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pressure injuries aka
pressure ulcers
decubitus ulcers
bedsores
pressure injuries
injuries to skin and underlying tissue from prolonged pressure
location of pressure injuries
bony areas: heels, ankles, hips, coccyx
pressure injury timing
hours to days
patients at risk of pressure injuries
patients with conditions that limit ability to change position
chornic conditions that decrease perfusion, wound healing and increase inflammation
limit ability to change positions
surgery
fractures
trauma
burns
chronic conditions that decrease perfusion, wound healing and increase inflammation
diabetes mellitus
cardiac issues
obesity
imporance of prevention of pressure injuries
once occur some never heal completely
stage 1 pressure ulcer
non-balancing
erythema
with intact epidermis
stage 2 pressure injuries
partial thickness involving dermis and epidermis
stage 3
full thickness extending through dermis into subcutaneous tissue
stage 4 pressure injury
deep tissue destruction extending through fascia and amy involve muscle, bone and tendon
treatment of pressure injury
keep clean
analgesia
prophylactic antibiotics
assess signs and symptoms
deep wound care
wound packing
wound irritation
follow unit protocol and patient orders
wound packing for wounds
deep draining
purpose of packing wounds
promotes healing and prevents superifical closure with an enclosed abscess
wound irritation
normal saline and antibiotics
antibiotic use with wounds
decrease evidence for use due to resistance
how to measure exercise capability
VO2 max measured in L/min
VO2 max measures
circulaiton capacity to heart and blood
lung capacity
oxygen delivery to working muscles and is extracted by muscles
CN commond center and hypothalmus in acitivity
brainstem and SNS
adrenal gland hormone secretion
local vasodilatory factors stimulated in high-output organs
CO increase
vasodilation location
coronary circulation
muscle
brain
CO increase to
match demans
Skeletal muscle co at rest
20%
skeletal muscle co in exerise
95%
Co increase other mechanism
diverted from other organs
energy stored form
ATP
energy is used from
stored ATP
aerobic metabolism of pyruvate
other sources of energy
muscle glycogen
fatty acids
creatinine phosphate
CO2 excretition
exhaled and evaporated via diaphoresis
evaporation causes
cooling
high water losses cause
dehydration
extreme dehydration
fluid shifts and hypovolemic shock
heat
a product of chemical reactions = warm/flushed appearance
anaerobic metabolism
When demand exceeds supply (short bursts of high output: long time
by product of aerobic metabolism
carbon monoxide and water
by product of anaerobic metabolism
lactate
fatigue
activity intolerance due to exhausted reserves
causes of fatigue
physiologic
psychologic
pathologic
unknown
physiologic
inadequate ATP to generate muscle activity
physiological fatigue has depleted
glucose and electrolytes
psychologic fatigue
inadequate CNS ability to generate activity
deleted in psychologic fatigue
neurotransmitters, enhanced inhibitory neurotransmitters
pathologic fatigue
disease treatment alterations to normal function
disease that alter noraml function
heart failure
anemia
renal failure
cancer
unknown causes of fatigue
chronic fatigue syndrome
types of fatigue
acute and chronic
acute
sudden onset, clear cause, rest leads to recovery
chronic
unclear onset and causality, rest does not lead to recovery, accumulates and interferes with ADLs causes other pathologies
other pathologies caused by chronic fatigue
depression
anxiety
IBS
chronic fatigue syndrome aka
myalgia encephalomyelitis
systemic exertion intolerance disease
ME
myalgia encephalomyelitis
SEID
systemic exertion intolerance disease
etiology hypothesis chronic fatigue syndrome
an infectious disease postdrome caused by an immune hyperactivity of inflammatory mediators (cytokines)
infection
infections that chronic fatigue syndrome
enteroviruses, coronaviruses
diagnosis of chronic fatigue syndrome
signs and symptoms
signs and symptoms of chronic fatigue syndrome
chronic fatigue longer than 6 months with effect on ADLs
post exercise malaise
unrefreshing rest/sleep
congnitive and orthostatic effects
other symtpoms might be present on health assessment but are not diagnostic due to inaiblity to differentiate from cause and effect
examples of other symptoms with chronic fatigue syndrome
pharyngitis
lymphadenopaty
myalgia
splenomegaly
treatment of chronic fatigue injuries
CBT
team approach to management of associated pathologies
acute injuries force
sudden force
acute injuries
fractures
contusions
articulation injuries
articulation injuries
strains
sprains
dislocations
chronic cause
overuse
chronic injuries
stress fractures (no time to heal from acute injury)
strains and sprains with no time to heal adequately
musculoskeletal system percent of body mass
70%
msk includes
bone
cartillage
soft tissue
soft tissue in
articulations (joints) weakest link
ligaments
tendons
muscle
soft tissue in ligaments
bone to bone connection attack articulating ends together
soft tissue tendons
muscle to bone connection, join muscle to bone periostium
sprain
mechanical overload of a joint
mechanical overload of joint causes
ligement injury=stretch, tear complete or incomplete associated bone injury potentially
signs and symptoms of sprain
pain
inflammation
contusion
decreased function
common sprains
ankle inversion
knee ACL and MCL
elbow
wrist
things that tear
ACL
strain
mechanical overload of muscle or muscle tendon complex
mechanical overload of muscle or muscle tendon complex
excessive stretch or contraction = tearing of fascia, muscle, joint structures
high risk supports
signs and symptoms of strain
inflammation
pain
increased pain with aggravating activity
common strains
muscles lower back
c spine
joints elbow and shoulder
itis an inflammation of tendon usually chronic in nature
strain can tear
achilles tendon
bicep tendon
lateral epicondylitis
overuse of the forearm muscles, inflaming the lateral epicondyle muscle/tendon complex
treatment strain and sprain
compressess
compression of affected area
drugs
rest
rehabilitation
surgery
prevention
treatment of strain and sprain dependent on
degree of injury
compresses sprain/strain
cold, warm/cold
cold timing
less than 48 hours for 15-20 minutes, allow to return to body temp, decrease inflammation
warm timing
more than 48 hours for 15-20 minutes/time; increase healing decreases stiffness
compression of affected area
supports and decreases inflammation
immobilization (splint, cast)
drugs sprain/strain
antispasmodics and NSAID
rest timing
weeks
rehabilitation
return to function and strength exersizes
surgery
asap if at all
prevention of sprain/strain
posture
exercise mechanics
warm up and cool down
limit overuse
antispasmodics
centrally acting muscle relexants for muscle spasm or chornic pain
moa antispasmodics
decrease somatic nervous system activity CNS interruption/depression
outcome of antispasmodics
decreased muscle spasm, CNS effects (drowsiness)
antispasmodic drugs
cyclobenzoaprine
methocarbamol
methocarbamol combo drugs
robaxacet
robaxisal
robaxacet
methocarbamol and acetaminophen
robaxisal
methocarbamol and ASA
treatment if pain becomes chronic
underlying cause and treatment
chronic pain clinics
CNS drugs
decrease the excitatory neurotransmitters
enhance serotonin
antispasmodies
chronic pain clinics
counselling; CBT
physiotherapy