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1. Latent Learning: Learning that occurs without obvious reinforcement and only becomes apparent when there is an incentive to demonstrate it. 2. Insight Learning: A sudden realization or “aha” moment in problem-solving, often involving the reorganization of information rather than trial-and-error learning. 3. Modeling: Learning by observing and imitating the behavior of others. 4. Vicarious Conditioning: Learning that occurs by observing others being rewarded or punished, influencing the observer’s behavior. 5. Social Learning Theory: A theory proposing that learning occurs through observation, imitation, and modeling, incorporating cognitive processes in addition to environmental influences. 6. Partial Reinforcement: A reinforcement schedule in which a behavior is not rewarded every time it occurs, making it more resistant to extinction. 7. Continuous Reinforcement: A reinforcement schedule in which every instance of a behavior is reinforced, leading to rapid acquisition but less resistance to extinction. 8. Learned Helplessness: A condition in which exposure to uncontrollable aversive events leads to a failure to act or escape, even when opportunities to do so are available. 9. Instinctive Drift: The tendency for an animal’s innate responses to interfere with conditioned behaviors. 10. Shaping: Gradually reinforcing successive approximations of a target behavior until the desired behavior is achieved. 11. Primary Reinforcer: A stimulus that is naturally reinforcing because it satisfies a biological need (e.g., food, water). 12. Law of Effect: Thorndike’s principle stating that behaviors followed by satisfying consequences are more likely to be repeated, while those followed by unpleasant consequences are less likely to occur. 13. Punishment: A consequence that decreases the likelihood of a behavior occurring again. 14. Reinforcement: A consequence that increases the likelihood of a behavior occurring again. 15. Operant Conditioning: Learning in which the consequences of a behavior determine the likelihood of its recurrence. 16. Habituation: A decrease in response to a stimulus after repeated exposure, demonstrating a basic form of learning. 17. Preparedness (Biological): The predisposition of certain species to learn associations that have survival value more easily than others. 18. Taste Aversion (Conditioned): A learned avoidance of a particular taste or food after it has been associated with illness or discomfort. 19. Counterconditioning: A technique where an undesirable response is replaced with a desirable one by associating the stimulus with a new response. 20. Higher-Order Conditioning: A process in which a conditioned stimulus becomes associated with a new neutral stimulus, creating a second conditioned stimulus. 21. Generalization: The tendency for stimuli similar to the conditioned stimulus to elicit the conditioned response. 22. Discrimination (Classical Conditioning): The ability to differentiate between similar stimuli and respond only to the conditioned stimulus. 23. Spontaneous Recovery: The reappearance of a conditioned response after extinction and a rest period. 24. Extinction: The weakening and eventual disappearance of a conditioned response when the conditioned stimulus is repeatedly presented without the unconditioned stimulus. 25. Conditioned Stimulus (CS): A previously neutral stimulus that, after being paired with the unconditioned stimulus, elicits a conditioned response. 26. Conditioned Response (CR): The learned response to the conditioned stimulus. 27. Unconditioned Stimulus (US): A stimulus that naturally triggers a response without prior conditioning. 28. Unconditioned Response (UR): The natural, reflexive response to the unconditioned stimulus. 29. Acquisition: The initial stage of learning during which the association between the conditioned stimulus and unconditioned stimulus is established. 30. Classical Conditioning: A learning process in which a neutral stimulus becomes associated with an unconditioned stimulus, resulting in a conditioned response.
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Chapter 6 - Learning
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Learning
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classical conditioning
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unconcious - chpater 16
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psychomotor stimulants
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pt 4: Adrenal gland structure Cortex (steroids) + medulla (catecholamines) Three adrenal cortex layers Zona glomerulosa, fasciculata, reticularis Zona glomerulosa Secretes aldosterone (mineralocorticoid) Aldosterone function Increases Na+ reabsorption, K+ excretion; raises blood pressure Aldosterone release triggered by Low BP, high K+, renin-angiotensin system, ACTH Renin-angiotensin system Low BP → renin → Ang I → Ang II → aldosterone + vasoconstriction Zona fasciculata Secretes cortisol (glucocorticoid) Cortisol function Increases glucose, suppresses immune system, maintains blood pressure High cortisol effects Immune suppression, muscle wasting, hyperglycemia Zona reticularis Secretes adrenal androgens Adrenal androgens Weak sex hormones contributing to puberty and libido Adrenal medulla hormones Epinephrine and norepinephrine Epinephrine effects Increases heart rate, metabolic rate Norepinephrine effects Increases vasoconstriction and blood pressure Pancreas function Exocrine (digestive enzymes) + endocrine (insulin, glucagon) Alpha cells in pancreas Produce glucagon Beta cells in pancreas Produce insulin Glucagon function Increases blood glucose via glycogenolysis and gluconeogenesis Insulin function Decreases blood glucose by increasing cellular uptake Somatostatin from pancreas Inhibits insulin and glucagon release Type 1 diabetes Autoimmune destruction of beta cells → no insulin Type 2 diabetes Insulin resistance; cells do not respond to insulin Effects of insulin Promotes glucose uptake, fat storage, glycogen formation Ovarian hormones Estrogen and progesterone Estrogen function Female development, menstrual cycle regulation Progesterone function Maintains uterus for pregnancy Testicular hormone Testosterone Testosterone functions Male traits, sperm production, libido Placental hormones hCG, estrogen, progesterone hCG function Maintains corpus luteum early in pregnancy Kidney hormone EPO (erythropoietin) EPO function Stimulates RBC production when oxygen is low Heart hormone ANP (atrial natriuretic peptide) ANP function Decreases blood pressure by reducing blood volume GI hormones Gastrin, secretin, CCK regulate digestion Skin hormone precursor Produces vitamin D precursor (cholecalciferol) Adipose hormone Leptin Leptin function Signals satiety and regulates metabolism Cushing’s syndrome Excess cortisol → moon face, buffalo hump, high glucose Addison’s disease Low cortisol/aldosterone → fatigue, low BP, hyperpigmentation Pheochromocytoma Adrenal medulla tumor causing excess epinephrine Conn’s syndrome Excess aldosterone → high BP, low K+ Hyperthyroidism symptoms Weight loss, heat intolerance, anxiety, fast heartbeat Hypothyroidism symptoms Fatigue, weight gain, cold intolerance Goiter Enlarged thyroid due to iodine deficiency or overstimulation Primary endocrine disorder Problem in the gland itself Secondary endocrine disorder Problem in pituitary or hypothalamus Calcitriol (active vitamin D) Increases Ca2+ absorption in intestines Endocrine disruptors Chemicals interfering with hormone actions
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