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what is AMD
destruction of macula (responsible for central vision)
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causes irrevesible central vision loss
2 types of AMD
DRY
WET
DRY AMD
non exudative
progressive thinning of macula
close vision tasks become more difficult
leads to progressive and painless vision loss
WET AMD
Exudatve and more severe form
rapid onset; progressed from the dry ama
characterized by the development of abnormal blood vesssels in or near the macula
-weak blood vessels, cause bledding and scarring
Etiology of AMD
retinal aging
genetic factors
ultraviolet exposure or UV Light exposure
DRY AMD
Drusen or yellowish extracellular deposits
WET AMD
growth of new, fragile blood vessels
scar formation
clinical manifestations
blurred ordarkneded vision loss
scotoma or blind spots in the visual field
meta morphopsia or distortion of vision
DIAGNOSTICS
Opthalmoscopy
primary dx procedure
presence of drusen is assessed
Amsler Grid Test
detects vision morphopsia
results provides a baseline for future comparison|
Fundus Photography
- determines extent and type of AMD
Drug mgt
SELECTIVE INHIBITORS OF ENDOTHELIAL GROWTH FACTOR
- slow vision loss in wet AMD
RANIBIZUMAB
BEVACIZUMAB
-injections are given at 4 to 6 weeks intervals
-ocular cohrence tomography
to determine retinal instability
medical management
photodynamic therapy
entails the use of vertiportin (Visodyne) IV and a cold laser to excite the dye
destroys abnormal blood vessels
Nursing Management
PT must be covered since exposure to sunlight could result in thermal burns.
diet must be rich in zinc to increase the subretinal fluid in the macula
smoking cessation
drive at day time at lower speeds