Exam 6 chickens, swine, pigmy goat, beef

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146 Terms

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Nose ring application

  • used to control bulls

  • insert in bulls 9-12 months of age

  • standing position in a chute under local analgesia

  • use self-piercing, non-rusting metal ring

  • inserted through the nasal septum about one inch from the tip of the nose

  • should not be used as the sole form of restraint

<ul><li><p>used to control bulls </p></li><li><p>insert in bulls 9-12 months of age </p></li><li><p>standing position in a chute under local analgesia </p></li><li><p>use self-piercing, non-rusting metal ring </p></li><li><p>inserted through the nasal septum about one inch from the tip of the nose </p></li><li><p>should not be used as the sole form of restraint </p></li></ul><p></p>
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indications for dehorning in cattle

  • prevent operator injury

  • prevent livestock injury

  • ease of working animals

  • less space required for housing and feeding

  • treatment of disease conditions

  • correction of behavioral problems associated with dominance

  • reduce carcass condemnation due to bruising associated with horn injuries

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horn anatomy and growth

  • The horn grows from a modified skin epithelium (buds)

  • buds are not attached to the skull before 2 months of age

  • frontal sinus opens to the cavity of the horn at 4 months

<ul><li><p>The horn grows from a modified skin epithelium (buds) </p></li><li><p>buds are not attached to the skull before 2 months of age </p></li><li><p>frontal sinus opens to the cavity of the horn at 4 months </p></li></ul><p></p>
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restraint and analgesia with dehorning in cattle

  • head tray or similar restrait device

  • head tie to one side with halter

  • cornual nerve block (conrnual) branch of the lachrymal nerve) - 5ml of 2% lidociane using 20-22 g 0.5-1% needle

  • local infiltration around base of horn

<ul><li><p>head tray or similar restrait device </p></li><li><p>head tie to one side with halter </p></li><li><p>cornual nerve block (conrnual) branch of the lachrymal nerve) - 5ml of 2% lidociane using 20-22 g 0.5-1% needle </p></li><li><p>local infiltration around base of horn </p></li></ul><p></p>
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Disbudding in cattle

  • caustic paste / caustic chemical injection

    • pastes contain NaOH, KOH or CaOH

    • injectable contains CaCI

      • apply at <1 week of age

      • do not use in beef calves or in group housed calves cover the horn bud with duct tape

      • clip hair around horn bud, use gloves

      • post op NSAIDs

  • Electrothermal (hot iron)

    • butane-powered, rechargeable or corded

    • calves up to 2 moths old

    • hot iron must fit comfortably over horn bud

    • apply for 5-10 seconds, depending on the size of the calf

<ul><li><p>caustic paste / caustic <strong>chemical </strong>injection </p><ul><li><p>pastes contain <strong>NaOH, KOH or CaOH </strong></p></li><li><p>injectable contains <strong>CaCI </strong></p><ul><li><p><strong>apply at &lt;1 week of age </strong></p></li><li><p>do not use in beef calves or in group housed calves cover the horn bud with duct tape </p></li><li><p>clip hair around horn bud, use gloves </p></li><li><p>post op NSAIDs </p></li></ul></li></ul></li><li><p>Electrothermal (hot iron) </p><ul><li><p>butane-powered, rechargeable or corded </p></li><li><p><strong>calves up to 2 moths old </strong></p></li><li><p>hot iron must fit comfortably over horn bud </p></li><li><p><strong>apply for 5-10 seconds, </strong>depending on the size of the calf </p></li></ul></li></ul><p></p>
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Barns dehorner

  • calves from 3 months to a year or more

  • remove 1-1.5 cm of skin at base

  • control hemorrhage

  • remove all pieces of bone frome horn

<ul><li><p>calves from 3 months to a year or more </p></li><li><p>remove 1-1.5 cm of skin at base </p></li><li><p>control hemorrhage </p></li><li><p>remove all pieces of bone frome horn </p></li></ul><p></p>
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Dehorning in adult cattle

  • dehorning cattle with large size horns is not recommended - sinusitis, skull fractures, pain

  • keystone dehorner

    • used for large horns of heifers, cows, and bulls

    • good restrait

    • local analgesia

    • post operative NSAIDs

<ul><li><p>dehorning cattle with large size horns is not recommended - sinusitis, skull fractures, pain </p></li><li><p><strong>keystone dehorner </strong></p><ul><li><p>used for large horns of heifers, cows, and bulls </p></li><li><p>good restrait </p></li><li><p>local analgesia </p></li><li><p>post operative NSAIDs </p></li></ul></li></ul><p></p>
8
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Horn tipping adult cattle

  • Adult cattle, bulls

  • cut distal 1/3 of the horn length

  • manual saw dehorners

  • electric saw dehorners

  • OB wire dehorning

<ul><li><p>Adult cattle, bulls </p></li><li><p>cut distal 1/3 of the horn length </p></li><li><p>manual saw dehorners </p></li><li><p>electric saw dehorners </p></li><li><p>OB wire dehorning </p></li></ul><p></p>
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cosmetic dehorn

  • preferred in animlas younger than 2 years old

  • show cattle to improve appearance of head

  • standing restraints, tranquilization, cornual nerve block

  • aseptic preparations

  • surgical removal of all germinal epithelium

  • use barn dehorner to cut horn from its base and bone rongeur to smoothen the bone edges

  • close skin the non absorbale, vertical mattress

  • remove skin sutures in 3 weeks

<ul><li><p>preferred in animlas younger than 2 years old </p></li><li><p>show cattle to improve appearance of head </p></li><li><p>standing restraints, tranquilization, cornual nerve block </p></li><li><p>aseptic preparations </p></li><li><p>surgical removal of all germinal epithelium </p></li><li><p>use barn dehorner to cut horn from its base and bone rongeur to smoothen the bone edges </p></li><li><p>close skin the non absorbale, vertical mattress </p></li><li><p>remove skin sutures in 3 weeks </p></li></ul><p></p>
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Disbudding goat kids

  • Electrothermal (hot iron)

    • Disbud kids before 2 weeks of age

    • restrain in a dehorning box

    • sedation/ analgesia

    • local block - ring block

    • nerve block conrual branch of the lachrymal nerve and infratrochlear branch of the ophthalmic nerve

  • post operative - NSAIDs, tetanus, antitoxin

  • complications

    • thermal meningitis

    • scurs and ingrown horns

<ul><li><p>Electrothermal (hot iron) </p><ul><li><p><strong>Disbud kids before 2 weeks of age </strong></p></li><li><p>restrain in a dehorning box </p></li><li><p>sedation/ analgesia </p></li><li><p>local block - ring block </p></li><li><p>nerve block conrual branch of the lachrymal nerve and infratrochlear branch of the ophthalmic nerve </p></li></ul></li><li><p>post operative - NSAIDs, tetanus, antitoxin </p></li><li><p>complications </p><ul><li><p>thermal meningitis </p></li><li><p>scurs and ingrown horns </p></li></ul></li></ul><p></p>
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Dehorning adult goats

  • Not recommended high risk of tetanus, sinusitis, myiasis, abortion, ketosis, and death

  • dehorning wound is difficult to close with sutures and takes months to heal

  • the goat looses social ranking

  • under GA

  • post op antibiotics, NSAIDs, bandage

<ul><li><p>Not recommended high risk of tetanus, sinusitis, myiasis, abortion, ketosis, and death </p></li><li><p>dehorning wound is difficult to close with sutures and takes months to heal </p></li><li><p>the goat looses social ranking </p></li><li><p>under GA </p></li><li><p>post op antibiotics, NSAIDs, bandage </p></li></ul><p></p>
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Descenting Goats

  • A view of the kid from the top of its head

  • (A) the area of applying the cauterizing unit for descenting

  • (B) the area of applying the cauterizing unit for dehorning

  • circles - surgical removal of glands under general anesthesia

<ul><li><p>A view of the kid from the top of its head </p></li><li><p>(A) the area of applying the cauterizing unit for descenting </p></li><li><p>(B) the area of applying the cauterizing unit for dehorning </p></li><li><p>circles - surgical removal of glands under general anesthesia </p></li></ul><p></p>
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Keratoconjuctivitis in sheep and goats

  • Chlamydophila percorum is the most common cause of pinkeye in sheep

  • Mycoplasma spp. also can cause pinkeye

    • very common

    • clinical signs: Epiphora, hyperemia, mucopurulent discharge, keratitis

    • history may indicate other problems in the herd: mastitis, polyarthritis, pleuropneumonia

    • Diagnosis: cytology, IFA, culture

    • treatment: topical antibiotics, systemic oxytetracycline, NSAIDs, protect the eye

<ul><li><p><strong>Chlamydophila percorum is the most common cause of </strong><span style="color: red"><strong>pinkeye </strong></span><strong>in sheep </strong></p></li><li><p><strong>Mycoplasma spp. </strong>also can cause pinkeye </p><ul><li><p><strong>very common </strong></p></li><li><p>clinical signs: Epiphora, hyperemia, mucopurulent discharge, keratitis </p></li><li><p>history may indicate other problems in the herd: <strong>mastitis, polyarthritis, pleuropneumonia </strong></p></li><li><p>Diagnosis: cytology, IFA, culture </p></li><li><p>treatment: topical antibiotics, <strong>systemic oxytetracycline, </strong>NSAIDs, protect the eye </p></li></ul></li></ul><p></p>
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Infectious bovine keratoconjunctivitis

  • pink eye, contagious ophthalmais, New forest disease

  • occurs primarily during summer months

  • all breeds susceptible

    • Bos tauru » than Bos indicus

    • Herefords and Hereford crosses

    • younger > older

  • DDx - foreign bodies, trauma, MCF, IBR, listeria

  • Moraxella bovis: gram negative bacillus

  • newer strains emerged: M. bovoculi

  • recently discovered cause of IBK: Mycoplasma bovoculi

  • Neisseria spp can also cause IBK

  • transmitted by handlers, mechanical vectors (face fly, house fly, stable fly)

