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Onset Course Duration outcome and treatment of Autism Spectrum Disorder
Onset: symptoms often recognized at 12-24 months, average age of diagnosis 5.5 years
Course: chronic and/or worsening
Duration: life long
outcome: dependent upon level of impairment
Treatment: Intensive treatments can be extremely effective, uses ABA (applied behavioral analysis), needs to be for a lot of hours
Key Symptoms of ASD
A: deficits in social communication and social interaction manifested by deficits in ALL:
social emotional reciprocity
nonverbal communication
developing, maintaining, and understanding social relationships
B: Restricted, repetitive patterns of behavior interests, or activities manifested by two of the following:
Stereotyped or repetitive motor movements, use of objects, or speech
insistence on sameness, inflexible adherence to routines, or ritualized patterns of verbal and nonverbal behavior
highly restricted, fixated interests that are abnormal in intensity or focus
Hyper- or hypoactivity to sensory input or unusual interest in sensory aspects of the environment
C: symptoms must be present in early developmental period
D: symptoms cause clinically significant impairment in social, occupational, or other important areas of functioning
E: not better explained by Intellectual Disability or Global Developmental Delay
Onset, course, duration, outcome and treatment of Specific learning disability
onset: during school age
course: chronic
duration : learning problems often persist into adulthood, particularly if they are not targeted for treatment in early elementary school
outcome: If untreated, these deficits persist over time
treatments: Use different interventions depending on problem area, basic reading treatment, guided oral reading, for example
Key Symptoms of Specific Learning Disability
A: difficulties learning and using academic skills that has persisted for 6 months
B: affected academic skills are below those expected for the individuals chronological age, and cause significant interference with academic performance or daily living confirmed by individually administered standardized achievement measures and clinical assessment
C: begins during school-age,
D: Learning difficulties are not better accounted for by intellectual disability or other problems
Problems with reading, writing, or math
Onset, course, duration, outcome, and treatments of ADHD
onset: must be before age 12, often see symptoms earlier
Course: Chronic
Duration: symptoms decrease, particularly hyperactive-impulsive, but impairment does still stay
outcome
Treatment:
Behavioral treatments: Parent training, classroom management, social skills training
uses ABC model to change behavior based on operant/social learning theory principles
Psychostimulants/nonstimulant medication
cognitive training
Combination of behavior and medication recommended for school-aged children
Key Symptoms of ADHD
developmentally inappropriate levels
duration of 6 months
cross-setting Occurrence of Symptoms
Impairment in major life activities
onset of symptoms/impairment by 12
Inattention symptoms (need 6 of 9 in kids)
fails to give close attention to details
difficulty sustaining attention
does not seem to listen when spoken to
does not follow through on instructions
difficulty organizing tasks or activities
avoids tasks requiring sustained mental effort
loses things necessary for tasks
easily distracted
forgetful in daily activities
Hyperactive-Impulsive (6 of 9)
Fidgets with hand or feet or squirms in seat
leaves seat in inappropriately
runs about or climbs excessively
has difficulty playing quietly
is “on the go” or “driven by a motor”
talks excessively
blurts out answers before questions are completed
has difficulty awaiting turn
interrupts or intrudes on others
Onset, course, treatments of ODD
onset: first symptoms can appear during preschool, but can see emergence through adolescence, if it’s a childhood-onset it’s more likely to turn into conduct disorder
Course: worsening symptoms with improvement after adolescence, unless conduct disorder develops
Treatment:
parent management training
problem solving skills
Multisystemic therapy (family, school environment, friendships, neighborhood)
Key symptoms of ODD
A pattern of angry/irritable mood, argumentative/defiant behavior, or vindictiveness lasting at least 6 months, needs at least 4 exhibited with an individual who is not a sibling
Angry/irritable mood
Argumentative/Defiant behavior
vindictiveness
Behavior must be developmentally inappropriate
children under 5 - behavior should occur on most days for 6 months
children over 5 - behavior occurs at least once a week for 6 months
Distress or functional impairment
Do no occur exclusively during the course of a psychotic, substance use, depressive or bipolar disorder
onset, course, outcome, and treatments of CD
onset: can be in childhood or adolescence
Course: depends on onset
Adolescent: might “grow out of it”
Childhood w/out CU/LPE: worsens over time
Childhood w/ CU/LPE: might later be diagnosed with APD, poor response to treatment
