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what can cause the shoulder to need replacement?
osteoarthritis
rheumatoid arthritis
humeral fractures
types of medical management
hemiarthoplasty
total shoulder replacement (TSR)
reverse total shoulder arthroplasty (RTSA)
hemiarthoplasty
replaces femoral head
total shoulder replacement (TSR)
The procedure involves replacing the head of the humerus (the "ball") and the glenoid (the "socket") with metal and plastic parts
reverse total shoulder arthroplasty (RTSA)
- reverses the normal anatomic relationship between the humerus head and the glenoid socket
- A ball is placed onto the socket & a cup or socket is placed on top of the humerus in place of the ball
postoperative considerations
1) soft tissue healing and proaction of the glenohumeral alignment are critical
2) pain, edema, and inflammation managed with physician-directed modalities
3) use of shoulder in sling or swathe for 3-4 weeks, weight bearing restricted 6-8 weeks
shoulder replacement PRECAUTIONS
-NO WEIGHT BEARING with surgical UE
-no lifting > 1-2 lbs
- avoid abduction past 45 degrees
- avoid ER past 30-60 degrees
-avoid IR past 60 degrees
- limit passive flexion 90-100
(not active just passive)
t/f OT can begin day 1 post if medically stable
true
OT goals
-restore safe occupational performance
- train ADLs/IADLs with adaptive strategies
- EDUCATE on PRECAUTIONS
-progressive UE functional mobility and strength
eval and intervention
1) asses motor, cognitive, and emotional factors
2) focus on ADLs, adaptive equipment, and compensatory methods (ex, dressing stick, one handed techniques)
3) progress AROM, strength, and activity tolerance
phase 1 (0-4 weeks) PP PHASE
-PROM only, pendulum exercise
goals: protect repair, manage pain and edema, and prevent stiffness.
(PROM ONLY within surgeon defined limits
Movements are restricted to the following ranges: shoulder flexion up to 90-100°, abduction up to 45°, and ER up to 30°. IR is limited to placing the hand on the abdomen only.
Once cleared by the surgeon, do Codman's pendulum exercises (gentle circular or lateral motions)
encourage (AROM) of distal joints (elbow, wrist, and hand) to support circulation and prevent edema.
During rest, the shoulder should be maintained in a neutral alignment
phase 2 (4-6 weeks) AAROM, light isometrics
Active-assisted range of motion (AAROM) and light isometric exercises
introduce AAROM - AROM transition
( table slides, dowel exercises, and incline board pushing)
flexion tolerance may increase to 70° and ER to 60°.
Begin active-assisted exercise: (Overhead pulleys, wall slides, and supine dowel movements)
Light isometric exercises, such as scapular retraction, can begin as tolerated;
IMPORTANT: lifting and weight-bearing activities remain restricted at this stage.
phase 3 (6-12 weeks) STRENGTHEN
Gradual strengthening, closed-chain activities.
- Progress to light strengthening:
- scapular stabilization (elevation, depression, retraction).
- shoulder adduction, flexion, and rotation with light weights.
-Closed-chain activities (e.g., wall push-ups, prone-on-elbows).
-Restore functional motion through all planes.
phase 4 (12+ weeks)
Full AROM, return to occupations
reverse total shoulder replacement protocol
Used for rotator cuff-deficient
shoulders.
PROM limited to 45°
flexion/abduction, neutral external
rotation.
Begin pendulum exercises ~day 1.
AAROM starts around 7-10 days if
stable.
Full AROM expected by 12 weeks if
healing progresses.
