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Amplification
increasing the magnitude of a signal
Amplifier Gain
exact magnitude of amplification
Amplifier Linearity
degree to which gain is constant across all input
magnitudes
Filters
reshape the acoustic waveform to eliminate selected energy above or below a certain frequency range
Importance of Acoustic Measurement
Can provide objective and noninvasive
analysis of vocal function
What are 5 Common Acoustic Measures
Fundamental frequency
Intensity
Perturbation measures
Ratio to signal-energy to noise
spectral or cepstral features
Fundamental Frequency
the rate of vibration of the vocal folds and is expressed in Hertz (Hz) or cycles per second (cps)
Intensity
referenced to sound pressure level (SPL) and
measured on a logarithmic decibel (dB) scale
Vocal intensity is the acoustic correlate of vocal
loudness
Perturbation Measures
Cycle-to-cycle variability in a signal (typically
measured from sustained vowel productions or
“extracted” vowels from connected speech).
• Jitter = cycle-to-cycle variability in frequency
(a.k.a. frequency perturbation, pitch
perturbation).
• Shimmer = cycle-to-cycle variability in amplitude
(a.k.a. amplitude perturbation).
• Calculation requires a quasi-periodic signal for
reliable/valid perturbation analysis (i.e., Type I
signal).
Perturbation Measures
Jitter vs. Shimmer
• Jitter = cycle-to-cycle variability in frequency
(a.k.a. frequency perturbation, pitch
perturbation).
• Shimmer = cycle-to-cycle variability in amplitude
(a.k.a. amplitude perturbation).
Glottal Fry
should be observed for when doing oral mech exam
Kourtney K voice
Diplophonia
the production of two separate tones during a single voice
Spasmodic Dysphonia (SD)
Adductor Spasmodic Dysphonia
Abductor Spasmodic Dysphonia
Mixed Spasmodic Dysphonia
Adductor Spasmodic Dysphonia
Strained-strangled voice with voice stoppages/spasms.
Abductor Spasmodic Dysphonia
Involuntary breathy bursts/spasms.
Mixed Spasmodic Dysphonia
Both strained voice stoppages and breathy bursts.
Vocal Fold Nodules
Age and Gender Trends:
– Children (boys > girls, 3:1 ratio),
– Adults (women > men, rare among post-pubescent and adult males).
• Possible Personality Factors (Women with Nodules):
– Extraverted (talkative),
– Socially Dominant,
– Stress Reactive (tense), Aggressive,
– Impulsive.
• Occupational factors: singers; professions with extended teachers).
Mild to Moderate Dysphonia
– Roughness, breathiness related to gaps anterior and posterior to
lesions, increased muscular tension.
• Severity of dysphonia varies depending upon:
– extent (size) of lesions,
– Length of time since onset (type of nodules, chronic vs. acute),
– Degree of accompanying inflammation.
• Treatment:
– First line = voice therapy,
– Surgical Removal by “skilled” laryngologist and only...
• If/when patient has been compliant with voice therapy, but did not
respond completely/satisfactorily,
• Surgery to be followed by post-surgical voice therapy
Vocal Fold Polyps
• Fluid-filled, exophytic lesion composed of gelatinous
material in SLLP with active blood supply (typically located
on middle third of the VF).
– Most often seen in adults,
– Often unilateral, but can be bilateral.
• Types:
– Sessile (blister-like),
– Pedunculated (attached to a stalk).
• Cause(s):
– Acute Vocal Trauma (i.e, phonotrauma).
• Voice Effects:
– Mild to severe dysphonia depending upon:
• Size, type, and location (and associated mass and stiffness effects),
• Degree of interference with glottic closure and VF vibration,
• Presence of hemorrhagic blood vessel “feeding the lesion.”
Treatment:
– Voice Conservation/Rehabilitation (primary)
– Phonosurgery (and voice rehab)
Vocal Fold Cysts
• Fluid-filled, typically unilateral, sessile lesions (sacs) on cephalic
surface or medial edge of the VF that can be:
– Congenital or Acquired.
