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allergic/hypersensitivity conditions
- erythema multiforme minor
- stevens johnson syndrome
- toxic epidermal necrolysis (TEN)
- atopic dermatitis
- nummular eczema
- uticaria (hives)
- lichenification
Erythema Multiforme Minor description
less extreme than stevens-johnson syndrome and TEN
Erythema Multiforme Minor treatment
self limiting
Erythema Multiforme Minor at risk
those taking drugs like sulfonamides, anticonvulsants, and NSAIDS
Erythema Multiforme Minor transmission
irritant exposure
Stevens-Johnson syndrome description
< 10% body surface
Stevens-Johnson syndrome treatment
- compresses
- antipruritic drugs
- topical anesthetic
Stevens-Johnson syndrome at risk
- children and younger adults
- can develop at any age
Stevens-Johnson syndrome transmission
irritant exposure
Toxic Epidermal Necrolysis (TEN) description
- >30% epidermal
detachment death/breakdown
- Skin falling off, needs
fluid report.
(if losing on skin probably losing on lungs)
Toxic Epidermal Necrolysis (TEN) treatment
- compresses
- antipruritic drugs
- topical anesthetic
Toxic Epidermal Necrolysis (TEN) at risk
- those with HIV infection
- certain genetic variations (HLA-B genes)
- a history of allergic reactions to specific medications
- those who take certain drugs like anticonvulsants, sulfonamides, and some HIV medications
Toxic Epidermal Necrolysis (TEN) transmission
irritant exposure
Atopic dermatitis description
- Erythema
- edema
- vesicle
- large bullae
Atopic dermatitis treatment
removal of irritant
Atopic dermatitis at risk
severe skin dryness
Atopic dermatitis transmission
irritant exposure
Nummular (discoid) eczema description
- Lesions are coin shaped
- papulovesicular patches
Nummular (discoid) eczema treatment
- reduce stress
- frequent bathing
- increase humidity
- Topical ointments
Nummular (discoid) eczema at risk
cold winter months
Nummular (discoid) eczema transmission
unknown
Urticaria (hives) description
- Pale, raised,itchy
papules or plaques
(superficial dermis).
- Looks wheal like.
Urticaria (hives) treatment
Antihistamins
Urticaria (hives) at risk
- females
- adults
Urticaria (hives) transmission
Irritant exposure:
foods/drinks/medications
Lichenification (chronic)
- thickening and hardening of the skin
- giving it a leathery appearance
- typically caused by chronic rubbing or scratching
Integumentary changes in the elderly
diminution
in subcutaneous
Integumentary changes in the elderly
thinning of
epidermal and dermal layers
Integumentary changes in the elderly
decreased
- melanocytes
- langerhans cells
- merkel cells
Integumentary changes in the elderly
decrease and thickening of
blood cells
Integumentary changes in the elderly
- keratoses (usually benign)
- actinic keratoses
- lentinges
- vascular lesions
- cherry angioma
- spider angioma
lentigines: macule
- age spots (freckle like)
- not sun exposure cause
vascular legions
- telangiectasia (vascular)
Pediatric skin conditions requiring interventions (all airborne but scarlet)
- roseola infantum
- rubella (german measles 3 days)
- rubeola (7 days measles)
- varicella
- scarlet fever
Roseola infantum
- no long term effects
- HHV-6 sixth disease
Rubella (German measles 3 days)
- gestational transmission can cause congenital rubella syndrome
- blindness/mental retardation
Rubeola (7-day measles)
- Koplik spots (buccal mucosa)
- Photosensitivitiy
varicella
chicken pox
scarlet fever
- not viral (staph/strep)
- skin lesions look like sand paper
Acanthosis nigricans
- characterize by pigmented hyperkeratosis that occur in skin flexures (axillary, perineal areas, back of neck)
- symmetric
- verrucous
- papillary lesions
Acanthosis nigricans associated w
- paraneoplastic syndrome
- clinical manifestations of metabolic syndrome
- often has to do w diabetes or blood sugars or thyroid issues
Know the warning signs of malignant melanoma ABCDE
- A: asymmetry
- B: border
- C: color (changes)
- D: diameter (changing)
- E: evaluation/elevation (growing)
Pressure injuries
- decubitus ulcers or bed sores
- ischemic lesions of the skin and underlying structures caused by unrelieved pressure
- not enough blood flow to lesion
Pressure injuries 4 factors contribute
- Pressure
- Shear force: moving pt. Bed to bed makes the skin move in an
opposite direction of the material
- Friction
- Moisture (bed bath, incontinence)
pressure injury prevention
- Turn patient every two hours
- Keep skin dry
- Check for incontinence
- Nutrition and hydration
- Helps with circulation
- Check for anemia
- Mobility
- Circulatory status
- no presence of shear and friction forces
- Meticulous skincare
- Food crumbs, IV tubing and other debris (checking for these)
pressure injury stage 1
non-blanchable erythema of intact skin
pressure injury stage 2
partial skin loss, the dermis has been broken into
pressure injury stage 3
- full thickness skin loss
- subcutaneous exposed
- rolled edge appearance
pressure injury stage 4
- full thickness skin and tissue loss
- exposed bone and or muscle
- can happen as result of not turning patient
pressure injury unstageable
- skin hasn't broken but it is bad
- based covered by slough and eschar
- base can not be determined