efficient Better for transportation, elimination of waste, heat, exchanges, etc 2.4 Plasma Membrane Small and nonpolar can pass through easily (oxygen and carbon dioxide) 2.5 Membrane Permeability Selectively permeable Transport proteins needed for larger polar molecules Cell wall - plants, fungi, and prokaryotes Provides extra support and protection 2.6 Transport Passive transport (high to low) Does Not require any energy Diffusion Osmosis Facilitated diffusion (uses proteins) Active transport (low to high) Require energy Exocytosis Moving things in or out Endocytosis 2.7 Facilitated diffusion Uses integral proteins Ex. aquaporins, ion channels, neurons Proteins also used for active transport 3.6 Cellular Respiration Glycolysis Within the cytoplasm Evidence of common ancestry because all organisms go through glycolysis Glucose to 2 pyruvates Energy investment phase and energy payoff phase Get pyruvate, ATP, and NADH Fermentation (ONLY IF NO OXYGEN) To reset everything Takes NADH and turns it back to NAD+ to keep running glycolysis Grooming Phase Modify and turn it into Acetyl CoA Kreb Cycle With in the matrix Making electron carriers (NADH and FADH2) Inner mitochondrial membrane Where the electron transport chain takes place 3.7 Fitness Max offspring Variation can increase fitness Unit 4 Cell Communications 4.1 Signal Transduction Pathway Autocrine (signal yourself) Paracrine (next to you) Endocrine (far from you) 4.2 Signal Transduction Pathway intro Reception → transduction → response Reception: ligand attacks to the receptor The process by which a cell detects a signal in the environment. Ex. ligand binds to G protein which activates Transduction: phosphorylation cascade and amplifies signal The process of activating a series of proteins inside the cell from the cell membrane. Response: The change in behavior that occurs in the cell as a result of the signal. Second messenger - first is ligand, second messenger is for amplification (cAMP - each can have their own phosphorylation cascades) 4.3 STP Responses Turn gene off/on Apoptosis Cell growth start/stop 4.4 changes to STP Mutations (respond too much or too little to the signal molecule attacking) Chemical can release that can interfere with your STP resulting with death 4,5 Feedback Respond to changes (homeostasis) Negative (reverse change) Positive (increasing the change) 4.6 / 4.7 Cell Cycle/ Regulation G1 - growth G1 checkpoint (determine if you go to S phase or to G0 non dividing state) S - DNA replication G2 - organelle replication and growth G2 checkpoint - make sure the cell is ready for division M phase - Mitosis PMAT Prophase - nucleus disappears Metaphase - lined up at the equator Anaphase - replicated chromosomes are split Telophase - move to opposite ends M-phase checkpoint - checks to make sure division is correct Cytokinesis - final split into 2 Cyclin increases during S and peaks at M Cdk binds with cyclin to produce mpf Level of cyclins lets cell know where it’s supposed to be Tells your cell you are at your full maturity ready to produce Unit 5 Heredity 5.1 / 5.2 Meiosis Increases genetic variation Crossing over (Prophase 1) Reduction division haploid (half the amount of genetic information) Random fertilization Nondisjunction (meiosis 1 all 4 cells are irregular / meiosis 2 half the cells are irregular) Independent Assortment Increases genetic diversity 5.3 Mendelian Genetics A = dominant allele a = recessive allele Genotype - combination of letters (AA, Aa, aa) Phenotype = looks Law of Segregation - Aa → A / a Law of Independent Assortment (Aa Bb → AB, Ab, aB, ab) Sex Linked Located on a sex chromosome Usually X Sex linked recessive is more common in males because they only have one X Sex linked dominant both can inherit easily Incomplete dominance - blending Codominance - both alleles expressed 5.5 Environmental Effects Ex. weather, pH of soil 5.6 Chromosomal Inheritance Mutation → inherited Some have no effect, negative effect, neutral effect, 6.1 Gene Expression and Regulation 6.1 DNA Double stranded Deoxyribose T RNA Ribose Single stranded U 6.2 Replication (S-Phase) 5’ → 3’ Ligase - binds the new bases together Helicase - unwinds the DNA DNA poly - put down the new bases Primase - makes primer Topoisomerase - stops DNA from getting overwind Leading - able to all go in one go Lagging - many primers and okazaki fragments 6.3 Transcription and Processing Nucleus RNA poly makes primary transcript (pre mRNA) from DNA Template strand is the one the DNA is using to build Non template strand one not being used RNA processing Introns are removed Exons are put together Add cap and tail for protection Alternative splicing 6.4 Translation Ribosome Reverse Transcriptase retroviruses Ex. HIV RNA genomes use reverse transcriptase to make DNA from RNA 6.5 Regulation of Gene Expression Signal to unpack the gene Transcribed (transcription factors differ by cells and allows different gens to