    • 75% unilateral eye lesion

    • epiphora, blephrospasm, photophobia

    • mucopurulent eye discharge, corneal abscess, blindness in severe cases only

    • central corneal ulcer

<ul><li><p>pink eye, contagious ophthalmais, New forest disease </p></li><li><p>occurs primarily during <strong>summer months </strong></p></li><li><p>all breeds susceptible </p><ul><li><p>Bos tauru » than Bos indicus </p></li><li><p><strong>Herefords and Hereford crosses </strong></p></li><li><p>younger &gt; older </p></li></ul></li><li><p>DDx - foreign bodies, trauma, <strong>MCF, IBR, listeria </strong></p></li><li><p><strong>Moraxella bovis: gram negative bacillus </strong></p></li><li><p><strong>newer strains emerged: M. bovoculi </strong></p></li><li><p><strong>recently discovered cause of IBK: Mycoplasma bovoculi </strong></p></li><li><p>Neisseria spp can also cause IBK </p></li><li><p>transmitted by handlers, mechanical vectors (face fly, house fly, stable fly) </p><ul><li><p>75% unilateral eye lesion </p></li><li><p>epiphora, blephrospasm, photophobia </p></li><li><p>mucopurulent eye discharge, corneal abscess, blindness in severe cases only </p></li><li><p>central corneal ulcer </p></li></ul></li></ul><p></p>
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Infectious bovine keratoconjunctivitis treatment

  • Long-acting oxytetracycline (2 injections of 20 mg/kg, IM or SC at a 48 to 72 hour interval

  • Subconjunctival injection with penicllin (2ml) with or without dexamtheazone (1mg or 0.5ml)

  • use 25g 5/8 needle

  • third eyelid flaps, tarsorrhaphy or eye patches

  • NSAIDs

  • prevention - vaccination, control flies

<ul><li><p><strong>Long-acting oxytetracycline (2 injections of 20 mg/kg, IM or SC at a 48 to 72 hour interval </strong></p></li><li><p><strong>Subconjunctival injection with penicllin (2ml) with or without dexamtheazone (1mg or 0.5ml) </strong></p></li><li><p>use 25g 5/8 needle </p></li><li><p><strong>third eyelid flaps, tarsorrhaphy or eye patches </strong></p></li><li><p>NSAIDs </p></li><li><p>prevention - vaccination, control flies </p></li></ul><p></p>
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Squamous cell carcinoma (SCC) or “cancer eye”

  • Economic importance

  • very invasive locally

  • may metastasize to the local lymph nodes

  • predisposing factors:

    • sunlight, age

    • non pigmented eyelids and conjunctiva (white faced cattle)

    • Herefords, simmentals and Holestein - Friesians

  • most common areas for tumors: the limbus (junction of the cornea and the sclera) the third eyelid, and on the upper and lower eyelid margins

  • treatment

    • small lesions less than 2 cm

      • cryosurgery

      • eyelid wedge resection

      • third eyelid resection

    • larger lesions (5cm or more): Enucleation

    • invasive lesions: culling

  • prognosis: 40-50% recurrence rate

<ul><li><p>Economic importance </p></li><li><p>very invasive locally </p></li><li><p>may metastasize to the local lymph nodes </p></li><li><p>predisposing factors: </p><ul><li><p>sunlight, age </p></li><li><p>non pigmented eyelids and conjunctiva (white faced cattle) </p></li><li><p><strong>Herefords, </strong>simmentals and Holestein - Friesians </p></li></ul></li><li><p>most common areas for tumors: the limbus (junction of the cornea and the sclera) the third eyelid, and on the upper and lower eyelid margins </p></li><li><p>treatment </p><ul><li><p>small lesions less than 2 cm </p><ul><li><p>cryosurgery </p></li><li><p>eyelid wedge resection </p></li><li><p>third eyelid resection </p></li></ul></li><li><p>larger lesions (5cm or more): Enucleation </p></li><li><p>invasive lesions: culling </p></li></ul></li><li><p>prognosis: 40-50% recurrence rate </p></li></ul><p></p>
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<p>Enucleation </p>

Enucleation

  • Surgical removal of the entire eyeball

  • indications

    • panophthalmitis

    • intra-ocular neoplasia

    • severe primary or secondary infection, eg infectious bovine keratoconjunctivitis

    • trauma or rupture of globe

  • restraint and analgesia

    • standing or recumbent

    • sedation 10-20mg xylazine IV

    • eyelid aknesia - auriculopalpebral nerve block: needle inserted front of the base of the at the end of zygomatic arch

  • post op care

    • systemic antibiotics for 5-7 days

    • NSAIDs for 3 days

    • tetanus prophylaxis as required

    • remove sutures in 3 weeks

  • complications

    • failure or inability to remove all neoplastic tissue (SCC)

    • massive intra-orbital hemorrhage

    • abscess formation

    • excessive dead space

    • failure to appose the skin margin without excessive tension on sutures

<ul><li><p>Surgical removal of the entire eyeball </p></li><li><p>indications </p><ul><li><p>panophthalmitis </p></li><li><p>intra-ocular neoplasia </p></li><li><p>severe primary or secondary infection, eg infectious bovine  keratoconjunctivitis </p></li><li><p>trauma or rupture of globe </p></li></ul></li><li><p>restraint and analgesia </p><ul><li><p>standing or recumbent </p></li><li><p>sedation 10-20mg xylazine IV </p></li><li><p>eyelid aknesia - auriculopalpebral nerve block: needle inserted front of the base of the at the end of zygomatic arch </p></li></ul></li><li><p>post op care </p><ul><li><p>systemic antibiotics for 5-7 days </p></li><li><p>NSAIDs for 3 days </p></li><li><p>tetanus prophylaxis as required </p></li><li><p>remove sutures in 3 weeks </p></li></ul></li><li><p>complications </p><ul><li><p>failure or inability to remove all neoplastic tissue (SCC) </p></li><li><p>massive intra-orbital hemorrhage </p></li><li><p>abscess formation </p></li><li><p>excessive dead space </p></li><li><p>failure to appose the skin margin without excessive tension on sutures </p></li></ul></li></ul><p></p>
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Papillomatosis (Fibropapillomas, warts)

  • Most common skin tumor in cattle

  • benign and self limiting

  • 6 and 24 months old most at risk for warts

  • associated with dehorning, ear tagging, tattooing devices

  • lesions are cauliflower like, rough, or rusty

  • some appear flatter, gray and have a broad based skin attachment, others have a pedunculated base

  • head, neck, brisket and sometimes udder and teats and eyelids are mostly affected

treatment:

  • spontaneous regression within 1-12 months

  • large wards may be surgically removed and their bases cauterized chemically

  • cryosurgery

  • crushing is also used to remove wars and may stimulate the immune system

  • lithium antimony thiomalate 6% solution 15 mL by deep IM injection every other day 4-6 times

prevention:

  • commercial or autogenous vaccines should be repeated at 7 day intervals

  • in show animals it takes at least 4 to 6 weeks before vaccination cause warts to regress

  • disinfection of surgical instruments

<ul><li><p>Most common skin tumor in cattle </p></li><li><p>benign and self limiting </p></li><li><p>6 and 24 months old most at risk for warts </p></li><li><p>associated with dehorning, ear tagging, tattooing devices </p></li><li><p>lesions are cauliflower like, rough, or rusty </p></li><li><p>some appear flatter, gray and have a broad based skin attachment, others have a pedunculated  base </p></li><li><p>head, neck, brisket and sometimes udder and teats and eyelids are mostly affected</p></li></ul><p>treatment: </p><ul><li><p>spontaneous regression within 1-12 months </p></li><li><p>large wards may be <strong>surgically removed and their bases cauterized chemically </strong></p></li><li><p><strong>cryosurgery </strong></p></li><li><p><strong>crushing </strong>is also used to remove wars and may stimulate the immune system </p></li><li><p>lithium antimony thiomalate 6% solution 15 mL by deep IM injection every other day 4-6 times </p></li></ul><p>prevention: </p><ul><li><p>commercial or autogenous vaccines should be repeated at 7 day intervals </p></li><li><p>in show animals it takes at least 4 to 6 weeks before vaccination cause warts to regress </p></li><li><p>disinfection of surgical instruments </p></li></ul><p></p>
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Dermatophytosis (Ringworm)

  • club lamb fungus in sheep and goats

  • common in diary young calves in group housing during winter months

  • Trichophyton verrusconsum is the most common pathogen

  • infection by contact

  • zoonotic disease

  • treatment

    • remove crusts and debs

    • topical application of 7% iodine solution

    • topical application of chlorine bleach (1:10 dilution)

    • sun exposure: UV rays from sunlight aid healing

<ul><li><p><strong>club lamb fungus </strong>in sheep and goats </p></li><li><p>common in diary young calves in group housing during winter months </p></li><li><p>Trichophyton verrusconsum is the most common pathogen </p></li><li><p>infection by contact </p></li><li><p>zoonotic disease </p></li><li><p>treatment </p><ul><li><p>remove crusts and debs </p></li><li><p>topical application of 7% iodine solution </p></li><li><p>topical application of chlorine bleach (1:10 dilution) </p></li><li><p>sun exposure: UV rays from sunlight aid healing </p></li></ul></li></ul><p></p>
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Club lamb fungus

  • treatment options

  • most effective treatment

  • prevention strategies

  • hygiene practices

  • avoid communal bathing areas

  • disinfect grooming tools and equipment regularly

  • early detection

  • isolate affected lambs promptly to prevent spread

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<p>Dermatophilosis </p>

Dermatophilosis

  • Rain rot; Rain scald; Streptothricosis in cattle, goats, lamas; lumpy wool in sheep

    • Dermatophilus congolensis - gram positive, non acid fast, facultative anerobic actinomycete

    • moist environmental conditions and long hair coats predisposes to infection

    • animlas housed outside during rainy seasons

    • clinical signs - matted tufts of hair

    • zoonotic disease

  • Diagnosis

    • bacterial smears

      • gram stain, new methylene blue, or Diff-Quik

      • parallel rows of gram positive cocci that look like railroad tracks or tramcar line

  • treatment

    • topical and systemic antibiotics

    • topical application of lime sulfur

    • keep animals dry

<ul><li><p>Rain rot; Rain scald; Streptothricosis in cattle, goats, lamas; lumpy wool in sheep </p><ul><li><p><strong>Dermatophilus congolensis - </strong>gram positive, non acid fast, facultative anerobic <strong>actinomycete </strong></p></li><li><p>moist environmental conditions and long hair coats predisposes to infection </p></li><li><p>animlas housed outside during rainy seasons </p></li><li><p>clinical signs - <strong>matted tufts of hair </strong></p></li><li><p>zoonotic disease </p></li></ul></li><li><p>Diagnosis </p><ul><li><p>bacterial smears </p><ul><li><p>gram stain, new methylene blue, or Diff-Quik </p></li><li><p>parallel rows of gram positive cocci that look like <strong>railroad tracks or tramcar line </strong></p></li></ul></li></ul></li><li><p>treatment </p><ul><li><p>topical and systemic antibiotics </p></li><li><p>topical application of lime sulfur </p></li><li><p>keep animals dry </p></li></ul></li></ul><p></p>
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Urticaria and Angioedema

