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Osteoarthritis
Slowly progressive deterioration of articular cartilage
Weight bearing joints (hip, knees)
where is OA typically seen?
previous accident
sports
other injury
known events/conditions that can be risk factors for OA [3]
inflammatory conditions that can impact joints
genetic conditions that can increase risk for OA;
pain
stiffness (in the ornign)
tender joints
deformity (late sign)
S+S of OA
If one side is effected, use of the other side increases to alleviate pain. Can lead to wear and tear of the other side and can progress to OA
How can OA become symmetrical over time?
would see narrowing of the joint space
What would a scan of OA joint look like?
look for pain, swelling, and muscoluskeletal assessment
ever had an injury?
Other co-morbidities (inflammatory
medications?
family hx
Health assessment questions to ask to determine OA diagnosis: [5]
increased WBC
increased ESR
increased c-reactive protein
lab results that can support OA diagnosis but cannot necessarily diagnose [3]
bone scan
CT
MRI
Xray
scans that can diagnose OA: [4]
improve/manage pain, optimize function, and prevent disability
goal for OA
heat/cold
activity that they can tolerate without pushing too far
lose weight
non-pharm lifestyle measures to treat OA:
massage
accupncutre
complementary and alternative therapies to treat OA: [2]
lowest dose possible to get maximal results
GOal for meds when managing OA:
can get worse over time/needs increase and can build a tolerance to meds over time
why want to give lowest dose possible to treat oa?
Know their normal to see if condiiton is improving/deteriorating
why is it important to get a baseline for OA assessments?
swimming
walking
light weight-bearing to strengthen joints
good exercises for people with OA:
fall hazards (rugs, bars, lightig, etc.)
proper footwear
home modifications for OA: [2]
Osteoporosis
bone disorder resulting in low bone density where the rate of boen reabsorption exceeds formation
spine
hips
wrist
Most common OP areas: [3]
primary OP
OP where there is no underlying cause, just something the person developed. Related to aging, poor bone development
seondary OP
OP caused by something else. Ex: long-term corticosteroids. When cause is removed can slow/stop the progression f the disease but not restore bone density
spontaneous fracture
normally the first sign of OP:
loss of height
back pain
restricted movement
fractures
pain on palpation
S+S of OP [5]
hip and spine
bones that give good representation of overall bone health: [2]
bone mineral density test (DEXA scan)
low-energy x-ray to evaluate bone density of hip and spine
need normal stress and strain on bones, the more the body gets used to repairing the bones
how can exercise treat OP?
Fosomax
medication often prescribed for OP
take on an empty stomach (absorption), with a full glass of water and sit up 30 mins after (reflux and esophageal damage)
things to educate patient on who is taking fosomax
reduction
Realigning fracture
Closed reduction
cast or manual reduction (no surgery)
Open reduction
Surgical reduction
Maintain alignment (immobilize)
priority for fractures:
cast
backslab
traction
how to maintain immobilization of fracture [3]
full weight baring
touch toe weight baring
partial weight baring
non-weight baring
levels of weight baring: [4]
infection
compartment syndroe
venous thromboembolism
fat embolism
elimination
complications associated with fractures: [5]
osteomyelitis
infection of the bone, occurs most often in open fractures
Can become systemic. Blood drawn from multiple sites to compare
Why are blood cultures taken for bone infection?
6 weeks around the clock IV antibiotics
treatment for osteomyelitis:
Surgical debridement
When the affected area is cut open and cleaned out. Whatever was causing the issue is removed.
keep site clean with sterile water/NS
How to prevent osteomyelitis:
Decrease in compartment size (tight cast/dressing)
Increase in compartment content (swelling, inflammaiton)
compartment syndrome is caused by two things:
intracompartmental pressure monitor
device that monitors pressure for compartment syndrome
fasciotomy
procedure done when intracompartmental pressure is too high. the fascia is opened to let eveything breath and get blood flow returned.
skin graft
long term antibiotics
vac dressing
wound left open
post op patient for fasciotomy gets:
redness
swelling
warmth
tightness in calf
signs of DVT [4]
early ambulation
anti-embolism stockings
anti-coagulants
monitor for S+S
how to prevent DVT [4]
low SPO2
SOB, winded
chest pain
coughing up blood
sign for pulmonary embolus [4]
anticoagulants
oxygen
treatment for pulmonary embolism:
fat embolism
when fat globule release from bone marrow into blood vessels and starts to cause problems
cutaneous petechiae: little bruises in upper chest area because it blocks capillaries
Sign that differentiates fat embolism:
dyspnea, increased RR, decreased SPO2
headache, confusion
tachycardia, chest pain
cutaneous petechiae
signs of fat embolism [4]
CT scan
how is fat embolism diagnosed?
until it is fixed/set properly to prevent fat embolism and keep the bone immobilized
how long are femur fracture patients on bedrest?
symptoms are treated and eventually body will absorb the fat
O2
fluids
pain/anti-anxiet meds
how is fat embolism treated?
reduces lung collapse and risk of pulmonary embolism
why is it important to have patients do lung exercises when in bed rest?
screen respiratory system for potential infection and make sure lungs are okay
why do patients get chest xray before surgery?
to catch any heart abnormalities and abnormal rhythms
why get preop ecg?
to catch infections, pregnancy, and check idney function
why get preop urines?
The person performing the operation
who gets consent before surgery?