Anaerobes; meningococci

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B. fragilis

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31 Terms

1

B. fragilis

gram negative opportunistic bacteria that has B-lactamases that confer penicillin resistance

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2

P. melaninogenica

gram negative diagnostic mark for oral squamous sarcoma

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3

P. melaninogenica and P. intermedia

gram negative, play a role in chronic adult periodontitis

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4

P. denticola

gram negative, involved in periodontal disease and can lead to soft tissue infections

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5

fusobacterium

possess potent endotoxin, normal inhabitant in oral, GI, and female genital tract

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6

F. nucleatum

commonly found in Acute Necrotizing Ulcereativee Gingivitis (ANUG), oral infections, lung abscess, and atherosclerotic plaques

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7

Acute Necrotizing Ulcerative Gingivitis (ANUG)

bleeding gingiva with abrupt onset, malaise/fever, excessive salivation and foul breath typically seen in people under 35

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8

gentle debridement, mouth rinses, antibiotics

treatment for acute necrotizing Ulcerative gingivitis

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9

survival and replication

significance of spore forming property of Clostridium

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10

C. Perfringens

Which organism is mainly responsible for causing gas gangrene?

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11

C. tetani

blocks inhibitory neurotransmitters, can be neonatally transferred through umbilical cord, immunizations only treatment option

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12

C. botulinum

block release of acetylcholine, causes paralysis of muscles, associated with infant botulism

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13

actinomyces

part of normal flora in oral, urinogenital, and GI tract, pathogeenic after trauma (ex: extraction)

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14

cerevicofacial actinomyces

pyogenic abscess “sulfur granules” may form, most commonly caused by A. israelii

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15

activation of the complement system

how does the immune respond to neisseria

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16

IgA protease and ploysaccharide capsule

virulence factors of N. meningitidis

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17

Ig A protease

virulence factor of N. gonorrhoeae

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18

meningitis

inflammation of the meninges

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19

H. influenzae, Strep. pneumoniae, Neisseria meningitidis

bacteria that cause bacterial meningitis

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20

through blood (hematogenous) or direct spread (i.e. surgery)

how can bacteria like neisseria meningitidis get into the CSF

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21

colonization, invasion with IgA protease, survival though capsule, crossing blood brain barrier

how does N. menengitidis enter the CSF in a hematigenous manner

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22

stiff calfs, can’t bend knees

kernigs sign of meningeal inflammation

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23

stiff neck, legs fold when neck bends

bruzinski’s signs of meningeal inflammation

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24

encephalitis

inflammation of brain parynchyma

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25

Meningoencephalitis

inflammation of meninges and parynchyma

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26

WBC increase

CSF cell count change with meningitis

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27

elevated protein

CSF protein change with meningitis

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28

low glucose

CSF glucose change with meningitis

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29

conjugate vaccine, mask, chemoprophylaxis after close exposure

strategies for preventing meningitis

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30

clostridium, actinomyces, lactobacillus

gram positive anaerobic bacteria

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31

Bacteroides, prevotella,fusobacterium, porphyrromonas

gram negative anaerobic bacteria

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