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90% vocal fold paralysis is
unilateral due to peripheral damage to vocal folds
Majority of vocal fold paralysis are
adductor type, can't close the glottal space fully
Recurrent laryngeal nerve (Vagus-CN X) - "wandering" nerve damage - flaccid dysphonia
May lead to unilateral true vocal fold paralysis
Unilateral True Vocal Fold Paralysis Etiology
May result from surgical trauma, tumors/neoplasms, trauma, CNS dysfunction, radiation, inflammatory, cardiovascular & unknown idiopathic factors
Unilateral True Vocal Fold Paralysis Voice Characteristics
May result in aphonia to normal voicing because the localization of paralysis will determine it,
Unilateral True Vocal Fold Paralysis Treatment
Surgery, Behavior therapy, Medical care
MANAGEMENT OF UNILATERAL VOCAL FOLD PARALYSIS goals
Improve voice and prevent
Aspiration
MANAGEMENT OF UNILATERAL VOCAL FOLD PARALYSIS Surgical
With recurrent laryngeal
nerve damage, it's a "wait & see" approach for 6-9 months; Medialization, Arytenoid adduction, Vocal fold augmentation, Reinnervation surgical approaches, Gene therapy
Medialization
moving vocal fold towards the middle
Arytenoid adduction
done if theres a large glottal gap
Vocal fold augmentation
fatten up paralyzed vocal cord so stronger cord can contact the closer paralyzed cord, temporary affect, requires reapplication
Reinnervation surgical approaches
to the recurrent laryngeal nerve,
Gene therapy
to the recurrent laryngeal nerve, ongoing research into potential side effects
MANAGEMENT OF UNILATERAL VOCAL FOLD PARALYSIS Behavioral Voice Therapy
Vocal function exercises, Resonant Voice Therapy, Lee Silverman Voice Treatment (LSVT)
Bilateral Abductor True Vocal Fold Paralysis Description
Life threatening condition when there is bilateral paramedian vocal fold paralysis, Variable voice symptoms
Bilateral Abductor True Vocal Fold Paralysis Etiology
Most commonly happens from surgical trauma, malignancies, endotracheal tube intubation neurological diseases, & idiopathic causes
Bilateral Abductor True Vocal Fold Paralysis Management
Management is a "wait & see" approach for 6-9 months, Pacing strategies may include electrical stimulation of vocal folds, Botox injections to obtain adequate glottal opening
Superior Laryngeal Nerve Paralysis Description
the vocal folds can appear normal or "rotated" at rest to weakened side. Vocal fold bowing and shortening, ipsilateral hyperadduction of the false fold
Superior Laryngeal Nerve Paralysis Symptoms
disruption of adduction and elongation for
increased pitch, weak breathy voice, hoarseness
Superior Laryngeal Nerve Paralysis Managment
wait & see" approach, surgically may include the fusion of the thyroid and cricoid cartilages
Spasmodic Dysphonia Description
focal laryngeal (in vocal folds) dystonia during phonation with unknown origin (can be adductor, abductor, or both dysphonias)
Adductor spasmodic dysphonia (85% of cases)
intermittent strained strangled quality
Abductor spasmodic dysphonia
intermittent breathiness
Spasmodic Dysphonia Symptoms
Perceived stoppages of voice are heard with vowel prolongation and connected speech, More severe during connected speech
Spasmodic Dysphonia Management
Botox injection; voice therapy may play some role in improvement
Essential Voice Tremor Description
Most common movement disorder, varying etiology, inherited and worsens with age, tremor affects the hands
Essential Voice Tremor Symptoms
Audible, rhythmic cycles of tremor can be heard during vowel prolongation, Cyclic tremor, Possible pitch or voice breaks
Essential Voice Tremor Management
with medication (beta-blockers, propranolol, and metoprolol)
Vocal fold paralysis
no movement
Vocal fold paresis
weakness of vocal folds, slight movement is preserved