NURS 225 exam 3

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Last updated 5:21 PM on 3/29/26
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101 Terms

1
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What is an aneurysm?

An abnormal bulging of an artery due to weakness in the vessel wall.

2
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What is a true aneurysm?

A bulging of an artery involving all three layers of the vessel wall.

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What is a pseudo-aneurysm?

A tear in the artery where blood collects between the middle and outer layers.

4
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What causes an abdominal aortic aneurysm (AAA)?

Hypertension, atherosclerosis, smoking, and connective tissue defects.

5
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Why is hypertension a risk factor for AAA?

It increases pressure on arterial walls, weakening them.

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Why does atherosclerosis contribute to AAA?

Plaque buildup weakens the vessel wall.

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What is a common early sign of AAA?

Often asymptomatic.

8
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What is a classic physical finding in AAA?

Pulsatile abdominal mass.

9
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What is distal embolization in AAA?

Clots break off and travel downstream.

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What happens as an AAA enlarges?

Abdominal pain that radiates to the back.

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What is the most dangerous complication of AAA?

Rupture causing rapid, life-threatening hemorrhage.

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What tests diagnose AAA?

Ultrasound and CT angiogram.

13
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How are small aneurysms managed?

Control blood pressure and monitor.

14
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When is surgery indicated for AAA?

When >5-6 cm (unless not a surgical candidate).

15
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What is endovascular repair?

A minimally invasive repair with a risk of leakage.

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What is open repair?

A more invasive surgery with higher mortality risk.

17
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What is the pathophysiology of aortic dissection?

Tear in the intima with blood collecting between intima and media.

18
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What are risk factors for aortic dissection?

Hypertension, Marfan syndrome, elderly age.

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What is the classic symptom of aortic dissection?

Tearing chest pain radiating to the back.

20
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What abnormal vital sign may be seen in an aortic dissection?

Unequal blood pressures.

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What does a chest X-ray show in aortic dissection?

CXR will show widened mediastinum

22
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What confirms aortic dissection diagnosis?

CT scan

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What is the priority treatment for Aortic Dissection?

Rapid blood pressure reduction.

24
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What medications are used for Aortic Dissection?

IV beta blockers (esmolol) and nitroprusside.

25
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What is the definitive treatment for aortic dissection?

Surgical repair

26
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What type of monitoring is required for aortic dissection?

Critical care monitoring.

27
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What is the nurse's role with medications for aortic dissection?

Titrate to control BP.

28
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What other nursing priorities exist for aortic dissection?

Pain management, ongoing assessment, psychosocial support, prep for surgery.

29
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What causes PAD?

Atherosclerosis of peripheral arteries.

30
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What is the main problem in PAD?

Impaired distal perfusion.

31
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What are risk factors for PAD?

Hypertension, smoking, diabetes, hyperlipidemia.

32
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What is intermittent claudication for PAD?

Pain with walking that is relieved by rest.

33
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What neurological symptom may occur for PAD?

Neuropathy.

34
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What happens to pulses in PAD?

They are diminished or absent.

35
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What are trophic changes in PAD?

Skin becomes pale, shiny, hairless, with thick nails.

36
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What severe complications can occur in PAD?

Arterial ulcers and gangrene.

37
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What type of pain occurs in PAD?

Burning or gnawing pain.

38
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What happens to pain when legs are elevated in PAD?

Pain increases (due to ischemia).

39
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What happens to leg color with elevation in PAD?

Pallor (pale).

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What happens when legs are lowered in PAD?

Rubor (redness).

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What happens to capillary refill in PAD?

Decreased.

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What is the temperature of affected limbs in PAD?

Cool.

43
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How are pulses assessed in PAD?

Presence, strength, rate, rhythm (may need Doppler).

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What skin findings are common in PAD?

Ulcers, shiny skin, thick nails, hair loss.

45
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What tests diagnose PAD?

Arterial duplex, ankle-brachial index (ABI), angiography.

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What is the first step in treatment in PAD?

Risk factor modification

47
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What lifestyle intervention is important in PAD?

Exercise

48
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What medications are used in PAD?

Antiplatelets and lipid-lowering drugs.

49
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What procedures may be needed in PAD?

Angioplasty with stent, bypass surgery, amputation.

50
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What is the most important lifestyle change for PAD?

