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aortic aneurysm
50% increase in normal diameter or >3cm in size
pseudo/false aneurysm
collection of blood/CT outside aortic wall
AAA
abd/back pain
pulsatile abdominal mass
aortic rupture
pulsatile abdominal mass
hypotension
sudden, severe abdominal pain
gold standard dx AA and aortic dissection
CTA
aortic dissection
MCC acute HTN emergency or chronic HTN
DeBakey
1: ascend and descend
2: ascend
3: descend, proximal to celiac artery
4: descend, distal to celiac artery
stanford
1: ascend +/- descend
2: only descend
aortic dissection
sudden, severe chest/back pain
hypo or hypertension
pulse/BP differences between limbs
PAD (chronic arterial insufficiency)
MCC tissue ischemia
PAD (chronic arterial insufficiency)
pulses decreased/absent
cool, pale skin
dusky red (rubor) on dependency
no edema
trophic skin changes
ulcer on toes
chronic venous insufficiency
MCC venous hypertension
chronic venous insufficiency
normal pulses
petechiae/brown pigmentation
marked dependent edema
ulcer on medial ankle
telangiectasias
PAD
ABI ≤ 0.9
acute arterial occlusion of a limb
MCC embolism
in situ PAD
acute arterial occlusion of a limb
5 Ps:
-pallor
-pain/ paresthesias (numbness/tingling)
-pulseless
-paralysis (muscles w/ no perfusion)
-polar (cold)
acute arterial occlusion of a limb
arterial US/doppler
virchow's triad
stasis blood flow
endothelial injury
hypercoagulability
DVT
unilateral
pain
swelling
red
warm
tender
DVT
+ homan's sign
superficial thrombophlebitis
pain/tender along vein
warm
red
hard vein
varicose veins
uni or bilateral
dull, ache, heavy
venous stasis skin changes (red-purple)
R sided endocarditis
IVDU
tricuspid
SA
L sided endocarditis
dental procedures
mitral valve
strep viridans, SA
endocarditis
F:ever (MC)
R:oth spots
O:sler nodes
M:umur (tricuspid regurg)
J:aneway lesions
A:nemia
N:ail bed hemorrhages
E:mboli
myocarditis
infectious or non infectious (lupus) causes
myocarditis
days-weeks after URI
new onset HF
pericardial friction rub
myocarditis
definitive: endomyocardial biopsy
dressler syndrome
persistent fever, tachycardia, pulsus paradoxus
rheumatic fever
pericarditis
erythema marginatum, SQ nodules
sydenham chorea
polyarthritis
pericarditis
pleuritic chest pain
worse w/ deep breathing
better leaning forward/sitting up
friction rub > LSB
pericarditis
diffuse ST ↑ and PR↓ in same leads
flat, inverted T waves
CXR: water-bottle sign
pericarditis
colchicine + NSAIDs x3 months
pericardial effusion
muffled heart sounds
low voltage QRS, alternans
CXR: water-bottle sign
cardiac tamponade
d/t malignancy, infection, or trauma
cardiac tamponade
Beck's Triad
pulsus paradoxus
tachycardia
cardiac tamponade
low QRS voltage, alternans
echo: swinging in pericardial cavity
CHF
1) primary insult
2) compensatory mechanism
3) maladaptation
RHF
systemic sx
↑JVD
bilateral LE edema
hepatomeg
nocturia
LHF
pulmmonary sx
cough (frothy)
SOB
bilateral basilar crackles
S3 gallop
displaced PMI
CHF
CXR: cardiomeg, pulmonary vessel cephalization, kerley B-lines, pleural effusions
HFrEF
LVEF <40%
previously systolic HF > ejection problem
HFpEF
LVEF >50%
previously diastolic HF > filling problem
ACC/AHA stage A
at risk HF
tx HTN and DM prn
ACC/AHA stage B
pre-HF (no s/s, but structural heart disease)
ACEI/ARB, BB
SGLT2 if DM
ACC/AHA stage C
structural heart disease and current/past HF sx
ARNI
BB
spironolactone
SGLT2
ACC/AHA stage D
refractory sx or recurrent admission d/t HF
heart transplant, LVAD, palliative care
BB in HF
carvedilol
bisoprolol
metoprolol succinate XL
ARNI in HF
sacubitril/valsartan (entresto)
MRA/aldosterone antagonist in HF
spironolactone
eplerenone
SGLT2 in HF
dapagliflozin (farxiga)
empagliflozin (jardiance)
tx acute CHF exacerbation
loops
vasodilator (nitro)
inotropes (dobutamide)
morphine
DO NOT START BB
type 1 MI
MI d/t CAD w acute thrombosis
type 2 MI
MI d/t increased O2 demand or decreased O2 supply
ACS initial tx
1) ASA 162-325mg IR tablet, chewable, non-coated (plavix if asa CI)
2) SL nitro
3) O2 prn
STEMI
ST elevation or new LBBB or new Q waves
elevated troponins
NSTEMI
normal, inverted T waves, or ST depression
elevated troponins
unstable angina
normal, inverted T waves, or ST depression
normal troponins
PCI
tx NSTEMI if <90 mins needle to door
fibrinolytics
tx NSTEMI if <30 mins needle to door
PCI
NSTEMI tx when 1-3 arteries are narrowed
CABG
NSTEMI tx when significant narrowing of LAD or narrowing of all 3 major coronary arteries
unstable angina
MCC: non-occlusive thrombus
CAVS
crushing chest pain relieved with nitro, cocaine can cause
CAVS
ergonovine challenge during angiography
liver
makes cholesterol
total cholesterol
<200
LDL
<160
<130 (mod risk heart disease)
<100 (heart disease, or DM, or high risk)
<70 (heart disease + DM)
HDL
≥60
M<40, F<50 = increased risk heart disease
triglycerides
<150
dyslipidemia
screening:
20-39 w/o known CVD if high risk
40-75 w/o known CVD (q5 years prn)
≥75: insufficient evidence
high intensity statins
atorvastatin 80 mg
rosuvastatin 20 mg
significant ASCVD and >75 y/o
mod statin
significant ASCVD and <75 y/o
high statin
40-75
LDL: 70-189
ASCVD ≥ 7.5%
mod-high statin
40-75 and LDL ≥90
OR
40-75, hx DM, and LDL: 70-189
high statin
MC type of heart disease
CAD
stable ischemic heart disease (SIHD)
stable angina
chest pain worse w exertion
relieved by nitro
2 months duration
CAD
gold standard dx: cardiac cath
SIDH
GDMT:
1) asa (81 mg)
2) BB
3) nitro prn
4) mod-high statin
SIDH if still sx after GDMT
titrate BB up
consider CCB and long-acting nitrates
secondary prevention of CAD
1) ASA
2) rivaroxaban (pts who need)
3) ACEI/ARBs
4) SGLT2 or GLP1 (if DM)
dilated CM
MC type of cardiomyopathy
dilated CM
ventricles enlarged
systolic dysfx
= congestive HF
restrictive CM
stiff myocardium, chambers same size
restrictive CM
MCC: amyloidosis
restrictive CM
HF
newly dx arrhythmia
S4
restrictive CM
echo: evidence of diastolic dysfx with restricted filling
CMRI: diffuse hyperenhancement of gadolinium image
cath: dip and plateu, square root sign
hypertrophic CM
LV wall > 1.5cm thick
hypertrophic CM
auto dominant
mitral regurg
young athletes > sudden death
loud S4
bifid pulse
tako-tsulo syndrome
ST↑, deep anterior T wave inversion
echo: LV apical dyskinesia