ATH501 Exam 1

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Therapeutic Modalities-Pain

Last updated 7:45 PM on 9/16/23
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216 Terms

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True or False: All modalities speed up the healing process

False: few to no modalities speed the healing process

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True or False: The body heals at its own pace
True
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modality

form of stress applied to body to elicit an involuntary physiological response

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applying different modalities at the __ time may _ recovery
wrong; hinder
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Stages of General Adaptation Syndrome
Alarm, Resistance, Exhaustion
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Alarm Stage
initial reaction to a change in homeostasis
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Resistance Stage
body adapts to stressor, using homeostatic resources to maintain tissue integrity
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Exhaustion Phase
occurs when body cannot handle stress; overuse injury
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Which stage of the General Adaptation Syndrome is the longest?
Resistance Stage
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Macrotrauma
harmful stress that causes injury
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Microtrauma
repeated, low intensity forces that cause stress fracture, chronic inflammation, and muscle soreness
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Magnitude of the modality being too great can…
further injury may occur
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Modalities being too low intensity or too short yields…
no benefits to the patient
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What tissues must energy pass through to reach target tissues?
epidermis, dermis, and adipose tissues
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Ischemia
lack of oxygen to a tissue
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Primary Injury
tissue destruction resulting directly from traumatic forces
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Secondary Injury
cell death by lack of oxygen, enzymatic damage, and mitochondrial damage
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Phases of Healing
acute inflammatory phase, proliferation phase, maturation
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Acute Inflammatory Phase
supply building phase; migration of phagocytes and fibroblasts, formation of granulation tissue, histamine released from damaged cells
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Purpose of Granulation Tissue in Acute Inflammatory Phase
isolates and localizes trauma from healthy cells
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Effects of Histamine Release
inc capillary permeability, swelling inc as proteins and water flow out into the tissues
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Proliferation Phase
inc number of fibroblasts, collagen and ground substance pool in trauma area to prep for rebuilding
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Components of Rebuilding Damaged Tissues
revascularization, wound contraction, wound remodeling
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Maturation Phase
number of fibroblasts, myofibroblasts, and macrophages return to preinjury level; reduction in capillary vascularity and water content; type 1 collagen replaces type 3 in collagen lattice
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Secondary hypoxic injury
causes cell death due to dec oxygen supply to area
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Decrease of Oxygen Supply can be caused by….
hemorrhaging, reduced blood flow, hematoma pressure, muscle spasm, hydropic swelling of damaged cells
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Secondary injury is first visible in what organelle
mitochondria
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How quickly can secondary injury be seen in the mitochondria?
within 30 min of primary trauma
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Swelling
inc volume in a body part or area; fluid buildup
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Why does capillary permeability increase after an injury?
to ease movement of fluids and matter out of the area
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Edema
build up of excess fluids and protein in interstitial joint space resulting from pressure imbalance around the cell membrane
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edema can obstruct __ and _ return
lymphatic; venous
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How can you reduce edema formation?
apply ice
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How is edema removed?

inc venous and lymphatic return, gravity, circulation, and compression

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Starlings Law
fluids move from areas of high concentration to low concentration
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Muscle Spasm
involuntary contraction of muscle fibers, stimulating mechanical and chemical pain receptors
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Causes of Muscle Spasm
direct trauma, dec oxygen, neurological dysfunction
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Chronic Inflammation
caused by low intensity irritants; can be developed w/o acute trauma
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Chronic Inflammation is a strong predictor of …
future disability
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What are the consequences of injury?
secondary injury, swelling, edema, muscle swelling, muscle atrophy, chronic inflammation
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Primary Pain Control
remove/reduce mechanical or chemical stimulation that triggers the nociceptors; encourages healing
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Secondary Pain Control
interrupts the transmission or interpretation of the noxious impulse; used for comfort between treatments
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Types of Therapeutic Modalities
thermal, electrical, mechanical, ultrasound, exercise, short wave diathermy
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Types of Medications
analgesics, anti inflammatories, anesthetics, opiod analgesics
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Sensory Pain
intensity, duration, location, frequency
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Cognitive/Affective Pain
unpleasant feelings
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Functional and Behavioral Pain
physical, social, emotional
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Problem Solving Approach
logic based technique that uses clinical exam finding, patient long term goals, and available evidence to decide a treatment plan
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Aspects of Treatment and Rehab Programming

knowledge of clinical exam skills, pathology, and physiology of treatment techniques, and knowing patient functionality, restrictions, and motivation