turn on) RNA editing Translation Polypeptide folding All need to go correctly or else the gene wont be expressed Acetylation of histones - adding acetyl group causes the DNA to be more loose making it easier to read Methylation of histones - adding methyl groups to the DNA causes it to be tighter and harder to read Enhancers - enhances transcription and causes it to occur more often Activators - dont bind to RNA poly it binds to the enhancer Depends of which genes and stage of development Epigenetics - one gene controls another gene Inducible Operon - usually off Repressor is bound to operon and lactose inactivates Repressible Operon - usually on Repressor is usually inactive, trp activates repressor 6.6 Gene Expression and Cell Specialization Promoter region (TATA box) alerts RNA poly that its a promoter region and where to attach Negative regulation - blocks promoter so RNA poly cant attach small RNA - can turn certain genes off 6.7 Mutations Increase normal gene function Decrease normal gene function Can lead to new phenotypes Cancer can be due to overproduction of growth factors, hyperactive proteins (requires many mutations Can have positive, negative, or no effect Causes of mutation Exposures Random Errors in DNA replication Increase or decrease in chromosome number Prokaryotes Transformation - pick up random DNA Transduction - virus accidentally is filled with bacterial DNA Conjunction - mating bridge/sex pilus 6.8 Biotechnology Electrophoresis - separates DNA by charge and size PCR - artificial DNA replication, increases amount of DNA sample Transformation - you make the bacteria take up a gene you're interested in Unit 7 7.1 Natural Selection natural / selective pressures decide survival Reproductive fitness (max out your kids) 7.2 Natural Selection Acts on phenotypes which can affect genotype Preferring brown fur over white decreases white fur allele frequency Environmental changes → selective pressures 7.3 Artificial Selection Humans select (ex. Dogs, livestock, etc) Convergent evolution - not closely related but because of similar environments you look alike Divergent - had a recent common ancestor but you started becoming separate Niche partitioning - choosing separate niches so you dont have to compete with others 7.4 Population Genetics Mutation - variety and evolution Genetic drift - random event that alters the gene pool Bottleneck effect - an event causes a large part of the population to die off and the remaining left repopulate with a different gene pool Founder effect - the og are there but some leave/get separated 7.5 Hardy Weinburg Large population No natural selection Random mating No mutation No gene flow P+q = 1 p2 + 2pq +q2 = 1 (AA) + (Aa) + (aa) = 1 7.6 Evidence of Evolution Fossils DNA (molecular homologies) Anatomy Vestigial structure (things we dont need anymore) (evidence of common ancestry) Biogeography (species are found all around the world)(kangaroos, genetic code, glycolysis) 7.7 Common Ancestry All Eukaryotes Membrane bound organelles Linear DNA and chromosomes Genes with introns 7.8 Continuing Evolution Genomic changes over time Continuous changes in fossils Evolution of antibiotic resistance Disease evolution 7.9 Phylogeny / Cladistics Phylogeny = included time Cladograms = just traits Shared characters Derived characters Molecular (DNA, proteins, amino acids) are more accurate than characteristics Parsimony - the one with the fewer events on it, the frewer you have the more likely it is 7.10 Speciesation Pre-zygotic Mechanical - parts dont match Gametic - egg doesnt match Geographical - dont live in the same place Temporal - ready to mate at different times Behavioral - specific type of mating display is not there Post-zygotic Hybrid sterility - the hybrid made is healthy but they cannot have children (mule) Hybrid breakdowns - the hybirds are okay but after a generation or two they cannot produce anymore Hybrid inviability - hybrid is produced but cannot survive long enough to reproduce Sympatric New species arrises in the original location Gradualism - slow steady evolution Allopstric Separation leads to speciation Punctuated - long periods of evolution with no change then rapid change 7.11 Extinction Can be natural or human caused If something goes extinct it can open up opprotunities for other species 7.12 Variation Genetic diversity Diversity of the ecosystem = inc biodiversity Less likey to be 7.13 Origins of Life on Earth No oxygen on earth 4.6 billion No ozone layer Tons of UV radiation High ocean levels Vooacanic eruptions RNA was the first genetic material DNA is dependant of RNA in

Dominance: Incomplete Dominance, Codominance and Overdominance

Lecture 6 - Codominance and Incomplete Dominance

Codominance and Incomplete Dominance

Incomplete Dominance, Co-Dominance and Lethal Alleles

Incomplete Dominance and Codominance

Autosomal, Sex-linkage, Co-dominance, Incomplete Dominance and Multiple Alleles