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Sono non visual
PT not NPO
???
Contracted gb wt stones
Absent
Porcelain gb
Sludge filled
Gb neoplasm filled lumen
Ectopic gb (wrong location)
Gas
Chronic cholecystitis (gb inflammation)
NPO - nil per os
Nothing by mouth
Small GB
Not NPO
Chronic cholecystitis
Hepatitis & Cirrhosis (decre. Gb vol. due to hepatocyte injury bile prod. decre.
Hypoplasia
Enlarged GB (hydrops)
Prolonged fast
IV hyperalimentation (too much fluid)
Cystic d obs. (can’t empty)
Courvoisiers GB
Diabetes (long insulin resistance)
Post vagotomy (cut vagus nerve)
Courvoisiers GB
enlg due to distal CBD obs.
Due to malignancy in panc hd ca, common duct, ca, duodenal ca, ampulla cater (2nd pt of duodenum) ca
Painless jaundice, elev serum bilirubin, abnorm LFTs
Gb wall thickness intrinsic (inside/direct) causes
Acute cholecystitis (diffuse)
Chronic cholecystitis (diffuse)
Gangrenous cholecystitis (asymmetrical)
Emphysematous cholecystitis (asymmetrical)
Adenomyomatosis (focal or diffuse)
Polyp (focal)
GB ca : primary or metastatic (focal)
Focal
Diffuse
Asymmetrical
GB wall thickness extrinsic(outside) causes (most cs diffuse thickening)
Rt side heart failure
Acute hepatitis
AIDS
Sepsis
As iris due to hypoalbumemia
Renal failure
GB wall thickness physiologic causes
Contracted GB after eating
Sludge
combo of calcium bilirubinate granules & cholesterol crystals
Sludge symptoms
RUQ pain, nausea, vomitting
Sludge causes
Biliary stasis secondary to prolong fast
Hyperalimentation (Pt not moving)
Cystic duct obstruction
Acute or chronic cholecystitis
Sludge sono
mobile/gravity dependent, non shadowing, low echo lumen
Tumefactive Sludge
longstanding causing sludge to not move, appears fluffier & organized (resemble pseudo tumor & gb neoplasm)
Sludge Ball
org of sludge into round, mobile, echogenic, non shadowing ball
Cholelithiasis risk factors
pregnancy, gestational diabetes, diet
estrogen therapy, oral bc
Female, Fat, Fertile, Flatulent, Forty, Fair
Cholelithiasis (gall stones) symptoms
RUQ pain, radiates to RT shoulder (fatty meals)
nausea, vomiting
may be asymptomatic
Diarrhea means not a gallstone
Cholelithiasis lab
ALP may be elevated if CD or CBD obstr.
non obstr. all values normal
Cholelithiasis sono
solitary or multiple
1.) strong echogenicity
2.) posterior shadowing
3.) mobile/ gravity dep
Cholelithiasis false positive structures
bowel gas, junct folds/ valves of heister, gb edge shadowing, past cholecystectomy scarring/ surg clips (confuse with contracted)
Cholelithiasis treatment
diet restrictions (low fat)
cholecystectomy (by laproscopy, early interv)
Cholelithiasis complications
1/3 pt develop acute calculous cholecystitis
high association with gb carcinoma
Acute Cholecystitis
inflammation of gb wall with vary degr necrosis & infection (caused by inflamed wall, also causes ulceration, swell, edema)
Necrosis
Dead tissue
Calculous acute cholecystitis
most common
Acalculous Acute cholecystitis