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Digoxin
rapid onset, narrow margin of safety
Digoxin dosing
every 12 hours, at same time, if dose missed within 4 hours can readminister, do not readminister if child vomits
Digoxin considerations
low K enhances effects, hold for HR in infants 90, child 70, teen 60
Ace inhibitor
reduce afterload, BP and HR before giving, teratogenic
Ace inhibitor drugs
PRIL
Beta blockers
vasodilation, not for asthma, BP and HR before giving
Beta blockers drugs
OLOL
Cardiac catheterizations
can be diagnostic or interventional
Cardiac catherizations pre op
ht/wt, history, NPO, allergy, s/s infection (diaper rash!), assess and mark bilateral pulses, baseline VS
Cardiac catheterization post op
pulse checks below, if bleeding apply pressure above site and call for help, flat in bed for 4 to 8 hours
Cardiac catheterizations teaching
keep covered 2 to 3 days, no tub baths, no strenuous activity, remove dressing next day, diet as tolerated, monitor for infection
Congestive heart failure
tachypnea (early sign) , SOB, swelling in legs and feet
Congestive heart failure
heart cannot pimp an adequatre amount of O2 to body; poor perfusion leads to oxygenation issues
Rheumatic fever
inflammatory autoimmune disease, from prior group A beta hemolytic strep infection, strep antibodies (inflammation of connective tissue) heart valves
Rheumatic fever who
symptoms appear 2 to 6 weeks after untreated, not fully eradicated strep infection, upper resp infection
Rheumatic fever presentation
recent strep infection, JONES criteria, minor criteria (fever, ESR, arthralgia, long PR interval, CRP, positive strep titers)
CRP and ESR high
inflammation indicator
JONES criteria *R fever
Joints (polyarthritis) Carditis (bleeding lesions around heart) Nodules (subcutaneous) Erythema marginatum (rash) Sydnham chorea (uncoordinated movements)
Rheumatic fever nursing care
prophylaxis antibiotics for dental work, VS monitor, control joint pain, bedrest during febrile phase, seizure precautions
Tetralogy of fallot
decreased pilmpmay blood flow
Tetralogy of fallot defects
pulmonary stenosis, overriding aorta, VSD, right ventricular hypertrophy
Tet spell
hypoxemia and hypercyanotic
Hypoxia
cyanosis, polycythemia, clubbing, squatting, resp distress, metabolic acidosis
Hypercyanotic spell
sudden, brought on by stress (crying illness) emergency, flex knees and hips, call for help, keep calm
Kawasaki disease
mucocutaneous lymph node syndrome, acute systemic vasculitis of unknown cause
Kawasaki disease who
young children, most common acquired heart disease in children, cardiac issues are most serious complication, not contagious
Kawasaki disease acute
FEVER, conjunctival hyperemia, enlarged cervical lymph nodes, irritable, red throat, swollen hands, rash, fever, dry lips
Kawasaki disease subacute
Begins when fever ends; cracked lips, peeling of hands/feet thrombocytosis, joint pain, continued irritability, cardiac manifestations
Kawasaki disease convalescent phase
normal appearance from outside, until LABS normal, IRRITABLE, signs of inflammation, may go on for months, joint pain and irritability
Kawasaki disease nursing care
fever control, monitor for s/s of IVIG transfusion reactions, child may be hypersensitive to touch/light/other stimulation
Kawasaki disease aspirin
BENEFIT outweighs risk of reye's disease, give in high dose
Kawasaki disease meds
aspirin prescribed for antipyretic and antiplatelet effect, high dose until no fever, low dose until platelet counts returns to normal
Kawasaki disease parent education
necessary frequent ECHOs, NO contact sports, irritability can last 2 months, no live vaccine for year after IVIG administration, s/s bleeding and cardiac complications
4.5 to 13.5
WBC
12 to 16
Hemoglobin
34 to 50
Hematocrit
140 to 440
platelet
Low platelet
excessive bleeding, bruising, pinpoint (petechiae) rash, low pulse ox
Low hemoglobin and hematocrit
low energy, pale
Sickle cell crisis
illness, fever, emotional or physical stress
HBG A
normal
HGB S
abnormally sickled shape
Pain plan
similar to asthma action plan but for sickle cell
Hydroxyurea SC
increase flexibility of RBC
Folic acid SC
help with RBC production
Oral penicillin prophylaxis SC
2m to 5 yrs due to risk of death from sepsis
Stool softener SC
as needed, sometimes routine due to pain meds
Sickle cell
use heat packs, avoid cold packs
Sickle cell infant
measure and girth
Sickle cell nutrition
high fat, high calorie, HYDRATION
Hemophilia
bleeding disorder, platelets are not affected = coagulation protein deficiency
Hemophilia A
VIII (8) deficiency of coagulation protein
Hemophilia B
IX (9) deficeicny of coagulation protein
Hemophilia CM
