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Attitudes and actions of the culturally competent nurse → cultural awareness
the nurse recognizes the enormous impact that culture has on patients’ health values and practices
Attitudes and actions of the culturally competent nurse → Cultural knowledge
help us to understand behaviors that might otherwise be misinterpreted. It helps nurses to establish rapport, ask appropriate questions, avoid misunderstandings, and identify the cultural variables that should be considered in the planning of nursing care
Attitudes and actions of the culturally competent nurse → Cultural encounters
directly engage in interactions with patients from culturally diverse backgrounds → multiple cultural encounters of diverse backgrounds will decrease the likelihood of stereotyping
Attitudes and actions of the culturally competent nurse
Cultural skill is the ability to perform a cultural assessment in a sensitive way
Exhibit cultural desire through patience, consideration, and empathy → giving the impression that you are willing to learn from the patient = hallmark of cultural desire
ADHD → symptoms
Hyperactivity and impulsivity
overlooks or misses details, work is inaccurate, difficulty remaining focused, mind seems elsewhere, starts task but quickly loses focus and is easily sidetracked, difficulty organizing tasks and activities, avoids, dislikes, or is reluctant to engage in tasks that require sustained mental effort, loses things necessary for tasks or activities, easily distracted by extraneous stimuli, often forgetful in daily activities
ADHD → pharmacological treatment
Stimulant drugs
Methylphenidate (Ritalin and others) and the mixed amphetamine salts (Adderall) are the most widely used
Autism spectrum disorder → symptoms
Deficits in social relatedness → disturbances in developing and maintaining relationships
Stereotypical repetitive speech, obsessive focus on specific objects, over adherence to routines or rituals, hyperreactivity or hyporeactivity to sensory input, and resistance to change
Pharmacological Tx of alcohol abuse disorder → Aversive conditioning: Disulfiram
Inhibits aldehyde dehydrogenase
Pt feels terrible when the consume ETOH when on this med
Pharmacological Tx of alcohol abuse disorder → Opiate antagonist therapies: Naltrexone
Blocks mu opioid receptors → contribute to the high and euphoria from drinking
Diminish positive reinforcement “reward” craving
Pharmacological Tx of alcohol abuse disorder → Glutamate-based interventions: Acamprosate
When ETOH is chronically used then withdrawn, state of glutamate hyperactivity occurs – with GABA deficiency
Pharmacological Tx of alcohol abuse disorder → GABAergic interventions: Topiramate, Gabapentin
Topiramate (anticonvulsant) inhibits glutamate release and enhances GABA (not FDA approved)
Addiction treatment → etiology
Block the reward center in the brain
Addiction treatment → principles
Abstinence and support are the main principles of treatment
Application of therapeutic communication and caring principals
Facilitate communication
Assist with problem solving
Develop insight
Promote independence and recovery
Educate
Increase accuracy of diagnosis
Yields collaborative decision making
Improves identification of pt emoticons, perceptions, apprehension, and pt and provider satisfaction
Reduces risk of malpractice and provider burnout
Delirium → symptoms
Occurs quickly, may last days or months, short-term memory impaired, erratic sleep, may have day-night sleep reversal, fluctuating consciousness, thought content matches LOC, thought process depends on LOC, lociacal alternating with illogical, slurred speech, visual and tactile hallucinations, anxious, fearful, may startle easily
Delirium → etilogy
Complications from acute medical conditions
Effects of drugs or meds
In combination with predisposing, non-modifiable risk factors (Dementia or hepatic disease) or precipitating, modifiable (infections, surgery)
Associated with: reduced acetylcholine, melatonin; increased dopamine, glutamate, norepinephrine
Personality disorders → symptoms
significant challenges in self-identity or self-direction
Have problems with empathy or intimacy within their relationships
Personality disorders → therapeutic interventions
Psychotherapy
Group therapy → noninvasive and supportive group
Violence and abuse → risk
Patients
Spouse
Stranger
They can get attacked
Violence and abuse → etiology
Genetics: more biologically predisposed to respond to life events with irritability, easy frustration, and anger
Neurobiological: associated with anger and aggression. Brain tumors, Alzheimer disease, temporal lobe epilepsy, and traumatic injury to certain parts of the brain result in changes to personality that could include increased violence
