PT 471C

relative flexibility

relative flexibility

increased mobility or frequency of movement in a joint adjacent to a body part with restricted mobility- stiff muscle, joint capsule, tendon, bone, ligament

the body compensates for restrictions by ____

increasing motion elsewhere to maintain function

when a joint develops abnormal compensatory motion in response to an adjacent stiffness, what happens to the joint and its supporting structures

stabilizing muscles, tendons, ligaments around the joint become too flexible or lax, providing insufficient stiffness or resistance to motion

what is a common issue with relative inflexibility

shear force

stiff muscle

goes through full range of motion, but often creates movement compensation when countered by a less stiff muscle

less stiff muscle

- fewer sarcomeres in parallel- relatively weaker: cannot overcome passive tension of antagonist- decreased excitability of alpha motor neuron pool

relative flexibility due to muscle dominance

muscles can become overly dominant in action, leading to movement fault- hamstrings > gluteus maximus for hip ext- spinal extensor > gluteus maximus for return from L/S flexion- lats > subscapularis for shoulder IR

secondary test

a correction of the movement to determine if a change occurs- with symptoms, range of motion, etc

when a muscle is short, you should ____

lengthen the muscle

when a muscle is stiff, you should ____

create more stiffness in muscles that prevent compensation

pathokinesiologic source

tissue of pathoanatomical structure believed to cause symptoms- ex: supraspinatus tendon

kinesiopathologic source

mechanical factor that causes tissue irritation- ex: insufficient scapular upROT

MSI diagnosis

determined by motion, direction, or alignments that most consistently elicit symptoms, and when corrected, decrease symptoms

MSI diagnostic name is based on:

- the motion that is performed in a less than idea manner- movement or postural alignment which the pt complains of pain and when corrected reduces their symptoms- ex: flexion syndrome

directional susceptibility to movement (DSM)

- repeated motions and sustained posture alter tissue and movement patterns- increase frequency of accessory and physiologic movements into a specific direction- stress/microtrauma/tissue damage- inflammation/pain- DSM increases potential for tissue injury

physiologic/osteokinematic hypermobility

- greater motion than ideal range- in relation to composite body area

accessory/arthrokinematic hypermobility

- greater translation of roll/glide/spin- in relation to joint name

frequency of motion hypermobility

- movement more often at a specific joint/muscle than adjacent joints/muscles during a functional activity- more motion than recommended for functional task

how to determine patient MSI diagnosis

primary test: unadjusted movementsecondary test: adjusted movement that improves mechanics and symptoms

fundamental principles of physical stress theory

1. changes in relative level of physical stress cause predictable response to all biological tissue2. 5 responses to physical stress: atrophy, maintenance, incr stress tolerance, injury, death3. inflammation renders tissue less tolerant to stress than prior to injury4. tissues are affected by history of recent stresses5. stress thresholds can be modulated based on: extrinsic/intrinsic factors, psychosocial factors6. level of exposure to physical stress is defined by magnitude, time, and direction of stress application

normal sequence of lumbar flexion

- initial motion from posterior sway of pelvis as hips flex- as hips flex, lumbar spine reverses lordosis- when lumbar motion completes, last of motion comes from hip flexion

abnormal lumbar flexion ROM

- greater than 25-30 degrees lumbar flexion- less than 80 degrees of hip flexion- more than 50% of L/S flex motion before initiation of hip flexion

return from lumbar flexion normal sequence

- initial motion from hip ext- equal portions of hip ext and L/S ext throughout motion

abnormal return from lumbar flexion

- initiation of motion with L/S ext before hip ext- exaggerated forward sway- overuse of L/S paraspinals and underuse of glutes can increase facet pressure

lumbar sidebend ROM normal sequence

- more thoracic than lumbar motion- smooth curve

abnormal lumbar sidebend ROM

- hinge points- straight lumbar spine- secondary test: block w/ your hand and see if symptoms or motion improves- positive sign for ipsilateral rotation if hinge point, stabilize above iliac crest

single leg stance common errors

- pelvic drop- trunk lean- pelvic lateral shift- pelvic IR

lumbar rotation normal ROM

- 13 degrees to ea side- majority of motion comes from thoracic

seated hip flexion normal movement

able to flex hip to 120 degrees without lumbar compensation and with neutral femur

abnormal seated hip flexion

- anterior/posterior pelvic tilt: flex/ext syndrome- pelvic rotation- trunk sidebend and/or rotation- femur ER/IR- stabilize pelvis to see if Sx change

