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functions of the integumentary system
Protection against infection, injury, and UV rays
Regulation of body temperature
Sensory perception
Synthesis of vitamin D
Excretion of waste products
skin and vitamin d metabolism
UV light exposure converts 7-dehydrocholesterol in the skin to vitamin D3 (cholecalciferol), which is then processed in the liver and kidneys to become active vitamin D
skin changes in patients
Type 2 Diabetes Mellitus: Delayed wound healing, increased risk of infection, diabetic dermopathy (brown, scaly patches), and acanthosis nigricans (dark, velvety skin patches).
Liver Disease: Jaundice (yellowing skin), spider angiomas, palmar erythema, and pruritus (itchiness)
cyst
closed sac filled with fluid or semi-solid material
macule
flat, discolored spot (freckle)
nodule
solid, raised lesion >1cm
papule
elevated, firm lesion <1cm
plaque
raised, flat-topped lesion >1cm
pustule
elevated lesion containing pus
vesicle
elevated, fluid filed lesion <1cm
annular
ring-shaped (ringworm)
linear
line formation (shingles)
polymorphous
varied shapes and sizes
satellite
smaller lesions near a larger central lesion
Braden Scale
Purpose: Assesses pressure ulcer risk
Factors Considered: Sensory perception, moisture, activity, mobility, nutrition, friction/shear
Score Ranges: 6-23 (Lower scores indicate higher risk)
aging and skin changes
thinner skin, reduced elasticity, slower wound healing, and increased dryness
xerosis
dry, itchy, scaly skin commonly seen in older adults
psoriasis
Pathophysiology: Autoimmune disorder causing rapid skin cell production
Clinical Manifestations: Red patches with silvery scales, itching, burning
Treatment: Topical corticosteroids, vitamin D analogs, phototherapy, biologics
diagnostic test
Wound/Bacterial Infection: Wound culture and sensitivity test
Fungal Infection: Potassium hydroxide (KOH) test
malignant skin growth indicators
Asymmetry, border irregularity, color variation, diameter >6mm, evolving characteristics (ABCDE rule)
staphylococcus skin infection
Clinical Manifestations: Redness, swelling, warmth, pain, and possible drainage
herpes simplex virus (hsv-1 and hsv-2)
Clinical Manifestations: Painful blisters or sores on lips (HSV-1) or genitals (HSV-2)
Treatment: Antiviral medications (e.g., acyclovir)
Cure: No cure; managed with medications
Spread: Direct contact with infected skin or fluids
Patient Teaching: Avoid sharing utensils, lip balm, or towels; practice safe sex
tinea infections
Pathophysiology: Fungal infection affecting skin, nails, or hair
Clinical Manifestations: Itchy, red, scaly patches; cracked or peeling skin
Treatment: Antifungal creams, oral antifungals
Patient Teaching: Keep skin dry, avoid sharing personal items
plaque psoriasis
Pathophysiology: Chronic autoimmune skin condition
Clinical Manifestations: Thick, red patches with silvery scales
Treatment: Topical agents, phototherapy, systemic medications
Cure: No cure; managed with treatment
Patient Teaching: Moisturize regularly, avoid triggers
wound closures
Primary: Immediate closure with sutures or staples
Secondary: Healing through granulation (left open)
Tertiary: Delayed closure after infection resolution
actinic keratosis
Description: Precancerous skin lesion caused by sun exposure
Potential Progression: Can develop into squamous cell carcinoma
Common Locations: Face, scalp, ears, and back of hands
basal cell carcinoma
Slow-growing skin cancer often appearing as a pearly nodule or ulcerated lesion
skin cancer risk reduction
Use broad-spectrum sunscreen, wear protective clothing, avoid tanning beds, and regularly examine skin for changes
MRSA (methicillin-resistant staphylococcus aureus)
Skin Conditions: Abscesses, cellulitis, and boils
bacterial skin/soft tissue infection
Clinical Manifestations: Redness, warmth, swelling, pain
Complicated Infection: Fever, chills, and systemic symptoms
acute vs. chronic
Acute Pain: Sudden, short-term, and resolves with healing
Chronic Pain: Persistent pain lasting longer than 3 months
allodynia
pain from non-painful stimuli
analgesic
pain-relieving medication
antipyretic
fever-reducing medication
hyperalgesia
increased sensitivity to pain
pain tolerance
maximum pain level a person can endure
OPQRST-AAA Pain Assessment
Onset, Provocation, Quality, Region/Radiation, Severity, Time
Associated symptoms, Aggravating factors, Alleviating factors
numeric pain intensity scale
rates pain 0-10
verbal rating scale
describes pain as a mild, moderate, or servere
visual analog scale
marking pain on a 10cm line
visual analog thermometer
thermometer-like pain indicator
wong-baker scale
faces scale for children and non-verbal patients
NSAIDs
Common Examples: Ibuprofen, naproxen
Help with both pain and inflammation
Antipyretic properties reduce fever
Acetaminophen (Tylenol) is not an NSAID; it reduces pain and fever but lacks anti-inflammatory effects
Side Effects of NSAIDs: GI irritation, kidney damage, cardiovascular risks
opioids
Common Drugs: Morphine, oxycodone, hydrocodone
Side Effects: Drowsiness, constipation, respiratory depression
Dangers: Risk of dependency and overdose
Patient Teaching: Use only as prescribed; avoid alcohol
Monitoring Parameters: Respiratory rate, pain level, sedation score
pain documentation
Always document the patient's reported pain score, not your perceived severity
heat application contraindications
Avoid if the patient has active bleeding, an acute injury, or lacks sensation
pain management
Pharmacological: Medications
Nonpharmacological: Massage, relaxation techniques, heat/cold therapy
WHO three-step ladder for pain management
Non-opioids (e.g., NSAIDs)
Mild opioids (e.g., codeine) with or without non-opioids
Strong opioids (e.g., morphine) for severe pain