Adaptations to Aerobic and Anaerobic Training

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35 Terms

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cardiorespiratory endurance

- Ability to sustain prolonged, dynamic exercise

- Improvements through multisystem adaptations (cardiovascular, respiratory, muscular, metabolic)

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endurance training

maximal endurance capacity = á VO2max

submaximal endurance capacity:

• Lower HR at same submaximal exercise intensity

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heart size

– With training, heart mass and LV chamber size

plasma volume = LV volume = EDV =  SV

– Volume loading effect vs pathology of hypertrophy from hypertension

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SV ↑ after training

– Resting, submaximal, maximal

–Plasma volume with training = EDV =         preload

– Resting and submaximal HR with training =   filling time = EDV

LV mass with training = force of contraction

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resting HR

– ↓ markedly (~1 beat/min per week of training)

– ↑ parasympathetic, â sympathetic activity in heart

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submaximal HR

– ↓ HR for same given absolute intensity

– More noticeable at higher submaximal intensities

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maximal HR

– No significant change with training (may ↓ slightly)

– ↓ with age

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HR recovery

– Faster with training

– Indirect index of cardiorespiratory fitness

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Cardiac output (Q)

– Little or no change at rest or during submaximal exercise with training

Maximal Qconsiderably (due to SV)

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fiber type

size and number of type I fibers

–Type IIa perform more like type I

–Type IIx may perform more like type IIa

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capillary supply

number of capillaries supplying each fiber

–May be key factor in VO2max

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myoglobin

myoglobin content by 75% to 80%

– Supporting oxidative capacity in muscle

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respiratory exchange ratio (RER)

at both absolute and relative submaximal intensities

dependent on fat, dependent on glucose

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lactate threshold

- ↑ to higher percentage of VO2max  

- ↓ lactate production, lactate clearance

– LT occurring at approx. same absolute [LA]

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resting and submaximal VO2

– Resting VO2 unchanged with training

– Submaximal VO2 unchanged or slightly with training

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Maximal VO2 (VO2max)

–Best indicator of cardiorespiratory fitness

substantially with training (15%-20%)

due to cardiac output and capillary density

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more

_ people over age 50 are engaged in sport and exercise today than 30 years ago.

– Recreation

– Competition

– More fit than older sedentary counterparts

• Performance declines with age.

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height with age

– Starting at 35-40 years

– Compression of intervertebral disks

– Poor posture

– Later: osteopenia, osteoporosis

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weight , then

- ↑ 25-45 years: physical activity, caloric intake

65+ years: loss of body mass, appetite

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body fat content

• tends to increase

– Active versus sedentary older adults: variation

– Older athletes body fat content

– Older athletes central adiposity

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fat free mass

- starting around age 40.

– Due (in part) to lack of activity

muscle, bone mass

– Sarcopenia (protein synthesis )

growth hormone, insulin-like growth factor 1

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bone mineral content

decreases

– Bone resorption > bone synthesis

– Due to lack of weight-bearing exercise

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body comp variables

– Body weight

– Percent body fat

– Fat mass

– Fat-free mass (FFM)

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training

• alters age-related changes in body composition.

- ↓ weight, percent body fat, fat mass

FFM (more likely with resistance training than with aerobic training)

– More in men than in women

• Biggest results come with diet + exercise.

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strength and neuromuscular function

decreases with age

– Interferes with activities of daily living.

– Manifests at about 50-60 years of age.

– Results from muscle mass.

• Strength offset by resistance exercis

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type ii fiber loss

• occurs with aging

– Decrease in type II motor neurons

– Higher percent type I fibers

• Training slows fiber-type change.

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size and number of muscle fibers

with age

– Size of both type I and type II

– Loss of 10% per decade after age 50

• Endurance training → no impact on decline in muscle mass with age

• Resistance training = muscle atrophy, muscle cross-sectional area

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reflexes

• slow with age.

– Exercise preserves reflex response time.

– Active older people ≈ young active people.

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motor unit activation

with age.

– Fewer MU activated = Force production

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exercise

• maintains muscle physiology.

– Number of capillaries is unchanged.

– Oxidative enzyme activity is only mildly reduced.

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mitochondrial function

• declines with age.

– Reduced mitochondrial protein synthesis

– Reduced maximal rate of ATP production

• May contribute to muscle atrophy.

– Increased intracellular oxidative stress interferes with myofilament function.

• Increased free radicals with aging

• Improved by exercise training.

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central and peripheral cardiovascular

decrements with age

• Reduced maximal HR

– Reduction varies considerably

– Electrical changes with age

– Increased parasympathetic NS influence

– Same for active and sedentary people

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maximal stroke volume (SV)

decreases with age

↓ contractility, response to catecholamines

– LV, arterial stiffening

– Decline in SVmax attenuated by exercise

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VO2max

with age due to â Qmax

– Due more to â HRmax, less to â SVmax

- Decline inVO2maxattenuated by exercise

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sedentary habits

• ↑ risk for vascular aging

– ↓ cardiac and arterial compliance

– ↑ narrowing of arteries

– ↑ vasodilation