Neurotransmitters: serotonin, dopamine, and gamma-aminobutyric acid (GABA), play a role in anger and aggression
Violence and abuse → symptoms
Burns
Fractures
Lacerations
Delay in seeking treatment
Inconsistent stories
Injury does not match age or development of child
High incidence of UTIs
Bruised and multiple bruises in various stages of healing
Red or swollen vulva
Rectal or vaginal trauma
Evidence of old, repeated injuries
Violence and abuse → communication technique with a child
Open ended questions (how did this happen, who takes care of you, what do you do after school, who are your friends, what happens when you do something wrong)
Avoid asking too many people asking the same questions
Violence and abuse → communication technique with parent or caregiver
Open ended questions (what arrangements do you make when you need to leave your child alone, how do you punish your child, when your infant cries for a long time, how do you get him/her to stop)
Do’s: conduct private interviews; be direct, honest, professional, attentive, and understanding; inform them you need to make a referral to CPS and explain process
Don'ts: try to “prove” accusations or demands; display horror, anger, or disapproval of parents or situation; place blame on or make judgments about the parents or child
Dementia → goal of treatment
Prevent further buildup of amyloid plaques
Keep what level of functioning they currently have
Dementia → general pharmacological intervention
Donepezil (enhances effects of available ACh by inhibiting acetylcholinesterase activity)
Rivastigmine (inhibits both acetylcholinesterase and butyrylcholinesterase; possible resulting in greater preservation of ACh activity)
Galabatmine (inhibits only acetylcholinesterase; also an allosteric modulator of nicotinic cholinergic receptors)
Memantine (NMDA receptor uncompetitive; antagonist; 5-HT3 receptor antagonist; does not alter ACh
Dementia → general non pharmacological interventions
Address unmet needs
Identify/modify environment and daily routine stressors
Caregiver support/training
Behavior modification
Group/individual therapy
Problem solving, distracting
Provide outlets for pent-up energy
Avoid behavior triggers
Increase social engagement
Relaxation techniques
Reminiscence, pet, and music therapy
Aromatherapy
Suicide and self harm → assessment
Be blunt
“Do you want to kill yourself”
Find out if they have a plan and if so, the means to carry out the plan
Assess everyone
Suicide and self harm → interventions
establishing a therapeutic alliance
written safety plan
careful administration of medication
Suicide and self harm → precautions
One-to-ones
Suicide and self harm → need for hospitalization
If they are a danger to self or others
Significant side effects of 1st gen SDAs
Sedation, anticholinergic effects, and dizziness upon standing
Dizziness and orthostatic hypotension
Significant side effects of 2nd gen SDAs
Increase metabolic syndrome risk.
They produce weight gain, hyperglycemia, and hyperlipidemia to varying degrees
Major actions of dopamine
Involved in fine motor movement
Involved in integration of emotions and thoughts
Involved in decision making
Stimulates hypothalamus to release hormones
Major actions of serotonin
Plays a role in sleep regulation, hunger, mood states, and pain perception
Hormonal activity
Plays a role in aggression and sexual behavior
Major actions of GABA
Plays a role in inhibition; reduces aggression, excitation, and anxiety
May play a role in pain perception
Has anticonvulsant and muscle-relaxing properties
May impair cognition and psychomotor functioning
Major actions of acetylcholine
Plays a role in learning and memory
Stimulates parasympathetic branch of autonomic nervous system for "resting and digesting" actions
Affects sexual and aggressive behavior
Major actions of glutamate
Excitatory
AMPA plays a role in learning and memory
Major actions of norepinephrine
Level in brain affects mood
Attention and arousal
Stimulates sympathetic branch of autonomic nervous system for “fight or flight” in response to stress
Somatic disorders: symptoms
Pain or fatigue, to the point of excessive concern
Preoccupation
Fear
Common symptoms for primary care visits → chest pain, fatigue, dizziness, headache, swelling, back pain, shortness of breath, insomnia, abdominal pain, and numbness
Eating disorders → symptoms
can wax and wane over the life span
Anorexia nervosa: low weight, amniorrhea, muscle weakening, constipation
Bulimia nervosa: normal to low body weight, tooth erosion, muscle weakening, gastric dilation, calluses, scars on hand (Russell sign)
Binge-eating: Dysfunctional eating pattern, eating in response to internal cues, sedentary lifestyle, attempts to hide weight gain, eats as a coping method
Eating disorders → intervention
Collaborate with other members of healthcare team to develop treatment plan.
