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skin trauma
contusion, ecchymosis, petechiae, abrasion, laceration
contusion
skin discoloration caused by blow, trauma, or suction
usually bleeding in superficial tissue
ecchymosis
appears similar to bruise, caused by leaking of blood into subcutaneous tissue
caused by trauma causing rupture of the deeper capillaries
petechiae
tiny red or purple spots on skin, caused by bleeding under skin
can be caused by trauma or bacterial infections
abrasion
scraping away superficial layers of skin usually by friction/shear
laceration
deeper than abrasion, irregular tear of skin d/t trauma
dermatitis
eczema, allergic or contact dermatitis
eczema
aka atopic dermatitis, chronic inflammatory disease that causes itching, dryness, redness, and inflammation of skin
go through periods of flares and remission, begins in childhood and improves with age, not contagious
eczema cause
unknown, family member with eczema, hay fever, or asthma increases risk
eczema treatment
daily skin care: lukewarm bath, unscented soaps, pat skin dry, moisturizing creams, protect from skin irritants
target inflammation: eliminate triggers, manage stress, avoid scratching/rubbing, maintain level indoor temp, get enough sleep
meds: corticosteroid creams, immunosuppressants, antihistamines
phototherapy: UVA or UVB light waves
allergic or contact dermatitis
mast cell response from coming into contact with allergen or irritant
bacterial infections
impetigo, cellulitis, necrotizing fasciitis, hidradenitis suppurativa
impetigo
superficial bacterial infection of epidermis, common in children, highly contagious, usually in areas not covered by clothing
bacteria enters through break in skin (most common staphylococcus aureus and streptococcus pyogenes)
impetigo s/s
starts as red area of skin that develops into pus-filled vesicle/blister, ruptures and leaves honey-colored crust
impetigo treatment
antibiotics, keep area covered
cellulitis
bacterial infection involving deeper layers of skin, most commonly from staphylococcus aureus and streptococcus pyogenes
cellulitis presentation
redness, edema, warmth to touch, tenderness
most common LE, typically unilat
cellulitis treatment
antibiotics, limb elevation to decrease swelling
necrotizing fasciitis
bacterial infection that results in death of body’s skin and soft tissue layers
life threatening: high mortality, sudden onset and rapid spread, biggest factor is time from onset to debridement
necrotizing fasciitis risk factors
poor immune function, obesity, alcoholism, IV drug use, PAD
necrotizing fasciitis sx
severe pain, fever, purple-colored skin in area affected, usually occurs in areas of decreased blood flow (limbs and perineum)
necrotizing fasciitis treatment
debridement surgery to remove infected tissue to clear margins, antibiotics
hidradenitis supprativa
noncontagious, inflammatory condition characterized by pus filled bumps or boils and tunnels in and under skin, often in axilla or inguinal area starting in hair follicles
HS cause
unknown, 1/3 have family member with HS
females > males, afr am > white, smokers and obese individuals, appears age 20-40
HS treatment
address lifestyle factors: smoking, weight loss
wear loose clothing to avoid friction, keep areas clean, warm compress, antibiotics
viral disorders
herpes 1 and 2, herpes zoster
herpes 1 and 2
herpes 1: cold sore, spread by direct contact, treat with antivirals
herpes 2: STI, vesicular, can be fatal to newborns, no cure but treat with antivirals
can occur anywhere
herpes zoster
reactivation of varicella virus (lies dormant in cerebral ganglia or ganglia of posterior nerve root), biggest risk is age
contagious to those that have not had chickenpox, prevention with shingles vaccine for 50+
herpes zoster symptoms
burning, itching, painful that develops into a vesicular
follows dermatome, considered disseminated if > 20 vesicules outside primary dermatome
herpes zoster treatment
antivirals within 72 hrs onset
herpes zoster complications
pain can last months-years (post-herpetic neuralgia), can cause serious eye issues if spreads to eye or involves CN III
warts
common, caused by HPV, spread through direct contact
hands, fingers, soles of feet most common
treated with cryotherapy, salicylic acid, OTC treatment
ringworm
fungal infection of skin, can affect humans and animals, spread through direct contact
sx: itchiness, ring shaped areas
