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Dr. ML (Emergencies and Urgencies pt 1/2)
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what may cause transient “loss of vision”?
more common (few sec)
papilledema
more common (few min)
amaurosis fugax
vertebrobasilar artery insufficiency
more common (10-60 min)
impending CRVO
ischemic optic neuropathy
glc
sudden change in BP
CNS lesion
optic disc drusen
giant cell arteritis
what may cause painless loss of vision for longer than 24 hrs?
sudden, painless (more common)
retinal artery or vein occlusion
ischemic optic neuropathy
vitreous hemorrhage
RD
optic neuritis
other retinal or CNS disease
gradual, painless (more common)
cataract
RE
open-angle glc
chronic retinal disease
chronic corneal disease
optic neuropathy/atrophy
what may cause painful loss of vision longer than 24 hrs?
acute-angle closure glc
optic neuritis
uveitis
corneal hydrops
non-physiologic visual loss
what may cause monocular double vision?
more common
RE
astigmatism → monocular
strab → binocular
corneal opacity/irregularity
cataract
less common
dislocated natural lens/lens implant
macular disease
RD
non-physiologic
what may cause binocular double vision?
intermittent
myasthenia gravis
intermittent decompensation of an existing phoria
constant
6th, 3rd, 4th nerve palsy
orbital disease
cavernou sinus/superior orbital fissure syndrome
status post ocular sx
status post-trauma
intranuclear ophthalmoplegia
vertebrobasilar artery insufficiency
other CNS lesions
spectacle problem
what may cause flashes of light?
RD, retinal break
PVD
migraine (diagnosis of exclusion)
rapid eye movements
trauma
less common
CNS disorders
retinitis
“true” anterior segment ocular emergencies
chemical burn
have pt start treating it on site - flush w water
corneal laceration
acute angle closure
orbital cellulitis
trauma-related issues - penetrating ocular injuries
vision threatening - corneal ulcer (microbial keratitis)
”true” posterior segment ocular emergencies
RD
CRAO (Central retinal artery occlusion)
arteritic ischemic optic neuropathy (AION, AAION)
CNIII palsy w blown pupil
bilateral optic nerve elevation (papilledema)
transient visual obscuration/amaurosis fugax
manipulation of the eye in susceptible individuals will induce a ____
induce a strong parasympathetic reaction
any bodily function controlled by parasympathetic system can be affected
may lose sphincter control, urinate, defecate during episode
vomiting common
pts don’t faint quietly, may lat 30-90 sec
potential signs and symptoms of fainting (vasovagal syncope)
tunnel vision
blurry vision
pale skin, cold clammy sweat
lightheadedness, nausea
dilated pupils
twitching
slow, weak pulse
types of seizures
focal seizures (60%)
generalized seizures
“grand mal” seizure (1-3 min, lose control)
epileptic seizures are rare
cal 911 if it lats more than 3 min
anti-seizure meds
1st line meds for tonic-clonic seizures
valprioc acid (Depakene, Depakote)
lamotrigine (Lamictal)
topiramate (Topamax)
1st line for partial seizures
carbamazapine (Tegretol)
phenytoic (Dilantin)
oxcarbazepine (Trileptal)
*phenobarbital most common for very young children
common signs and symptoms of heart attack (MI)
chest pain/discomfort
upper body discomfort (arm, back, jaw, etc)
SOB (shortness of breath)
cold sweat, feeling tired for no reason, nausea
listen and look, then CAB (compressions, airway, breathing)
types of strokes
hemorrhagic
ischemic
“stroke to eye”
signs and symptoms of stroke
FAST
Facial drooping
Arm weakness
Speech difficulty
Time to call emergency services
define anaphylaxis
severe type I allergic response
pt exposure, snacks/food in office
injection of epi-pen
clean area, prepare tissue, hold leg firmly near injection site and inject perpendicularly (usually upper of middle of outer thigh), swing and push auto-injector firmly until it clicks, hold at least 3 sec
normal BP? elevated BP?
normal: less than 120 and less than 80
elevated: 120-129 and less than 80
stage 1 HTN? stage 2 HTN? hypertensive crisis?