  • Hypersensitivity reactions

    • Urticaria (hives) appears as skin wheals or mucous membrane swellings as a result of dermal edema

    • Angioedema tends to imply larger swelling or plaques of edema that involve subcutaneous tissue

    • variety of drugs, feeds, and other stimuil may evoke hypersensitivity reactions

    • rapid onset

    • severe respiratory and cardiovascular signs

    • milk allergy is common in jersery cows at drying off

  • treatment

    • remove inciting cause

    • Antihistamines, NSAIDs with or without coricosteroids

<ul><li><p>Hypersensitivity reactions </p><ul><li><p>Urticaria (hives) appears as skin wheals or mucous membrane swellings as a result of dermal edema </p></li><li><p>Angioedema tends to imply larger swelling or plaques of edema that involve subcutaneous tissue </p></li><li><p>variety of drugs, feeds, and other stimuil may evoke hypersensitivity reactions </p></li><li><p>rapid onset </p></li><li><p><strong>severe respiratory and cardiovascular signs </strong></p></li><li><p><strong>milk allergy is common in jersery cows at drying off </strong></p></li></ul></li><li><p>treatment </p><ul><li><p>remove inciting cause </p></li><li><p><strong>Antihistamines, NSAIDs with or without coricosteroids </strong></p></li></ul></li></ul><p></p>
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Photosensitization

Primary:

  • occurs when a photodynamic agent is either ingested, injected or absorbed through the skin

  • Examples: hypericin (St. John’s wort), fagopyrin (Buckwheat), furocoumarins (psoalens) from various toxic plants

Congential photosensitization resulting in aberrant pigment synthesis

  • bovine erythropoietic porphyria

  • bovine protoporphyria

secondary (hepatogenous) photsensitization

  • the most common type

  • impaired hepatic detoxification and excretion of the phototoxic agent phylloerythrin

  • hepatotoxic plants containing pyrrolizidine alkaloids

  • blue green algae

  • molds such as Aspegillus sp, fusarium sp, and pithomyces sp

Diagnosis

  • clinical signs - erythema and edema of hairless, nonpigmented areas of skin

  • evidence or history of exposure to photosensitizing agents or hepatotoxins

  • Evidence of liver damage, elevated SDH, GGT, ALP, direct bilirubin

prognosis is poor for cattle with hepatogenous photosensitization

<p>Primary: </p><ul><li><p>occurs when a photodynamic agent is either ingested, injected or absorbed through the skin </p></li><li><p>Examples: hypericin (St. John’s wort), fagopyrin (Buckwheat), furocoumarins (psoalens) from various toxic plants </p></li></ul><p>Congential photosensitization resulting in aberrant pigment synthesis </p><ul><li><p>bovine erythropoietic porphyria </p></li><li><p>bovine protoporphyria </p></li></ul><p>secondary (hepatogenous) photsensitization</p><ul><li><p>the most common type  </p></li><li><p>impaired hepatic detoxification and excretion of the phototoxic agent <strong>phylloerythrin </strong></p></li><li><p>hepatotoxic plants containing <strong>pyrrolizidine alkaloids</strong></p></li><li><p>blue green algae </p></li><li><p>molds such as Aspegillus sp, fusarium sp, and pithomyces sp </p></li></ul><p>Diagnosis </p><ul><li><p>clinical signs - erythema and edema of hairless, <strong>nonpigmented areas of skin </strong></p></li><li><p>evidence or history of exposure to photosensitizing agents or hepatotoxins </p></li><li><p><strong>Evidence of liver damage, elevated SDH, GGT, ALP, direct bilirubin </strong></p></li></ul><p>prognosis is poor for cattle with hepatogenous photosensitization </p><p></p>
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LIce

  • 2 types

    • sucking lice - larger and take blood meals

    • biting lice - feed on dead skin and feathers

    • causes pruritus, restlessness, and excessive licking

    • blood loss anemia in calves

  • treatment

    • organochlorine, organophosphorus, pyrethroid, coumaphos, diazinon and permethrin

    • Ivermectin injectable gets sucking lice but not biting spp

<ul><li><p>2 types </p><ul><li><p>sucking lice - larger and take blood meals </p></li><li><p>biting lice - feed on dead skin and feathers </p></li><li><p>causes pruritus, restlessness, and excessive licking </p></li><li><p>blood loss anemia in calves </p></li></ul></li><li><p>treatment </p><ul><li><p>organochlorine, organophosphorus, pyrethroid, coumaphos, diazinon and permethrin </p></li><li><p><strong>Ivermectin injectable gets sucking lice but not biting spp </strong></p></li></ul></li></ul><p></p>
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Ticks

  • 2 types: soft and hard

    • inflammation, itching, and swelling at the site of the bite

    • anemia, weight loss, and even death

    • ticks can also create wounds that can become infected if not taken care of

    • spread diseases

      • Dermacentor - tansmits anaplasmosis

      • Cattle fever tick (Rhinpicephalus annulatus) - transmits babesia (reportable)

<ul><li><p>2 types: soft and hard </p><ul><li><p>inflammation, itching, and swelling at the site of the bite </p></li><li><p>anemia, weight loss, and even death </p></li><li><p>ticks can also create wounds that can become infected if not taken care of </p></li><li><p><strong>spread diseases </strong></p><ul><li><p><strong>Dermacentor - tansmits anaplasmosis </strong></p></li><li><p>Cattle fever tick (Rhinpicephalus annulatus) - <strong>transmits babesia (reportable) </strong></p></li></ul></li></ul></li></ul><p></p>
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Mange

  • occur during colder months, in houses animals with close contact

  • causes skin irritation, hide damage, reduced gain

  • reportable in most states

  • several species

    • Psoroptes cuniculi - ear mites in sheep and goats

    • Chorioptes bovis - tail or foot or red mange, pruritic and nodular lesions, most common type of mange in cattle

    • Demodex - non pruritic nodular lesions containing the mite, cigar shaped mite

  • Diagnosis

    • microscopic examination of skin scrapes

    • scrape the edges of the lesion using scalpel

    • wet scraping with 10% potassium hydroxide

  • treatment

    • cattle

      • use approved products

      • injectable or pour-on formulations of doramectin, ivermectin, and moxidectin

      • 2 to 3 topical treatments separated by 2-3 weeks

    • sheep and goats

      • hot lime sulfur spray or dip. treatment should be repeated every 12 days if needed

<ul><li><p>occur during colder months, in houses animals with close contact </p></li><li><p>causes skin irritation, hide damage, reduced gain </p></li><li><p><strong>reportable in most states </strong></p></li><li><p>several species </p><ul><li><p>Psoroptes cuniculi - <strong>ear mites in sheep and goats </strong></p></li><li><p><strong>Chorioptes bovis - tail or foot or red mange, pruritic and nodular lesions, most common type of mange in cattle </strong></p></li><li><p><strong>Demodex - non pruritic nodular lesions containing the mite, cigar shaped mite </strong></p></li></ul></li><li><p>Diagnosis </p><ul><li><p>microscopic examination of <strong>skin scrapes </strong></p></li><li><p>scrape the edges of the lesion using scalpel </p></li><li><p>wet scraping with 10% potassium hydroxide </p></li></ul></li><li><p>treatment </p><ul><li><p>cattle </p><ul><li><p>use approved products </p></li><li><p>injectable or pour-on formulations of doramectin, ivermectin, and moxidectin </p></li><li><p><strong>2 to 3 topical treatments separated by 2-3 weeks </strong></p></li></ul></li><li><p>sheep and goats </p><ul><li><p><strong>hot lime sulfur spray or dip. treatment should be repeated every 12 days if needed </strong></p></li></ul></li></ul></li></ul><p></p>
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Obligatory or Primary Myiasis (Screwworm)

  • the fly larvae are completely parasitic

  • invade small skin wounds or mucus membranes

  • Eradicated in USA using the sterile male release program

  • occasional outbreaks in ares on Mexico border (Recently in Florida in Deer)

  • reportable disease

<ul><li><p>the fly larvae are completely parasitic </p></li><li><p>invade small skin wounds or mucus membranes </p></li><li><p>Eradicated in USA using the sterile male release program </p></li><li><p><strong>occasional outbreaks in ares on Mexico border (Recently in Florida in Deer) </strong></p></li><li><p>reportable disease </p></li></ul><p></p>
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Facultative Myiasis

  • Blow fly strike or maggots

    • more common problem in sheep less in goats and cattle (breech region in sheep)

    • castration and tail wounds, clipping wounds, head wounds on fighting rams

    • open wounds that are infected and smelly attract flies (wound strike or maggots)

  • treatment

    • wound care

    • Larvicidal agents - ivermectin or organophosphates

  • prevention

    • shearing regularly before the summer: shearing controls outbreaks

    • crutching (ie wool is shorn between the legs and around the tial)

    • Mulesing (ie operation to remove folds of skin from the hindquarters of the sheep) welfare issue

    • topical application of quarternary ammonium, phenols, caustic soda or plastic clips on the hindquarter of sheep

    • proper care of surgical and traumatic wounds - Ivermectin given at time of sx provides residual protection for 16-20 days

    • insecticide sprays or ointments

    • vaccination

<ul><li><p>Blow fly strike or maggots </p><ul><li><p>more common problem in sheep less in goats and cattle (<strong>breech region in sheep) </strong></p></li><li><p>castration and tail wounds, clipping wounds, head wounds on fighting rams </p></li><li><p>open wounds that are infected and smelly attract flies (<strong>wound strike or maggots) </strong></p></li></ul></li><li><p>treatment </p><ul><li><p>wound care </p></li><li><p>Larvicidal agents - <strong>ivermectin or organophosphates </strong></p></li></ul></li><li><p>prevention </p><ul><li><p>shearing regularly before the summer: shearing controls outbreaks </p></li><li><p><strong>crutching (ie wool is shorn between the legs and around the tial) </strong></p></li><li><p><strong>Mulesing (ie operation to remove folds of skin from the hindquarters of the sheep) welfare issue </strong></p></li><li><p>topical application of quarternary ammonium, phenols, caustic soda or plastic clips on the hindquarter of sheep </p></li><li><p><strong>proper care of surgical and traumatic wounds - Ivermectin given at time of sx provides residual protection for 16-20 days </strong></p></li><li><p>insecticide sprays or ointments </p></li><li><p>vaccination </p></li></ul></li></ul><p></p>
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Hypodermiasis (Warbles, Grubs)

Warble or heel flies of cattle:

