Lab Exam-2

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Last updated 12:56 AM on 3/28/24
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289 Terms

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<p>1</p>

1

sinoatrial node

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<p>2</p>

2

atrioventricular node

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<p>3</p>

3

atrioventricular bundle

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<p>4</p>

4

bundle branches

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<p>5</p>

5

Purkinje fibers

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function of SA node

generates action potentials

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function of AV node

impulses pause before moving to AV bundle

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function of AV bundle

electrical connection between left and right ventricles

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function of bundle branches

conducts impulses to the ventricles

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function of purkinje fibers

depolarizes the contractile cells of the ventricle

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p wave represents

atrial depolarization

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QRS complex represents

ventricular depolarization

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T wave represents

ventricular repolarization

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U wave

small wave associated with repolarization

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P-R segment represents

Time interval from end of atrial depolarization to beginning of ventricular depolarization

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S-T segment represents

Time interval from end of ventricular depolarization to beginning of ventricular repolarization

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P-R interval represents

Time interval from beginning of atrial depolarization to beginning of ventricular depolarization

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Q-T interval represents

From beginning of ventricular depolarization to end of ventricular repolarization

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on EKG: a big square is ___ seconds

0.2

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on EKG: a small square is ___ seconds

0.04

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normal P wave should be no more than ___ to ___ seconds in duration

0.08 to 0.10 seconds

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in adults, the normal QRS duration is ___ seconds or less

0.12

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T wave duration in seconds

0.1- 0.25

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PR segment should be no longer than ___ second

0.1

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<p>red (middle)</p>

red (middle)

QRS complex

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<p>pink/purple</p>

pink/purple

ST segment

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<p>green</p>

green

PR segment

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<p>blue</p>

blue

QT interval

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<p>orange</p>

orange

PR interval

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Heart rate from EKG formula

# of small boxes * 0.04/ 60

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impact of exercise on EKG

all intervals shorten; increased conduction and increased heart rate shorten duration of intervals

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Location of sound: aortic valve

second intercostal space, right sternal border

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Timing of sound: aortic valve

heard during S2

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Location of sound: tricuspid valve

fifth intercostal space, left sternal border

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Timing of sound: tricuspid valve

heard during S1

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Location of sound: mitral valve

fifth intercostal space, mid-clavicular line

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Timing of sound: mitral valve

heart during S1

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Location of sound: pulmonary valve

second intercostal space, left sternal border

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Timing of sound: pulmonary valve

heard during S2

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S1 makes ___ sound

lub

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S2 makes ___ sound

dub

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The parasympathetic nervous system releases _______ to affect heart rate.

acetylcholine

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A cholinergic drug that worked the same as acetylcholine would _______.

be an agonist and decrease heart rate.

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Norepinephrine affects the heart rate by _________.

increasing the rate of depolarization and increasing the frequency of action potentials.

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The __________ receptor binds norepinephrine and epinephrine.

B-1 adrenergic

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Pilocarpine is a cholinergic drug, an acetylcholine agonist. Predict the effect that pilocarpine will have on heart rate.

decrease HR

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Atropine is another cholinergic drug, an acetylcholine antagonist. Predict the effect that atropine will have on heart rate.

increase HR

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What increases the heart rate and mimics the sympathetic nervous system?

epinephrine

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Individuals with weakened hearts need to allow maximum time for venous return and increased stroke volume and would therefore most likely benefit from ______.

increased force of contraction and decreased heart rate

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Pilocarpine decreased the heart rate. Typical of cholinergic agonists, it _________.

decreased the frequency of action potentials.

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The effect of atropine was to ________.

mimic the sympathetic nervous system.

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The modifiers tested that decrease the heart rate were ______.

digitalis and pilocarpine.

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To increase the heart rate, the best choices would be ______

epinephrine and atropine.

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normal BP

120/80

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instruments used to get BP

stethoscope, Sphygmomanometer

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causes for increased BP

increased resistance, vasoconstriction, increased viscosity

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T/F: Longer vessels have higher peripheral resistance than shorter vessels.

true

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causes for decreased BP

decreased resistance, vasodilation, decreased viscosity

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Cardiac output formula

CO= SV x HR

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systolic pressure

the maximum pressure exerted on the wall of an artery (during ventricular contraction)

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diastolic pressure

The minimum pressure exerted on the wall of an artery (during ventricular relaxation)

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<p>e</p>

e

aortic arch

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<p>g</p>

g

thoracic aorta

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<p>h</p>

h

abdominal aorta

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<p>c</p>

c

right common carotid

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<p>p</p>

p

left common carotid

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<p>o</p>

o

brachiocephalic trunk

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<p>q</p>

q

left subclavian artery

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<p>r</p>

r

left axillary artery

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<p>s</p>

s

left brachial artery

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<p>u</p>

u

left ulnar artery

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<p>t</p>

t

left radial artery

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<p>m</p>

m

right common iliac artery

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<p>n</p>

n

right internal iliac artery

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<p>w</p>

w

left femoral artery

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<p>x</p>

x

left popliteal artery

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<p>y</p>

y

left anterior tibial artery

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<p>z</p>

z

left posterior tibial artery

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<p>i</p>

i

celiac trunk

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<p>J</p>

J

superior mesenteric artery

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<p>l</p>

l

inferior mesenteric artery

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<p>u</p>

u

superior vena cava

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<p>ee</p>

ee

inferior mesenteric vein

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<p>ff</p>

ff

superior mesenteric vein

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<p>s</p>

s

left brachiocephalic vein

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<p>t</p>

t

left subclavian vein

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<p>e</p>

e

right axillary vein

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<p>d</p>

d

right internal jugular vein

89
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<p>gg</p>

gg

left common iliac vein

90
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<p>hh</p>

hh

left internal iliac vein

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<p>m</p>

m

right femoral vein

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<p>n</p>

n

right great saphenous vein

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<p>r</p>

r

right small saphenous vein

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<p>2</p>

2

abdominal aorta

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<p>4</p>

4

left common iliac artery

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<p>6</p>

6

right internal iliac artery

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<p>1</p>

1

right common carotid artery

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<p>2</p>

2

right subclavian artery

99
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<p>3</p>

3

right axillary artery

100
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<p>4</p>

4

right brachial artery