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Anorexia physical problems:
__________
Constipation
Cold intolerance
Lanugo hair on body
Loss of body fat
Muscle atrophy
Hair loss
Dry skin
Dental caries
_______ edema
Bradycardia, arrhythmias
Orthostasis
Enlarged parotid glands and hypothermia
Electrolytes imbalance
Amenorrhea, pedal
Bulimia weight is usually
Normal
Anorexia and Bulimia Biologic factors
•Genetic vulnerability
•Disruptions in the nuclei of the _________ relating to hunger and satiety
•Neurochemical changes (norepinephrine, serotonin); not known if these changes cause disorders or are result of eating disorders
Hypothalamus
Anorexia and Bulimia Developmental factors:
No ________ , bc of enmeshment (lack of clear boundaries) with family
Cultural belief
Body image disturbance
Control
Anorexia Nervosa
•Onset usually between the ages of 14 and 18
•Denial early on; depression and _______ with progression; isolation; medical complications
•Treatment: often difficult; client is resistant, uninterested, denies problem
Lability
Can promote weight gain in clients with anorexia nervosa
Amitriptyline, Cyproheptadine
Antipsychotic effect on bizarre body image distortions and weight gain,
Problematic side effects
Olanzapine
Anorexia
Ssris may have effectiveness but need
Close monitoring for weight loss
Bulimia Nervosa
•Onset: late adolescence or early adulthood (average age of 18–19 years)
•Binge eating frequently begins during or after dieting
•Possible restrictive eating between binges
•Clients aware eating behavior is pathologic; go to great lengths to hide
Treatment
•_______ most effective
CBT
Bulimia nervosa
Psychopharmacology: antidepressants
Desipramine, imipramine, amitriptyline, nortriptyline, ________, __________
Phenelzine, fluoxetine
Difficulty identifying and expressing feelings
Alexithymia
Do not ask pt w anorexia if they are sad, instead ask them to
Describe feelings
Raise client awareness, regain sense of control, dairy of all food eaten
Bulimia self monitoring
Therapy most effective for anorexia
Family
Syndrome involving disturbance of consciousness with change in cognition
Usually develops over short period
Etiology: almost always results from identifiable physiological, metabolic, or
cerebral disturbance or disease or from drug intoxication or withdrawal
Delirium
Delirium meds
Sedation, antipsychotic
Delirium appearance and Motor behavior:
Restless, speech problems
Delirium mood and affect
Unpredictable mood shifts
Delirium thought process and content
Disorganized, fragmented
Initial sign of delirium is an
Altered loc
Delirium judgment and insight
Impaired
Delirium roles and relationships
Inability to fulfill
Delirium self-concept
Frightened
Delirium physiological considerations
Disturbed sleep wake cycles, fail to perceive internal body cues
Delirium
Managing client’s confusion:
______________ (calling by name, referring to time of day or expected activity); speaking in low, clear voice; use of touch;
avoiding sensory overload
Orienting cues
Deterioration of language function
Aphasia
Impaired ability to execute motor functions despite intact motor abilities
Apraxia
Inability to recognize or name objects despite intact sensory abilities
Agnosia
Repeating words or sounds over and over
Palilalia
Only dementia that has abrupt onset
Rapid changes w plateau
Vascularized dementia
Medications for degenerative dementias:
____________ like donezepil
cholinesterase inhibitors