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Seizures
Arrhythmia in the nerve cells- defined as episodes due to abnormal electrical activity in the the nerve cells, a localized area or all of the brain is involved
Types of seizures
generalized- petite mal and tonic clonic (whole brain is affected) , focal, and epilepsy- chronic seizure condition
Seizures are caused by
tumors, cvd, cns, infection, withdrawal, fever in children, head injury, hypertension, hypoxemia, and metabolic/ toxic conditions, need to find underlying cause !
what to give during a seizure
Lorazepam
medication given after seizure
Iv infusion phosphenytoin
seizures are dx by
eeg , ct and MRI
med mgmt for seizures
Carbamazepine , anticonvulsants, diazepam benzodiazepines, and surgery- if epilepsy is due to a tumor or abscess
DX of epilepsy
two or more seizures in a lifetime, unprovoked recurring seizures
Nursing mgmt durring seizures
prevent injury and aspiration , time the seizure, loosen clothing, protect head, implement seizure precautions low bed, side rails up, padded rails, don’t restrain, monitor airway,
position of seizure patient
turn them on their side
document seizure
length of seizure, warning sign, movements during, any incontinence , paralysis following
Education
Taking their meds daily at the same time and get serum labs drawn regularly , dont put anything in their mouth !!
Carbamazepine
anti convulsant med worried about bone marrow suppression and drawing labs
Absence seizures petit mal time
lasts 2-15 seconds
s/s of absence seizures
staring, eye fluttering, automatisms - if prolonged, lip smacking, picking at clothes and fumbling
post absence seizures s/s
amnesia, no confusion and promptly resumes activity - these patients act like nothing happened post seizure
Generalized tonic clonic seizures time
1-2 minutes
signs and symptoms of generalized tonic- clonic seizures
crying, falls, tonicity, rigidity, cloniciity , jerking , incontinence of urine potentially cyanosis, tongue is often chewed
Tonic clonic patients may experience
an Aura ; taste, smel, sound and sight
post tonic clonic seizure s/s
amnesia, confusion, and deep sleep. involves both hemispheres of the brain causing both sides of the body to react - these patients are affected post seizure !
Status epilepticus time
lasts 5+ minutes or involved serial seizures without full recovery of consciousness in between
tx for status epilepticus
stop the seizure, give iv benzodiazepines, then an anticonvulsant, medical emergency
EEG
measure electrical activity of the brain - appears as wavy lines like a ekg strip- tall waves called spikes indicate brain cells are firing strongly -means pt is going to have a seizure
spinal cord injury
defines as the result of concussion, contusion , laceration or compression of the spinal cord
primary sci
initial trauma - permanent damage
secondary sci
result of ischemia, hypoxia or hemorrhage that destroys nerve tissue- can be treated if caught early
sci caused by
mvas, falls, violence, and sport injury
risk factors sci
alcohol, drug use, and male gender about 80% of sci
major cause of death in sci
PNA, PE and sepsis
SCI dx by
xray and ct initially, then mri potential ligamentous injury can be present without bony injury , myelogram -if mri is contraindicated
s/s of sci
depend on location of injury
at or above c4
respiratory paralysis and tetraplegia
c5-6
paralysis of leg/wrists/hands, weakness of shoulders, loss of bicep reflex
c6-c7
paralysis of legs/wrists/ hands
c8-T1
horner syndrome and paralysis of legs
T1 and conus medullaris
paralysis of legs
med mgmt for sci
methylprednisolone, riluzole - prevents further damage to nerves , eeg( bradycardia and asystole common), oxygen (hypoxia can worsen neuro deficits), spinal surgery
nursing mgmt sci
monitor respiratory status , changes in motor and sensory function , temperature, monitor for bladder retention and distention, assess skin integrity and traction and pin care
complication of sci
spinal shock, neurogenic shock- risk for within first 24-72 hours and autonomic dysreflexia- always at risk