Smoking cessation

51
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What daily habit is important for PAD?

Inspect feet daily

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Why protect feet for PAD?

Prevent injury due to poor circulation.

53
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Who should patients see for PAD?

Who should patients see?

54
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What type of system is the venous system?

Low pressure, high volume.

55
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What helps venous blood flow?

Valves and muscle activity.

56
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What are the three components of Virchow's Triad?

Venous stasis, endothelial damage, hypercoagulability.

57
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What is DVT?

deep clot in the vein

58
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Why is DVT dangerous?

High risk of embolization (can cause PE).

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What percentage of surgical patients are affected for DVT?

About 5%.

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What are the main symptoms of DVT?

Edema, erythema, pain.

61
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How can DVT be prevented?

Mobilization, ankle pumps, compression devices (SCDs), TEDS, anticoagulation.

62
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How is DVT diagnosed?

Venous Doppler.

63
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How is DVT treated?

Anticoagulation.

64
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What is an IVC filter?

A device to prevent clots from reaching the lungs.

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What must nurses monitor for?

Signs of pulmonary embolism (PE).

66
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What causes venous insufficiency?

Incompetent valves causing backflow and edema.

67
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What are key assessment findings for venous insufficiency?

Edema and brown skin discoloration.

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How is venous insufficiency treated?

Compression, leg elevation, low sodium diet, skin care.

69
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What is the normal conduction pathway of the heart?

SA node → AV node → Bundle of His → Right & Left bundle branches → Purkinje fibers

70
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What is the function of the SA node?

Acts as the natural pacemaker and initiates electrical impulses

71
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What is the function of the AV node?

Delays conduction to allow ventricles to fill before contraction

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What do Purkinje fibers do?

Conduct impulses to ventricles causing contraction

73
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What is the difference between electrical and mechanical events in the heart?

Electrical = conduction of impulses; Mechanical = actual contraction/pumping

74
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Can electrical activity occur without effective mechanical contraction?

Yes (example: ventricular fibrillation)

75
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What are structural causes of arrhythmias?

Accessory pathways, conduction defects, ischemia/infarction, hypertrophy

76
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What are systemic causes of arrhythmias?

Acid-base imbalance, electrolyte imbalance, hypoxia, thyroid disease

77
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What external factors can cause arrhythmias?

Drug toxicity (including antiarrhythmics), alcohol

78
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What is the most important principle when evaluating an arrhythmia?

Treat the patient, not just the monitor

79
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What should you assess to evaluate perfusion?

LOC, vital signs, pulse ox, skin, respiratory status

80
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Why is identifying the underlying cause important for an arrhythmia?

Because treatment depends on correcting the cause

81
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What are the main treatments for arrhythmias?

Correct cause, drugs, electricity, ablation

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What is a key principle about antiarrhythmic drugs?

they can cause or worsen arrhythmias

83
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What is atrial fibrillation?

Chaotic atrial electrical activity with no organized contraction

84
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What is lost in atrial fibrillation?

Atrial kick

85
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Why is atrial fibrillation dangerous?

Risk of clot formation → stroke

86
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What conditions can cause atrial fibrillation?

CAD, ischemia, valve disease, COPD, atrial damage

87
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How is atrial fibrillation treated?

Rate control, anticoagulation, antiarrhythmics, cardioversion, ablation

88
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What is a heart block?

Delay or interruption of conduction through the AV node

89
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What happens in 2nd degree heart block?

Some P waves do not conduct to ventricles

90
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What happens in 3rd degree heart block?

No association between atrial and ventricular conduction

91
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What is the treatment for severe heart blocks with low HR?

Pacemaker

92
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What characterizes ventricular tachycardia?

Rapid rate with wide QRS

93
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Can V-tach have a pulse?

Yes or no

94
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What can V-tach deteriorate into?

Ventricular fibrillation

95
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Causes of V-tach?

MI, CAD, electrolyte imbalance, prolonged QT, antiarrhythmics

96
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Treatment of stable V-tach?

Medications

97
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Treatment of unstable V-tach with pulse?

Cardioversion

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Treatment of pulseless V-tach?

CPR and defibrillation

99
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What is ventricular fibrillation?

No ventricular contractions and no cardiac output

100
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How quickly do brain cells begin to die in V-fib?

4-6 minutes

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