RTSR PHASE 1 (0-2 WKS)
PROM (Flex less than or equal to 90, abd less than or equal to 45)
codeman's pendulums only with surgeon approval
AVOID: ADD+IR
RTSR PHASE 2 (2-6 wks)
Gentle AAROM; light deltoid/scapular isometrics;
ER less than or equal to 30°
RTSR PHASE 3 (6-12 wks)
Controlled AROM, light resistance (deltoid focus);
avoid resisted IR or extension
what is rotator cuff tears
reversible inflammation and microtearing (tendinitis/tendinosis) to partial or full-thickness disruptions of the tendon
Supraspinatus
initiates abduction
infraspinatous
external rotation
teres minor
assists with ER and humeral head stabilization
subscapularis
internal rotation an anterior stability
rotator cuff tears epidemiology
- most frequent cause of shoulder pain in adults
-40-70 yrs
-dominant shoulder affected > 60% of cases
- degenerative tears> traumatic tears in individuals >50
injury spectrum rotator cuff tear
intrinsic degeneration
extrinsic impingement
overuse and repetitive overhead activity
acute trauma
intrinsic degeneration
age related collagen disorganization, reduced vascular supply
extrinsic impingement
mechanical compression beneath the coraco-acromial arch or acromial spurs
overuse and repetitive overhead activity
micro trauma exceeding repair capacity
acute trauma
falls on outstretch hand or lifting injury in younger patients
classification types
tendinopathy
partial thickness tear
full thickness tear
massive tear
tendinopathy
degenerative changes without discontinuity of fibers
-pain with overhead use; strength preserved
partial thickness tear
incomplete disruption of tendon fibers on articular or bursal side
(1-3 cm)
-pain> weakness
night pain common
plain will be greater than weakness
full thickness tear
complete discontinuity from tuberosity insertion
weakness and drop arm sign
crepitus
(3-5)
massive tear
>5 cm or 2 tendons involved
-loss of active elevation
pseudo paralysis (because not necessarily of a nerve it is just because it is so weak)
rotator cuff tear clinical presentation
dull aching pain in lateral (supraspinatous tension is there) shoulder
night pain when lying on affected side (compression in adduction)
weakness with abduction/externla rotation
difficulty with ADLs (grooming, reaching overhead)
neers/hawkins-kennedy test
purpose: impingement screening
positive finding: pain elicited near end range flex/IR
Drop arm test
purpose: full thickness tear
positive finding: inability to control slow lowering
empty can (Jobe)
purpose: supraspinatus weakness
positive finding: pain/weakness in scaption
lift-off test
purpose: subs cap tear
positive finding: inability to lift hand from back
RTC rehab primary goals
-protect surgical repairs
- prevent selective capsular hypo mobility
- gradually restore neuromuscular control and function
RTC evidenced based framework
-early passive motion minimizes adhesions (avoid aggressive stretching)
-progression based on TISSUE healing, not a timeline
-frequesnt reassessment for signs of overload (night, pain, inflammation)
RTC phase 1 (0-6 weeks) PROTECTIVE/PASSIVE MOTION PHASE
-PP PHASE
-immobilization: spring 4-6 weeks with intermittent pendulums and PROM in pain free range
- emphasize postural correction and scapular setting
-PROM: begin flexion/scaption - progress to 90/90 ER by 4 wks
-AROM: NONE!! avoid active contraction of repaired tendon
-mods: cryotherapy 3xday/ IRF (currents) PRN (as needed) for pain
-precautions: no behind the back or unsupported bicep activity
-TE: Pendulums, wrist/hand mobility, sub-max pain-
free isometrics (Flex, Abd, IR, ER at 4-6 weeks).
RTC phase 2 (6-12 weeks) ACTIVE
Active-assisted to active motion
-initiate AAROM-AROM with emphasis on scapular mechanism
- begin isometric and light isotonic as tolerated
- avoid compensatory upper trap dominance
RTC phase 3 (12 + weeks)
- strength and function restoration
-progressive resistance exercises for RTC (progress dumbbells, theraband) and scapular stabilization
-integrate CKC (WEIGHT BEARING) and task specific training
-pain free ROM and > 4/5 strength
RTC phase 3 (12 + weeks) TE focus:
Scapulothoracic + GH exercise
• Isotonic IR/ER in scaption.
• Sidelying ER, rhythmic stabilization, OKC/CKC progression.
• Emphasize lower trap and serratus anterior activation.
• Perturbation training, 8 weeks unsupported biceps, lower trap exercises,
unsupported triceps
rtc prognosis
-small tears respond well to conservative management
-full-thickness repairs regain greater than or equal to 80-90% ROM and strength with compliance
-POOR outcomes associated with non-compliance, re-tear or persistent pain syndrome
-
adhesive capsulitis
it is characterized by progressive pain and loss of A/PROM due to capsular fibrosis and thickening
Primary (idiopathic) adhesive capsulitis
no known precipitating event, spontaneous onset of pain and stiffness
common in Middle Aged women without trauma
secondary adhesive capsulitis
occurs after shoulder injury or surgery associated with systemic or neurologic condition
-post RTC repair, fracture, stroke, or prolonged immobilization.