  • reported in sheep, goats, deer, humans, dogs, cats,

  • hide damage, decreased milk production, weight loss

  • decreased amount of feeding time due to irritation and gradding

entire life cycle is 1 year

  • larvae migrate to the submucosa of the caudal thirds of the esophagus

  • larvae reac the epidural adipose tissue between the dura mater and periosteum of the thoracolumbar vertebrae

treatment

  • treat cows between July 1 till end of October

  • to prevent adverse reactions cattle should NOT be treated between December and March

  • pour on products - ivermectin, moxidectin, organophosphates

adverse reactions

  • esophageal inflammation could choke

  • temporary or permanent paralysis

  • anaphylactic reactions are common

<p>Warble or heel flies of cattle: </p><ul><li><p>reported in sheep, goats, deer, humans, dogs, cats, </p></li><li><p>hide damage, decreased milk production, weight loss </p></li><li><p>decreased amount of feeding time due to irritation and gradding </p></li></ul><p>entire life cycle is 1 year </p><ul><li><p><strong>larvae migrate to the submucosa of the caudal thirds of the esophagus </strong></p></li><li><p><strong>larvae reac the epidural adipose tissue between the dura mater and periosteum of the thoracolumbar vertebrae </strong></p></li></ul><p>treatment </p><ul><li><p><strong>treat cows between July 1 till end of October </strong></p></li><li><p>to prevent adverse reactions cattle should NOT be treated between <strong>December and March </strong></p></li><li><p>pour on products - <strong>ivermectin, moxidectin, organophosphates </strong></p></li></ul><p>adverse reactions </p><ul><li><p>esophageal inflammation could choke </p></li><li><p>temporary or permanent paralysis </p></li><li><p>anaphylactic reactions are common </p></li></ul><p></p>
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Sheep ked

  • not a tick but a wingless blood suckling fly

  • keds will bite humans producing painful result

  • spreadby contact; long wooled breeds are more susceptible

  • heavy infestation common in autumn and winter

  • loss of condition, anemia, irritation, biting and rubbing, damaged skin and low quality fleece

  • diagnosis by inspection of affected wooly areas looking for wingless fly

  • treatment using pour on products - ivermectin, moxidectin, organosphosphates

<ul><li><p>not a tick but a wingless blood suckling fly </p></li><li><p>keds will bite humans producing painful result </p></li><li><p>spreadby contact; long wooled breeds are more susceptible </p></li><li><p>heavy infestation common in autumn and winter </p></li><li><p>loss of condition, anemia, irritation, biting and rubbing, damaged skin and low quality fleece </p></li><li><p>diagnosis by inspection of affected wooly areas looking for wingless fly </p></li><li><p>treatment using pour on products - ivermectin, moxidectin, organosphosphates </p></li></ul><p></p>
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Pygmy goats as pets

  • Origin: West Africa

  • Introduced to the U.S. in the 1950s for research and exhibition

  • gained popularity as pets and livestock due to small size, friendly nature, anda adaptability

  • lifespan: 10-15 years

<ul><li><p>Origin: West Africa </p></li><li><p>Introduced to the U.S. in the 1950s for research and exhibition </p></li><li><p>gained popularity as pets and livestock due to small size, friendly nature, anda adaptability </p></li><li><p>lifespan: 10-15 years </p></li></ul><p></p>
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Pygmy goat internal parasites

  • types: GI nematodes, flukes, lungworms

  • clinical signs

    • weight loss, poor growth, anemia, poor hair coat, submandibular edema (bottle jaw,) diarrhea

    • specific signs (coughing from lungworms)

  • risk factors: environmental conditions, stocking density

  • diagnosis fecal exmiantion, fecal egg count

<ul><li><p>types: GI nematodes, flukes, lungworms </p></li><li><p>clinical signs </p><ul><li><p>weight loss, poor growth, anemia, poor hair coat, submandibular edema (bottle jaw,) diarrhea </p></li><li><p>specific signs (coughing from lungworms) </p></li></ul></li><li><p>risk factors: environmental conditions, stocking density </p></li><li><p>diagnosis fecal exmiantion, fecal egg count </p></li></ul><p></p>
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treatment of internal parasites in goats

  • administer only when clinical signs are evident. avoid overuse

  • assess anemia severity using FAMACHA

  • combination treatments are more effective than single drug treatments in reducing the survival of resistant worms, especially if used early

  • examples: albendazole + levamisole

  • monitor drug efficacy using fecal egg count

  • consider breeding for inherent resistance

Copper oxide wine particles (COWPs):

  • COWPs are tiny copper oxide rods

  • used as a supplement to reduce barber pole worm infections in sheep and goats

  • best used selectively for animals showing clinical signs of hemonchosis (FAMACHA score 4)

  • combination treatment examples:

    • Albendazole + COWPs

    • Levamisole + COWPs

decision-making tools for deworming:

  • FAMACHA system: specifically for blood feeding parasites like barber pole worm

  • five point check

    • eye: check for anemia

    • jaw: look for bottle jaw

    • back: assess body conditon score (BCS)

    • tail: observe for dags

    • coat: inspect for coat condition

  • happy factor

    • performance indicators: use average daily gain and or milk production

<ul><li><p>administer only when clinical signs are evident. avoid overuse </p></li><li><p>assess anemia severity using FAMACHA</p></li><li><p>combination treatments are more effective than single drug treatments in reducing the survival of resistant worms, especially if used early </p></li><li><p>examples: albendazole + levamisole </p></li><li><p>monitor drug efficacy using fecal egg count </p></li><li><p>consider breeding for inherent resistance </p></li></ul><p>Copper oxide wine particles (COWPs): </p><ul><li><p>COWPs are tiny copper oxide rods </p></li><li><p>used as a supplement to reduce barber pole worm infections in sheep and goats </p></li><li><p>best used selectively for animals showing clinical signs of hemonchosis (FAMACHA score 4) </p></li><li><p>combination treatment examples: </p><ul><li><p>Albendazole + COWPs </p></li><li><p>Levamisole + COWPs </p></li></ul></li></ul><p>decision-making tools for deworming: </p><ul><li><p>FAMACHA system:  specifically for blood feeding parasites like barber pole worm </p></li><li><p>five point check </p><ul><li><p>eye: check for anemia </p></li><li><p>jaw: look for bottle jaw </p></li><li><p>back: assess body conditon score (BCS) </p></li><li><p>tail: observe for dags </p></li><li><p>coat: inspect for coat condition </p></li></ul></li><li><p>happy factor </p><ul><li><p>performance indicators: use average daily gain and or milk production </p></li></ul></li></ul><p></p>
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psoroptic mange (ear mange)

  • Caused by P. cuniculi

  • typically infests the ears of goats but can spread to the head, neck, and body

  • clinical signs: scaling, crusting, inflammation, alopecia, ear scratching, head shaking, and rubbing of ears and head

  • prognosis is good with appropriate treatment

  • treatment

    • ivermectin, moxidectin or eprinomectin

    • Coumaphos, toxaphene, lime sulfur and phosmet

    • repeat treatment at least twice 5 to 7 days aprat

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Chorioptic mange

  • C. caprae

  • lesion distribution: scrotum, distal limbs in sheep; lower limbs, abdomen, hindquarters in goats

  • clinical signs: alopecia, erythema, excoriation, crusting; pruritus, restlessness.

  • treatment

    • ivermectin, moxidectin or eprinomectin

    • repeat treatment at least twice, 5 to 7 days apart

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Demodectic mange

  • D. caprae

  • lesion distribution: face, limbs, back; severe folliculitis, secondary pyoderma

  • diagnosis: deep skin scraping; examination of exudate for mites

  • treatment: ivermectin, moxidectin, or eprinomectin

  • repeat treatment at least twice 5 to 7 days apart

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Lice

  • Clinical signs: pruritus, alopecia, anemia, skin damage, loss of condition, decrease in production (both milk and wool)

  • treatment:

    • Coumaphos

    • malathion

    • injection of avermectin anthelmintics

    • avermectin are useful in treating sucking lice but have limited efficacy against biting lice

    • repeat treatment at least twice 5 to 7 days apart

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Dermatophilosis (Streptothricosis, Lumpy wool disease, rain scald, rain rot)

  • causative agent: Dermatophilus congolensis baterium

  • transmission: through carrier animals, biting insects, abrasion, and moisture

  • predisposing factors: skin damage, moisture, weakened immune system

  • clinical signs: papules and pustules coalesce, forming “paintbrush lesions”

  • diagnosis: gram stain, histopathology, culture

  • treatment:

    • topical agents like iodine

    • oxytetracycline

  • prevention: maintain dry conditions, good nutrition and control ectoparasites; vaccines have limited effectiveness

<ul><li><p>causative agent: Dermatophilus congolensis baterium </p></li><li><p>transmission: through carrier animals, biting insects, abrasion, and moisture </p></li><li><p>predisposing factors: skin damage, moisture, weakened immune system</p></li><li><p>clinical signs: papules and pustules coalesce, forming “paintbrush lesions” </p></li><li><p>diagnosis: gram stain, histopathology, culture </p></li><li><p>treatment: </p><ul><li><p>topical agents like iodine </p></li><li><p>oxytetracycline </p></li></ul></li><li><p>prevention: maintain dry conditions, good nutrition and control ectoparasites; vaccines have limited effectiveness </p></li></ul><p></p>
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Staphylococcal Dermatitis

  • Nonpruritic lesions on head and face

  • alopecia, papules, crusts, erosions or ulcers, exudation, erythema, hyperpigmentation, thickening of the skin.