– Embedded in SLLP, but often extend into ILLP and DLLP (i.e., the vocal ligament).
• No clear etiology, but...
– Mucous gland blockage, and/or
– Phonotrauma
Treatment: Surgical excision/dissection of the cyst off of the vocal ligament (from a superior and lateral approach to avoid scarring of the vocal fold)
Reinke’s Edema
• SLLP becomes filled with viscous, gelatinous fluid.
• “Polypoid degeneration” is a severe form of edema wherein the
entire membranous VF is filled with fluid.
• Etiologic Factors:
– Chronic Phonotrauma,
– Smoking.
• Vibratory Effects
– Increased mass and stiffness.
• Voice Effects:
– Signature low pitch and husky hoarseness described as a “whiskey”
or “smoker’s” voice.
• Surgery (accompanied/preceded by smoking cessation
program).
– Pre- and post-operative voice therapy.
Granuloma
unilateral or bilateral, vascular and inflammatory
exophytic lesions related to tissue irritation in the posterior larynx typically on medial surface of arytenoid cartilage(s).
S/S include:
– Pain, sore throat, with or without voice change (i.e., Posterior site of
lesion(s), may not affect VF vibration).
Two Primary Etiologies:
– “Mechanical” or “Chemical” tissue irritants of posterior larynx...
• Mechanical = endotracheal intubation,
• Chemical = laryngopharyngeal reflux.
– Persistent Voice Misuse...
• Pressed, low-pitched voice with excess tension.
Contact Ulcer
ulcerated lesion on the same site often on
opposite side of granuloma (cup/saucer relationship).
S/S include:
– Pain, sore throat, with or without voice change (i.e., Posterior site of
lesion(s), may not affect VF vibration).
Two Primary Etiologies:
– “Mechanical” or “Chemical” tissue irritants of posterior larynx...
• Mechanical = endotracheal intubation,
• Chemical = laryngopharyngeal reflux.
– Persistent Voice Misuse...
• Pressed, low-pitched voice with excess tension.
Fundamental Freq =’s…
Pitch
S/z ratio
an assessment used to look at respiration
times the longest duration of /s/ and /z/ phonemes to analyze phonation and respiration capabilities
Typical ratio with vocal fold problems is around 1.4 and would want the ratios to be the approximately the same
Phonation threshold pressure (PTP)
amount of pressure needed for VF vibration
Subglottal Pressure
the pressure of the air coming up through the vocal chords from beneath.
Laryngeal Resistance
the quotient of peak intraoral pressure
(estimated from the production of an unvoiced
/p/) divided by the peak flow rate (measured
from the production of a vowel /i/) produced in
a repeated train of /pi/ syllables.
Intended to reflect overall resistance of the
glottis and estimates laryngeal valving function
(cm H20/l/sec).
– Hyperfunction (valve too tight): LR too high
– Hypofunction (valve too loose): LR too low
– Normal (as compared to normative values)
How do the intrinsic laryngeal muscles work?
Synergistically
Cricoarytenoid Joint
serves to abduct and adduct the vocal folds
Cricothyroid Joint
serves to abduct and adduct the vocal folds
5 layers of the vocal folds
epithelium, lamina propria (superficial, intermediate, deep), vocalis muscle
Epithelium
Outermost, mucosal layer, thin pliable capsule
– Thin layer of slippery mucous lubrication needed for
vocal folds to oscillate best
• Mucociliary blanket covers epithelium
– Mucinous layer (outermost viscous protective layer)
– Serous Layer (watery layer with cilia)
• Epithelium
– Exposed to environmental influences
• Humidity
• Dehydration
• Pollution
• Reflux
• Basement Membrane Zone (BMZ)-transition zone
Lamina Propria
• Three layered Structure
– Superficial layer (SLLP)
– Intermediate (middle) layer (ILLP)
– Deep layer (DLLP)
• Each layer composed of distinct concentrations of
fibrous proteins (connective tissue)
– Elastin (allows tissue to deform/stretch)
• Most concentrated in SLLP and ILLP
– Collagen (less stretch, but tolerates stress and provides
tensile strength)
• Most concentrated in ILLP and especially in DLLP
– As progress from superficial to deep layers of the LP there
is increasing density/stiffness
– LP vibrates passively in response to aerodynamic forces
Lamina Propria continued
Superficial layer (SLLP) - a.k.a. “Reinke’s Space”
– Loose and Flexible
– Soft, slippery, gelatin-like substance
– Vibrates significantly during phonation
• Intermediate (middle) layer (ILLP)*
– Mostly elastin fibers (some collagen)
– Also vibrates during phonation
• Deep layer (DLLP)*
– Mostly collagen fibers (fewer elastin fibers)-most dense layer of LP
– Interspersed with muscle fibers to join LP to underlying vocalis
muscle
*Note: The combined intermediate and deep layers of the lamina
propria is also known as the “Vocal Ligament”.
