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Light
electromagnetic energy measured and described in wavelengths
hue
color; determined by the wavelength of light
intensity
amount of energy in light/ sound; determined by waves amplitude (ex. neon has higher than baby yellow)
Amplitude
the height of a wave
Cornea
dome shaped covering on front of the eye (where light enters) - transparent and PROTECTIVE
Pupil
black part; adjustable opening through which light passes (size controlled by iris)
Iris
ring of muscle tissue; colored portion'; controls pupils’ size
iris expand/ pupil contract
bright light
iris contract/ pupil expand
dark light
lens
transparent structure behind pupil which changes shape to focus on images (malfunction = need glasses)
Accommodation
process in which the lens changes structure to focus light on the retina
Vitreous Humor
thick transparent liquid in the eyeball which helps to give shape (clearness = lets light through)
Retina
light-sensitive inner back surface of the eye where vision happens (contains photo-receptors/ cones and rods)
rods
help see peripheral vision and black, gray, and white
cones
central vision that helps to see color (many at the fovea)
fovea
little dip in the retina where the lens focuses the light (central focus area)
Bipolar and Ganglion cells
help to TRANSDUCE light waves into neural impulses for optic nerve
Optic Nerve
Sends neural impulses from the eye to the brain
Blind Spot
where the optic nerve connects to the eye, no neurons here so cannot see!
Acuity
the sharpness of one’s vision (measure by vision chart)
Myopia (near)
easier to see close objects (light projected in front of fovea)
Hyperopia (far)
easier to see further objects (light projected behind fovea)
Astigmatism
blurring of image at retina (intense glare, typically at night) - caused by irregular shape of cornea/ lens
Retinopathy
damage to blood vessels of retina (blood cells leak into vitreous humor and cause floaters)
Glaucoma
fluid pressure builds up in eye/ eye swelling, causes damage to optic nerve (blurred vision and loss of peripheral vision)
Macular Degeneration
comes with age; dark spots in center of vision because there is less cell replication and degeneration of eye cells
Cataracts
with age; clouding of the lens (white in eye)
rods and cones - b and g cells - optic - opp thalamus - visual cortex
Visual info processing path
Parallel Processing
the brain’s natural mode of processing several things at once (see vision subdivisions, more than just color to see)
Vision subdivisions
movement, depth, form, and color
Hubel and Weisel
scientists who proved vision subdivisions
color vision
the colors one can see determined by amount of cones (humans have low/ limited while other animals can have more/ humans see rainbow only)
Young-Helmhoitz Trichromatic Theory
a theory of vision that the retina has THREE CONES which BLEND to let us see all other colors. These cones are red, green, and blue. If we cannot see a color, it is because it cannot be made from one of those three cones.
Color Deficiency/ Blindness
cones in the eye don’t function properly
Protanope
red cones don’t function right (hard to see red, looks brown)
Deuteranope
green cones don’t function right (green color is hard to see) - MOST COMMON!
Tritanope
Blue cones don’t work right (blue color is hard to see)
Afterimage
after viewing a color for a long period of time, then it is removed, one can see the COMPLIMENTARY COLOR! Think of the bee (black and white seems colorful)
Opponent Process Theory
theory of vision in which our ability to see color is controlled by 3 RECEPTOR complexes. These receptors are tuned ON / OFF, like a switch and only ONE can be activated at a time. Ex. red receptor suppresses green because of such different hue/ amplitude = not at same time.