Involve patient and/or significant others in the treatment plan.
Work with team to set a target weight.
Consult with a dietitian to determine caloric intake necessary to attain and/or maintain target weight → encourage patient to discuss food preferences with dietitian.
Mania symptoms
Happiest, most excited, and most optimistic people you could meet
They feel euphoric and energized
They eat and sleep little, if at all, and are in perpetual motion
They feel so important and powerful → take horrific chances and engage in hazardous activities
As the mania intensifies, individuals may become psychotic → hallucinations, delusions, and dramatically disturbed thoughts
Differences in mood disorders → fear
Threat is known, external, definites, conflictual
Differences in mood disorders → anxiety
Threat is unknown, internal, vague, and conflictual
Mild: perceptual field → heightened, flexible focus, aware
Moderate: perceptual field → narrowed, focused on source, less aware and attentive
Sever: perceptual field → greatly reduced, focused on specific detail, scattered attention; no instruction
Panic: perceptual field → unable to attend to environment, lost focus; no instruction
Differences in mood disorders → OCD
Obsessions: unwanted, intrusive, persistent, ideas, thoughts, impulses or images that cause anxiety or distress
Compulsions: unwanted, ritualistic behavior the individual feels driven to perform to reduce anxiety
Exist along continuum
Differences between perceptual/psychosis disorders
Schizoaffective disorder → psychotic disorder with co-occurring mood disorder
Schizophrenia disorder → psychosis symptoms are independent of any mood disorders
Bipolar disorder with psychosis → psychosis symptoms start with increased severity of maina, and get worse as maina worsens
Depression with psychotic features → psychosis symptoms start with increased severity of depression, and get worse as depression worsens
Symptoms of schizophrenia → positive
The presence of symptoms that should not be present.
Hallucinations, delusions, paranoia, or disorganized or bizarre thoughts, behavior, or speech
Symptoms of schizophrenia → negative
The absence of qualities that should be present
Inability to enjoy activities (anhedonia), social discomfort, or lack of goal-directed behavior
Alcohol intoxication → symptoms
Drowsiness, N/V, slowed reaction, blackouts
Opioid intoxication → symptoms
psychomotor retardation, drowsiness, slurred speech, altered mood (withdrawn to elated), and impaired memory and attention
Physical → miosis (pinpoint pupils) and decreased bowel sounds. Respiratory rates and blood pressure are reduced and heart rates are normal to low
Alcohol and opioid intoxication → assessment priorities
Assess VS
BP, HR, RR
Assess airway
Pulse ox and RR
Maintain airway
Alcohol intoxication → risks
undergoing withdrawal to the point of psychosis → considered a medical emergency because of the risks of unconsciousness, seizures, and delirium
Withdrawal seizures → may occur within 12 to 24 hours after alcohol cessation. These seizures are generalized and tonic-clonic
Alcohol withdrawal delirium → medical emergency that can result in the death in 20% of untreated patients → pneumonia, renal disease, hepatic insufficiency, or heart failure
Opioid intoxication → risks
Death due to respiratory arrest due to respiratory depressant effect of the drug
OD → S/S: unresponsiveness, slow respiration, coma, hypothermia, hypotension, and bradycardia.
Three symptoms—coma, pinpoint pupils, and respiratory depression—are strongly suggestive of overdose
PTSD → symptoms
Illusions and delusions
Paranoia and auditory pseudohallucinations
Vivid flashbacks to trauma
PTSD → etiology
Prior trauma history or psychiatric history
Persistent stress response with amygdala hyperactivity, sympathetic NS, hypothalamic-pituitary-adrenal axis involvement
PTSD treatment → non pharmacological
Eye movement desensitization and reprocessing (EMDR)
Cognitive processing therapy (CPT)
Prolonged exposure (PE)
Specific cognitive behavioral therapy, narrative exposure therapy (written or oral) and brief electric therapy
PTSD treatment → pharmacological
SSRI and SNRI
Sertraline (Zoloft); Paroxetine (Paxil); Venlafaxine (Effexor); Fluoxetine (Prozac)
PTSD treatment → others
Prozan for nightmares
Clonidine, Propranolol (help alleviate hyperarousal or physical symptoms)