Vocalis Muscle
Fifth histological layer (most dense)
• Forms the “body” of the vocal fold and
provides:
– Tone
– Stability
– Mass
• Vocalis still oscillates during vocal fold
vibration (but not as much as cover and
transition layers of VF)
How do lengthening and thickening relate to pitch
More lengthening = high pitch
More thickening = low pitch
Stroboscopy
A special lighting technique wherein a stroboscopic light
flashes at specific moments to form a composite vibratory
cycle, derived from many single points along multiple
cycles.
• The strobe light flashes at a phase point in VF vibration that is slightly faster than the Fo. Each successive flash captures a series of separate images, sampled from different points of VF vibration.
• Stroboscopy produces an apparent (not real!!) slow motion effect.
• Like other measures, significant aperiodicity in the voice, compromises the clinical utility of stroboscopy because a “stable” fundamental frequency can not be extracted.
What is the most common type of imaging?
stroboscopy
What does the GRBAS measure?
G: Grade or a judgment of how rough the voice sounds
R: Roughness is a judgement of how irregular and noisy the voice sounds; it should relate to aperiodicity in the vibratory cycle
B: Breathiness is a judgement of how much additional airflow is perceived; it should relate to higher minimum airflow during the glottal cycle
A: Asthenia is a judgement of how weak the voice sounds; it should relate to the sound pressure level of the voice
S: Strain is a judgement of how compressed or hyperfunctional the voice sounds
GRBAS vs. CAPE-V
CAPE-V is more reliable
Whats the difference between GERD and LPR?
GERD is a backflow of stomach contents into the esophagus; LPR is backflow from stomach all the way into the throat (it is aerosolized, essentially a mist of relflux
Muscles of Inspiration
- Diaphragm
– External Intercostals
– Sternocleidomastoids
– Scalenes
– Pectoralis (major and minor)
Muscles of Expiration
-Internal Intercostals
– Rectus Abdominis
– Transverse Abdominis
– Internal Obliques
– External Obliques
Extrinsic LARYNGEAL MUSCLES
Suprahyoids vs. Infrahyoids
• Suprahyoid Muscles - Raise the larynx
– Stylohyoid
– Mylohyoid
– Digastrics (anterior and posterior bellies)
– Geniohyoid
Infrahyoid Muscles - Lower the hyoid and
larynx
– Thyrohyoid
– Sternothyroid
– Sternohyoid
– Omohyoid
Intrinsic Laryngeal Muscles
Five Intrinsic Laryngeal Muscles
• Affect the position, length, and tension of the
vocal folds
– Change position of the cartilage framework that
houses the vocal folds.
– Alter the length, tension, and shape of the vocal
fold edge.
– Change the shape of the glottal opening between
the vocal folds.
intrinsic laryngeal muscles cont
Cricothyroid m. (CT)-Tensor
• Thyroarytenoid m. (TA)-Adductor
• Lateral Cricoarytenoid m. (LCA)-Adductor
• Interarytenoid m.(IA)-Adductor
– Transverse (Horizontal)
– Oblique (Crossed)
• Posterior Cricoarytenoid m. (PCA)-Abductor
Superior Laryngeal Nerve Paralysis (External Branch):
Unilateral
• Unilateral ESLN paralysis = Unilateral Cricothyroid muscle dysfunction.