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Flashcards covering key concepts from Chapter 11 (Hemodynamic Monitoring) and Chapter 12 (Methods to Improve Ventilation) of Pilbeam’s Mechanical Ventilation, 8th Edition.
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Pulmonary artery waveform. The waveform is obtained from a pulmonary artery catheter, which is used to measure pressures in the pulmonary artery. (https://i.imgur.com/YOUR_IMAGE_ID.png)
Preload represents the filling pressure of the ventricle at the end of diastole before the contraction occurs. It reflects the volume of blood stretching the heart muscle at the end of filling.
Right ventricular end-diastolic pressure (RVEDP) is typically used to indicate right ventricular preload, which is the pressure in the ventricle at the end of diastole. This measure helps assess the degree of stretch on the right ventricular muscle before contraction.
Ejection fraction is used to estimate the contractility of the ventricles. It represents the percentage of blood pumped out with each contraction and is an indicator of how well the heart is functioning as a pump.
The ejection fraction is calculated as stroke volume divided by end-diastolic volume. In this case, EF = \frac{40}{125} = 0.32.
Systemic vascular resistance (SVR) is an indication of left ventricular afterload, which is the resistance the left ventricle must overcome to circulate blood. SVR reflects the constriction or dilation of blood vessels.
Venous return determines the preload and is the amount of blood returning to the heart from the venous circulation. It affects the degree of ventricular filling and subsequent cardiac output.
Increase left ventricular afterload because Systemic vascular resistance (SVR) increases with vasoconstriction or an increase in blood viscosity. Increased afterload makes it harder for the heart to eject blood.
Strain gauge transducer is the main component of a hemodynamic monitoring system because it converts physiological pressure into an electrical signal that can be measured and displayed.
The transducer in the invasive vascular monitoring system converts the fluid pressure to an electrical signal allowing continuous monitoring of blood pressure.
For accurate arterial line monitoring, the catheter tip must face upstream to correctly sense arterial pressure, and the transducer should be level with the catheter tip to negate hydrostatic pressure effects.
Accept the CVP reading because the phlebostatic axis is the correct anatomical reference point for CVP measurement, so no adjustment is needed. Thus, the reading should be accepted as is.
If there is a dampened waveform along with the inability to aspirate blood from the catheter, the catheter is most likely occluded or clotted off causing the catheter to be non functional. Therefore, because of risk of thromboembolism the catheter needs to be removed and a new site chosen.
The Allen test determines if there is collateral circulation to the hand. If the Allen test is greater than 15 seconds, this could indicate inadequate collateral circulation and you should not insert the catheter at that site. Choose the other wrist since the left hand has to be tested to determine if the collateral circulation is adequate there.
Phlebitis is not a common complication of systemic catheterization. Common complications include thrombosis, infection, and bleeding.
Central venous pressure (CVP) estimates right ventricular preload, referring to the filling pressure of the right ventricle.
Confirmation of placement of a central venous pressure catheter is done with a Chest radiograph. To confirm proper placement and rule out complications such as pneumothorax, a chest radiograph is required.
Hypovolemia, or low blood volume, can cause a low pulmonary artery occlusion pressure (PAOP), which reflects left atrial pressure and indirectly, left ventricular end-diastolic pressure.
When inserting a pulmonary artery catheter, the subclavian and antecubital veins may require a surgical cut down to allow access for catheter insertion because of anatomical challenges or vessel condition.
During the introduction of a pulmonary artery catheter, the waveform that is visible on the monitor represents the location in the Right Atrium. (https://i.imgur.com/YOUR_IMAGE_ID.png)
During the insertion of a pulmonary artery catheter, advancing the catheter into the intrathoracic vessels can cause damage, so when the catheter is positioned here, the balloon needs to be inflate with air to allow flow directed insertion to continue.
For pulmonary artery catheter insertion, choose internal jugular and subclavian veins to avoid insertion sites where phlebitis is present, reducing risk of complications and infection.
Excessive movement can lead to catheter knotting, which requires catheter removal and potential vascular damage.
A pulmonary artery catheter must be wedged in Zone 3 because this is where pulmonary arterial pressure is greater than alveolar pressure , which is greater than venous pressure. The other zones do not have this relationship.
The range for the time a pulmonary artery catheter should be inflated before pulmonary artery occlusion pressure is measured is 15-30 seconds. You should not inflate longer than this, otherwise it could cause pulmonary infarction.
The formula for calculating CaO2 is: (Hgb × 1.34 × SaO2) + (PaO2 × 0.003). (9 × 1.34 × 0.96) + (97 × 0.003) = 11.6 + 0.29 = 11.9 vol%.