  • diagnosis: culture, histopathologic examination

  • treatment:

    • Antibiotics

    • wash affected skin with iodine or chlorhexidine shampoo

    • topical antiseptic

    • antibiotic ointment (Oxytetracycline/polymyxin B)

  • prevention: prevent facial injuries, control fly populations, isolate affected animals

<ul><li><p>Nonpruritic lesions on head and face </p></li><li><p>alopecia, papules, crusts, erosions or ulcers, exudation, erythema, hyperpigmentation, thickening of the skin. </p></li><li><p>diagnosis: culture, histopathologic examination </p></li><li><p>treatment: </p><ul><li><p>Antibiotics</p></li><li><p>wash affected skin with iodine or chlorhexidine shampoo </p></li><li><p>topical antiseptic </p></li><li><p>antibiotic ointment (Oxytetracycline/polymyxin B) </p></li></ul></li><li><p>prevention: prevent facial injuries, control fly populations, isolate affected animals </p></li></ul><p></p>
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Dermatophytosis (Ringworm)

  • Caused by Trichophyton verrucosum

  • infection by contact

  • zoonotic disease

  • treatment

    • remove cruts and debris

    • topical application of 7% iodine solution

    • topical application of chlorine bleach (1:10 dilution)

    • sun exposure: UV rays from sunlight aid healing

<ul><li><p>Caused by Trichophyton verrucosum </p></li><li><p>infection by contact </p></li><li><p>zoonotic disease </p></li><li><p>treatment </p><ul><li><p>remove cruts and debris </p></li><li><p>topical application of 7% iodine solution </p></li><li><p>topical application of chlorine bleach (1:10 dilution) </p></li><li><p>sun exposure: UV rays from sunlight aid healing </p></li></ul></li></ul><p></p>
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Bacterial pneumonia

  • Pasteurella multocida, Mannheamia hemolytica, Mycoplasma spp

  • symptoms: fever (>104F) coughing, nasal discharge, difficulty breathing, weakness, death

  • Diagnosis:

    • auscultate the lungs for abnormal lung sounds such as crackles or wheezes

    • thoracic radiographs

    • collect a tracheal wash sample for bacterial culture

  • treatment: antibiotics, NSAIDs, supportive care

  • prevention: good nutrition, sanitation, proper ventilation, vaccination

<ul><li><p>Pasteurella multocida, Mannheamia hemolytica, Mycoplasma spp </p></li><li><p>symptoms: fever (&gt;104F) coughing, nasal discharge, difficulty breathing, weakness, death </p></li><li><p>Diagnosis: </p><ul><li><p>auscultate the lungs for abnormal lung sounds such as crackles or wheezes </p></li><li><p>thoracic radiographs </p></li><li><p>collect a tracheal wash sample for bacterial culture </p></li></ul></li><li><p>treatment: antibiotics, NSAIDs, supportive care </p></li><li><p>prevention: good nutrition, sanitation, proper ventilation, vaccination </p></li></ul><p></p>
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Viral pneumonia

Parainfluenza type 3 (PI3)

  • member of the paramyxovirus family

  • clinical signs: coughing, nasal discharge, fever rare

  • diagnosis: virus isolation

  • treatment: supportive

  • prevention: vaccination with live intranasal vaccine may reduce disease incidence

Adenovirus:

  • clinical signs: mild, severity increases with secondary bacterial infection

  • diagnosis: virus isolation or paired serology samples

  • treatment: supportive

  • prevention: vaccination with live intranasal vaccine may reduce disease incidence

respiratory syncytial virus (RSV):

  • clinical signs: anorexia, fever, conjunctivitis, cough, tachypnea, tachycardia

  • diagnosis: necropsy findings, observation of syncytial cells on histopathologic examination, immunoperoxidase staining

  • treatment: supportive

  • prevention: use of a commercial cattle monovalent modified live virus vaccine against RSV

Caprine Herpesvirus:

  • linked to rhinitis, vulvovaginitis, abortions

  • may induce mild rhinitis, tracheitis

  • diagnosis by PCR

  • treatment: supportive

  • control: emphasize environmental and stress management

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Keratoconjuctivitis

  • caused by Chlamydophila spp, and Mycoplasma spp

    • very common

    • clinical signs: Epiphora, hyperemia, mucopurulent discharge, keratitis

    • history may indicate other problems in the herd: mastitis, polyarthritis, pleuropneumonia

    • diagnosis: cytology, IFA, culture

    • treatment:

      • topical antibiotics

      • oxytetracycline

      • NSAIDs

<ul><li><p>caused by Chlamydophila spp, and Mycoplasma spp </p><ul><li><p>very common </p></li><li><p>clinical signs: Epiphora, hyperemia, mucopurulent discharge, keratitis </p></li><li><p>history may indicate other problems in the herd: mastitis, polyarthritis, pleuropneumonia </p></li><li><p>diagnosis: cytology, IFA, culture </p></li><li><p>treatment: </p><ul><li><p>topical antibiotics </p></li><li><p>oxytetracycline </p></li><li><p>NSAIDs </p></li></ul></li></ul></li></ul><p></p>
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Caprine arthritis encephalitis (CAE)

  • caused by: Lentivirus

  • clinical signs: progressive paresis in kids, arthritis in adults, agalactia, chronic weight loss

  • transmission: vertical (colostrum, milk) and horizontal (blood, mucus)

  • diagnosis by serologic testing (ELISA, AGID) PCR

  • treatment: pain medications

  • prevention:

    • isolate kids immediately after birth

    • feed kids pasteurized colostrum

    • test kids for CAEV every 6 months and cull positive animals

    • biosecurity - new additions should be quarantined and tested within 60 days of arrival

    • chemical disinfection of equipment using phenolic and quaternary ammonium compounds.

<ul><li><p>caused by: Lentivirus </p></li><li><p>clinical signs: progressive paresis in kids, arthritis in adults, agalactia, chronic weight loss </p></li><li><p>transmission: vertical (colostrum, milk) and horizontal (blood, mucus) </p></li><li><p>diagnosis by serologic testing (ELISA, AGID) PCR </p></li><li><p>treatment: pain medications </p></li><li><p>prevention: </p><ul><li><p>isolate kids immediately after birth </p></li><li><p>feed kids pasteurized colostrum </p></li><li><p>test kids for CAEV every 6 months and cull positive animals </p></li><li><p>biosecurity - new additions should be quarantined and tested within 60 days of arrival </p></li><li><p>chemical disinfection of equipment using phenolic and quaternary ammonium compounds. </p></li></ul></li></ul><p></p>
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Paratuberculosis (Johne’s Disease)

  • Caused by Mycobacterium avium paratuberculosis

  • transmission: vertical (colostrum, milk) fecal-oral, in utero, milk

  • clinical signs: weight loss, submandibular edema, weakness; diarrhea less common

  • diagnosis: fecal PCR testing, necropsy (enlarged lymph nodes, thickened intestine)

  • treatment: no cure

  • prevention:

    • maintain a closed herd

    • kidding area cleanliness

  • vaccinations

    • there is no vaccine approved for goats

    • USDA-approved vaccine for cattle is effective in reducing the risk for shedding in lactating herd

<ul><li><p>Caused by Mycobacterium avium paratuberculosis </p></li><li><p>transmission: vertical (colostrum, milk) fecal-oral, in utero, milk </p></li><li><p>clinical signs: weight loss, submandibular edema, weakness; diarrhea less common </p></li><li><p>diagnosis: fecal PCR testing, necropsy (enlarged lymph nodes, thickened intestine) </p></li><li><p>treatment: no cure </p></li><li><p>prevention: </p><ul><li><p>maintain a closed herd </p></li><li><p>kidding area cleanliness </p></li></ul></li><li><p>vaccinations </p><ul><li><p>there is no vaccine approved for goats </p></li><li><p>USDA-approved vaccine for cattle is effective in reducing the risk for shedding in lactating herd </p></li></ul></li></ul><p></p>
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Caseous lymphadenitis (CL; cheesy gland)

  • Caused by: Corynebacterium pseudotuberculosis

  • transmission: skin breaks, contact with pus, contaminated fomites

  • forms: internal (chronic weight loss) and external abscesses

  • diagnosis: radiographs, culture, or serologic testing (SHI test)

  • antimicrobials (intralesional and or systemic)

    • treatment is extra-label; strict adherence to withdrawal times is required

  • systemic antibiotics

    • long term procaine penicillin G and Rifampin have shown some success

    • tulathromycin reaches effective concentrations in walled-off abscesses

  • drainage and isolation

  • environmental decontamination

  • strict biosecurity measures

  • vaccination: there is no vaccine licensed for goats

    • sheep vaccine - not 100% effective but reduces disease prevalence and incidence

  • shearing precautions

  • environmental hazard reduction remove barbed wire and such

<ul><li><p>Caused by: Corynebacterium pseudotuberculosis </p></li><li><p>transmission: skin breaks, contact with pus, contaminated fomites</p></li><li><p>forms: internal (chronic weight loss) and external abscesses </p></li><li><p>diagnosis: radiographs, culture, or serologic testing (SHI test) </p></li><li><p>antimicrobials (intralesional and or systemic) </p><ul><li><p>treatment is extra-label; strict adherence to withdrawal times is required </p></li></ul></li><li><p>systemic antibiotics </p><ul><li><p>long term procaine penicillin G and Rifampin have shown some success </p></li><li><p>tulathromycin reaches effective concentrations in walled-off abscesses </p></li></ul></li><li><p>drainage and isolation </p></li><li><p>environmental decontamination </p></li><li><p>strict biosecurity measures </p></li><li><p>vaccination: there is no vaccine licensed for goats </p><ul><li><p>sheep vaccine - not 100% effective but reduces disease prevalence and incidence </p></li></ul></li><li><p>shearing precautions </p></li><li><p>environmental hazard reduction remove barbed wire and such </p></li></ul><p></p>
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Clostridium tetani infection (tetanus)

  • an anaerobic spore forming bcterium found in soil and manure

  • clinical signs

    • lockjaw

    • muscle spasms, especially in neck, limbs, and back

    • sawhorse stance

    • difficulty breathing

    • seizures and possible death

  • Diagnosis: based on clinical signs and history of injury

  • treatment:

    • tetanus antitoxin, antibiotics (penicillin/tetracycline)

    • muscle relaxants, sedatives

    • supportive care

  • prevention

    • vaccination (tetanus toxoid)

    • proper wound care

  • prognosis: poor

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Pregnancy toxemia

  • Cause: high energy demand late in pregnancy, especially with multiple kids

  • clinical signs and diagnosis:

    • neurological signs such as depression, recumbency, tremors, star-gazing, incoordination, circling, and teeth grinding

    • confirmed by detecting increased urine and blood ketone concentrations

  • treatment

    • in early cases

      • oral propylene glycol 2-3 ounces twice daily

      • B vitamins, calcium borogluconate

      • transfaunation

      • feed energy rich diet

    • severe cases in late gestation require C-section, IV fluids with dextrose and B vitamins

  • prevention:

    • avoid excessive weight gain early in pregnancy

    • increase grain in final month

    • provide low stress environment

<ul><li><p>Cause: high energy demand late in pregnancy, especially with multiple kids </p></li><li><p>clinical signs and diagnosis: </p><ul><li><p>neurological signs such as depression, recumbency, tremors, star-gazing, incoordination, circling, and teeth grinding </p></li><li><p>confirmed by detecting increased urine and blood ketone concentrations </p></li></ul></li><li><p>treatment </p><ul><li><p>in early cases </p><ul><li><p>oral propylene glycol 2-3 ounces twice daily </p></li><li><p>B vitamins, calcium borogluconate </p></li><li><p>transfaunation </p></li><li><p>feed energy rich diet </p></li></ul></li><li><p>severe cases in late gestation require C-section, IV fluids with dextrose and B vitamins </p></li></ul></li><li><p>prevention: </p><ul><li><p>avoid excessive weight gain early in pregnancy </p></li><li><p>increase grain in final month </p></li><li><p>provide low stress environment </p></li></ul></li></ul><p></p>
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Coccidiosis in the neonate

  • clinical signs: thin kids, diarrhea, sudden death around ~3 weeks old.

