Common Maternal Complications

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Vocabulary-style flashcards covering definitions and key concepts from the lecture notes on postpartum hemorrhage and hypertensive disorders in pregnancy.

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51 Terms

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Postpartum hemorrhage (PPH)

Excessive bleeding after birth. ACOG defines it as cumulative blood loss >1,000 mL or with signs of hypovolemia; vaginal delivery normal blood loss is <500 mL, but bleeding of any amount can be PPH.

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early vs late post partum hemorrhage

early is within 24

late is 24-48 hours

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Four Ts (causes of PPH)

Tone (uterine atony) (the uterus is boggy) THE MOST COMMON ISSUE

Trauma (lacerations hematomas, inversion, rupture)

Tissue (retained placenta or invasive placenta)

Thrombin (coagulopathies)

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what do you do with the blood

quantify it (to keep track of blood loss)

1ml of blood is 1gram of weight

keep even wet gauze

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Uterine atony

Lack of uterine muscle tone leading to heavy postpartum bleeding; the primary and most common cause of PPH.

BIG BOGGY UTERUS

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what causes the uterus to end up boggy or struggle to tighten back up

big baby

more than 1 baby pregnancy

too much amniotic fluid

infection of the amniotic fluid or uterus

prolonged labor

retained placenta

more than 4 vaginal births

episiotomy

hematoma

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Tissue (in the context of PPH)

Retained placental tissue or placenta accreta / increta/ percreta causing ongoing bleeding.

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Thrombin (coagulopathy)

Disorders of coagulation that contribute to PPH (bleeding due to impaired clotting).

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Risk factor: magnesium sulfate

Magnesium therapy in preeclampsia can contribute to uterine atony and bleeding risk.

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Risk factor: coagulopathy

Bleeding disorder that worsens postpartum hemorrhage.

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Risk factor: cesarean birth (urgent/emergent)

Cesarean delivery increases risk of PPH compared with vaginal birth.

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Risk factor: uterine inversion

Turned inside out uterus after delivery; a cause of heavy bleeding requiring immediate management.

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Assessment finding of post partum hemorhage: lochia

Postpartum uterine bleeding discharge; monitor for color, amount, and clots

the clots bigger than a quarter

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Assessment finding: Systemic

Tachycardia: Elevated heart rate that may accompany hemorrhage.

Hypotension: Low blood pressure indicating possible shock from bleeding.

Oliguria: Low urine output, a sign of reduced perfusion in hemorrhage.

Cool clammy skin

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post partum hemorrhage secondary signs

womens bodies have so much blood that systemic signs such as low blood pressure would not show up till things get really bad

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Blood loss thresholds (PPH signs)

1000 mL: significant hemorrhage by ACOG criteria;

higher volumes correlate with worsening clinical signs (e.g., 1500–2500+ mL associated with progressive shock).

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subinvolution

• Delayed return of enlarged puerperal corpus to normal size and function

Common with secondary or late PPH

Causes

• Retained placenta

• Infection

• Malposition

• Myomas

• Gestational trophoblastic disease

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symptoms of subinvolution

• Pelvic discomfort or backache

• Bleeding from an enlarged, boggy, tender uterus

• Lochia does not change from rubra

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meidcation for early PPH

Pitocin: 0-40 U in 500/1000 ml crystalloid IV, or 10 units IM

Methergine: can not give to people with hypertension (meth gives you hypertension) 

Hemabate: Can not give to people with asthma, they get mad crazy diarrhea as a side effect

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Cytotec for ealy PPH

800-100mcg rectally to control hemorrhage and stimulate uterine contractions.

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Tranexamic acid (TXA) for early PPH

• Helps decrease blood loss by maintaining blood clots and

preventing the breakdown of fibrin

1 gm loading dose of IV TXA over 10 minutes (1 mL/min) upon

diagnosis of excessive blood loss

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Bakri balloon

Intrauterine balloon tamponade device used to control postpartum hemorrhage; commonly filled with normal saline (e.g., 500 mL) to apply internal pressure and stop bleeding

<p>Intrauterine balloon tamponade device used to control postpartum hemorrhage; commonly filled with normal saline (e.g., 500 mL) to apply internal pressure and stop bleeding</p>
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The jada for PPH

is a flexible drainage device designed to control uterine bleeding by providing suction and creating negative pressure which collapses the uterus

made by engineering students

<p>is a flexible drainage device designed to control uterine bleeding by providing suction and creating negative pressure which collapses the uterus </p><p>made by engineering students</p>
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B-Lynch suture

A uterine compression suture technique used surgically to control atony when other measures fail.