subcostal angle

- normal: 90 degrees- > 100: short rectus + internal oblique, long external oblique- < 75: short external obliques

paraspinal asymmetry

when one side of lumbar area is 1/2 inch larger, considered positive for lumbar rotation

supine hip ER normal movement

- no pelvic or lumbar ROT within first 50% of movement- start with one knee extended- go to hooklying if too difficult- test for rotation in pelvis: stabilize to see if Sx improve

lumbar movement dysfunction classifications

- lumbar flexion- lumbar extension- lumbar flexion rotation- lumbar extension rotation- lumbar rotation

treatment of lumbar movement dysfunction (Maluf 2000)

- correct lumbar movement dysfunction by limiting direction specific motion or alignment that elicits painful symptoms- address impairments in muscle force and joint flexibility

MSI exercise principles

- perform modified versions of movement tests - goal to modify movement by decreasing preferred movements of the limb or trunk- emphasize correct performance over number of reps- avoid reproduction of Sx

lumbar extension/rotation syndrome sources of Sx

- facet dysfunction- spinal stenosis- spondylolisthesis- spinal instability- DDD- L/S OA

L/S ext/rotation syndrome relative flexibility and stiffness impairments

lumbar spine is excessively flexible into extension and ROTpossible stiff/short:- hip flexors/rectus femoris: anterior tilt- hip abductors: lateral pelvic tilt- TFL: pelvic rotation- latissimus dorsi: anterior tilt, extensionnot stiff enough:- abdominals: posterior pelvic tilt- lateral abdominal/obliques: lateral tilt, pelvic rotation- gluteal muscles: posterior tilt, return from flexion

L/S ext/rotation muscle recruitment patterns

- dominance of L/S extensors without counterbalance of abdominals for posterior pelvic tilt- return from standing: L/S extensor dominance over hip ext- dominance of hip flexors over abdominals for leaning forward, sitting up- eliminating hip flexor muscle activity may reduce Sx

L/S extension syndrome flexibility/stiffness impairments

L/S more flexible into extensionshort/stiff:- hip flexors: anterior tilt- lats: anterior tilt, lumbar extensionnot stiff enough:- gluteals: return from flexion- external obliques/abdominals: posterior tilt

L/S rotation syndrome tissue sources

- pain with lumbar rotation and sidebend- DDD- spondylolisthesis- OA- spinal instability- facet dysfunction

L/S extension syndrome muscle recruitment

- hip flexors dominant over abdominals- L/S paraspinals dominant over hip extensors

L/S rotation syndrome flexibility/stiffness impairments

L/S more flexible into rotation and side bendshort/stiff:- L/S paraspinals: lateral flexion/SB- hip abductors: lateral flexion, lateral pelvic tilt- TFL: pelvic rotationnot stiff enough:- lateral abominals/obliques: cannot control rotation- transverse abdominis

L/S flexion/rotation syndrome tissue sources

- herniated disc- DDD- OA- spinal instability

limited hip motion in L/S flex/rot syndrome

- usually not due to hamstring stiffness/shortness- limited hip motion result of influence of hamstrings to exert control over weight of the trunk

corrected forward bend for L/S flexion syndrome

place hands on elevated table and bend forward by flexing hips

L/S flex/ROT syndrome flexibility/stiffness impairments

L/S more flexible into flexion and rotationshort/stiff:- hamstrings- glutes- rectus abdominis: more support than back ext- TFL: pelvic ROTnot stiff enough:- L/S paraspinals- obliques: cannot control rotation

L/S flex/rot syndrome muscle recruitment patterns

- rectus abdominis dominant over back extensors for providing trunk support- abdominals have poor control of lumbopelvic rotation: obliques

