16: respiratory emergencies 17: cardiovascular emergencies
the most common symptom of a respiratory emergency is:
shortness of breath
the respiratory system can be divided into three portions:
upper airway
lower airway
lungs and accessory structures (work with first two portions to allow the oxygenation of body cells and the elimination of carbon dioxide from the bloodstream
the upper and lower airway are separated by:
the vocal cords (or glottic opening)
what is the primary purpose of the upper and lower airways?
the conduction of air into and out of the lungs
normal breathing is defined differently for each individual patient group based on:
age
preexisting disease
the following findings are consistent with a patient who is breathing adequately:
an intact (open) airway
normal respiratory rate
good rise and fall of the chest
normal respiratory rhythm
breath sounds that are present bilaterally
chest expansion and relaxation that occurs normally
minimal-to-absent use of accessory muscles to aid in breathing
if no other condition or injury is involved, the following should also occur in a patient who is breathing adequately:
(along with open airway, normal respiratory rate, chest rise/fall, breath sounds, etc.)
normal mental status
normal muscle tone
normal pulse ox reading
normal skin condition findings
abnormal factors that are present in certain pulmonary (lung) conditions can decrease the efficiency of gas exchange across the alveolar-capillary membrane, including the following:
increased width of the space between the alveoli and blood vessels
lack of perfusion of the pulmonary capillaries from the right ventricle of the heart
filling of the alveoli with fluid, blood, or pus
T/F: the inspiratory and expiratory centers in the medulla and pons in the brainstem are accessory structures that are part of the respiratory system
true - they exert nervous control of breathing
receive info about the oxygen and carbon dioxide content of the bloodstream from special sensors in the vascular system
stretch receptors in the walls of the lungs provide information to the brainstem to prevent accidental overexpansion injuries, and irritant receptors in the walls of the bronchioles detect the presence of abnormalities such as excessive fluid, toxic fumes, or smoke, or significant air temperature changes
_____ receptors (near the alveoli) can play a role in the feeling of shortness of breath the patient can experience and can also promote shallow and rapid breathing
juxta-capillary
detect when the alveolar-capillary beds become abnormally engorged with blood because of heart failure
to achieve the most accurate interpretation of breath sounds, it is important to auscultate in the appropriate fashion:
whenever feasible, have the patient sit upright and, while using the diaphragm end of your stethoscope over bare skin, instruct the patient to take a deep breath through the mouth and breathe out passively through the mouth
(you may need to instruct the patient a few times to make no airway or vocal sounds while he does this)
ensure the tubing of the stethoscope does not touch any surface because this can produce extraneous sounds and be mistaken as abnormal breath sounds
place the head of the stethoscope on the patient’s thorax, and listen through the entire phases of inhalation and exhalation
if necessary, listen to a few of the pts breaths at each auscultation location to ensure your interpretation of any abnormal breath sound
finally, listen to sounds on one location of the body and then listen to the exact location on the other side (mirror location)
before continuing
T/F: never auscultate over clothing
true
three basic types of abnormal breath sounds that are early indicators of impending respiratory distress include:
wheezing
ronchi
crackles
wheezing
high-pitched, musical, whistling sound that is best heard initially on exhalation but can also be heard during inhalation in more severe cases
indication of swelling and constriction of the inner lining of the lower airways (primarily the bronchioles
usually heard in asthma, emphysema, and chronic bronchitis, pneumonia, congestive heart failure, and other conditions when they cause bronchoconstriction
T/F: in wheezing patients with severe obstruction of the lower airways by bronchoconstriction and inflammation, wheezing can be significantly diminished or absent
true - because the velocity of air movement through the bronchioles is no longer sufficient to produce the wheezing sound
ronchi
AKA course crackles
snoring or rattling noises heard on auscultation
indicate obstruction of the larger conducting airways of the respiratory tract by thick secretions of mucus
often heard in chronic bronchitis, emphysema, aspiration, and pneumonia
a characteristic: quality of sound changes if the person coughs or sometimes even when the person changes position
crackles
AKA rales
bubbly or crackling sounds heard during inhalation
associated with fluid that has surrounded or filled the alveoli or small bronchioles
crackling sound is commonly associated with the alveoli and terminal bronchioles “popping” open with each inhalation