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OB hemorrhage cart

• Quick access to emergency supplies

• Refrigerator for meds

• Establish necessary items and par levels

• Label drawers/compartments

• Include checklists

• Develop process for checking and restocking

• IV pressure bags

• Sutures for B-lynch and modified B-lynch techniques

• Bakri balloon

• 500 cc fluid for filling

• Bag for drainage collection

• Kerlex roll

• Vaginal pack

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nursing management of PPH

• Call for help

• Assess and massage uterus properly

Ensure 18 G IV, LR with ongoing Pitocin

• Emergency Cart

• Weigh pads; monitor bleeding

• Assess lochia for color, amount, and clots

• Administer medications/ blood/ oxygen

• Monitor vital signs

• Insert Foley

• Assign runner, recorder, CRNA, family liaison

• Collect lab work as ordered

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lacerations and hematomas

• Injuries to the labia, perineum, vagina, and cervix

Hematoma

  • Cardinal sign is pain not relieved by analgesics

Lacerations

  • Bleeding despite firm uterus

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Episiotomy laceration degrees

First-degree: skin and vaginal mucosa;

Second-degree: includes perineal muscles;

Third-degree: extends to anal sphincter;

Fourth-degree: extends into rectum itsself and mucosa.

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Vulvar/vaginal hematomas

Bleeding into vulvar or vaginal tissues often associated with forceps delivery;

presents with rectal pressure and severe pain.

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Uterine inversion management

Discontinue pitocin, call for help, start IV fluids,

Consider what medications you have available to relax uterus, manually inverse uterus;

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Retained placental tissue

• Normal separation within 15 min

• 95% separation within 30 min

• Retained placenta

• Not separated within 30 min of start of 3rd stage

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Abnormal placental implantation (accreta/increta/percreta)

Placenta adheres to/penetrates the uterus abnormally:

accreta (adhesion to myometrium)  A for ADHERE

increta (into myometrium), I for INTO

percreta (attaches through uterus)

<p>Placenta adheres to/penetrates the uterus abnormally:</p><p></p><p><strong>accreta </strong>(adhesion to myometrium)&nbsp;<u> A for ADHERE</u></p><p><strong>increta </strong>(into myometrium), <strong>I for INTO</strong></p><p><strong>percreta </strong>(attaches through uterus)</p>
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question

A nurse is caring for a client 2 hr after a spontaneous vaginal

birth and the client has saturated two perineal pads with blood

in a 30-min period. Which of the following is the priority nursing

intervention at this time?

• Palpate the client’s fundus

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activity

4 causes: Tone, trauma, tissue, thrombin

Which of the 4 t’s the most common cause of PPH: tone

Name 3 risk factors: big baby, multiple babies, long labor, too much amniotic fluid, infection

3 medications used: pitocin, hemabate, methergine

3 nurisng interventions for hemorrhage: fundal massgae, start IV, call for help, weighing blood, getting hemorrhage cart

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4 types of hypertension

  • Chronic Hypertension–CHTN

  • Gestational Hypertension-GHTN

  • Preeclampsia- (AKA Pre-e or older slang term was ‘Toxemia’)

  • Preeclampsia with severe features

  • Eclampsia

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why is hypertension bad in pregnancy

Narrowed blood vessels

Placental insufficiency/ restricted blood flow/ poorly oxygenated uterus

Oligohydramnios = low amniotic fluid

IUGR, Small for gestational age baby

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Chronic hypertension (CHTN) in pregnancy

• Present BEFORE pregnancy

  • May or may not be diagnosed already

• 140s/90s typical

• If new onset HTN develops prior to 20 weeks, it is chronic hypertension.

• No protein in the urine

• Increased BPs remain high 12 weeks after birth

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Gestational hypertension (GHTN)

New-onset hypertension after 20 weeks of gestation without proteinuria or severe features; resolves postpartum.

• HTN with BP >140s/90s

• 2 occasions, 4 hours apart

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Preeclampsia

HTN that develops >20 weeks

• Hypertension 140s/90s with

Proteinuria in a urine specimen- 300+ mg in a 24 hr specimen, Or 0.3+ protein/creatinine ratio spot check

Resolves by 12 weeks postpartum

Common risk factors

• Multifetal gestation

• History of preeclampsia

• Chronic hypertension

• Preexisting diabetes and/or thrombophilias

• Women with a new partner

• Paternal factors

2% to 7% of healthy pregnancies

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signs of preclampsia

headache that doesnt go away

trouble breathing

swelling in legs, hand, face

nausea

sudden wieght gain of 2-5 pounds

changes in vision, blurriness, flashing lights seeing spots

<p>headache that doesnt go away</p><p>trouble breathing</p><p>swelling in legs, hand, face</p><p>nausea</p><p>sudden wieght gain of 2-5 pounds</p><p>changes in vision, blurriness, flashing lights seeing spots</p>
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Preeclampsia with severe features