outcome: depends on onset
Adolescent: better response to treatment
Childhood w/out CU/LPE: moderate response to treatment
Childhood w/ CU/LPE: might later be diagnosed with APD, poor response to treatment
Treatment:
parent management training
problem solving skills
Multisystemic therapy (family, school environment, friendships, neighborhood)
Key symptoms of CD
patterns of behavior where the basic rights of others or major age-appropriate societal norms or rules are violated
needs 3 or more of the following criteria in the past 12 months, at least 1 criterion present in the past 6 months
Aggression to people and animals
Destruction of property
Deceitfulness or Theft
Serious violation of rules
specific types: Childhood-onset type, adolescent-onset type, unspecified onset (not enough info on when first symptom began)
Subtype: limited prosocial emotions/Callous unemotional traits
3 broad areas where children can show impairments in specific learning disability
Reading: word reading accuracy, reading rate of fluency, reading comprehension
Writing: Spelling accuracy, grammar and punctuation accuracy, clarity or organization of written expression
Mathematics: number sense, memorization of arithmetic facts, accurate fluency calculation, accurate math reasoning
ASD symptoms and impairments
Social communication/interaction impairments
lack of social-emotional reciprocity, deficits in social relationships, deficits in nonverbal communication
Restrictive, repetitive patterns of behavior
impairments in social, occupational or other important areas of current functioning
Self- Injurious behaviors, definition, theories for cause, and treatments
Repetitive movements of the hans, limbs, or head in a manner that can, or does, cause physical harm or damage to the person
communication
endogenous opioid release
Reinforcement/ extinction
differential reinforcement of other behavior (DRO)
Has to be consistent
Is environment specific
Noncontingent reinforcement (NCR)
Can’t stop reinforcing self-injuries altogether because they’ll intensify to try and get a (extinction burst). Have to reward not doing the self-injurious behavior (DRO)
Best and worst outcomes for ASD
Dependent on level of impairment
Better prognosis with higher IQ (>70), better verbal skills, and early intervention
Less severe autism symptoms, improved educational outcomes, increased likelihood of functioning within normal limits in later in life
Poor prognosis for IQ<50 or no communicative language by age 5-6
2 hypothesis (with evidence) for increase in ASD diagnoses
Increased awareness (parents and physicians)
ID turns into ASD diagnosis
When to use harsh punishments
there is an immediate physical danger to the individual or to others
the scientific literature suggests the restrictive procedure would be effective
less restrictive intervention would be ineffective or harmful
the restrictive treatment is discontinued when its benefits can be maintained through a less restrictive means
the treatment is reviewed and approved by a peer and human rights committee, and the individual’s guardian provides consent
ADHD diagnostic criteria and presentations
two symptom types, Hyperactivity-Impulsivity and Inattention, 3 presentations, Hyperactive-impulsive subtype, combined subtype, inattentive subtype
developmentally inappropriate levels
duration of 6 months
cross-setting Occurrence of Symptoms
Impairment in major life activities
onset of symptoms/impairment by 12
Inattention symptoms (need 6 of 9 in kids)
fails to give close attention to details
difficulty sustaining attention
does not seem to listen when spoken to
does not follow through on instructions
difficulty organizing tasks or activities
avoids tasks requiring sustained mental effort
loses things necessary for tasks
easily distracted
forgetful in daily activities
Hyperactive-Impulsive (6 of 9)
Fidgets with hand or feet or squirms in seat
leaves seat in inappropriately
runs about or climbs excessively
has difficulty playing quietly
is “on the go” or “driven by a motor”
talks excessively
blurts out answers before questions are completed
has difficulty awaiting turn
interrupts or intrudes on others
validity concerns of ADHD presentations
Are presentation distinctions warranted?
largely do not differ on core neuropsychological and neurobiological variables and share common genetic risk factors
Is hyperactive/impulsive subtype real?
Is the diagnostic paradigm developmentally appropriate?
Is there a separate disorder for some of the children diagnosed with ADHD inattentive type?
Gender differences real?
Sluggish cognitive tempo/ cognitive disengagement syndrome
Symptoms:
daydreaming/spacey/stares
slow information processing
hypoactive/lethargic/sluggish
Easily confused, mentally “foggy”
poor focused/ selective attention
erratic retrieval - Long-term memory
socially reticent/uninvolved
Rarely comorbid of ODD/CD
less likely to respond to psychostimulants
more likely to evince perceptual or motor control deficits
Possibly greater family history of anxiety disorders and LD (?)