  • diagnosis: fecal flotation test to detect oocysts of Eimeria spp

  • treatment:

    • amprolium

    • decoquinate

    • benzeacetonitrile compounds

    • parenteral sulfadimethoxine

    • oral sulfadimethoxine

    • oral sulfadimidine

<ul><li><p>clinical signs: thin kids, diarrhea, sudden death around ~3 weeks old. </p></li><li><p>diagnosis: fecal flotation test to detect oocysts of Eimeria spp </p></li><li><p>treatment: </p><ul><li><p>amprolium </p></li><li><p>decoquinate </p></li><li><p>benzeacetonitrile compounds </p></li><li><p>parenteral sulfadimethoxine </p></li><li><p>oral sulfadimethoxine </p></li><li><p>oral sulfadimidine </p></li></ul></li></ul><p></p>
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Navel ill and septic joint

  • cause: infection of the umbilicus

  • clinical signs: swollen umbilicus, swollen joints, reluctance to move, failure to gain weight

  • treatment:

    • systemic antibiotics: Florfenicol or tylosin

    • joint lavage with saline solution

    • intra-articular injection of antimicrobials (Ceftiofur, penicilli, amikacin)

    • regional limb perfusion of antibiotics (ceftiofur, penicillin, amikacin)

    • surgical removal of the umbilicus

  • prevention: Dip umbilicus with 7% iodine immediately after birth; maintain a clean birthing area

<ul><li><p>cause: infection of the umbilicus </p></li><li><p>clinical signs: swollen umbilicus, swollen joints, reluctance to move, failure to gain weight </p></li><li><p>treatment: </p><ul><li><p>systemic antibiotics: Florfenicol or tylosin </p></li><li><p>joint lavage with saline solution </p></li><li><p>intra-articular injection of antimicrobials (Ceftiofur, penicilli, amikacin) </p></li><li><p>regional limb perfusion of antibiotics (ceftiofur, penicillin, amikacin) </p></li><li><p>surgical removal of the umbilicus </p></li></ul></li><li><p>prevention: Dip umbilicus with 7% iodine immediately after birth; maintain a clean birthing area </p></li></ul><p></p>
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Contagious ecthyma (sore mouth)

  • cause: orf virus

  • clinical signs: painful lesions around mouths of kids, on teats of does; may lead to anorexia and starvation

  • transmission: direct contact, fomites

  • treatment:

    • parenteral antibiotics

    • topical antibiotics

    • repellents and larvicides to prevent myiasis

    • NSAIDs for pain

    • supportive care

  • prevention: vaccine is live and should only be administered if the disease is present

<ul><li><p>cause: orf virus </p></li><li><p>clinical signs: painful lesions around mouths of kids, on teats of does; may lead to anorexia and starvation </p></li><li><p>transmission: direct contact, fomites </p></li><li><p>treatment: </p><ul><li><p>parenteral antibiotics </p></li><li><p>topical antibiotics </p></li><li><p>repellents and larvicides to prevent myiasis </p></li><li><p>NSAIDs for pain </p></li><li><p>supportive care </p></li></ul></li><li><p>prevention: vaccine is live and should only be administered if the disease is present </p></li></ul><p></p>
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Polio

  • causes:

    • primary: Thiamine deficiency, sulfur toxicity

    • secondary: excessive grain, rumen acidosis, improper feeding

  • clinical signs:

    • stargazing, circling, head pressing, blindness, nystagmus, convulsions, death in 24-72 hours if untreated

  • treatment:

    • thiamine: 0.25cc/10pound body weight, 3-4 times daily

  • prevention:

    • diet: high roughage, low concentrate

    • avoid moldy feeds, excessive carbs

    • supplementation: thiamine, probiotics, brewers yeast

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Resuscitation of kids following dystocia

  • they may look normal initially but crash 2-4 hours later

  • need to be aggressive in therapy

  • if depressed: measure bicarb and correct deficit or use BD of 10

  • soludelta-dortef (1 mg/lb) injection

  • consider oxygen even if no respiratory difficulty and normal color

  • dehydrated, non-diarrheic (septic, hasnt nursed): hypoglycemic

    • if <8% dehydrated, mild depression, still walking: 150-250 mls oral calf electrolytes without bicarb

    • If >8% dehydrated, depressed, recumbent: correct fluid deficit with balanced electrolyte solution

    • follow up with milk

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Castration

  • castration of males reduces offensive odor

  • should be delayed to allow musculoskeletal development

  • early castration (before 3 months) hinder penile development and urethral diameter leading urolithiasis

  • aseptic techniques, proper pain management and post-operative care are essential for animal well being

  • surgical castration under general anesthesia is preferred

  • complications can include hemorrhage, infection, and tetanus

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dystocia goats

  • causes: fetal malpositioning, fetal disproportion, multiple fetuses, incomplete cervical dilation (ringwomb), uterine inertia, and uterine torsion.

  • C-section common in pygmy goats

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ringwomb

  • failure of the cervix to adequately dilate resulting in dystocia

  • causes:

    • late gestation hormonal imbalance

    • nutritional factors

    • genetic predisposition

  • treatment

    • early intervention

      • IM administration of estrogen

      • followed by SQ hourly xytocin (10 units)

    • manual dilation: gradual manual dilation with adequate lubrication after the second oxytocin dose

    • most effective treatment: Cesarean section for rapid and successful resolution

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Foot rot goats

  • cause:

    • Fusobacterium necrophorum and Dichelobacter nodosus

    • wet and muddy conditions promote infection

    • overgrown hooves increase susceptibility

  • signs

    • lameness, swelling, pain, reluctance to walk

    • necrotic lesions in the interdigital space with a foul odor

  • diagnosis: clinical signs physical exam

  • treatment

    • systemic antibiotics: penicillin streptomycin or oxytetracycline

    • foot bathing in formalin or zinc sulfate

  • prevention

    • quarantine new animals before flock introduction

    • maintain dry and clean barns

    • regular hoof trimmig and foot care

    • provide balanced nutrition with essential minerals

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goat hoof trimming

  • untrimmed hooves are prone to developing lameness, splayed toes, and foot rot

    • hooves are trimmed regularly preferably every 4 to 6 weeks

    • hoof care

    • conduct regular foot baths, especially during wet seasons to maintain hoof health

<ul><li><p>untrimmed hooves are prone to developing lameness, splayed toes, and foot rot </p><ul><li><p>hooves are trimmed regularly preferably every 4 to 6 weeks </p></li><li><p><strong>hoof care </strong></p></li><li><p>conduct regular foot baths, especially during wet seasons to maintain hoof health </p></li></ul></li></ul><p></p>
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Deworming program for goats

  • conduct regular fecal exams (every 3-4 months) to monitor parasite load

  • deworm strategically based on fecal egg counts

  • rotate pastures to reduce exposure to parasite eggs and larvae

  • deworm 3 to 4 times annually (every 3-4 months)

  • deworm does before kidding and during the last month of pregnancy

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goat vaccination program

  • Core vaccinations

    • CDT (Clostridium perfringens type C + D and tetanus)

    • vaccination for does

      • vaccinate 30 days before giving birth

      • administered as two shots 3-4 weeks apart

    • vaccination for kids

      • from vaccinated does

        • first dose at 5-6 weeks of age

        • booster 3-4 weeks later

      • from uncertain vaccinated history does

        • first dose at 7-21 days of age

        • booster 3-4 weeks alter

        • option to give tetanus antitoxin at birth or castration

  • optional vaccinations

    • caseous lymphadenitis vaccine if CL is common in the area

    • rabies if there is a risk or it is required by local laws

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Foot trimming swine

  • indications

    • to prevent lameness and ensure proper locomostion

    • to treat overgrown hooves, cracks, or foot infections

  • done as needed typically every 6-12 months in mature pigs or when lameness is oberved

  • procedure

    • restrain using a chute, sling, or sedation

    • use hoof shears, a rasp, or an angle grinder

    • remove overgrown hoof material carefully avoiding damage to sensitive tissues

    • treat any visible lesions, cracks, or infections with antiseptics or anibiotics

<ul><li><p>indications </p><ul><li><p>to prevent lameness and ensure proper locomostion </p></li><li><p>to treat overgrown hooves, cracks, or foot infections </p></li></ul></li><li><p>done as needed typically every <strong>6-12 months </strong>in mature pigs or when lameness is oberved </p></li><li><p>procedure </p><ul><li><p>restrain using a chute, sling, or sedation </p></li><li><p>use <strong>hoof shears, a rasp, or an angle grinder </strong></p></li><li><p>remove overgrown hoof material carefully avoiding damage to sensitive tissues </p></li><li><p>treat any visible lesions, cracks, or infections with antiseptics or anibiotics </p></li></ul></li></ul><p></p>
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Tusk trimming (boars)

  • to prevent injuries to handlers, other pigs, or sows during breeding

  • every 6-12 months, depending on the growth rate

  • use a wire saw, bolt cutters, or an electric grinder

  • restrain with sedation

  • trim the tusks just above the gum line, avoiding damage to the pulp cavity to prevent pain or infection

  • disinfect tools before and after use

  • apply antiseptic to the trimmed area

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Tail docking

  • to prevent tail-biting which can lead to infections and abscesses in pigs

  • perform tail docking within the first 3-7 days of life

  • use sterilized equipment such as a sharp cutter, scissors, or a cauterizing device

  • leave about 1-2 cm of tail to minimize nerve damage

  • apply an antiseptic to prevent infection

  • docking tails too short lead to rectal prolapse

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Needle tooth removal

  • to prevent injuries to littermates and sows udder during nursing

  • perform within the first 24-48 hours of life

  • use sterilized sharp clippers or grinders to clip the sharp tips of the 8 needle teeth (corner deciduous incisors and canines)

  • avoid clipping too close to the gum to prevent damage and infection

  • disinfect tools between litters to prevent disease transmission

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OHE

  • performed in pot belly pigs

  • indications

    • to prevent unwanted pregnancies

    • to prevent ovarian and uterine tumors

    • therapeutic treatment of chronic uterine infection tumors

  • age 6-8 weeks optimal

  • linea alba incision

  • use 3 clamp vessel ligation technique

  • subcuticular closure

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Pseudorabies (Aujeszky’s disease)