L/S flexion syndrome tissue sources

- herniated disc- lumbar strain- DDD

femoral anterior glide syndrome

- inadequate posterior glide of femur during hip flexion due to taut posterior capsule, stretched anterior capsule- altered PICR of hip flexion- causes iliopsoas tendonitis- femur impinges on anterior tissue of joint capsule- development associated with hip extension activities stretching the anterior joint capsule

femoral anterior glide syndrome relative flexibility/stiffness impairments

short/stiff:- TFL- hamstrings (medial)- hip ER: prevent posterior glide of femurnot stiff enough:- iliopsoas: prevent anterior capsule from getting pinched- glute max/medius

femoral anterior glide syndrome muscle recruitment pattern

- TFL dominant over iliopsoas and posterior glute medius- hamstrings dominant over glute max- medial hamstrings > lateral hamstrings causing hip IR with knee ext

hip adduction syndrome

- with or w/o IR, w/ IR most common- women > men- piriformis syndrome- ITB syndrome- result of insufficient performance of hip abductors

hip adduction + IR syndrome flexibility/stiffness impairments

short/stiff:- hip adductors- TFL: dominant as hip abdnot stiff enough:- glute med/max- hip ER

hip add + IR syndrome muscle recruitment pattern

- hip add dominant over hip abd- TFL + sartorius may be used to abd

hip extension with knee extension

- insufficient gluteus maximus or quads- contraction of hamstrings to help quads with extension- pain at ischial tuberosity - HS origin- likely hamstring strain

hip lateral rotation syndrome

- insufficient participation of hip ER- shortened piriformis with sciatica- dominance of hip ER mm over IR- short/stiff hamstrings, quads, hip ER mm

femoral accessory mobility motion hypermobility

- rotation and excessive accessory motion of anterior/superior glide occur during hip flex/ext- hypermobility- potential labral tear- excessive compression of hip by rectus femoris and hamstring stretching in hip flexion/extension

femoral accessory motion hypermobility flexibility/stiffness impairments

- rectus femoris and hamstrings stiff/short- iliopsoas, hip rotators, gluteus medius not stiff enough

femoral accessory motion hypermobility muscle recruitment

- hamstrings dominant as hip extensors- TFL + rectus femoris dominant hip flexors

hip flexion arthrokinematics

- spin- anterior roll, posterior glide

hip extension arthrokinematics

- spin- posterior roll, anterior glide

hip IR arthrokinematics

anterior roll, posterior glide

hip ER arthrokinematics

posterior roll, anterior glide

external oblique actions

- posterior pelvic tilt bilaterally- contralateral trunk rotation w/ CL internal oblique- ipsilateral pelvis rotation w/ CL internal oblique- ipsilateral side bend

iliopsoas muscle actions

- hip flexion- anterior pelvic tilt- upper lumbar extension- lower lumbar flexion- ER and ABD

erector spinae actions

- spinal extension- anterior pelvic tilt- pelvis lateral tilt

latissimus dorsi action on spine

- pelvis anterior tilt- L/S extension as compensatory motion when short, or if abdominals not stiff enough to counterbalance

QL muscle action

- ipsilateral side bend- lateral pelvic tilt

TFL actions

- hip flex, IR, abd- can cause pelvic rotation if short/stiff, also resulting in lumbar rotation

gluteus maximus actions

- hip ext + ER- upper half abd- lower half add

spurt muscle

- origin away from joint- insertion close to joint center- ex: piriformis, glutes, iliacus, psoas

shunt muscle

- origin near the joint- insertion away from joint- causes shear/compression force if not countered by local muscles- ex: hamstrings, TFL, sartorius

the hip greater trochanter will move ____ when hamstrings are dominant muscle for hip extension

anteriorly- will maintain its position or move posteriorly if glutes and piriformis are prime movers

Van Dillen article hip ROM + LBP

- is hip PROM different in people with or w/o LBP for people who participate in rotation related sports- group with LBP had less total hip ROT than group w/o LBP- LBP group displayed more asymmetry of total hip ROT between right and left- directional demands of sport may be important factor to address or consider in interventions

Lewis article hip forces

Increases in anterior hip forces due to muscle can be attributed to:- decreased force contribution of glutes in hip ext- decreased force contribution of iliopsoas during hip flex- increasing hip extension anglecompensations:- semimembranosus, TFL, sartorius activation increases with decreased glute activation- TFL and sartorius activate to offset ER and add torques- TFL, sartorius, and add longus dominate hip flex instead of iliopsoas

femoral lateral glide syndrome

- laxity of hip abductors cause femoral head to glide laterally to the point of subluxation- popping hip with hip add/IR- excessive hip add, IR with movements- short/stiff TFL, weak glute med/external rotators

tibiofemoral rotation w/ valgus syndrome

excessive IR and add of femur compared to tibia- may impact ITB, tibiofemoral J, patellofemoral J- PFPS and ITB syndrome- resting femur IR or tibia ER- posterior pelvic tilt indicates weak glutes- potential XS pronation of feet

tibiofemoral rotation w/ valgus flexibility/stiffness impairments

short/stiff:- TFL: femur IR or tibia ER- tibia ERnot stiff:- hip ER:- hip abd- tibia IR

tibiofemoral rotation w/ varus syndrome

varus thrust in gait- medial compartment tibiofemoral J at risk- large knee adduction moment arm, increase force thru medial J compartment- toe out gait may potentially relieve Sx- femoral IR + knee hyperext- potential supinated/flat foot