stage 1: 130-139 or 80-89
stage 2: 140 or higher or 90 or higher
hypertensive crisis: higher than 180 and/or higher than 120
immediate management of ocular chemical injuries
lavage for 30 min
topical anesthetic q15-20 min
litmus eval (pH paper)
immediate management of corneal laceration/open globe
fox shield the eye
photodocument for meicolegal purposes
avoid topical meds and seek immediate repair
signs and symptoms or acute angle closure
pain, photophobia
blurred vision
halos around lights
severe headache, nausea, vomiting
closed angle in involved eye
acutely elevated IOP
corneal edema
narrow angle in the other eye
injection, mid-dilated pupil, shallow chamber
immediate management of acute angle closure
gonio and IOP eval
in-office pressure lowering
signs and symptoms of orbital cellulitis
blurred vision
red eye, pain
fever
diplopia, headache
associated with
sinus infection
orbital fracture
focal infection around the eye
secondary to complications of orbital sx or retained FB
signs
eyelid edema
erythema, warmth, tender
chemosis, injection, proptosis
restriction or pain with eye movement
management of orbital cellulitis
immediate
look for APD, EOM, extensive hx
refer
blood work, CBC with differential
CT scan of orbits and sinuses
lumbar puncture and neuro consult
should improve in about 1 day
signs and symptoms of RD
flashes, floaters
curtain or shadow moving in vision
vision loss
pigment/RBC in anterior vitreous
PVD
decreased IOP in affected eye
movement of tissue within eye
ddx of RD
PVD
with retinal tear
with retinal tear and vitreous heme
rhegmatogenous RD
management of RD
immediate referral for eval with retinal specialist
pt will need bed rest until surgical repair
what are some emergencies may require calling 911?
vasovagal syncope (fainting)
epileptic seizures
cardiac and respiratory arrest
stroke
malignant hypertension/hypertensive crisis
anaphylaxis
management of vasovagal syncope (fainting)
don’t let the pt fall
can handle in office without EMS assistance
never leave the pt alone
protect yourself with gloves when contacting bodily fluids
turn pt head or lean them over slightly while holding garbage can under their face
what triaging questions can be asked?
why are you interested in getting eye exam?
emergency or routine exam?
symptoms? when did they start?
*PFE = not an emergency
urgency vs. emergency
urgency = need to see them STAT
emergency = refer out IMMEDIATELY
what minimum 4 points need to be asked during an emergency red eye case hx?
TADD
Time of day - when does it occur (morning, evening)
Association - what are you doing when it happens
Duration - how long it happens (once in awhile, nonstop)
Description - which eye
what subjective pertinent negatives need to be documented in an emergency red eye case?
± chemical to eye
± CL wear
± previous occurrence
± pain (with or without light)
± discharge
± redness
look see feel for red eye
look
redness, swelling, whiteness or spots, discharge
see
blurry vision, blacked out area, distortion, light changes, vision coming and going
feel (ask pt)
pain, itching, light sensitivity, FBS
tips for taking BCVAs in an emergency red eye case
monocular for both eyes
don’t remove CL if they wore them in
even if the red eye only affects one eye, check the unaffected eye as the baseline
if VA worse than 20/30 do PH
in an emergency red eye case, pupils testing and EOMs will show:
pt will feel pain when the pupil restricts, pain on eye movement
pain on direct (red eye is OD, light in OD) = ulcer
pain on consensual (red eye is OD, light in OS) = severe ulcer (cells/flare)
pertinent negatives of an emergency red eye (post seg)
± staining
± AC reaction
± follicles or papillae
± preauricular nodes
± flashes/floaters
± holes/tears
when would you perform a DFE for an emergency red eye?
if indicated based on hx → metal on metal FB, flashes/floaters, decreased BCVA w/o ant seg finding
usually dilate 1 eye only
BIO + scleral depression if flashes/floaters, vitreo-retinal tuft, peripheral lattice, holes, Shafer’s sign
management of globe protrusion (non emergency “emergency”)
globe leaves socket, still attached by optic nerve
SL manipulation, CL A/R, lid eversion
systemic hx: TED, ocular tumor, other causes of proptosis
stay calm, undo lid manipulation
lean pt back (use gravity to advanatage)
gently massage globe with lids until it returns to socket
_____ is unilateral, painless, severe vision loss
central retinal artery occlusion (CRAO)
occurs in seconds
if cilioretinal artery → less vision loss