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Manifestations of Right Sided Heart Failure
- congestion of peripheral tissues
- dependent edema and ascites
- liver congestion -> signs related to impaired liver function
- GI tract congestion -> anorexia, GI distress, weight loss
Manifestations of Left Sided Heart Failure
- Decreased cardiac output -> activity intolerance and signs of decreased tissue perfusion
- pulmonary congestion
- impaired gas exchanges -> cyanosis and signs of hypoxia
- pulmonary edema -> orthopnea -> cough with frothy sputum and paroxysmal nocturnal dyspnea
Findings with Ischemia
- pain
- ECG abnormality
- Metabolic abnormality
- LV dysfunction
- Vasoactive substrates
What to look at during examination
breathing pattern, color, edema, oximeter, JVD, changes in extremities, weight gain
What to listen to during examination
breath sounds, heart sounds, blood pressure
What to feel for during examination
edema, breathing pattern, pulse (rate, rhythm, force)
New York Heart Association Class 1
cardiac disease without limitations
New York Heart Association Class 2
cardiac disease slight limitation
- physical activity results in fatigue, palpitations, dyspnea (SOBOE), and anginal pain
New York Heart Association Class 3
Cardiac disease marked limitation
- OK at rest, less than ordinary activity causes symptoms of fatigue, palpitations, dyspnea, and anginal pain
New York Heart Association Class 4
cardiac disease but instability to perform physical activity w/o discomfort
- fatigue, palpitations, dyspnea, and anginal pain may be felt at rest
- physical activity intensifies symptoms
97-99 SPO2 relationship with 90-100 PaO2 S&S
none
95 SPO2 relationship with 80 PaO2 S&S
none
90 SPO2 relationship with 60 PaO2 S&S
tachycardia, tachypnea, restlessness
85 SPO2 relationship with 50 PaO2 S&S
incoordination, impaired judgement, labored respirations, confusion
80 SPO2 relationship with 45 PaO2 S&S
tachycardia, tachypnea, restlessness, incoordination, impaired judgement, labored respirations, confusion
Medical interventions of Cardiac Muscle Dysfunction and Ischemic Heart Disease
fundamental treatment is directed at pathophysiologic cause
- improve hearts pumping ability -> beta blockade, vasodilator therapy
- Control sodium intake -> diet, heart healthy, low cholesterol and fat
- Water retention -> diuretics, increase urine output
PT Assessment of Cardiac Muscle Dysfunction and Ischemic Heart Disease
- when did symptoms begin?
- stable, worse, what brings on symptoms (rest, activity)
- chest pain, claudication, SOBOE, sleeping, history
- activity level (FITT principal)
- objective measures / labs -> ECG, Echo, blood gas levels, auscultation, 6MWT, questionnaires, physical appearance, RR, breathing pattern
Inpatient PT care for Cardiac Muscle Dysfunction and Ischemic Heart Disease
- flexibility exercises, cycle ergometry, and treadmill ambulation
- 30 min, 3-5 days / wk, 2-4 weeks, 50-70% of peak cycle work rate
- improvements have been reported in decreased symptoms, improved HR and exercise tolerance
- energy conservation
- self-management
- transfers
- education
Home Care PT Interventions for Cardiac Muscle Dysfunction and Ischemic Heart Disease
- flexibility exercises, cycle ergometry, and walking
- 20-60 minutes, 3-7 days / wk, 50-80% of peak cycle heart rate or O2
- improvements have been reported in decreased symptoms, improved HR, blood pressure, and exercise tolerance via GXT
- self-management of sx, diet, activity, energy conservation, ther ex / act
- ADLs / IADLs
Rehabilitation Centers PT interventions for Cardiac Muscle Dysfunction and Ischemic Heart Disease
- aerobic exercise beneficial
- various methods of aerobic activity
- most use cycle ergometry
- 20-60 min, 3-7 days / wk, 2-57 months, 40-90% of peak cycle heart rate or VO2
- improvements have been reported in decreased symptoms, improved HR, blood pressure and exercise tolerance via GXT
SLE for PT with Chronic Heart Failure
Unilateral exercise better than 2-legged exercise
- severe CHF
- limited exercise tolerance
- low cardiac output especially with exercise
Strength Training with CHF
10 repetitions for 2-4 months OR 60-80% of maximum voluntary contraction
- progression is slow with weight training
Breathing Exercises with CHF
- inspiratory muscle training -> hand-held device
- limited data on yoga -> results positive for decreasing dyspnea
Criteria for modification or termination of exercise
- marked dyspnea or fatigue
- RR > 40 breaths per min
- development of S3 heart sound
- increase in pulmonary crackles
- decrease in HR or BP of > 10 bpm or mmhg during steady state or progressive exercise
- diaphoresis, pallor or confusion
Exercise-Induced Changes in Myocardial Function
- effects on: HR, systolic BP, rate-pressure product, oxygen demand via decreased energy demand, changes in myocardial oxygen consumption (demand vs delivery)
Disease Specific Effects on Heart Rate
- chronotropic incompetence
abnormal heart rate recovery
Chronotropic Incompetence
failure to reach 85% of APMHR in the absence of Beta-Blockers or other medications with similar effects
Abnormal Heart Rate Recovery
a decrease in HR of < 12 bpm in 1 minute of ACTIVE RECOVERY or < 22 bpm at 2 minutes of supine recovery
Disease specific effects on Blood Pressure
in the absence of B-blockers or ACE inhibitors, a failure of systolic BP to rise in proportion to exercise intensity
Phase 1 of Cardiac Rehabilitation
inpatient phase was introduced in the 1960s and consists of the early graded mobilization of the stable cardiac patient to the level of activity required to perform simple household tasks
Phase 2 of Cardiac Rehabilitation
consists of outpatient monitored exercise and risk factor reduction. This multidimensional approach gained popularity in the 1970s and became well structured in the 1980s
Phase 3 of Cardiac Rehabilitation
maintenance phase consists of home- or gymnasium-based exercise with the goal of continuing the risk factor modification and exercise program learned during phase II