bleeding episodes and lab findings of clotting times, identification of specific coagulation deficiency, abnormal bleeding response to trauma or surgery, bruising, abnormal bleeding response to trauma or surgery, frequent epistaxis
Hemophilia sign
bleeding cascade
Joint bleeding care
RICE (rest, ice, compress, elevate)
No ROM
DURING bleeds
Hemophilia
no contact sports but encourage activity
Hemophilia care
joint bleed care (RICE), no ROM during bleeds, replace clotting factor (prophalaxis), monitor for hematuria, prevent bleeds, at risk for intracranial hemmorrge
ADHD
inattention, overactivity, impulsive behavior
ADHD dx
early is imperative, made by self report, teacher observation and parent report (Must be in more than one setting)
ADHD test
vanderbilt screening
ADHD care
behavioral therapy and meds, limit setting, create routine and consistency, praise positive behavior, CONSISTENCY is key
ADHD meds
addreall and ritalin
ADHD med ADR
appetite suppression, nervousness, insomnia
Febrile seizure
unusual after age 5
Hydrocephalus
increased level of CSF within the ventricles of the brain causing increased ICP/head enlargement
Hydrocephalus types
obstructive (non communicating), communicating (impaired absorption, communicating but not the way it should)
Ventriculoperitoneal shunt
internal, straw to drain excess CSF into abdomen to be absorbed and excreted
Ventriculoperitoneal shunt complications
infection, can move around, frequent revisions as child grows
Ventriculoperitoneal shunt pre op
NPO, position for comfort, protect skin integrity, family support and education (educate on signs of increased ICP)
Ventriculoperitoneal shunt post op
VS, neuro checks, position flat on operative side, monitor s/s increased ICP, measure HC, comfort measures, strict I&O, antiemetics due to risk of vomit, bowel sounds present before PO feeds, risk of infection (antibiotics necessary), assess for leakage, peritonitis, malfunction/occlusion
Hydrocephelus infant
high shrill cry, irritability, lethargy, poor feeding
Hydrocephalus toddler
headache, lack of appetite, falling over/lack of coordination
Hydrocephalus older child
alteration in LOC
Meningitis
inflammation of meninges and CSF
Meningitis CM
acute onset, muscle pain, confused with flu, neck pain, increased ICP, toxic appearance, photosensitive
Meningitis dx
lumbar puncture
Meningitis IV antibiotics
priority intervention, start immediately (definitive diagnosis will take 72 hours) (rifampin, cetrifaxone, ciprofloxcin)
Meningitis antibiotics also for
direct contact with patient (family members, direct exposure to patients secretions = ate/slept/stayed in the home; flights over 8 hours)
(Meningitis) Brunzinski sign
life head when supine, positive if patient flexes knees
(Meningitis) kernig's sign
flex hip to 90, extend knee, positive if patient experiences back pain
Meningitis spinal fluid
decreased glucose, cloudy appearance, increased WBC, increased protein
Meningitis care
strict I&O, NPO, IV fluid, frequent vitals, droplet and seizure precautions, quiet calm enviorment, HOB slightly elevated
Seizure care
time of onset, aura/behavior before, monitor for apnea, cyanoisis, post pehavior, nothing in mouth, turn if risk for aspiration
Seizure long term care
medications (taise threshold of excitability, sleepy is a side effect, taper when discontinuing), vagal nerve stimulator, ketogenic diet, surgery
Reye syndrome goal
minimize cerebral perfusion and control increasing ICP
Reye syndrome
acute encephalitis, swelling in the liver and brain (found by liver biopsy)
Reye syndrome CM
fever, vomiting, dehydration, general malaise, progressive neuro deterioration, increased ammonia levels, history of viral illness in 4 to 7 days
Reye prevention
NO aspirin in kids
Reye syndrome treatment
rest, decreased stimulation, frequent VS, Q2 neuro checks, seizure, Vitamin K, IV glucose, anticonvulsants, diuretics, strict I&Os, miralax
Reye syndrome labs
elevated liver function, prolonged PT & PTT, hypoglycemia, elevated ammonia level tx, normal bilirubin
Depression
family history, traumatic event, sad expression, withdrawing from friends and fam, tearful, wt loss or gain
Depression meds
tricylic antidepressant, SSRI
Congenital clubfoot treatment
serial casting immediately after birth, new cast q 1 to 2 weeks or surgery if not corrected in 3 to 6 m
Congenital clubfoot
diagnosed at birth, forefoot pointed / midfoot turned out / hindfoot varus / ankle equinus
Congenital clubfoot goal
flat food for panless walking
Duchenne Muscular dystrophy
degeneration of muscle fibers (ALS in kids)
Duchenne Muscular dystrophy hallmark
persistent gowers maneuver (tripod and climb to stand) AFTER age 3
Duchenne Muscular dystrophy who
almost all males, X linked recessive trait, develops 3 to 5yrs