SLHS 408 Neurogenic Swallowing Disorders

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Last updated 8:25 PM on 3/18/26
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29 Terms

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neurogenic swallowing disorders

occur when there is damage to the central or peripheral nervous system

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isolation

some swallowing disorders occur in _____ while others are a symptom of a larger disease process

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dysarthria

swallowing disorders are often classified within _____ categories: speech disorders attributed to a weakness, slowness, or incoordination

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  • cognitive

  • oral

  • pharyngeal/laryngeal

  • esophageal

multiple systems are involved such as:

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  1. flaccid

  2. spastic

  3. ataxic

  4. hypokinetic

  5. hyperkinetic

what are the 5 major types of impairment

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lower motor neuron damage

what is the etiology of flaccid dysarthria

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  • weakness (could also be paralysis)

  • hypotonia

  • diminished reflexes

  • fatigue

what are the symptoms of flaccid dysarthria

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  • brainstem CVA

  • muscular dystrophy

  • ALS

  • Myasthenia Gravis

  • Guillain-Barre

what diseases may cause flaccid dysarthria

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will depend on site of damage, but generally, weak muscle response can mean:

  • poor chewing, movement of bolus (oral residue)

  • weakness of velopharynx (nasal regurgitation)

  • poor closure of larynx (aspiration)

  • poor propulsion (squeezing) of the bolus downward (pharyngeal residue)

how does flaccid dysarthria impact the swallow

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upper motor neuron damage

what is the etiology of spastic dysarthria

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  • weakness

  • loss of fine, skilled movements

  • increased tone

  • increased reflexes

  • spasticity

what are the symptoms of spastic dysarthria

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  • CVA

  • anoxic brain injury (ex. cerebral palsy)

what diseases may cause spastic dysarthria

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will depend on site of damage, but generally the hypertonicity, weakness, and spasticity will cause:

  • poor chewing, movement of bolus (oral residue)

  • poor timing and coordination of specific movements that initiate swallow

  • poor closure of larynx (aspiration)

  • weakness/improper movement of velopharynx (nasal regurgitation)

  • poor propulsion (squeezing) of the bolus downward and now may have spasms in the esophagus

how does spastic dysarthria impact the swallow

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cerebellar damage

what is the etiology of ataxic dysarthria

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  • slowness and inaccuracy of movement

  • difficulty with coordination of movement

  • tremor

what are the symptoms of ataxic dysarthria

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  • degenerative diseases

  • CVA

  • tumors

what diseases may cause ataxic dysarthria

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  • biting tongue/cheek when eating

  • poor respiratory coordination during swallowing (can lead to aspiration)

how does ataxic dysarthria impact the swallow

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basal ganglia dysfunction

what is the etiology of hypokinetic dysarthria

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  • slowness of movements

  • diminished movements

  • sensory impairments

  • rigidity

  • tremors

what are the symptoms of hypokinetic dysarthria

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Parkinson’s Disease

what diseases may cause hypokinetic dysarthria

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  • drooling

  • tremor of oral musculature impacts oral prep and transit phase

  • decreased frequency of swallow (aspiration)

  • decreased movement of structures that close the larynx and protect the airway (aspiration)

how does hypokinetic dysarthria impact the swallow

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basal ganglia dysfunction

what is the etiology of hyperkinetic dysarthria

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  • abnormal, rhythmic movements

  • irregular and unpredictable movements

  • slow, involuntary movements

what are the symptoms of hyperkinetic dysarthria

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Huntington’s Disease

what diseases may cause hyperkinetic dysarthria

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  • motor control across all phases of the swallow are affected—any number of impairments can be seen

how does hyperkinetic dysarthria impact the swallow

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mixed impairments

any combination of two or more of the single impairment types is possible; neurologic disease is distributed across two or more dimensions of the nervous system; one type may predominate

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combined neurologic events

  • multiple CVAs

  • PD + CVA

  • advancement of degenerative neurologic disease (ALS, MS)

what causes a mixed impairment

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Amyotrophic Lateral Sclerosis (ALS or Lou Gehrig’s disease)

  • half of the motor neurons have already died before symptoms even appear

  • etiology (unknown)

    • 5-10% have familial type

    • protein aggregation

      • TDP-43

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  • LMN: fasciculations, muscle atrophy, weakness

  • UMN: spasticity, hyperreflexia, emotional lability

what are the signs of ALS

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