bases of the lungs posteriorly reveal crackles first because of the natural tendency of fluid to be puled downward by gravity
can indicate pulmonary edema or pneumonia
this type of breath sound typically does not change with coughing or movement
T/F: both ronchi and crackles can change in sound quality of the person coughs or when they move/change position
false - only ronchi is has this characteristic while crackles typcally don’t
wheezing can indicate
asthma
emphysema
chronic bronchitis
pneumonia
congestive heart failure
other conditions causing bronchoconstriction
ronchi (course crackles) can indicate
chronic bronchitis
emphysema
aspiration
pneumonia
crackles (rales) can indicate
pulmonary edema
pneumonia
failing to breath adequately, even for short periods of time, can result in hypoxemia
hypoxemia is:
decreased oxygen in the blood stream
failing to breath adequately, even for short periods of time, can result in hypercarbia
hypercarbia is:
increased carbon dioxide in the blood stream
dyspnea
shortness of breath
apnea
complete respiratory arrest (no longer breathing)
hypoxia is:
when the cells of the body are not getting an adequate supply of oxygen in which cellular dysfunction and death occur
indications of hypoxia
shortness of breath
abnormal upper airway sounds
faster/slower breathing rates
poor chest rise/fall
other symptoms of respiratory distress
T/F: if adequate breathing and gas exchange are not present, the lack of oxygen causes the body cells to begin to die
true - some cells become irritable when they are hypoxic, causing the cells to function abnormally
ex: hypoxic cardiac cells become irritable and begin to send out abnormal impulses, leading to cardiac dysrhythmias (abnormal heart rhythms)
the following findings commonly occur in pts with respiratory distress
subjective complaint of shortness of breath
restlessness and anxiety
tachycardia (early finding) or bradycardia (later finding)
tachypnea
pale, cool, clammy skin (early finding) or cyanosis (later finding)
abnormal respiratory pattern
wheezing, ronchi, or crackles
difficulty or inability to speak
muscle contractions
altered mental status (anxiety and confusion = early finding; aggressive or complacent behavior and sleepiness = later finding)
abdominal breathing
excessive coughing
tripod position
pulse ox reading <94%
tachycardia is a _______ finding with respiratory distress
early
bradycardia is a ______ finding with respiratory distress
later
pale, cool, and clammy skin are __________ findings with respiratory distress
early
cyanosis is a ______ finding with respiratory distress
later
bronchoconstriction
significant narrowing of the lower airways, which include the bronchi and bronchioles, from inflammation, swelling, or constriction of the muscle layer
AKA bronchospasm
bronchoconstriction - this narrowing causes a drastic increase in:
resistance to airflow in the lower airways
makes inhalation and particularly exhalation extremely difficult and producing wheezing
beta 2 agonist bronchodilator
in aerosol form
can be inhaled during episode of breathing difficulty caused by bronchoconstriction
designed to relax the bronchi and bronchiole smooth muscle, causing dilation, which results in a decrease in airway resistance and increase in the effectiveness of moving air in and out of the alveoli, better gas exchange and a relief from the S&S
breathing difficulty can also be a symptom of injuries to:
the head. face, neck, spine, chest, or abdomen
(can also be caused by cardiac compromise, hyperventilation, associated with emotional upset, and various abdominal conditions)
cardiac compromise
when the heart isn’t getting enough oxygen
T/F: the sensation of shortness of breath occurs when the metabolic demands of the body are not met
true - this typically results from an inadequate amount of oxygen delivered to the cells to allow for normal aerobic metabolism to continue
the sensation of shortness of breath occurs when the metabolic demands of the body are not met. Dyspnea is usually caused by one of the following:
mechanical disruption to the airway, lung, or chest wall that prevents effective mechanical ventilation
examples of conditions that can cause a disruption in mechanical ventilation include:
airway obstruction
flail chest
chest muscle weakness
neuromuscular disease
lung collapse
stimulation of the receptors in the lungs. Such stimulation produces a sensation of shortness of breath.
conditions that stimulate the receptors include:
asthma
pneumonia
congestive heart failure
inadequate gas Exchange at the level of the alveoli and capillaries causing a decrease in the oxygen content in the blood or a rise in the level of carbo dioxide
this can occur because of any of the following:
ventilation disturbance
perfusion disturbance
both a ventilation and a perfusion disturbance
a gas exchange disturbance
flail chest
2 or more adjacent ribs fractured in two or more places
dyspnea can be caused by inadequate gas exchange at the level of the alveoli and capillaries causing a decrease in the oxygen content in the blood (hypoxemia) or a rise in the level of carbon dioxide in the blood (hypercarbia).