  • herpes virus

  • affcts all ages

  • clinicals signs

    • suckling piglets: sudden death

    • growers and weaned piglets: nervous signs

      • tremors, incoordination, dog-sitting, paddling, convulsions, coma and death

  • finishing and adults; respiratory signs

  • sows: reproductive failure

  • clinical symptoms:

    • giving birth to weak piglets

    • foaming at the mouth

    • severe neurological disorders

    • aborted fetus

    • hemorrhagic spot on the renal cortex

    • cerebral hemorrhage and congested meninges

    • pulmonary hemorrhage

    • liver with mulitple small focal areas of necrosis

<ul><li><p>herpes virus </p></li><li><p>affcts all ages </p></li><li><p>clinicals signs </p><ul><li><p>suckling piglets: sudden death </p></li><li><p>growers and weaned piglets: nervous signs </p><ul><li><p>tremors, incoordination, dog-sitting, paddling, convulsions, coma and death </p></li></ul></li></ul></li><li><p><strong>finishing and adults; respiratory signs </strong></p></li><li><p><strong>sows: reproductive failure </strong></p></li><li><p>clinical symptoms: </p><ul><li><p>giving birth to <strong>weak piglets </strong></p></li><li><p><strong>foaming at the mouth </strong></p></li><li><p>severe neurological disorders </p></li><li><p>aborted fetus </p></li><li><p>hemorrhagic spot on the renal cortex </p></li><li><p>cerebral hemorrhage and congested meninges </p></li><li><p>pulmonary hemorrhage </p></li><li><p>liver with mulitple small focal areas of necrosis </p></li></ul></li></ul><p></p>
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Swine influenza

clinical symptoms:

  • fever, coughing, pneumonia spreading rapidly, rapid return to normality within 7-10 days

  • reproductive failure: return to estrus, abortions, reduced litter size, infertility in boars

Epidemiology:

  • endemic in most herds

  • intermittent bouts of respiratory disease and infertility

Diagnosis: virus isolation in nasal swabs, serology

prevention strategies: biosecurity measures, vaccination

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Porcine reproductive and respiratory syndrome (PPRS)

  • affects all ages

  • causes significant economic loss

  • clinical symptoms:

    • pneumonia in weaners:

      • mild coughing

      • sneezing

      • increased respiratory rates

      • death rate up to 15%

    • reproductive failure in sows

      • late term abortion

      • premature birth

      • prolonged anoestrus

      • agalactia

  • clinical signs and lesions

    • normal duroc crossbred pig

    • normal wild boar

    • red discoloration in ears of a domestic duroc crossbred pig infected with PPRS

    • normal lung

    • pulmonary hyperplasia and consolidation in wild boar infected with PPRS

    • normal heart

    • cardiac hemorrhages and edema in a wild boar infected with PPRS

<ul><li><p>affects <strong>all ages </strong></p></li><li><p>causes <strong>significant economic loss </strong></p></li><li><p>clinical symptoms: </p><ul><li><p>pneumonia in weaners: </p><ul><li><p>mild coughing </p></li><li><p>sneezing </p></li><li><p>increased respiratory rates </p></li><li><p>death rate up to 15% </p></li></ul></li><li><p>reproductive failure in sows </p><ul><li><p>late term abortion </p></li><li><p>premature birth </p></li><li><p>prolonged anoestrus </p></li><li><p>agalactia </p></li></ul></li></ul></li><li><p>clinical signs and lesions </p><ul><li><p>normal duroc crossbred pig </p></li><li><p>normal wild boar </p></li><li><p><strong>red discoloration in ears of a domestic duroc crossbred pig infected with PPRS </strong></p></li><li><p>normal lung </p></li><li><p><strong>pulmonary hyperplasia and consolidation in wild boar infected with PPRS</strong></p></li><li><p>normal heart </p></li><li><p><strong>cardiac hemorrhages and edema in a wild boar infected with PPRS </strong></p></li></ul></li></ul><p></p>
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Mycoplasma pneumonia (enzootic pneumonia) pigs

  • endemic in most herds

  • clinical presentation

    • acute pneumonia: fever, cough, respiratory distress, high mortality across all ages

    • chronic cases: prolonged cough and breathing difficulty (thumps)

  • lung lesions: consolidation of the anterior, cardiac, intermediate, and anterior diaphragmatic lung lobes

  • diagnosis methods

    • PCR (polymerase chain reaction) serology

  • treatment: medicated feed

  • prevention strategies

    • maintain an EP disease-free herd

    • implement biosecurity measures

    • vaccination

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Actinobacillus pleuropneumonia (APP)

  • affects pigs 8 to 16 weeks of age

    • respiratory distress

    • cyanosis of the ears

    • sudden death with hemorrhage from the nose (dxx bacillus anthracis)

  • diagnosis

    • bacterial culture from lesions

    • PCR

    • serology (ELISA)

  • treatment

    • antibiotic injections

    • medicated feed or water for the rest of the herd

  • prevention

    • biosecurity measures

    • vaccination

    • medicated feed

<ul><li><p>affects pigs 8 to 16 weeks of age </p><ul><li><p>respiratory distress </p></li><li><p><span style="color: red"><strong>cyanosis of the ears </strong></span></p></li><li><p><span style="color: red"><strong>sudden death with hemorrhage from the nose (dxx bacillus anthracis) </strong></span></p></li></ul></li><li><p>diagnosis </p><ul><li><p>bacterial culture from lesions </p></li><li><p>PCR </p></li><li><p>serology (ELISA) </p></li></ul></li><li><p>treatment </p><ul><li><p><strong>antibiotic injections </strong></p></li><li><p><strong>medicated feed or water for the rest of the herd </strong></p></li></ul></li><li><p>prevention </p><ul><li><p>biosecurity measures </p></li><li><p>vaccination </p></li><li><p>medicated feed </p></li></ul></li></ul><p></p>
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Greasy pig disease (Exudative epidermitis; Staphylococcus hyicus)

  • affects all ages

  • septicemia and toxemia cause death

  • dark patches of flaky, greasy skin lesions

  • diagnosis - bacterial culture

  • treatment - antibiotic injection, spray affected pigs several times with 10% bleach, chlorhexidine or dilute iodine

  • prevention - biosecurity, autogenous vaccine

<ul><li><p>affects <strong>all ages </strong></p></li><li><p>septicemia and <strong>toxemia cause death </strong></p></li><li><p><strong>dark patches of flaky, greasy skin lesions </strong></p></li><li><p>diagnosis - bacterial culture </p></li><li><p>treatment - antibiotic injection, spray affected pigs several times with 10% bleach, chlorhexidine or dilute iodine </p></li><li><p>prevention - biosecurity, <strong>autogenous vaccine </strong></p></li></ul><p></p>
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Glassers disease (Haemophilus parasuis)

  • anthrax-like disease with high mortality and sudden death in sows and suckling pigs

  • Young growing pigs - meningitis, middle ear infection, pleuropneumonia, pericarditis and peritonitis

  • diagnosis - bacterial culture

  • treatment - antibiotics injections, medicated feed or water for the rest of the herd

  • prevention - biosecurity, autogenous vaccine, medicated feed.

<ul><li><p><strong>anthrax-like disease </strong>with high mortality and sudden death in sows and suckling pigs </p></li><li><p>Young growing pigs - <strong>meningitis, middle ear infection, pleuropneumonia, pericarditis and peritonitis </strong></p></li><li><p>diagnosis - bacterial culture </p></li><li><p>treatment - antibiotics injections, medicated feed or water for the rest of the herd </p></li><li><p>prevention - biosecurity, autogenous vaccine, medicated feed. </p></li></ul><p></p>
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Colibacillosis (Enterotoxigenic E. coli)

  • severe watery diarrhea, dehydration in piglets 2-4 days of age with high mortality

  • older pigs are not affected

  • inflammation of the small intestine with watery, yellow, content

  • the stomachs of dead piglets is full of milk

  • diagnosis - bacterial culture from feces or rectal swabs

  • treatment - antibiotic injection, oral electrolytes, mediated feed for the herd

  • prevention - hygiene in maternity, all in / all out system, autogenous vaccines, commercial vaccines

<ul><li><p><strong>severe watery diarrhea, dehydration in piglets 2-4 days of age </strong>with high mortality </p></li><li><p>older pigs are not affected </p></li><li><p>inflammation of the small intestine with <strong>watery, yellow, content </strong></p></li><li><p>the stomachs of dead piglets is <strong>full of milk </strong></p></li><li><p>diagnosis - <strong>bacterial culture from feces </strong>or rectal swabs </p></li><li><p>treatment - antibiotic injection, oral electrolytes, mediated feed for the herd </p></li><li><p>prevention - hygiene in maternity, <strong>all in / all out system, </strong>autogenous vaccines, commercial vaccines </p></li></ul><p></p>
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Edema disease

  • caused by hemolytic E coli that produce Shiga toxin 2e (stx2e also known as verotoxin 2e or VT2e)

  • affects wearner pigs (days to few weeks after weaning)

  • associated with dietary changes overfeeding poor hygiene

  • swollen face edema of the eyelids, nose, and ears

  • CNS signs - dullness, blindness, head pressing, lateral recumbency, paddling leg movement, coma, death

  • diagnosis - detect stx2e in feces by ELISA, culture of E. coli that are positive for stx2e and f18 antigens

  • treatment - antibiotic oral or injection

  • prevention - improve hygiene, medicated feed, oral vaccination

<ul><li><p>caused by <strong>hemolytic E coli </strong>that produce<span style="color: red"> <strong>Shiga toxin 2e (stx2e also known as verotoxin 2e or VT2e) </strong></span></p></li><li><p>affects wearner pigs (days to few weeks after weaning) </p></li><li><p>associated with <strong>dietary changes overfeeding </strong>poor hygiene </p></li><li><p><span style="color: red"><strong>swollen face edema of the eyelids, nose, and ears </strong></span></p></li><li><p><strong>CNS signs</strong> - dullness, blindness, head pressing, lateral recumbency, paddling leg movement, coma, death </p></li><li><p>diagnosis - <strong>detect stx2e in feces by ELISA, </strong>culture of E. coli that are positive for <strong>stx2e and f18 antigens </strong></p></li><li><p>treatment - antibiotic oral or injection </p></li><li><p>prevention - improve hygiene, medicated feed, oral vaccination </p></li></ul><p></p>
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Enterotoxemia C. perfingens

  • bloody diarrhea and death in piglets hours to few days in age

  • hindquarters are soiled with bloody feces

  • pasty pink diarrhea

  • diagnosis - bacterial culture, demonstration of the beta toxin in the intestinal contents by ELISA

