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myopia
nearsighted
Cant see MY own hand
hyperopia
farsighted
astigmatism
imperfect eye curvature
presbyopia
normal loss of accommodation with age
PRESBY- Older age → PRESident
hordeolum
sty
chalazlon
chronic inflammatory granuloma of. sebaceous gland
warm compress/lancet
blepharitis
chronic bilateral inflammation of a lid margin
prevent: hand washing, antibiotics with bacterial
conjunctivitis
infection of the conjunctive (pink eye)
viral/allergic
eye crusted shut → bacteria
keratitis
infection of the cornea
more SEVERE than rest
aggressive treatment with antibiotics
ex: contact lenses, makeup products, hand hygiene
cataracts
very common in old age
cloudy lens
may be in one or both eyes
can get surgically removed
are visible to the eye.
what cause cataracts
diabetes
certain medications
blunt penetrating trauma to the eye
symptomology of cataracts
gradual vision loss
abnormal color perception
GLARE***** (scattering of light, worse at night when pupils dilate)
after ABC are established
when someone comes in with visual acuity problems, DO A VISUAL ACUITY TEST!!!!!!!!!!
interocular pressure NORMAL
10-21
cycloplegic
paralysis of accommodation and dilation
phacoemulsificaton
most common type of surgery
vibrations to dissolve clouded lens (small incisions or tears)
post op of cataracts
meds: antibiotic and corticosteroids drops (to prevent inflammation/infection)
can come off once eye fully healed.
SURGERY ON ONE EYE AT A TIME!
have to have a ride home after procedure (OUTPATIENT)
implementation of cataracts
no specific preventative measures
wear sunglasses, avoid unnecessary radiation (tanning beds)
antioxidant vitamins (C and E); adequate nutrition
pros and cons education.
if increased/intense pain
CONTACT PROVIDER ASAP
could be a sign of intraocular pressure
can damage optic nerve
lead to permanent vision loss/blindness
ambulatory care
most have little visual impairments
review instructions with patient and caregiver
if significant visual impairments, activity and environment modifications (Bending, coughing, bearing down)
use of eye patch: altered depth perception, fall precautions
CRAIG the crusty dog
Color perception abnormal
Reduced vision
Age-related
I need a ride after surgery
Glare
retinopathy
microvascular damage to the retina, blurred vision; progressive loss of vision
Most common with HTN or diabetes mellitus
diabetes retinopathy
can prevent with tight glucose control!
nonproliferative: loss of CENTRAL VISION, most common.
proliferative: advanced disease, severe vision loss,
treatment: laser photocoagulation
hypertensive retinopathy
blockages in retinal blood vessels from hypertension.
treatment: lower BP to restore vision
papylodemia
MEDICAL EMERGENCY → swelling of optic disc and nerve, causes sudden vision loss
retinal detachment
retina detached from where supposed to be. fluid accumulation between layers.
related to trauma
MEDICAL EMERGENCY
breaks, holes (spontaneous), tears (aging or trauma)
********EXPERIENCE COBWEB/HAIRNET in field of vision*******
risk factors for retinal detachment
age
eye trauma
recent cataract surgery
family/personal
severe myopia
manifestations of retinal detachment
photopsia
floaters
COBWEB/HAIRNET in field of vision
UNTREATED: LEAD TO BLINDNESS IN INVOLVED EYE
person comes in with suspected retinal detachment
VISUAL ACUITY TEST
laser photocoagulation
light beam makes inflammatory reaction
helps push retina up against skin behind to reattach.
cryoplexy
freezing → SCAR
freeze, thaw (ON REPEAT) - seals break.hole
scleral buckling
band placed around globe
band around eye to keep it attached
more things around to push scleral toward detached retina
scleral buckles inward
retinal detachment intraocular procedures
pneumatic retinopexy: intravitreal injection of has to form bubble to close retinal break
keep patient in prescribed position.
PATIENT CAN BE AT RISK FOR RETINAL DETACHMENT IN OTHER EYE.
any symptoms following procedure, needs to be seen ASAP!
retinal detachment postoperative considerations
bedrest/activity restrictions
medications: analgesia and topical
patient education
promote the use of eyewear to protect from eye damage (trauma)
sports/mowing/woodworking
age-related macular degeneration (ARMD)
most common cause of irreversible CENTRAL VISION LOSS
ARMD Dry (nonexudative)
atrophy of macular cells
more common
slow, progressive, painless loss of vision
close vision tasks become harder for them.
ARMD wet (exudative)
more severe, abnormal blood vessels develop in or near macula
RAPID ONSET OF VISION LOSS; AMD related blindness
more wet had dry first
catch it dry before it leads to wet
ARMD manifestations
acute vision loss, blurred or darkened vision, scotomas (blind spots), and metamorphopsia (visual distortion)
ARMD test
VISUAL ACUITY TEST
AMSLER GRID
DRUSEN SPOTS
in ARMD
small, yellow deposits of fatty proteins (lipids) that accumulate under the retina
BIP LIPS = LIPIDS
use an ophthalmoscope to look for drusen in the fundus
ARMD interprofessional care
medications directly injected every 4-6 weeks into vitreous cavity to stop new vessel formation and slow vision loss.
photodynamic therapy: use dye and laser to damage abnormal blood vessels.
patient must avoid sunlight and intense light for 5 days.