Clinical presentation of adhesive capsulitis (symptoms)
- gradual, insidious onset, night pain, stiffness
-pain localized anteromedially or into long head of biceps
Clinical presentation of adhesive capsulitis (AROM/PROM)
-Restriction (capsular pattern, ER/ABD/IR/FLEXION)
-substitution with scapular elevation
Clinical presentation of adhesive capsulitis (All limitation )
reaching overhead, grooming, dressing, fastening bra, throwing activities
what is KEY symptom in adhesive capsulitis
equal loss of active and passive ROM in a capsular pattern
Assessment: AROM/PROM comparison
purpose: determine global restriction
positive finding: similar limitation in both
Assessment: ER at 0 and 90 ABD
purpose: identify capsular pattern
positive finding: markedly limited ER
Assessment: Scapulohumeral rhythm observation
purpose: assess compensation
positive finding: excessive scapular elevation
Stages freezing (painful) stage
time frame: 10-36 weeks
process: synovial inflammation, capsular distension
clinically: severe pain, night pain, progressive loss of ROM
frozen (adhesive) stage
time frame: 4-12 months
process: fibroplasia, thickened capsule (contracture of coracohumeral ligament)
clinically: marked stiffness, pain subsides
Thawing (Recovery) Stage
time frame: 5-24 months
process: remodeling of collagen fibers and gradual restoration of motion
clinically: minimal pain, slow ROM return
therapeutic management for the freezing stage
goals: reduce pain/inflammation
MAKE AVAILABLE ROM without provoking symptoms
you can use ultrasounds, cryotherapy, IFC. then you can do grade 1 and 2 joint mobs, oscillations. for exercises you can do pendulum, table slides, wand ER to tolerance
educate on sleeping position and activity mods.
therapeutic management for the frozen stage
goals: increase capsular extensibility
restore functional ROM gradually
you can do grade 3-4 joint inferior and posterior mobs. to stretch you can do wand flexion, ER/IR at 90 degrees, and, posterior capsular stretch (slow prolonged holds for these) (moist heat pre stretch)
for strengthening you can do Isometrics, light isotonic for RTC and scapular stabilizers (cryotherapy post)
therapeutic management for thawing stage (resolution)
goals: regain full ROM and restore strength and endurance
reinstate functional movement patterns and participation
grade 5 joint mobs
advanced therEx: PNF DS patterns, wall slides, theraband series (emphasize eccentric control)
Functional retraining: reaching tasks, dressing simulation, work conditions.
management approach for adhesive capsulitis (NSAIDs/Corticosteroids)
pain dominate phase (short term pain relief combined with therapy)
management approach for adhesive capsulitis (manipulation under anesthesia MUA)
severe ROM loss > 6 mo
with this there is rapid ROM gain but risk capsular tear/fracture
management approach for adhesive capsulitis (arthroscopic capsular release)
failed conservative tx > 9 mo
high success when followed by intensive rehab
Type 1 (Scapular dysfunction kibler classifciatdon)
Interior angle dysfunction
Type 2 (Scapular dysfunction kibler classifciatdon)
Medial border dysfunction
Type 3 (Scapular dysfunction kibler classifciatdon)
Superior dysfunction
Type 4 (Scapular dysfunction kibler classifciatdon)
normal
type 1 Scapular dyskinesia info
-scapula inferior angel tilts posteriorly in sagittal plane
- increased in the hands on hips position or during eccentric lowering/control
TREATMENT: strengthen lower traps, stretch pec minor
type 2 Scapular dyskinesia info
-entire medial border winging off thoracic walls
- increased in the hands on hips position or during eccentric lowering/control
TREATMENT: strengthen the serratus anterior
type 3 scapular dyskinesia info
-excessive elevation of the superior border of the scapula with arm elevation
TREATMENT: inhibit UPPER TRAPS, STRENGTHEN LOWER TRAPS.
TREAT CAUSE: treat GH hypo mobility
instability
-hypermobility is NOT instability
- instability causes SYMPTOMS
-laxity is not the same as instability
-WE DONT TREAT LAXITY, we treat instability
clinical instability
Patient's inability to keep the humeral head centered in the glenoid fossa
functional instability
can be due to capsular/ligamentous instability
Generalized Ligamentous Laxity
Test
Beighton Scale
1. Hyperflexion of CMC and MCP Joint (Thumb
to Volar Wrist)
2. Hyperextension of 2nd MCP joint
3. Hyperextension of Elbows
4. Hyperextension of Knees
RTC impingement
Age: >40
Mechanism of Injury (MOI): Repetitive overhead activities, overuse of arm in unaccustomed position
Pain- night
Pain- lateral shoulder
Pain- Palpation
Pain- overhead activities
Painful arc (0-45 degrees is painless, 45-120 degrees IMPINGEMENT SYNDROME)
note* 120-170 painless, 170-180 (acromio-clavicular joint arthritis)
types of impingement syndromes
-primary (hypo mobile)
-secondary (hypermobile)
-internal
subacromial space
space between the humeral head and coracoacromial arch
structures at risks- supraspinatus, long head of bicep, subacromial bursa
T/F upward rotation/posterior tilt protect the subacromial space
true
Primary (hypo mobile)
bursal side (superior surface/top)
typically older than 40 years old
-STRUCURAL NARROWING ( acromion type, osteophytes
TREAT cause: mobilize joint, restore posture, progressive exercise
The rotator cuff tendons (especially the supraspinatus) are mechanically compressed under the acromion, coracoacromial ligament, or acromioclavicular (AC) joint during arm elevation.
