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definitions of abnormality - failure to function adequately
can’t cope with demands of everyday life
Rosenhaan + Seligman = observer discomfort, unpredictability, irrationality, maladaptiveness
+ serves as threshold to identify mental illness
- not the whole picture (Comer)
- exceptions - e.g. Harold Shipman murdered patients, still able to hold down job
- cultural relativism
- unclear cause + effect
definitions of abnormality - deviation from ideal mental health
Jahoda proposed 6 criteria:
positive attitudes towards self
self-actualisation
resistance to stress
personal autonomy
accurate perception of reality
environmental mastery
+ comprehensive explanation, used as checklist so less subjective
+ real-world app. in diagnosis
- self-actualisation is difficult to achieve (2%)
- possible benefits of stress
- cultural relativity
definitions of abnormality - statistical infrequency
behaviour that deviates numerically from the majority- extreme ends of normal distribution curve
+ reliable + easy to measure due to statistical nature
+ used in diagnosis of intellectual disabilities
+ measurement is objective- diagnosis without prejudice
- historical validity - stats always changing
- cultural relativity
- some undesirable characteristics are common (e.g. depression) and vice versa
definitions for abormality - deviation from social norms
people who don’t conform to a set of moral codes in the society in which they live
implicit = unspoken conventions, e.g. queueing
explicit = enforceable laws, e.g. robbery = illegal
- historical context - norms change over time, e.g. homosexuality
- political influence - Cohen- Japanese striving for industrial success viewed those who didn’t comply as mentally ill, sent to ‘loony bins’
- context - some behaviours only viewed as abnormal in certain situations, e.g. day drinking
phobias- characteristics
emotional:
disproportional anxiety
panic
behavioural:
avoidance
freezing
fainting
cognitive:
recognition of irrational fear
resistance to rational arguments
phobias - behavioural explanation
two process model:
classical conditioning - initiation = traumatic exp. with stimulus leads it to be associated with fear, leads to phobia, e.g. Little Albert (Watson et al)
operant conditioning - maintenance = negative reinforcement when stimulus is avoided, fear is gone
+ research support - Seligman ‘biological preparedness’
+ success of treatment
- reductionist
- deterministic
phobias - behaviourist treatments
systematic desensitisation = relaxation techniques, hierarchy of fears
flooding = surround patient with phobia and no escape until fear subsides, unethical (duh)
+ focus on behaviour - avoids labelling and stigma
+ acknowledges cultural and individual diff
- ethical issues
- relevance, many tests conducted w animals
depression - characteristics
emotional (5 for > 2 weeks):
extreme sadness
lack of pleasure in activities
anger
behavioural:
reduced/increased activity and energy
agitation/restlessness
change in sleep patterns or eating behaviour
cognitive:
irrational NATs
negative triad
self-fulfilling prophecy
depression - cognitive explanation
Beck - cognitive vulnerability:
fundamental errors in logic
negative self schemas
negative triad - self, world, future
+ practical app. in CBT
- does not include all aspects
Ellis - rational thinking:
A = activating event, B = belief, C = consequence
musturbatory thinking
+ practical application in CBT
- not all depression is triggered by specific event
diathesis stress model = genetic predisposition + env. stressors - > high risk, lower if only one or none of these things
depression - cognitive treatments
CBT:
thought catching
identifying NATs
reality testing
behavioural techniques
hw tasks
cognitive restructuring
REBT (Ellis):
Activating event
Belief
Consequence
Disputing
Effective replacement
OCD - characteristics
emotional:
distress from obsessions/compulsions
awareness + embarrassment of behaviour
behavioural:
compulsions
compulsions:
obsessions
anxiety due to uncontrollable behaviour
OCD - biological explanations
genetics:
Miguel et al = 87% monozygotic vs 47% others
genetically predisposed to OCD
+ research support - Nestadt’s twin studies
+ objective research
- family studies could be explained by same environment (confounding)
candidate genes (COMT = dopamine, SERT = serotonin)
polygenetic, Taylor found 230 genes
neural:
rise in dopamine in prefrontal cortex, drop in serotonin in basal ganglia
evidence from brain scans
Rapport + Wise - hypersensitive basal ganglia
+ advances in technology = evidence
- basal ganglia - > compulsions, not obsessions
OCD - biological treatments
drug treatments - anti anxiety/antidepressants
SSRIs (e.g. fluoxetine):
target serotonin by blocking serotonin transporters, raise serotonin levels
trycyclics (e.g. onafranil):
targets noradrenaline by blocking reabsorption
side effects, e.g. heart seizures, impotence
anti-anxiety drugs (e.g. diazepine):
targets GABA to slow activity of CNS
addictive properties, last resort
+ cheap + less time-consuming
+ advanced technology- objective, scientific
- can’t cure OCD but lessens symptoms
- ethical issues of side effects