TFRVar flexibility/stiffness impairments

stiff/short:- TFL: femoral IRnot stiff:- hip ER- hip abd

tibiofemoral hypomobility syndrome

limitation of knee motion due to degenerative changes or immobilization- may be due to arthrokinematics or mm extensibility

tibiofemoral hypomobility stiffness impairments

short/stiff:- hip flexors- hamstrings- ankle PFnot stiff:- glute max- hip ER- gastroc- quads

knee extension syndrome

knee pain associated w/ quad dominance or stiffness resulting in excessive pull on patella- patella superior glide/patella alta- patellar tendinopathy, quad strain- osgood schlatters

knee extension syndrome secondary tests

- inferior glide of patella during movement- allow tibia to advance forward over foot

knee extension syndrome stiffness impairments

short/stiff:- quads: pull patella superiorlynot stiff:- patellar tendon: - glutes- hamstrings: imbalance puts more demand on quads

knee hyperextension syndrome

knee pain associated with impaired knee extensor mechanism- dominance of hamstrings, poor performance of glutes and quads- pain with prolonged standing, rapid knee ext activities- PFPS- fat pad syndrome/Hoffa

knee hyperext syndrome stiffness impairments

short/stiff:- gastroc- hamstringsnot stiff:- glutes- quads: strong w/ MMT, not poor functional use

patella lateral glide syndrome

impaired patellar relationship in femoral groove- imbalance between VMO and vastus lateralis- movie goer syndrome: pain w/ prolonged knee flexion w/ sitting- secondary diagnosis w/ tibiofemoral ROT or hyperext- PFPS- patellar lateral tilt or glide

patella lateral glide stiffness impairments

short/stiff:- TFLnot stiff:- VMO: impossible to measure- glute med- hip ER

pronation syndrome stiffness impairments

short/stiff:- gastroc/soleus leading to impaired ankle DF- FHL limiting 1st MTP extnot stiff:- ankle PF: poor recruitment leads to pronation post MSt- hip ER: IR of femur can lead to pronation- foot intrinsics: cannot maintain arch during WB activity- limited talocrural DF

supination syndrome

rigid foot with little ability to absorb shock- absence of pronation in initial stance phase- whip to medial side of foot in push off- limited talocrural DF and MTP motion- pain in late stance due to eccentric loading at achilles tendon- pain at fibular mm during WB activities as mm works to eccentrically control supination

what muscles pronate the foot

none- gravity is pronator, most of muscles of the foot act as supinators and eccentrically control pronation

fibularis longus closed chain action

pulls 1st MTP down to the ground

windlass effect

from MSt to TSt full body weight transfers to MTP heads- MTP extends, plantar tissues tighten, arch elevates- navicular moves dorsally, causing PF of first ray- with 1st MTP passive ext there should be arch elevation

lower extremity cutaneous map

rearfoot varus

calcaneal inversion in subtalar neutral- uncompensated: lateral heel contact, limited eversion and pronation in gait- compensated: bring medial side of foot to the ground, stressing posterior tibialis as it tries to control pronation- associated with genu valgum

forefoot varus

medial side of forefoot higher than lateral- most common intrinsic cause of mechanical pain/dysfunction in the foot- compensated: increase force laterally at 4/5th met heads- uncompensated: excessive pronation of subtalar J to help medial foot hit the ground, keeps pronation throughout stance phase

effect of compensated forefoot varus

- foot mm fire out of sync, midtarsal joints hypermobile- abnormal stress on peroneus longus and forefoot- dorsiflexed, hypermobile first ray- first ray cannot contribute to propulsion, 2nd and 3rd met heads get excessive loading

compensated forefoot valgus

- excessive and rapid supination of subtalar joint- abnormal supination cannot adapt to uneven surfaces, increasing risk of inversion sprains- poor shock absorption at IC and necessary tibia IR for knee flexion compromised, leading to other problems