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Growth hormone (GH)
anterior pit
increases
growth
Free Fatty Acid (FFA) mobilization
gluconeogenesis: synthesizing glucose from noncarbohydrate precursors (lactate, glycerol, amino acids) primarily in the liver and kidney to maintain blood glucose levels during fasting or intense exercise
decreases
glucose uptake
Controed by: Hypothalamic GH-releasing hormone,
inhibits: hypothalamic somatostatin
Stimulation: exercise, stress, low blood glucose
Exercise: increaseses GH
Adrenocorticotrophic Hormone (ACTH)
anti pit
increase
synthesis & secretion of cortisol
controlled by :
hypothalamic ACTH-releasing hormone
stimulation:
stress
bone break
heavy exercise
burns
Exercise effect unknown
Antidiuretic Hormone (ADH) or Vasopressin
posterior pit
decreases
water loss at kidneys
increases
peripheral resistant (resistant against water retention)
controlling factors:
hypothalamic neurons
stimulation;
plasma volume
plasma osmolarity
ADH increases when exercising
cortisol
anti-pit
adrenal cortex
increases
glconeogenesis
free fatty acid mobilization
protein synthesis
decreases
glucose utilization
controlling factors:
ACTH
stimulation
stress
bone breaks
heavy exercise
burns
High-intensity exercise increases (spikes) cortisol levels
Light-intensity exercise decreases cortisol levels
Aldosterone
adrenal cortex
increases
potassium secretion
Sodium reabsorption at kidney
controlling factors:
plasma potassium concentration
reninangiotensin system
stimulation
low blood pressure and plasma volume
elevated plasma potassium
sympathetic activity to kidney
Exercise: increases aldosterone
epinephrine/neuroepinephrine
adrenal gland
adrenal medulla
increases:
glycogenolysis
free fatty acid mobilization
heart rate
stroke volume
peripheral resistance
controlling factors:
output of baroreceptors
glucose receptor in the hypothalamus
brain and spinal centers
stimulus
low blood pressure
low blood glucose
too much emotional stress
Exercise increases epinephrine/neuroepinephrine
insulin
pancreas
increases
glucose amino acid
free fatty acid uptake into tissue
controlling factors
plasma glucose and amino acid concentrations
autonomic nervous system
stimuli
elevated
plasma
glucose
amino acid concentrations
decreased by :
epinephrine/norepinephrine
Exercise decreases insulin
glucagon
pancreas
increases
glucose
free fatty acid mobilization
gluconeogenesis
controlling factors
plasma glucose
amino acid concentrations
autonomic nervous system
stimuli
low plasma glucose
low amino acid concentration
elevated epinephrine/norepinephrine
Exercise increases glucagon