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diverticulum
AN OUT-POUCHING MUCOSA AND SUBMUCOSA THAT PROTRUDES TO A WEAK PORTION OF MUSCULATURE OF ESOPHAGUS
sliding hiatal hernia (type 1)
paraesophageal hiatal hernia
2 types of hiatal hernia
sliding hiatal hernia
JUNCTION BETWEEN THE STOMACH AND THE ESOPHAGUS SLIDES UP THROUGH THE HIATUS
90% of hiatal hernia are this type
paraesophageal hiatal hernia
NO SLIDING UP AND DOWN A PORTION OF THE STOMACH REMAINS STUCK IN THE CHEST CAVITY
less common
HEAVY LIFTINGĀ
HARD COUGHINGĀ
Ā HARD SNEEZINGĀ
Ā PREGNANCY AND DELIVERYĀ
Ā VOMITINGĀ
Ā CONSTIPATIONĀ
OBESITY
causes of hiatal herniaĀ
HEART BURNĀ
Ā REGURGITATIONĀ
Ā DYSPHAGIAĀ
Ā SENSE OF FULLNES
S/S hiatal hernia
CHEST X-RAYĀ
Ā BARIUM X-RAYĀ
Ā UPPER ENDOSCOPY
Dx hiatal hernia
PPI
ANTACIDĀ
Tx hiatal hernia
NISSEN FUNDOPLICATION
Sx hiatal hernia
INCOMPETENT LOWER ESOPHAGEAL SPHINCTER (LES)
 ⢠AGING
 ⢠C-A-S-H (COFFEE, ALCOHOL, SPICY & HOT FOODS)
 ⢠HIATAL HERNIA
causes of GERD
remain upright
during sleep; semi-fowler or two pillow
position GERD
sense of fullness
nausea & vomitingĀ
dysphagia
ptyalism (hypersalivation)
S/S GERD
give ice chips (if vomited or NPO)
antiemetic : metoclopramide, ondansetron, granisetron
intervention nausea & vomiting GERD
chew sugarless gum / hard candy
toothbrushing
intervention ptyalism GERD
flex neck
thicken liquid
feed slowly
intervention dysphagia GERD
POSTPRANDIALĀ (occurs after meal)
Ā SHORT LIVEDĀ
Ā ASYMPTOMATICĀ
Ā NO NOCTURNAL SX
physiological GERD (how it should function)
SYMPTOMSĀ
Ā MUCOSAL INJURYĀ
Ā NOCTURNAL SX
pathologic GERD (abnormal)
ulcer
high HCL =?
antacid
to neutralized the acid(HCL)
BARIUM SWALLOWĀ
Ā ENDOSCOPY
Ā PH MONITORINGĀ
Ā ESOPHAGEAL MANOMETRY
Dx GERD
ANTACID
H2 RECEPTOR BLOCKERĀ
Ā FUNDOPLICATION
Tx GERD
Reflux and regurgitation
Hallmark of IngestionĀ
Epithelial Layer
Lamina Propria
Muscularis Mucosa
3 Layers of Mucosa
Epithelial Layer
Absorbs & secretes mucus & digestive enzymes
Lamina propria
blood & lymph vessels
muscularis mucosa
smooth muscle -> breaks down food
gall bladder
stores & concentrates bile produced by the liver
pancreas
releases insulin
pancreatic lipase
breaks down fats into fatty acids and glycerol
pancreatic amylase
breaks down carbohydrates into sugar
proteases
break down proteins into smaller peptides
Achalasia
rare disorder, malfunction of nerves that control the movement of food through the esophagus
jackhammer esophagus
diffuse esophageal spasmĀ
type III achalasia
3 types of esophageal spasmĀ
jackhammer esophagus
hypercontractile esophagus (most common)
diffuse esophageal spasm
spasm are normal in amplitude
type III achalasia
characterized by LES obstructionĀ
dysphagiaĀ
regurgitation
chest discomfort
weight loss
pyrosis (heart burn)
achalasia symptoms
dysphagia
hallmark sign of achalasia
esophageal manometry
definite diagnosis of achalasia
endoscopy
basic diagnosis achalasia
endoscopy
esophageal manometry
esophagram
Dx achalasia
⢠CANNOT BE REPAIRED
⢠EAT SLOWLY, DRINK PLENTY OF FLUID WHILE YOU EAT
⢠CHEW FOOD THOROUGHLY
⢠STAY UPRIGHT WHILE EATING AND AT LEAST 1 HOUR AFTER EATING
⢠DRINK A FULL GLASS OF WATER WITH PILL
Tx achalasia
isosorbide dinitrate or CCB
botox injection - easier to swallow after 6-12 mths
pneumatic balloon dilation
pharmaco achalasia
hiatal hernia
THE UPPER PART OF THE STOMACH PUSHES THROUGH AND OPENING IN THE DIAPHRAGM, AND UP INTO THE CHEST. PROTRUSION OF THE UPPER PART OF A STOMACH.
liver
makes bile
Epiglottis
Seals airway off
IngestionĀ
DigestionĀ
Absorption
Excretion
Main Function of GI System
laparoscopic
heller myotomy -> fundoplication
surgical achalasia
hiatal hernia
DIAPHRAGM HAS A LARGER OPENING THAN NORMAL
A PORTION OF THE UPPER STOMACH SLIPS UP OR PASSES THROUGH THAT HIATUS AND ENTER INTO THE CHEST
achalasia
⢠DECREASED OR ABSENT PERISTALSIS IN THE DISTAL PORTION OF ESOPHAGUS
⢠NORMAL PATTERN OF SWALLOWING DOES NOT OCCUR
⢠LOWER ESOPHAGEAL SPHINCTER MUSCLE DOES NOT RELAX PROPERLY
⢠THUS, PREVENTS THE PASSAGE OF SWALLED FOOD
TeethĀ
TongueĀ
Salivary glands
Liver
Gallbladder
Pancreas
Accessory Organs