1/185
Looks like no tags are added yet.
Name | Mastery | Learn | Test | Matching | Spaced |
---|
No study sessions yet.
post partum hemorrhage
greater than 500 mL
symptoms of post partum hemorrhage
decreased BP
increased pulse
restlessness
decreased urine output
when does early post partum hemorrhage occur
within 24 hours following delivery
what is early (primary) post partum hemorrhage related to?
poor uterine tone (atony)
lacerations
episiotomy
retained placental fragments
hematoma
uterine rupture
problems with placental implantation
coagulation disorders
what can uterine atony be related to?
over distension
prolonged labor
grandmultiparity
medications
prolonged 3rd stage
preeclampsia
operative delivery
retained placental fragments
what is the management for atony related hemorrhage
massage
IV access
infusion of crystalloid
blood products
bimanual massage
meds: pitocin, methergine
D&C
arterial embolization
uterine packing
ligation of the uterine or iliac artery
hysterectomy
risks with lacerations and post partum hemorrhage
nulliparity
epidural
precipitous delivery
macrosomia
operative delivery
pitocin
post partum hemorrhage: retained placental fragments
partial separation of the placenta
may be associated with massaging the fundus prior to separation
placenta should be inspected
uterine exploration
possible D&C
post partum hemorrhage: hematoma
injury to a blood vessel from birth trauma or bleeding of a repair site allows for collection of blood
risks of hematoma in PPH
preeclampsia
first full term delivery
precipitous labor
operative delivery
vulvar varicosities
management for hematomas in PPH
depends on size - may reabsorb or I&D
ice/heat
antibiotics
PPH: uterine rupture risks
prior surgery
fetal malpresentation
grandmultiparity
operative vaginal delivery
pitocin induction
PPH: management of uterine rupture
surgery
fluids
blood replacement
late (secondary) PPH
occurs between 24 hours and 6 weeks post partum
what is late PPH related to?
retained placenta
subinvolution
what is the management of late PPH?
methergine
antibiotics
possible D&C
puerperal infection
infection of the reproductive tract occurring within 6 weeks following delivery
puerperal morbidity
a temperature of 38C or higher for any 2 of the first 10 days PP, after first 24 hours
who is at greater risk for a puerperal infection?
women who deliver via C/S
what are the risk factors for reproductive tract infections
C/S delivery
diabetes
PPROM
chorioamnionitis
multiple vaginal exams
lapses in aseptic technique
compromised health status
internal monitoring
trauma
retained placenta/manual removal
endometritis
inflammation of the endometrial lining
signs/symptoms of endometritis
bloody vaginal discharge
foul smelling vaginal discharge
uterine tenderness
fever
tachycardia
causative agents: GBS, chlamydia, E.coli
what is the management for endometritis
broad spectrum antibiotics
perionitis
reproductive tract infection can spread to the entire peritoneal cavity
potential sites of wound infections
C/S incision
laceration/episiotomy
assessment for a wound infection
redness
foul drainage
warmth
approximation
UTI related to?