glaucoma
can be caused by cataracts
NO CURE
catch early → can be fixed (REGULAR EYE EXAMS)
causes PERIPHERAL VISION LOSS
glaucoma pathophysiology
aqueous production (inflow) and aqueous reabsorption (outflow) must be balanced to maintain IOP.
inflow > outflow = increased IOP
increased IOP → permanent loss of vision
primary open-angle glaucoma
outflow of aqueous decreased; drainage channels clogged → optic nerve damage
develops slowly
IOP = 22-32
acute closure glaucoma
reduced outflow from angle closure
sudden, onset
painful
pressure leads to nausea and vomiting
see colored halos around lights
IOC over 50!!!!
optic nerve starts to atrophy.
focus of glaucoma
decrease IOP to prevent blindness
low enough to prevent optic nerve damage (blindness)
AACG
OCULAR EMERGENCY
immediate intervention
miotics → work to decrease production of fluid in eyes.
Myotic eye drops = pupil constricts and angle to open to allow outflow. USE IV diuretics, glycerin liquid
PATIENT WITH GLAUCOMA + COPD/ASTHMA
-olol beta blockers
can affect patient systematically
do not give if patient has heart block or bradycardia
otits media
infection in middle ear,
seen in children (antibiotics)
severe = ear tubes
presbycusis
TYPE OF SENSORY NEURAL HEARING LOSS
hearing loss associated with aging
tinitus
ringing in ears.
come along with some hearing loss
medications causing tinitus
monitor patient → relation to ototoxicity
figure out the meds and stop them!
nystagmus
eye shaking
babies/ neuro patients
inner ear
plays a big role in keeping us steady.
pt with inner ear infection → off balance and more prone to falls
Meniere’s Disease
progressive disorder
inner ear
usually affects 1 ear. can affect both
cause: UNKNOWN
Meniere’s disease symptoms
excess fluid and pressure → hearing and balance problems
disability - sudden, severe attacks of vertigo, nausea, vomiting, sweating: unpredictable
prior to attack: fullness in ear, tinnitus, and muffled hearing
some experience feelings of being pulled to the ground (DROP ATTACKS)
last hours, several times/year; variable
what to start with on a susptected pt with Meniere’s disease
HEARING TEST
glycerol test: pull fluid out of inner ear and improve symptoms
medications for Meniere’s disease
NO CURE
treating symptoms
corticosteroids, antihistamines, anticholinergics → draw fluid out, decrease production of fluid
benzodiazepines→ sedatives
vertigo: bed rest, sedation, antiemetics, or antivertigo
surgical interventions Meniere’s disease
between attacks: diuretics, corticosteroids, low-sodium diet, and stress reduction
decompression (shunting fluid away from inner ear)
vestibular nerve section (Cut nerve)
gentamicin injections (go through tympanic membrane, results in inner ear damage → save for severe cases)
Meniere’s disease nursing care
dark, quiet room
avoid sudden movements
close eyes during vertigo
avoid fluorescent, flickering lights and TV
emesis basin (to catch vomit)
PRN MEDS
patient safety for Meniere’s disease
fall precautions
montior intake and output
patient education: protect from injury/fall
NO SWIMMING UNDER WATER
NO CLIMBING HIGH SURFACES
Conductive hearing loss
external and middle ear
decrease sound intensity and/or distortion
impair transmission of sound
SOUND IN HEAD = LOUD
hear best in noisy environment
CAUSES:
infection
wax in ear
perforation of ear drum
narrowing of external canal
HEARING AIDS ARE HELPFUL!!!!!!!!!!!
Sensorineural hearing loss
inner ear
distortion or faintness of sound
alter the ability to understand speech
complete hearing loss
can lead to misunderstanding by others
SOUNDS MUFFLED; DIFFICULT TO UNDERSTAND, ESPECIALLY HIGH-PITCHED SOUNDS
audiogram: loss in decibel levels
HEARING AIDS MAKE SOUNDS LOUDER BUT NOT CLEARER
caused by:
impairment of the inner ear
vestibulocochlear nerve (CN VIII) damage
congenital and hereditary factors
noise exposure
aging (presbycusis)
Meniere’s disease
Trauma
ototoxicity
central hearing loss
impaired auditory pathways in the brain
problems understanding the meaning of words heard
conductive hearing loss expectations
audiogram → better hearing through BONE than AIR
patient often speaks softly
identify and treat cause
sensoineural hearing loss ototoxic drugs
aspirin - oral
antibiotics - slow, IV
loop diuretics - LASIX, IV PUSH, be careful. hearing loss
weber test
stem of vibrating tuning fork placed on midline of skull or forehead
pt asked to indicate where sound is best heard
conductive loss in 1 ear: sound will be heard louder in that ear
sensorineural: sound is louder in the normal (unaffected) ear
conductive hearing loss RINNE TEST
bone conduction is longer than air conduction
sensorineural hearing loss RINNE TEST
both air conduction and bone conduction are reduced; AC is longer
hearing loss and deaf patients
ALWAYS GET AN INTERPRETER
use visual aids- need to validate understanding
irritability and frustration with speech and understanding
withdrawal, depression, cognitive decline as advances
noise is the
most preventable cause of hearing loss
limit exposure of loud noises throughout lifespan
cochlear implant
DIRECTLY ADVANCES CN VIII
gerontologic considerations
elderly not wanting to improve as they age.
reluctant to use new technology
diminished ability to understand speech
cannot differentiate consonants
consonant sounds have HIGHER frequency