Hypertension 160+/110+ and Proteinuria with one or

more:

o Thrombocytopenia

o Impaired liver function (do LFTS)

o New onset renal insufficiency (decreased GFR check labs)

o Pulmonary edema

  • swelling all over the body not just legs or arms

o New onset cerebral disturbance (severe headache)

o New onset of visual disturbances

• Increased risk of placental abruption, IUGR, and stillbirth

• Resolves by 12 weeks postpartum

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Eclampsia

Seizure activity or coma (definitive)

• No history of preexisting (seizure-related) disorder

• Eclamptic seizures can occur before, during, or after birth

Due to cerebral edema or cerebral hemorrhage

• Possible placental abruption

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interprofessional care

• High-risk patients can receive low-dose aspirin in 1st trimester

• Accurate BP’s at Clinic visits

Assess for

• HTN

• Edema / large sudden weight gain

• DTR’s/clonus

• Proteinuria

• Symptoms of severe features

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gestational hypertension and pre-e care management

Maternal and fetal assessment done at home

• NST, BPP weekly

• Fetal Movement Counts (4 or more in 1 hr)

o Amnioic fluid index and Eestimated fetal weight

o Labs weekly (liver, urine, kidney (BUN, creatinine, GFR), CBC

o BP twice/week

o Daily weights, did they gain like 5 pounds in one day

Regular diet with adequate protein

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pre e with severe features

• Inpatient management

Meds – Mag sulfate, antihypertensives, and betamethasone (if <34 weeks) helps mature babies lungs when they early

Goal BP is like 140/90 not 120/80

Continuous fetal monitoring

• Careful VS, Strict I/Os, careful assessment

• Bed rest, rails up, quiet darkened room

After Mag, inpatient expectant management versus delivery

• 37 weeks+ gestation = delivery

• 34-37 weeks-determine how ‘bad’ is the PreE, do we need to consider delivery

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Magnesium sulfate therapy- High risk medication alert

Goal is to lower risk for seizure activity

• Keep mom pregnant just a little longer + Neuro benefit to baby (less risk for cerebral palsy)

• Double-check system

• 4-6g over 15-30 min loading dose, then 2g per hour until 24 hours after delivery

o Maintain serum level 4-7 mEq/L

• Educate: PT will feel nauseated, malaise, warm, flushed, diaphoretic, and sedated

• Excreted by kidneys (check renal function closely)

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Magnesium sulfate toxicity monitoring

• Hourly Mag toxicity checks

o Foley with urimeter (atleast 30ml per hour)

o Respiratory Rate! if toxic level will be less than 12

o Reflexes/DTRs, they will have hyperreflxia, if absent too much magnesium sulfate

• Discontinue if no DTRs, slurred speech, poor respiratory rate, cardiac symptoms

• Calcium Gluconate available!!

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Magnesium toxicity

• TOXIC WHEN SERUM LEVELS ARE TOO HIGH:

• Monitor VS, Q30 minutes to hourly

• Monitor for RR<12 breaths/minute

• Hourly I&O’s, need UO>30 ml/hr

Monitor for signs of end organ damage

• Monitor DTR’s and clonus

Antidote: Calcium Gluconate

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seizure activity

Pad side rails for all patients at risk for seizures

• Call for help! Do not leave the bedside

• Protect airway - Turn to side

• Time the seizure

After seizure

o Administer O2 – pulse ox

o Suction as needed

o IV, foley, mag sulfate, monitor VS and fetus, stat labs, emergency meds available (hydralazine, labetalol, nifedipine, calcium gluconate),

Consider a stat delivery

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Interventions

• Control of Blood Pressure

• Antihypertensive medications are indicated when the systolic BP exceeds 160 mm Hg or the diastolic BP exceeds 110 mm Hg

Postpartum care

• Vital signs, intake and output, DTRs, level of consciousness

• Magnesium sulfate infusion is continued after birth for seizure prophylaxis as ordered, usually for 24 hours

Future health care

• Significant risk of developing preeclampsia in a future pregnancy (women with severe features)

• Increased risk for developing chronic hypertension and cardiovascular disease later in life

• Educate clients on lifestyle changes

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Aspirin for preeclampsia prevention

Low-dose aspirin in the first trimester for high-risk patients to reduce the risk of developing preeclampsia.