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Parameters for Orthostatic hypotension
SBP should decrease 20 mmHg or more from supine to standing and/or DBP decreases 10 mmHg with 10-20% increase in HR
Troponin
enzyme released from damaged/dead heart muscle that is used in diagnosis of acute coronary syndrome (MI)
Cholesterol level is used for what
Screening of heart disease
Expected reference range for cholesterol
less than 200 mg/dL
LDL is
Bad cholesterol, transports cholesterols to the body's cells from the liver
LDL expected range
Less than 130 mg/dL
Triglyceride levels are used to measure?
Client's risk for heart disease
Triglyceride expected range? (Male and female)
Male: 40 - 160 mg/dL
Female: 35 - 135 mg/dL
HDL is?
Good cholesterol, protects coronary arteries from heart disease by transporting cholesterol from body's cell to liver
HDL expected ranges for male and female?
Female: Greater than 55 mg/dL
Male: Greater than 45 mg/dL
Chest x-ray
Useful for evaluating heart failure and pulmonary edema
What is a transthoracic echocardiogram used for
a non invasive way to measure ejection fraction (Percentage of blood expelled from left ventricle)
-Patient lays on left side for about an hour
Normal Ejection fraction
65%, 35% of blood stays in ventricle
Stress echocardiogram
Examines the coronary arteries efficiency when the heart is placed under stress (exercise or drug induced)
Transesophageal echocardiogram
Ultrasound probe is inserted into the esophagus then pointed towards the heart
(requires moderate sedation)
What does the transesophageal echocardiogram do
removes interference from chest wall and lungs, and can help visualize valve disorders and thrombi
electrocardiogram is used for what
Monitoring electrical activity of the heart
- Used to diagnose dysrhythmias, chamber enlargement, myocardial ischemia, injury or infarction, monitor effects of electrolyte imbalances
- ECG is priority assessment when patient states chest pain
Which is better 12 lead to 3-5 lead
12 lead is for more detailed diagnostic
Exercise/medication stress test
Evaluates heart's response to physical stress in context of cvd
Coronary angiography and angioplasty
Insertion of a catheter through a major vein or artery into the heart or coronary arteries. Angioplasty is the inflation of a balloon and metal stent to hold the vessel open
- Uses contrast medium and fluoroscopy to identify coronary blockages and evaluate blood flow in coronary arteries
Angiography and angioplasty uses which arteries
Femoral, brachial, radial
Nursing considerations before and after coronary angiography and angioplasty
Before: NPO for 8 hours before, evaluate renal function tests, hold metformin 48 hrs before and after contrast
After: Reperfusion can cause dysrhythmias for hours after procedure, watch for hematoma at the insertion site, patient must keep extremity straight and avoid hip flexion for 6 hours after procedure
- patient with stent will require anticoagulation for 12 months
Complications of coronary angiography and angioplasty
Hematoma at insertion site (usually groin)
(Perform neurovascular assessment)
- Embolism
- Kidney injury
- Cardiac dysrhythmias
Risk factors for atherosclerosis (Modifiable and Non modifiable)
Modifiable: Hypertension, poor diabetes management, total cholesterol greater than 200, LDL greater than 130, HDL less than 45-55, triglycerides greater than 160, metabolic syndrome, obesity, smoking, alcohol
Non Modifiable: age, ethnicity, gender (Men at greater risk than women), genetics, family htx, family htx
Statins (Simvastatin, Atorvastatin, Rosuvastatin)
Cholesterol medication
Inhibits synthesis of cholesterol in liver, decreases LDL and increases HDL
What to monitor for when taking Statins?
Liver function tests (ALT and AST) and for rhabdomyolysis (Muscle pain and dark urine)
-PTT may increase with combined use of warfarin
Fibric acid derivatives?
Fenofibrate and Gemfibrozil
- Used to remove very LDL
- Lowers triglycerides increases HDL
What to monitor for when takin fibric acid derivatives
S/S of bleeding with combined use of warfarin
- Increased risk for rhabdomyolysis and elevated liver enzymes when combined with statins
Antiplatelets
Aspirin: Decreases platelet coagulation
What to monitor for when taking aspirin
GI upset, GI ulceration, tinnitus
Peripheral arterial disease
Progressive thickening and narrowing of arterial walls due to atherosclerosis
Chronic venous insufficiency
trapping and stasis of blood in the extremities due to faulty valves and endothelial injury by inflammation
Arteriography
Injection of contrast medium to visualize areas of decreased arterial flow
Exercise stress test
Claudication (calf cramping) during exercise can indicate peripheral arterial disease
Ankle brachial index (ABI)
A doppler is used to determine ankle systolic BP that is divided by brachial systolic BP
What is the expected normal range for PAD when doing the ABI
0.9-1.3, less than 0.9 in either leg is diagnostic for PAD
Risk factors for PAD
HTN, hyperlipidemia, DM, smoking, obesity, sedentary lifestyle, age over 65, genetic predisposition
Signs and symptoms of PAD
Intermittent claudification
- Numbness of burning pain, especially when legs elevated
- Relief of pain when legs are dangled/dependent
- Delayed cap refill
- Weakened non palpable pulses
- Loss of hair on calf, ankle, shin
- Dry, scaly, taut shiny skin
- Pallor when elevated
- Redness of extremity when dangled
- Muscle atrophy
- Ulcerations that look hole punched round and smooth
Medications for PAD?