  • treatment - medicated feed, oral electrolytes, antitoxin

  • prevention - hygiene, medicated feed, vaccination

<ul><li><p><strong>bloody diarrhea </strong>and death in piglets hours to <strong>few days in age </strong></p></li><li><p>hindquarters are soiled with bloody feces </p></li><li><p><strong>pasty pink diarrhea </strong></p></li><li><p>diagnosis - bacterial culture, demonstration of the <strong>beta toxin </strong>in the intestinal contents by ELISA </p></li><li><p>treatment - medicated feed, oral electrolytes, <strong>antitoxin </strong></p></li><li><p>prevention - hygiene, medicated feed, vaccination </p></li></ul><p></p>
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Clostridium difficle enteritis (neonatal tyflocolitis)

  • yellow pasty to watery diarrhea in piglets less than 7 days of age

  • the colon is swollen with edema and the contents of the lage intestine is pasty to watery and yellow

  • diagnosis - toxin detection by PCR or ELISA

  • treatment - medicated feed, oral electrolytes antitoxin

  • prevention - hygiene, vaccination, medicated feed

<ul><li><p>yellow pasty to watery diarrhea in piglets <strong>less than 7 days of age </strong></p></li><li><p>the colon is swollen with edema and the contents of <strong>the lage intestine is pasty to watery and yellow </strong></p></li><li><p>diagnosis - toxin detection by PCR or ELISA </p></li><li><p>treatment - medicated feed, oral electrolytes antitoxin </p></li><li><p>prevention - hygiene, vaccination, medicated feed </p></li></ul><p></p>
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Salmonella choleraesuis

  • all ages are affected

  • necrotizing enterocolitis with rectal strictures

  • septicemia/endotoxemia, cyanosis

  • diagnosis - bacterial culture, serotyping by PCR

  • treatment - isolation of affected animals injectable antibiotics, oral electrolytes

  • prevention - biosecurity, medicated feed, vaccination

<ul><li><p>all ages are affected </p></li><li><p><strong>necrotizing enterocolitis with </strong><span style="color: red"><strong>rectal strictures </strong></span></p></li><li><p>septicemia/endotoxemia, <strong>cyanosis </strong></p></li><li><p>diagnosis - <strong>bacterial culture, </strong>serotyping by PCR </p></li><li><p>treatment - isolation of affected animals injectable antibiotics, oral electrolytes</p></li><li><p>prevention - biosecurity, medicated feed, vaccination </p></li></ul><p></p>
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Swine dysentery - brachyspira hyodysenteriae

  • colitis with watery, bloody diarrhea and death in growers and finishers

  • diagnosis - bacterial culture from feces, fecal smear, PCR on feces

  • treatment - antibiotics injection or oral

  • prevention - biosecurity, medicated feed for the herd, no vaccines

<ul><li><p><strong>colitis with watery, bloody diarrhea </strong>and death in growers and finishers </p></li><li><p>diagnosis - bacterial culture from feces, fecal smear, PCR on feces </p></li><li><p>treatment - antibiotics injection or oral </p></li><li><p>prevention - biosecurity, medicated feed for the herd, <strong>no vaccines </strong></p></li></ul><p></p>
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Ileitis

  • 4 forms

    • Porcine intestinal adenopathy (PIA)

    • regional ileitis (RI)

    • necrotic enteritis (NE)

    • proliferative hemorrhagic enteropathy (PHE; bloody gut) - Massive bleeding into the small intestine, pale pigs, black tarry feces, sudden death

  • lesions - hose-like thickening of the mucosa of the terminal ileum

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transmissible gastroenteritis (TGE)

  • coronavirus

  • diarrhea and vomiting across all age groups

  • severe watery diarrhea and more mortality in younger pigs

  • lesions - distension of the small intestine with foamy, yellow, odoriferous fluid and milk curds. the intestinal wall is very thin and transparent

  • improve immunity in pregnant sows and gilts by feeding them guts and gut contents of dead piglets

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Streptococcus porcinus (Jowl abscesses)

  • purulent lymphadenitis of head and neck in weaners and finishers

  • external drainage of purulent exudate

  • diagnosis - bacterial culture

  • treatment - antibiotic injections

  • prevention - biosecurity

<ul><li><p><strong>purulent lymphadenitis of head and neck in weaners and finishers </strong></p></li><li><p>external drainage of purulent exudate </p></li><li><p>diagnosis - bacterial culture </p></li><li><p>treatment - antibiotic injections </p></li><li><p>prevention - biosecurity </p></li></ul><p></p>
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Leptospirosis

  • sows and gilts - abortions, stillbirths infertility

  • weaners and growers - jaundice, acute death

  • tx - medicated feed

  • prevention - biosecurity, vaccinaiton

<ul><li><p>sows and gilts - abortions, stillbirths <strong>infertility </strong></p></li><li><p>weaners and growers - <strong>jaundice, </strong>acute death </p></li><li><p>tx - medicated feed </p></li><li><p>prevention - biosecurity, <strong>vaccinaiton </strong></p></li></ul><p></p>
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Brucellosis

  • females - infertility, late-term abortions, stillbirths, weak piglets, vulval discharges

  • boars - swollen testicles, infertility

  • treatment - none

  • prevention - biosecurity no vaccine for swine

<ul><li><p>females - <strong>infertility, </strong>late-term abortions, stillbirths, weak piglets, vulval discharges </p></li><li><p><strong>boars - swollen testicles, infertility </strong></p></li><li><p>treatment - none </p></li><li><p>prevention - biosecurity no vaccine for swine </p></li></ul><p></p>
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Iron toxicity

  • muscle necrosis and death within 24 hours after iron injection

  • acute lameness within 2 hours after iron injection

  • injection site become dark and swollen

  • heavy breathing, pale

  • lesions - muscles appear pale due to necrosis of the muscle fibers, myocardial infarction

<ul><li><p><strong>muscle necrosis and death within 24 hours after iron injection </strong></p></li><li><p><strong>acute lameness within 2 hours after iron injection </strong></p></li><li><p>injection site become dark and swollen </p></li><li><p>heavy breathing, pale </p></li><li><p><strong>lesions - muscles appear pale due to necrosis of the muscle fibers, myocardial infarction </strong></p></li></ul><p></p>
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Cryptorchidectomy

  • retained testicles (ridgelings, one-nutters)

  • important in show barrows due to mounting behavior

  • very helpful to know which side is retained if animal is unilaterally castrated

  • NEVER do a unilateral castration on a cryptochid and leave the undescended testicle

  • testicle usually located dorsally between inguinal canal and kidney

  • make 2-3 incision on a abdomen of pig on side of retained testicle

  • feel for testicle within abdomen and exteriorize

  • ligate spermatic cord

  • close abdomen

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Scrotal hernias

  • hereditary condition ~1% incidence in industry

  • often found at time of castration

  • ddx - hydrocele, scirrous cord, hematoma

  • treatment

    • taping method: 1 inch elasticon tape around legs in figure 8 pattern after castration (puts pressure on inguinal rings) remove tape in 4-5 days

  • surgical correction

    • needs to be performed prior to castration

<ul><li><p><strong>hereditary</strong> condition ~1% incidence in industry </p></li><li><p>often found at time of castration </p></li><li><p>ddx - hydrocele, <strong>scirrous cord, </strong>hematoma </p></li><li><p>treatment </p><ul><li><p><strong>taping method: 1 inch elasticon tape around legs in figure 8 pattern after castration (puts pressure on inguinal rings) remove tape in 4-5 days </strong></p></li></ul></li><li><p>surgical correction </p><ul><li><p>needs to be performed prior to <strong>castration </strong></p></li></ul></li></ul><p></p>
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umbilical hernias / infected navels

  • hernias often start with umbilical infection or abscess

  • not hereditary

  • only repaired in valuable animals

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Disease organisms get to other farms by one of three ways 90% of the time what are those 3 things

  • Vehicle

  • poultry equipment

  • people

  • airborne transmission is not really a thing

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What does every poultry place has no matter what

  • Coccidiosis

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<p>What are the 3 foot baths </p>

What are the 3 foot baths

  • 1st soapy water

  • 2nd disinfectant

  • 3rd soapy water

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The development of an infectious disease depends on three variables:

  • resistance of the bird

  • virulence of the disease organism

  • dosage of the organism

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what is the egg really for?

  • support the development of the chicken embryo

why is nutrient make-up ideal?

  • it must contain everything needed from fertilization until after chick hatches

why developed as a food for humans

  • cheap protein source

  • ideal protein make-up

  • tastes good

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where does fertilization of the egg occur

  • Infunidbulum

<ul><li><p>Infunidbulum </p></li></ul><p></p>
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with the light method when do you know if the egg is fertilized or not

  • if the yolk is high then not fertilized

  • if fertilized the yolk would be at the bottom by the albumen

<ul><li><p>if the yolk  is high then not fertilized </p></li><li><p>if fertilized the yolk would be at the bottom by the albumen </p></li></ul><p></p>
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What does it look like in the yolk if it was fertilized

  • appearance of a donut look in the embryo

  • appearance of tissue development

<ul><li><p>appearance of a donut look in the embryo </p></li><li><p>appearance of tissue development </p></li></ul><p></p>
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infectious bursal disease

  • an acute very contagious viral disease of young chickens characterized by destruction of immature lymphocytes in the BF and to a lesser extent in other lymphoid organs

  • recognized in all poultry-producing areas of the world

  • one of the most important diseases in concentrated broiler producing areas (often not recognized subclinical form)

  • resistant to disinfectants and environmental factors

  • can persist for months in contaminated houses

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transmission of infectious bursal disease

  • virus shed in feces

  • feed, water, litter become contaminated

  • infection by ingestion of virus

  • easily transmitted (mechanically) between farms

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subclinical form of infectious bursal disease

  • infection of susceptible chickens less than 3 weeks of age:

    • no clinical signs, but permanent and severe immunosuppression

    • more economically important form

    • majority of field infections are subclinical

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clinical form of infectious bursal disease

  • infection of susceptible chickens 3-6+ weeks of age:

    • sudden onset, rapid increase in mortality

    • clinical signs include ruffled feathers, diarrhea, vent pecking, dehydration, trembling, depression, transient immunosuppression

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infectious bursal disease lesions

  • initially, the BF is swollen 2 to 4 days after infection

  • appears inflamed, edematous and hyperemic, possibly hemorrhagic

  • at 5 days bursa is normal in size

  • after 5 days the BF diminishes in size rapidly (atrophies) grey in color

  • hemorrhages may be present in thigh and pectoral muscles

  • kidneys may be swollen

  • severe cases - all follicles affected simultaneously

  • less severe cases - scattered follicles affected spread to other follicles