Adult Communication Disorders: Aphasia

expressive language, receptive language, written expression, repetition, reading comprehension

aphasia results in language abilities from brain damage that can affect up to these 5 modalities

CVA: ischemic, hemorrhagic, aneurysm, TBI, brain tumor, surgical trauma, infection, seizures

what are the etiologies of aphasia

anomia

word finding

anosognosia

not knowing what you don’t know. people are unaware of their deficits

prosopagnosia

difficulty with faces

agrammatism

difficulty with grammar

logorrhea

word vomit n

neologism

made up words

perseveration

repetition of a verbal or physical behavior

phonemic paraphasia

changing one phoneme to change meaning s

semantic paraphasia

changing one word for another every time

Alexia/agraphia

difficulty with reading/difficulty with writing

brocas, transcortical motor, global

nonfluent aphasias

wernickes, transcortical sensory, conduction, anomic

fluent aphasias

broca’s aphasia

name the aphasia: client comes in with halting, telegraphic speech but functionally intact receptive language. they have a hard time with word and phrase repetition but are aware of their deficits

transcortical motor

name the aphasia: client comes in with halting, telegraphic speech but functionally intact receptive language. their repetition is preserved and their articulation is intact

global aphasia

name the aphasia: client is non-verbal, and has difficulty with single word utterances, there is minimal receptive language and impaired repletion, they also present with several other deficits in cognition and motor abilities

wernicke’s aphasia

name the aphasia: client comes in with impaired repletion and logorrhea. their have severely impaired reception of langauge and they are unaware of their deficits

transcortical sensory

name the aphasia: client comes in with minimal receptive language but their repetition is intact. they have visual deficits and are unaware of their deficits

conduction

name the aphasia: client comes in with relatively intact receptive and expressive language but impaired repetition. the client is highly aware of their deficits

anomic

name the aphasia: client comes in with intact repetition but impaired receptive and expressive language. they are highly aware of their deficits and are quick to self repair words

progressive nonfluent aphasia

degenerative pathology of the frontal and temporal lobes that causes reduced MLU, anomia, slowed rate, but mostly intact receptive language

semantic dementia

fluent primary progressive aphasia that causes uninhibited speech, pragmatic deficits, and anomia

crossed aphasia

damage to the language dominant hemisphere is ipsilateral to the dominant writing hand and therefore spares the dominant hand

case history, functional communication, connected speech, standardized assessment, non-standardized assessment

assessment of aphasia

primary progressive aphasia and crossed aphasia

what are two types of atypical aphasia

Boston Diagnostic test of aphasia, western aphasia battery, communication activities of daily living

what are some standardized assessments for aphasia

spontaneous recovery for about 6 months, rationale for intervention, caregiver and patient education and training, and neuroplasticity

what are some treatments of aphasia

restorative, social, and compensatory

what are the three types of aphasia therapy s

Schuell’s stimulation, melodic intonation therapy, constraint induced, errorless learning therapy

what are some types of restorative aphasia therapy

AAC

what is an example of a compensatory aphasia therapy

group treatments and training communication partners

what are two social aphasia therapy techniques