The formula for calculating CaO2 is: (Hgb × 1.34 × SaO2) + (PaO2 × 0.003). (17 × 1.34 × 0.93) + (64 × 0.003) = 21.2 + 0.192 = 21.4 vol%.
The Fick equation is: CO = VO2 / (CaO2 – CvO2). CO = 340 / (17.3 – 12.8) = 7.6 L/min.
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Cardiac Index (CI) is calculated as heart rate (HR) x stroke volume (SV) / Body surface area (BSA). Therefore, CI = 80 x 55 / 2.8 = 1.6 L/min/m2
Stroke Index (SI) is calculated as cardiac output (CO) / heart rate (HR) / body surface area (BSA). Therefore, SI = 3.7 / 90 / 1.7 = 24 mL/m2.
Stroke Volume (SV) is calculated as cardiac output (CO) / heart rate (HR). Therefore, SV = 4.5 / 110 = 41 mL. The stroke volume index (SI) is stroke volume (SV) / body surface area (BSA). Therefore, 41 / 1.3 = 31.5 mL.
There are 60 seconds in one minute. Thus, if there are 80 beats in one minute, then the duration of one beat is 60 / 80 = 0.75 seconds. Then 0.75 * 1.3 ~ 1 second.
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Pulmonary Vascular Resistance (PVR) is calculated as (mean PAP – PAOP) / CO. In this case, PVR = ((67+25)/2) – 18) / 5.7 = 253 dyne.
Pulmonary Vascular Resistance (PVR) is calculated as (mean PAP – PAOP) / CO. In this case, PVR = (((40-22)/2) – 12) / 4.8 = 267 dyne.
Systemic Vascular Resistance (SVR) is calculated as (mean arterial pressure – CVP) / CO. In this case, (((100+50)/2) – 17) / 5.7 = 698
Systemic Vascular Resistance is (mean arterial pressure – CVP) / CO. In this case, the map is ((156+80)/2) -19 / 4.8 = 1438
In mitral valve stenosis, narrowing of the mitral valve elevates left atrial pressure, increasing pulmonary artery occlusion pressure (PAOP). A PAOP of 20 mm Hg is typical.
A CVP of 16 mm Hg suggests right heart failure. Elevated central venous pressure (CVP) indicates impaired right ventricular function and fluid overload, suggestive of right heart failure.
Hypervolemia is the only answer that increase systemic vascular resistance. Because the blood volume is high, it is increasing the pressure.
Mean arterial pressure (MAP) is the average arterial pressure during a single cardiac cycle. A MAP of 18 mm Hg is not within normal limits. You want the MAP to be >60mmHG for perfusion.
Catheter whip. A high cardiac output can cause a pulmonary artery catheter to move excessively in the pulmonary artery, leading to 'catheter whip'.
Pulmonary infarction is caused from Advancing a pulmonary artery catheter into a smaller artery. It may occlude that vessel, leading to tissue ischemia and possibly pulmonary infarction.
The PA waveform trend decreases during the inspiratory phase of spontaneous breathing. The increasing negative intrathoracic pressure increase venous return during spontaneous breathing causing the PA waveform to decrease.
Intubated patients. The patients hemodynamics need to be improved, therefor Decrease the PEEP incrementally and recheck hemodynamic measurements. High PEEP can increase intrathoracic pressure, thereby reducing venous return and cardiac output, this will improve the patients hemodynamics.
Cardiogenic pulmonary edema CVP 5 mm Hg, PAP 24/13 mm Hg, and PAOP 21 mm Hg. Clinical data can be used to identify cardiogenic pulmonary edema. Hemodynamic measurements reveal elevation of PAP and PAOP .
ARDS. CVP 3 mm Hg, PAP 21/10 mm Hg, and PAOP 8 mm Hg. Clinical data can be used to identify Acute respiratory distress syndrome. The patients has low CVP and PAOP .
Ventricular contractility can be estimated by the ejection fraction, which is the fraction of blood ejected by the ventricle relative to the end-diastolic volume.
An elevated right arterial pressure may mean there is Cardiac tamponade, a condition with fluid accumulation around the heart that impairs its ability to pump effectively. This will cause a back up of volume and increase pressure.
When increasing minute ventilation. Formula is: New VE = Old VE * Old PaCO2 / Desired PaCO2. New VE = 5.5 * 58 / 45 = 7.1 L/min.
The formula in calculating the set rate. New rate = (Old PaCO2/Desired PaCO2) X (Present Rate) New rate = (28 / 40) X 16 = 11/min.