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Outcomes
success of surgery, therapeutic modalities, and/or exercise to solve Px restrictions
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Disease-oriented approach
treats pathology or injury directly
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Patient-oriented approach
focuses on meaningful recovery based on Px preference
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Clinician Based Measures
assesses impairments
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Patient Based Measures
self-evaluations of activity limitations and restrictions
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Outcome Scales should have values consistent with…
determining improvement in patient condition
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Minimum Detectable Change (MDC)
smallest clinically significant change in scores after two trials; determines efficacy
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Minimally Clinically Important Change (MDIC)
smallest change that is important/beneficial to the patient; measures responsiveness
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Evidence-based Practice
provide most efficient and effective treatment to maximize Px outcomes
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Factors to Consider while Choosing a Modality
can the modality directly or indirectly affect target tissues, produce physiological response required to promote healing, and does it improve outcomes better than placebo/no intervention
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PICO question
patient, intervention, comparison, outcomes
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You should always validate the use of modalities with…
research to back it up
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Step 1 of Problem Solving Approach
obtain Px medical history
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Step 2 of Problem Solving Approach
identify Px activity limitations and restrictions
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Step 3 of Problem Solving Approach
Prioritize Px problems
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Step 4 of Problem Solving Approach
Establish treatment goals
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Step 5 of Problem Solving Approach
Review evidence for treatment
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Step 6 of Problem Solving Approach
Plan therapeutic interventions and treatment
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Step 7
reexamine Px and assess outcomes
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How to use Medical Histories
review existing records (tests, operative reports, referrals) to identify contraindication
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Problem Recognition
conduct examinations and interviews with Px prior to each treatment to assess effectiveness of treatment and to adapt as needed
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Always consider the ____ relationship between Px complaints to best aid the Px
cause and effect
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Long Term Goals
identify and quantify final outcome/ restore participation
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Short Term Goals
goals for periods of 2 weeks or less at a time, projects progress to help achieve long term
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Treatment Planning
applying knowledge of physiology, modalities, and exercise to resolve problems and reach goals
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Final Modality Decisions should be based on…
scientific evidence, personal comfort, the modality itself, and patient input
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Patient Self Treatment
educate Px on injury, treatment, and benefits; no more than 3 exs; provide clear written and visual instructions
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Reexamination
track Px progress towards goals, develop new treatment, and readminister outcome measurements
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If patient does not reach short term goals…
create new ones
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If patient does not reach long term goals…
identify why they are struggling to reach it
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Influences on Px Care
treatment setting, experience of staff, equipment, insurance constraints, Px time for treatment, Px support system, Px motivation
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pain
unpleasant sensory and emotional experience associated with actual or potential tissue damage
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Nociception
neural process of encoding and processing noxious stimuli
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Battlefield conditions
athlete being so focused on competition that magnitude of the injury is not immediately processed
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pain threshold
level of noxious stim required to alert an individual to a potential threat to tissue
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pain tolerance
measures how much pain a person can withstand
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pain is processed by…
higher brain centers
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First Order Neurons
peripheral nerves
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Second Order Neurons
transmit stimuli up spinal cord to high brain centers
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Higher Centers of the Brain
cerebral cortex, amygdala, sensory homunculus
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Hyperalgesia
following injury, a normally non-painful stim easily activates nociceptors
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Primary Hyperalgesia
initial damage to the site
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Secondary Hyperalgesia
increased pain around injured tissue
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Primary Control Theory
resolving the physiological pain triggers; dec mechanical or chemical irritation
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Secondary Pain Control
targets transmission and perception of pain
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Melzak and Walls Gate Control Theory-Ascending Mechanism
inc non-nociceptive stim from peripheral to the spinal cord to elicit pain relief
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Melzak and Walls Gate Control Theory-Descending Mechanism
small diameter nociceptive path and stim are transmitting to higher centers; initiated by specific higher center structures
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Endogenous Opiates
hypothalamus controls release of beta-endorphins that inhibit pain perception
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Chronic Pain
pain that extends beyond normal length of injury or illness; pain in absence of a pathology
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Referred Pain
pain presenting in an area outside of the place of injury
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Peripheral Nerve Root Pain
peripheral nerve damage that destroyed all nerve fibers causing loss of function distal to the site