Executive functions and working memory
executive functions: a person’s capacity to effectively perceive, process, and use information in order to sole problems and attain long-term goals (RE-WATCH VIDEO)
planning, organization, prioritizing, initiating tasks
Self-monitoring, self-inhibition
shifting attention
working memory: a cognitive system with a limited capacity that can hold information temporarily
how do we know that executive functions are implicated in ADHD
Children with ADHD exhibit an under-aroused prefrontal cortex
AND a structurally underdeveloped brain
is movement functional in ADHD
yes movement is functional for children with ADHD
Study shows that children with ADHD perform better on a working memory task when they move more
Children without ADHD performed worse the more they moved
Behavioral treatments: how they are theorized to work, evidence
Based on operant/social learning theory principles (uses ABC model to change behavior)
Parent training: parents are taught skills to help with poor attention, organization, compliance, independence, emotional regulation. Focus on positive strategies. Skills include: praise, rewards, using effective instructions, scaffolding peer interactions, prompting for emotional regulation skills, selective attention/planned ignoring, removing privileges, contingency management programs
Also uses classroom training and child skills training
behavioral alone has moderate benefits
best for impairments like social skills, organizational functioning, emotion regulation, compliance
stimulant medications: how they are theorized to work, evidence
Psychostimulants such as methylphenidate (MPH)
dopamine and norepinephrine reuptake inhibitors
promote availability of dopamine in PFC and other areas
most effective when combined with behavioral interventions
cognitive training: how they are theorized to work, evidence
based on concept of neuroplasticity: repeated practice or use can “rewire” the brain
mainly commercially available programs
all effects were duet to placebo effect, with significant variability among studies
ABC model
Antecedent
Behavior
Consequence
Uses this in therapy to set up child with ADHD for success
Coercive parenting cycle and patterson’s model
coercion: persuading someone to do something by using force or threats
parent derides, demeans, or diminishes children and teens by continually putting them in their place, putting them down, mocking them, or holding power over them va punitive or psychologically controlling means
results in coercive parent-child cycle: negative reinforcement teaches parent and child ways to act
Think about the cycle image in slides
Know how the etiology, symptom presentation, and response treatment for Adolescent-onset CD
etiology: exaggeration of normal autonomy-seeking processes of adolescence
relative to TD teens, more rebelliousness and rejection of conventional values
influence by peer groups, poor parental supervision, not associated with hostile parenting
Symptom presentation: less aggression, less criminality/antisocial behavior in adulthood, fewer dispositional risk factors (CU, emotional dysregulation, impulsivity)
Response to treatment : good
Know how the etiology, symptom presentation, and response treatment for child-onset CD without CU
Etiology: hostile, coercive parenting
emotional dysregulation
diathesis (fearlessness, impulsivity) Stress (poor parental supervision, low parental warmth, coercive parenting)
symptom presentation and course: symptoms appear early in childhood, worsen
antisocial and criminal behavior in adulthood
increased aggression (reactive/impulsive rather than provocative)
more mood symptoms, emotional dysregulation, emotional reactivity
high anxiety, distressed by own behavior, appropriate empathic reaction
response to treatment: moderate
Know how the etiology, symptom presentation, and response treatment for Child-onset CD With CU
Etiology: highly heritable (genes for serotonin and oxytocin systems implicated)
warm parenting style and appropriate parental involvement may be protective, but studies are inconsistent
Symptom presentation and course
lack of concern about own behavior, poor empathic skills
constrained display of emotions; emotional hyporeactivity
low effort, more severe conduct problems, more aggressive, stable sx
highest rates of APD in adulthood
Response to treatment: poor, some say worsening symptoms
less susceptible to punishment techniques due to low emotional response to sanctions
Callous unemotional traits/limited prosocial emotions; implications for etiology and treatment
they predict: violent sexual offenses
adult measures of psychopathy at ages 18-19, even after controlling for early CD problems and other risk factors
more severe CD problems, violence, aggression, & delinquency: a more seer and stable pattern of antisocial behavior
CU traits are usually stable into early adulthood
Thoughts to be a precursor to psychopathy traits in adulthood
ADHD vs SLD
SLD: associated psychological correlates: setting - SLD not impaired outside of academic setting
ADHD vs ASD
constellation of symptoms
Overlap in inattention and social dysfunction, but different quality
ADHD vs ODD
Forgetfulness vs defiance
ADHD Dx- only is refusal is solely in situations that demand sustained effort and attention or demand that the individual sit still
ODD vs CD
can meet criteria for BOTH ODD and CD
aggression, antisocial behavior, violating others’ rights = CD
Emotional dysregulation in ODD, not CD
treatments for ODD/CD (at least 3)
Parent management training
uses techniques like using token economies, practicing time out, and a daily school behavior report card
Problem solving skills training
skills like feeling recognition and anger management
Multisystemic therapy (MST)
targets the many “systems” that impact youth: Family, school environment, friendships, neighborhood
Works closely with the parents and child for 3-5 months in their home and community
All treatments dependent on severity of problems