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Bethanechol1
holinergic agonist; stimulates bladder contraction to help voiding.
Bethanechol
Increases gastrointestinal motility to relieve symptoms.
Pilocarpine1
Induces miosis, which lowers intraocular pressure by allowing fluid
drainage.
Pilocarpine
Stimulates salivary and lacrimal gland secretion.
Pyridostigmine
Reversible AChE inhibitor; increases acetylcholine at neuromuscular
junctions to improve muscle strength
Ecothiopate
Irreversible AChE inhibitor, reducing intraocular pressure by increasing
acetylcholine.
Pralidoxime and Atropine
Pralidoxime reactivates AChE, and atropine blocks excessive muscarinic effects
Atropine
Anticholinergic, increases heart rate by blocking parasympathetic input to
the heart.
Physostigmine
Reversible AChE inhibitor, increases acetylcholine to counteract
anticholinergic toxicity.
Scopolamine
Anticholinergic, blocks central muscarinic receptors to prevent nausea
Scopolamine
Blocks central muscarinic receptors for long-lasting antiemetic effect.
Tiotropium
Long-acting muscarinic antagonist (LAMA), provides prolonged
bronchodilation.
Ipratropium
Short-acting muscarinic antagonist (SAMA), providing bronchodilation by
inhibiting parasympathetic-induced bronchoconstriction.
Benztropine
Blocks muscarinic receptors in CNS, reducing tremor and rigidity.
Oxybutynin
Antimuscarinic agent, relaxes detrusor muscle.
Epinephrine
Stimulates alpha and beta receptors, providing bronchodilation and
vasoconstriction.
Dobutamine
Beta-1 agonist, increases cardiac contractility and output.
Oxymetazoline
Alpha-1 agonist causing vasoconstriction, but prolonged use leads to
rebound.
Methyldopa
Central alpha-2 agonist, reduces sympathetic tone, lowering BP.
Albuterol
Beta-2 agonist, provides rapid bronchodilation.
Salmeterol
Long-acting beta agonist (LABA), provides sustained bronchodilation.
Mirabegron
Beta-3 agonist, relaxes detrusor muscle.
Ephedrine
Mixed-acting sympathomimetic; stimulates alpha and beta receptors and
releases norepinephrine.
Phenoxybenzamine
Non-competitive alpha blocker; prevents excessive vasoconstriction by
blocking catecholamine effects.
Tamsulosin
Selective alpha-1A blocker; relaxes smooth muscle in prostate and
bladder neck, improving urinary flow with minimal BP effects
Prazosin
Alpha-1 blocker; treats both urinary symptoms and hypertension by
causing vasodilation.
Propranolol
Non-selective beta-blocker; reduces heart rate and inhibits conversion of
T4 to T3
Metoprolol or Bisoprolol
Selective beta-1 blocker; decreases heart rate and cardiac workload,
improving survival in heart failure.