spinal bcord injury between c3 and c5
will impact breathing
Horner syndrome
constricted pupils, ptosis, and facial anhidrosis
emergency mgmt sci
1st cervical collar 2nd log roll onto back board 3rd run tests to determine if its an SCI ( any twisting or moving can cause permanent damage)
spinal shock
results in sudden depression of reflex activity in the spinal cord aka areflexia that occurs below the level of injury
s/s of spinal shock
paralyzed , flaccid, absent reflexes - below level of injury , bradycardia, hypotension, bowel bladder dysfunction * distention and paralytic ileus- often occurs within the first 2-3 days after sci and resolves within 3-7 days
med mgmt of sci
maintain a mean arterial pressure map of 85 or higher during hyperacute phase and NG tube for decompression * paralytic ileus and bowel distention
Neurogenic shock
develops due to the loss autonomic nervous system function below the level of the lesion - affects vital organs
s/s of neurogenic shock
decrease in bp, hr, cardiac output, peripheral edema, peripheral vasodilation, lack of ability to perspire in paralysed locations - monitor for fever - most of concern bc it affects ANS
Autonomic dysreflexia aka autonomic hyperreflexia
defined as acute life threatening emergency that occurs due to exaggerated autonomic response to stimuli that are harmless in people without sci - occurs after spinal shock has resolved
risk factors for autonomic dysreflexia
patients with cord lesions above T6- the sympathetic visceral out flow level, after spinal shock has subsided
Triggers for autonomic dysreflexia
distended bladder (most common cause )- insert cath drain bladder , distention or contraction of visceral organs, constipation-impaction, or stimulation to skin tactile, pain thermal stimuli, pressure injury
s/s of autonomic dysreflexia
severe pounding headache, sweaty, confused hypertension , diaphoresis, nausea, congestion, and bradycardia
mgmt of autonomic dysreflexia
remove and avoid triggering stimulus, IV hydralazine tx htn
nursing mgmt for autonomic dysreflexia
place in sitting position hob 90 degrees and legs lower - lowers bp, empty bladder with catheter, manual removal of fecal mass , admin topical anesthetic 10-15 min prior , if not can enhance the dysreflexia, assess skin, increase fluids and fiber, remove cold drafts
complications of autonomic dysreflexia
due to htn, retinal hemorrhage, hemorrhagic stroke, MI, seizures , emergent situation usually happens years after sci !
post spinal surgery
pain mgmt, cervical collar , no prone , no driving, immobilize neck
tx for spinal cord alterations
conservative rest and meds, steroids , surgery is last option . NSAIDS good medication ice
spinal cord alterations assessment
neuro exam - DTR deep tendon reflex
Brain tumor
usually discovered after patient has had seizure
blood brain barrier
control what comes in and out of brain , 98% of meds do not pass blood brain barrier , if tumor there it is difficult to tx with meds very hard to penetrate barrier
clinical manifestations of neurological tumors
based on location, change in loc, visual disturbances loss of hearing seizures, headache, hormonal effects, increased icp and herniation
temozolomide
med mgmt for brain tumor- oral chemo that is able to cross blood brain barrier
Brachytherapy
seed that is placed directly into cancer, worry about skin integrity - stay away from children and pregnant women
Corticosteroids
dexamethasone - tx for brain tumor , relieves headache and altered loc
pre op brain tumor
prophylactic meds antibiotics to reduce potential infection , diazepam for anxiety , antiseizure, mannitol diuretics fluid restriction ,baseline needed! , ct, mri, angiography or doppler flow studies
assessment post of care brain tumor
neuro assessment compare baseline , VS, loc, CSF leak, cerebral edema, icp , periorbital edema, gag reflex, motor function
CSF leak post off brain tumor
fluid, dressing post nasal drop, salty taste= finger stick strip
nursing intervention post op care brain tumor
cool compress, elevate hob, suction with caution, position, regulate temp, ROM