Usually due to bony changes or degenerative anatomy (degenerative changes).
Contributing factors:
Acromial shape (hooked or curved acromion)
Bone spurs
Thickened coracoacromial ligament
AC joint osteophytes
secondary (hypermobile)
articular side (age 15-40)
INSTABILITY and LAXITY
DO NOT stretch, focus on DYNAMIC stabilization and neuromuscular control
Description:
Occurs due to glenohumeral joint instability (excessive humeral head translation) that reduces the subacromial space during motion.
The issue is not bony but due to muscle imbalance, weakness, or poor motor control of the rotator cuff and scapular stabilizers
Common in: Younger, more active individuals or athletes.
Contributing factors:
Scapular dyskinesis
Weak rotator cuff or scapular stabilizers
Capsular laxity
Poor posture or repetitive overhead activities
internal impingement
articular side, posterior cuff contact with glenoid rim
Cause: Posterior impingement of rotator cuff tendons inside the joint.
Description:
The undersurface (articular side) of the supraspinatus and infraspinatus tendons are pinched between the humeral head and the posterior-superior glenoid rim during extreme abduction and external rotation (e.g., throwing motion).
Common in: Overhead athletes (e.g., baseball pitchers, swimmers).
type 1 acromion shape
flat- 91% success rate
type 2 acromion shape
smooth curve- 66% success rate
type 3 acromion shape
anterior hook- 50% success rate (posible surgical cases)
NEER classification (rotator cuff impingement stage) stage 1
edema and hemorrhage (good)
age less than 25
REVERSIBLE
pain with overhead activity
inflammation of bursa
NEER classification (rotator cuff impingement stage) stage 2
fibrosis and tendonitis (bad)
age 25-40
thickened bursa/tendon
recurrent pain
night pain
needs meds
inflammation of tendon
NEER classification (rotator cuff impingement stage) stage 3
bone spurs and ruptures (ugly)
greater than 40 years old
-not all full thickness require repair
rupture
t/f Subacromial pressure increase with IR, decrease with ER
true
Internal rotation (IR) rolls the greater tuberosity of the humerus upward and closer to the acromion.→ This narrows the subacromial space and increases pressure, compressing the supraspinatus tendon and bursa.
External rotation (ER) moves the greater tuberosity away from the acromion.→ This widens the subacromial space and reduces pressure, relieving impingement.
strengthening ER at __ degrees reduces pressure
0
SAIS clinical presentation
Painful arc: 60°-120° abduction
• Lateral shoulder pain; night pain
• Pain on palpation & overhead activity
• Weakness due to pain inhibition
• Functional impact: reaching, dressing, lifting, typing
• dentify if hypomobile (stiff/painful) or hypermobile (unstable/fatigued)
differential diagnosis
RTC tear - Weakness > pain
Biceps tendinopathy - Pain with
resisted supination
Adhesive capsulitis - Global
restriction
SAIS intervention phase 1
Goals: Decrease pain, maintain mobility
Modalities: Ice, ultrasound, kinesiotaping
Pendulum, gentle PROM
Postural correction & ergonomics
Avoid overhead/provocative tasks
Primary: gentle mobility
Secondary: stabilization only
SAIS intervention phase 2
Goals: Restore dynamic stability & strength
• Scapular stabilization: wall slides, serratus punches, prone retraction
• RTC strengthening: ER/IR with bands, sidelying ER
• Posterior capsule stretching
• Add rhythmic stabilization, proprioceptive drills
Isometric dynamic endurance progression
SAIS intervention phase 3
Goals: Reinforce coordinated functional movement
Combine core + scapular activation (planks, quadruped reaches)
Simulate occupational tasks (reaching, typing, lifting)
Ergonomic & joint protection education
Gradual overhead retraining
What is the purpose of the glenoid labrum?
it is a ring of tough fibrous cartilage that surrounds the glenoid fossa. it deepens it to create a more secure fit
and for stability
the biceps long head anchors at the?
superior labrum
Wha can detach the labrum
excessive traction or compression
A healthy labrum
supports overhead stability