this can occur because of:
a ventilation disturbance
an inadequate amount of oxygen-rich air entering the alveoli and passing across the alveolar membrane to the capillary
a perfusion disturbance
an inadequate amount of blood traveling through the pulmonary capillaries which decreases the number of red blood cells available to pick up the oxygen and transport it to the cells
both a ventilation and a perfusion disturbance
in the lungs, leading to hypoxemia and hypercarbia
a gas exchange disturbance
from an increased amount of interstitial fluid in and around the alveoli
T/F: regardless of the cause, a complaint of breathing difficulty requires your immediate intervention. If severe hypoxia is present, time is critical because of the detrimental effects of severely or prolonged low oxygen levels on all cells and organs.
true
A breathing disturbance in a patient can be categorized in one of three ways:
respiratory distress
respiratory failure
respiratory arrest
it is critical during your primary assessment that you immediately identify in which category the breathing disturbance falls. This is a life-threatening condition, and your assessment must be made decisively and quickly
a patient who has difficulty breathing but has an adequate tidal volume and respiratory rate is said to be in
respiratory distress
typically, a pt in this condition has a normal minute ventilation compensation in the tidal volume (breathing deeper) and/or the respiratory rate (breathing faster)
because the tidal volume and respiratory rate are still adequate, the pt is compensating but there are still signs of respiratory distress so you should still administer oxygen
in a patient experiencing respiratory distress, you should administer oxygen via
nasal cannula at 2 lpm to increase or maintain the SpO2 reading at 94% or higher
oxygenation should be based on the patient’s oxygenation status as measured and primarily guided by the pulse oximeter instead of using predetermined devices and flow rates for all patients
a patient who presents with moderate to severe respiratory distress and who is awake and alert can benefit from
continuous positive pressure ventilation (CPAP)
a patient in respiratory distress presenting with a severely decreased SpO2 reading and obvious signs of severe hypoxia may benefit from
higher concentrations of oxygen delivered by nonrebreather mask at 15 lpm
T/F: the SpO2 in pregnant women should be maintained at the highest level possible
true - to maintain adequate oxygenation of the fetus
these pts must receive a high concentration of oxygen via a NRB mask regardless of the SpO2 reading
respiratory failure
when the tidal volume or respiratory rate is adequate and no longer can provide an adequate oxygenation of the cells
inadequate tidal volume
or an inadequate respiratory rate
or both
in a pt experiencing respiratory failure, if the tidal volume decreases or the respiratory rate increases or decreases significantly, you must
provide immediate PPV and oxygenation with a BVM device or other ventilation device with supplemental oxygen
if a pt with inadequate breathing is not treated promptly, it is likely that he will deteriorate to respiratory arrest
respiratory arrest
the complete cessation of breathing effort or the patient experiences agonal breathing
no tidal volume
no respiratory rate
pt may have agonal respirations in which there is a sudden gasping respiratory for a long period of apnea
agonal breathing occurs when
the brainstem reflex causes a gasping breath in an otherwise apneic patient
T/F: respiratory arrest can lead to cardiac arrest in minutes if not properly managed
true - because of a lack of oxygen delivery to the brain and heart
your priority in treating patients with breathing difficulty is to
determine if the pt is in respiratory distress and in need of only oxygen therapy
or
if he is in respiratory failure or respiratory arrest in which he needs immediate ventilatory assistance with a BVM or other ventilation device and supplemental oxygen
T/F: respiratory arrest and failure are treated the same way
true - with positive pressure ventilation and supplemental oxygen (because the tidal volume and/or rate is inadequate - they don’t only need oxygen, they also need help to actively breath)
treat patients in respiratory arrest by
immediately beginning PPV with a BVM and supplemental oxygen connected to the BVM
treat patients in respiratory failure by
immediately beginning PPV with a BVM and supplemental oxygen connected to the BVM
treat patients in respiratory distress by
providing supplemental oxygen to maintain an SpO2 95% or greater
respiratory distress
adequate tidal volume and adequate respiratory rate that produces an adequate minute and alveolar ventilation