Aspirin and statins
patient education for PAD
Encourage heat but not directly on extremity
- Avoid cold, stress, caffeine, nicotine, crossing legs, socks that are too tight
- Do not elevate legs above level of the heart, DO dangle to increase blood flow
Manifestations of Chronic venous insufficiency
Brown leathery skin, edema, ulcers over medial malleolus that are irregular in shape, pain
nursing care for chronic venous insufficiency
Wound care (hydrocolloid dressings), elastic stockings, avoid standing or sitting for prolonged periods, elevate legs above level of heart for at least 20 minutes 4-5 times per day,
Set of conditions that lead to VTE formation
Virchow's triad
Venous stasis, endothelial damage, hypercoagulability
Venous stasis
Dysfunctional valves and backward blood flow and/or inactive extremity muscles cause pooling of blood in extremity
Endothelial damage
From chronic inflammatory states, damage activates platelets and clotting factors
Hypercoagulability
Imbalance in clotting mechanisms by almost any inflammatory condition
Nursing care for VTE
No SCD if active DVT, avoid massaging site, elevate legs for edema, patient should ambulate, administer anticoagulation meds, monitor signs for P.E
Heparin
Inhibits thrombin and multiple clotting factors
How is heparin administered
IV (For active DVT) or SQ (Prevention)
What to monitor for when taking heparin
aPTT, platelet counts for heparin induced thrombocytopenia
Antidote to heparin in case of excessive bleeding
Protamine sulfate
Low molecular weight heparin
Enoxaparin, can be used at home
How is low molecular weight heparin administered
SQ
Warfarin
Interferes with vitamin K effects on clotting
How is warfarin administered
Oral
What to monitor for when taking warfarin
Therapeutic effect may take up to 5 days, overlap heparin/Lovenex while starting
- Monitor PT and INR
Antidote for warfarin
Vitamin K
Nursing care of patients on anticoagulants
Bleeding precautions, electric shavers/clippers, soft and gentle toothbrush, signs of GI bleed to report, avoid aspirin, ginko, garlic, NSAIDs, avoid forcefully blowing nose, stool softeners to avoid straining
- Avoid leafy green vegetables if taking warfarin
- Avoid IM and venipunctures
- Assess for unexplained mental status changes as this can indicate cerebral bleeding
Primary HTN
Idiopathic (no known cause)
Secondary HTN
Identifiable cause of hypertension
Elevated BP
120-129/less than 80
Stage 1 HTN
130-139/80-89
Stage 2 HTN
Over 140/ Over 90
Risk factors for HTN
Age, alcohol, diabetes, elevated lipids, ethnicity, excess sodium, family htx, gender, obesity, sedentary lifestyle, stress, tobacco use
Causes of secondary HTN
Diabetes, kidney disease, corticosteroid use, pregnancy, oral contraceptives, endocrine disorders, many prescription and illicit drugs
S/S of HTN
Bounding pulse, headache, facial flushing, dizziness, fainting, retinal/vision change, nocturia, proteinuria
Purpose of diuretics
Promotes sodium and water excretion, reduce plasma volume and reduce vascular response to catecholamines
What does thiazide diuretics cause
Hydrochlorothiazide- Hypokalemia and hypercalcemia
Loop diuretics causes waht
Furosemide- Hypokalemia, greater diuretic effect than thiazide but shorter duration
Potassium sparing diuretics can cause?
Spironolactone- Hyperkalemia
What to monitor when taking diuretics
K, Na, Ca levels, orthostatic hypotension, assess for muscle weakness, irregular pulse, dehydration and Intake and output
Ace inhibtors (Pril)
What to monitor for when taking ACE inhibitors
Angiotensin 2 receptor blockers (ARBs) (sartans)
patient education for ARBs
Beta blockers (lol)
What to monitor for when taking beta blockers
Direct vasodilators
Hydralazine and Nitroglycerine
Hydralazine
Nitroglycerine
Stable angina
Intermittent chest pain that occurs over a long period with the same pattern of onset, duration, and intensity
-Duration less than 15-20 mins
What triggers stable angina
Exercise, emotional stress
What relieves stable angina
Rest or nitroglycerin
patient teaching for nitroglycerin
Give sublingually as tablet or spray when pain occurs
- If no relief of symptoms in 5 minutes after first dose, call 911
-May repeat every 5 minutes for max of 3 doses
- Cannot be given if patient has recently used sildenafil (Viagara) or similar
What must be monitored when taking nitroglycerin
BP, can cause headaches and hold if BP is too low
S/S of unstable angina in women compared to men
women feel more fatigue while men feel like heavy pressure on chest.
Interventions for acute coronary syndromes
1) Assess ABC
2) Upright position
3) O2
4) Assess heart and breath sounds
5) 12 lead ECG
6) NOMA
7) Blood draw for troponin and electrolytes