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Rumen optimal pH, substances that decrease it, and VFAs that predominate in both scenarios
6.2-7
Ethanoic acid (acetate)
pH decreases with starch and sugar
Propanoic acid (propionate) + butanoic acid (butyrate)
3 reasons for acute ruminal acidosis
EMERGENCY - poor/guarded prognosis
Sudden ingestion of large amounts of fermentable carbs (eg lots of grain)
Concentrate overfeeding
Sudden lack of forage/straw bedding (can be eaten)
4 treatments for acute rumenal acidosis
Slow IV of 5% NaOH (5L/450kg over 30 mins)
IV of isotonic (0.9%) NaCl (150mL/kg over 6-12h)
Oral Mg(OH)2 (500g/450kg)
Rumenotomy or stomach tube rumen lavage
Traumatic procedure, not very successful
Cause of subacute rumen acidosis
Diet over long to medium term:
Suboptimal fiber levels and/or
High carb (grain) levels
Signs of SARA (4)
Low milk fat/yields
Inappetance
Occasional laminitis
Repeated epistaxis or haemoptysis (coughing up blood)
Caudal vena cava syndrome from inflamed rumen → liver
Basically diagnostic of chronic SARA
How does SARA lead to caudal vena cava syndrome? (5)
Rumen wall damage → bacteria go into portal circulation to liver
Or direct transfer from cranial rumen to liver where they touch
Liver abscesses
Septic embolism passes to caudal vena cava → lungs
Damage to pulmonary artery (and smaller vasculature)
Describe the 3 stages of distension seen in rumen bloat (tympany).
Mild - left sublumbar fossa
More - whole left flank
Severe - distended whole abdomen
Why does rumen bloat sometimes lead to sudden death?
Rumen pushes against diaphragm + lungs → respiratory distress
Name 3 plants which can cause frothy bloat.
(soluble leaf proteins → foaming properties:)
Clovers (the worst one)
Legumes
Alfalfa
(Cereal rich diets or rich pastures)
Name 2 grazing methods to minimise risk of frothy bloat.
Buffer feeding - feed before turning out to decrease risk of lush pasture overfeeding
Restriction (eg with temporary fence or strip grazing) so less rich pasture can be grazed
Name 3 ways to treat frothy bloat.
Remove cow from feed source
Antifoaming agent or surfactant
Poloxalene (bloatguard)
Mineral or corn oil
BIRP (simethicone)
Sternal recumbency to encourage eructation
List 4 causes of free gas (gaseous) bloat.
Excessive carb intake → acidosis
Oesophageal obstruction
Eating quickly, esp when trough space is limited
Milk fever (hypocalcaemia)
Lateral recumbency (blocks cardiac sphincter)
List 4 methods of treating gaseous bloat.
Ca2+ injection - stimulates rumenal contractions and eructation
Sit the cow up to potentially unblock cardiac sphincter
Relieve gas
Stomach tube
Trocar and cannula on L paralumbar fossa
Rumenotomy
Especially useful if rumenal acidosis is the cause
List 4 potential causes of oesophageal obstruction (aside from the obvious one).
Lesions of oesophageal groove → damage to vagus (vagal indigestion syndrome)
Blockage of cardiac sphincter (Actinobacillus or papilloma)
Tetanus
(theoretically enlarged mediastinal lymph nodes)
List 3 potential signs of traumatic reticuloperitonitis.
Local/diffuse peritonitis
Pericarditis (if pericardium is penetrated) → heart failure
Palpate jugular pulse
Splashing sound over heart
Liver abscesses
List 3 tests potentially used to diagnose traumatic reticuloperitonitis.
Withers pinch → pain vocalisations
Pole test → metal pole pulled up below sternum → pain vocalisations
Eric williams test (difficult) → pain signs just before reticulorumen cycle
List 4 treatments for traumatic reticuloperitonitis.
Conservative
Tie animal up with front feet higher than back feet
Parenteral (non GIT) antibiotics
Pain relief
Rumenotomy
Magnet (bolus gun) into reticulum - only magnetic metals!
(slaughter)
Describe the mechanism of LDA.
Abomasum (normally on right) slides underneath rumen → trapped between rumen and left body wall
Which cows does LDA typically happen in?
High producing dairy cows within 6 weeks after calving
List 9 causes of LDA.
Excess VFAs from rumen to abomasum → makes it floppy and mobile
Poor management over transition period
Low → high energy diet change made too quickly
Insufficient fiber
Bloat
Milk fever
Retained foetal membranes
Rumen collapse
Pneumoperitoneum
Vagal indigestion syndrome
Genetic factors
LDA clinical signs (4)
Raised blood/urine ketones
(variable) drop in milk yield
(variable) change in appetite
(variable) decreased BCS and reduced rumen contractions
LDA treatment methods (4)
Conservative - roll slowly onto right lateral recumbency and then onto back
Roll + toggle - suture abomasum back into place
toggle = plastic on end of suture
R flank laparotomy with R omentoplexy/pyloroplexy (Utrecht method) → hard
Paramedian laparotomy + ventral abomasoplexy + omentoplexy (hard)
Describe the mechanism of RDA and how it can lead to severe disease.
Abomasum moves dorsally and then undergoes torsion or volvulus → trapped between liver and body wall
Ischaemic necrosis due to blood supply cutoff
List 6 potential pathologies that may present as pings.
Rumen bloat (L)
LDA (L)
RDA (R)
Abomasal torsion (R)
Caecal dilation and torsion (R)
Gas in spiral colon (R, no clinical significance)
Where does pinging happen during caecal dilation? What potential risk is there?
Caudal right paralumbar fossa
Volvulus in free caudal end of caecum → ischaemia
Treatment of caecal dilation
Medical fluids + high fiber diet
Surgery if persistent
Treatment of caecal volvulus
Surgical draining - cut into apex
List the steps of a right sided omentoplexy. (5)
Reach cranially across rumen with sterile hand
Pop L abomasum to let gas out
Grab abomasum from below rumen by hand
Put it back on R side
Stitch omentum to muscle layers in incision
‘Sow’s ear’ floppy flap of omentum preferred for suturing
Holds the abomasum into place
Aftercare for right sided omentoplexy (5)
Isolation for rest
Antibiotics - abdominal contamination
Fiber only diet for 5 days
Monitor
Pneumoperitoneum can present as a ping but is harmless
Pain relief (not for too long)
Not NSAIDs - irritates abomasum
In ketosis (acetonaemia), which 2 ketone bodies increase in the blood?
Beta hydroxybutyrate (3 hydroxybutanoate)
Acetoacetate (3 oxobutanoate)