ringworm treatment
typically can treat with OTC antifungal creams
immune disorders
psoriasis, scleroderma, dermamyositis, pyoderma gangrenosum
psoriasis
chronic disease in which skin cells multiply too quickly resulting in red, scaly patches of skin from overactive immune system
psoriasis risk factors
family hx, more common adults
psoriasis s/s
thick red skin with silvery scales, often itches or burns, typically on elbows, knees, scalp, or trunk
periods of flares and remissions
psoriasis treatment
no cure, can lead to psoriatic arthritis
topical therapy (corticosteroid or coal tar), injected or oral steroids, methotrexate, phototherapy
scleroderma
autoimmune connective tissue that leads to fibrosis of skin and other parts of body resulting in thickened, hard skin
morphea
related to scleroderma, patches of firm, oval shapes that stay in one area
linear scleroderma
lines of thickened, different colored skin that extend down the arm or leg
types of scleroderma
localized: only involves skin and structures directly beneath
systemic: involves many systems in the body
scleroderma cause
unknown, female > male, ages 30-50, more severe afr am
scleroderma treatment
depends on form, typically medications (corticosteroids, analgesics, and immunosuppressants)
PT and OT to prevent contractures, exercises, joint protection
CREST syndrome
calcium deposits, Raynaud’s, esophageal dysfunction, sclerodactyly, teleangiectasis
associated with scleroderma
scleroderma precaution
as blood vessels become damaged can develop pulm arterial HTN leading to right HF
PT/OT need to closely monitor BP and watch for SOB
dermatomyositis/polymyositis
disorders of body’s connective tissue, causes weakness of arms and legs
unknown cause, thought to be immune reaction of body against own tissue, women > men, ages 5-15 or 40-60
dermatomyositis sx
skin rash, swelling around eye (may look bruised or purplish), swelling at base and sides of fingernails, splitting of skin on fingers
dermatomyositis treatment
corticosteroids, immunosuppressants
pyoderma gangrenosum
rare chronic inflammatory disease, poorly understood
unknown cause, autoimmune diseases found in 50% of cases, often see acute exacerbation in response to minor trauma
pyoderma gangrenosum
usually starts as painful nodule, progress unpredictably (slowly progress into larger and spreading wounds, explosive destruction of skin with rapid spread and pain, fever, hemorrhagic blisters, purulent drainage)
erythematous halo with dusky red purple border, painful, purulent exudate
most common sites for PG
LE, trunk - buttocks and abdomen
PG treatment
corticosteroids, conservative debridement, topical therapy to maintain moist wound bed
benign growths
seborrheia keratosis, actinic keratosis, moles
seborrheia keratosis
age spots
brown, black or tan growths
usually on face, neck, chest, and back
actinic keratosis
rough scaly patch on skin, usually caused by years of sun exposure, see after age 40
can lead to squamous cell carcinoma
moles
small dark spots caused by cluster of melanocytes
usually round or oval, < 1/4 “ in diameter, flat or slightly raised
most harmless but can become cancerous
skin cancers
basal cell, cutaneous squamous cell, melanoma, karposi’s sarcoma
most common type of CA in US, more common people with lighter skin but more deadly darker skin
skin cells grow and multiply in unorderly and disorganized fashion
skin CA risk factors
sun exposure, family hx, tanning bed use, use of drugs that increase sensitivity to light
cSCC: immunosuppressed, cigarette smoking
melanoma: increased moles, fair or light skinned
basal cell carcinoma
most common, begins in basal cells on sun-exposed areas, grows slowly so usually curable
shiny bump, open sores, red patch, areas that look like scars, or crusty area with depression in middle
squamous cell carcinoma
begins in squamous cells on sun-exposed areas
begins as firm pink/red nodule or scaly, crusted flat lesion, sometimes described as pimple that increases in size
melanoma
most serious, can metastasize, can develop anywhere
brown pigmented patch or bump, mole that changes in color, size, or bleeds
ABCDE
asymmetry, border, color, diameter, evolving
to identify melanoma
Kaposi’s sarcoma
cancer that forms on lining of blood and lymph vessels, in those that are immunocompromised (HIV increases risk)
purplish spots on legs, feet, or face
caused by human herpesvirus 8