postpartum diuresis
increased bladder capacity
decreased bladder sensitivity
catheterization
bacteria
retention
over distention
inability to empty bladder because of trauma
management for over distention
catheterize
encourage patient to empty bladder
cystitis
lower urinary tract infection (bladder)
pyelonephritis
upper urinary tract infection (kidney)
management of UTI
urine C&S
antibiotics
pyridium
mastitis
inflammation of lobular connective tissue in breast
generally unilateral
more frequent among nursing mothers
may progress to an abscess
S/Sx of mastitis
red, painful, swollen breast
causes of mastitis
milk stasis
bacterial invasion
trauma
obstruction of ducts
failure to empty breasts
treatment of mastitis
frequent breastfeeding
supportive bra
ice packs
meds: antibiotics/analgesics
venous thrombosis
formation of a blood clot
thrombophlebitis
inflammation, leading to formation of a clot
causes of thrombophlebitis
hypercoagulability
venous stasis
vessel injury
thromboembolic disease factors in PP period
increased clotting factors
release of thromboplastin
increased fibrinolysis inhibitors
risk factors for thromboembolic disease
C/S
immobility
smoking
prior thrombus
varicose veins
diabetes mellitus
AMA
multiparity
anemia
inherited disorders
superficial thrombophlebitis signs/sx
tenderness
localized warmth and redness
normal temp or low grade fever
management of superficial thrombophlebitis
heat
elevation
analgesics
bed rest
elastic compression stockings
deep vein thrombosis (DVT) signs/sx
edema
tenderness
pale limb color
low grade fever, followed by spike in temp
chills
management of DVT
often requires heparin therapy
in addition to superficial thrombophlebitis management
PP MMD: post partum depression sx
sadness
crying
sleep pattern disturbances
changes in appetite
difficulty concentrating and making decisions
feelings of worthlessness and inadequacy
lacks interest in pleasurable activities
lack of interest in appearance
risk factors of post partum depression
primiparity
ambivalence about pregnancy
PP blues
prior hx of PP depression
lack of social support/family relationships
complications
loss of newborn
domestic violence
management of PP depression
antidepressants
psychotherapy
educate patients about PPD prior to discharge
PP psychosis sx
irrational thinking
delusions
hallucinations
confusion
agitation
confusion
agitation
hyperactivity
insomnia
PP psychosis risk factors
previous PP psychosis
hx of bipolar disorder
obsessive personality
social factors
management of PP psychosis
safety is the priority!
often hospitalized
meds
psychotherapy and cognitive behavioral therapy
assistance with newborn
perinatal loss
death of a fetus or infant from conception through 28 days following birth
fetal death
loss after 20 weeks
causes of fetal death
could be unknown
asphyxia
congenital malformations
placental abruption/ previa
cord accidents
growth restriction
alloimmunization
preeclampsia
diabetes
lupus
thyroid disorders
renal problems
risk factors for fetal death
nullipara
multiple gestation
obesity
infection
clotting disorder
maternal physiologic implications
infection DIC (disseminated intravascular coagulation)
what is disseminated intravascular coagulation (DIC)
thromboplastin is released from fetal tissues, activating the clotting system
how is DIC managed?
address underlying condition
blood transfusions
anticoagulation
post birth evaluation
postmortem studies may give cause of demise
autopsy
chromosomal studies
cultures
visual examination
blood tests
x-ray/MRI
bereavement
to suffer loss
grief
reaction to loss
mourning
process of incorporating the loss
reactions to loss
shock
numbness
confusion
denial
anger
protest
guilt
loneliness
dual process model
loss and restoration
need to address the loss
regain balance in light of loss
attachment theory
the intensity of grief can be assessed by considering attachment to the anticipated baby
frameworks for attachment theory
planning pregnancy
confirming pregnancy
accepting pregnancy
fetal movement
accepting fetus as an individual
giving birth
seeing baby
touching baby
caring for baby
meaning reconstruction
redefining life after a loss
search for meaning
the caring theory
knowing- understanding the loss as it pertains to the parents
being with- ability to give of yourself to the family
doing for- do for the family as you would want others to do for you
enabling- help the family through events surrounding loss
maintaining belief- believing that the parents can get through this loss
caring for grieving family: RESPONDING acronym
R = recognition and validation of loss
E = emotional availability
S = spiritual and cultural accommodation
P = physical presence
O = open communication
N = normalization of grief reactions
D = decision making assistance
I = interdisciplinary involvement
N = nonjudgmental attitude
G = genuine caring
considerations for grieving family
room placement
acknowledge loss
continuity of care
encourage questions
if desired, allow time with infant
remove tubes/wires
bathe and dress
pictures
footprints/handprints, lock of hair
memory box
risk factors associated with greater neonatal morbidity and mortality
low socioeconomic status
no prenatal care
exposure to teratogens
preexisting maternal conditions
age and parity
pregnancy complications
IUGR: symmetrical (proportional)
restriction of growth in size of organs, body length, and head circumference
d/t long term conditions
IUGR: asymmetrical (disproportional)
birth weight is below 10th%
head and/or body length remain normal
often d/t impaired uteroplacental blood flow
complications of the SGA/IUGR newborn
hypoxia
aspiration syndrome
hypothermia
hypoglycemia
polycythemia
large for gestational age (LGA)
weighs more then the 90th%
correlations: diabetes, genes, multiparity, male infants
complications of the LGA
birth trauma r/t cephalopelvic disproportion
induction of labor
cesarean section
hypoglycemia
polycythemia
hyperviscosity
a newborn of a diabetic mother may be…
SGA or LGA
complications of the newborn of the diabetic mother
hypoglycemia
hypocalcemia
hyperbilirubinemia
birth trauma
polycythemia
respiratory distress syndrome (RDS)
congenital malformations
hypoglycemia in a baby
less than 40-45 mg/dl
signs/sx of hypoglycemia
tremors/seizures
apnea
cyanosis
temperature instability
poor feeding
hypotonia/lethargy
hypoglycemia frequency for baby
blood sugar checks done with every feed (2-3 hours) and want a few stable measurements before discontinued
prematurity
preterm: newborn delivered prior to 38 weeks
incidence: 12%
concern: prematurity of all systems
complications of prematurity
cardiovascular and respiratory
at risk for RDS
inadequate amounts of surfactant
pulmonary vessels not fully developed
at risk for PDA
prematurity: thermoregulation
great body surface area
little subcutaneous faat
thin skin
less flexion
prematurity complication: GI/GU
poor sucking
poor swallowing
poor gag reflex
small stomach capacity
difficulty absorbing fat
calcium and phosphorus deficiency
increased BMR and oxygen needs
at risk for necrotizing enterocolitis
immature kidneys
decreased GFR
limited ability to concentrate urine
prematurity complication: hepatic and hematologic
at risk for hypoglycemia
low iron stores
at risk for hyperbilirubinemia
prematurity complications: neuro
rapid brain development during the 3rd trimester
at risk for: intraventricular hemorrhage, intracranial hemorrhage, apnea (cessation of breathing for at least 20 secs - may be r/t bradycardia)
prematurity: possible long term complications
SIDS
respiratory infections
neurologic problems
auditory problems
speech problems
retinopathy of prematurity
prematurity expected care
cardio/respiratory monitoring and function
meet growth and development needs
nutrition
maintain fluid and electrolyte balance
thermoregulation
infection prevention
promote bonding
postmaturity
born after 42 completed weeks gestation
postmaturity complications
hypoglycemia
meconium aspiration
seizures
polycythemia
congenital anomalies
cold stress
newborn of the substance-abusing mother
born to a mother who is dependent on drugs or ETOH
complications include congenital anomalies and/or developmental problems
fetal alcohol syndrome
exposure to ETOH can cause physical, behavioral, and cognitive problems
appearance: short stature, microcephaly, thin
long term complications of fetal alcohol syndrome
impulsive behavior, cognitive involvement, speech problems, learning disabilities
drug abuse
may be legal or illegal drugs
risks to baby: asphyxia, infection, LGA or SGA, low APGAR scores, respiratory distress, congenital anomalies, behavioral problems. withdrawal
long term problems with newborns of substance-abusing mother
developmental, cognitive, social, behavioral, GI, and respiratory problems
care given for newborn of substance-abusing mother
promote postnatal care
newborn drug screen
social service consult
medication administration
reduce withdrawal symptoms
abstinence scoring
small, frequent feedings
monitor GI status
swaddle, pacifier, calm infant
skin protection
decrease stimulation
phenylketonuria (PKU)
phenylalanine amino acid disorder - unable to convert excess phenylalanine to tyrosine
leads to cognitive disabilities
galactosemia
carbohydrate metabolism problem
homocystinurea
deficiency of cystathionine beta synthase