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what are the avenues of dry heat exchange?
convenction , radiation, conduction , evaporation
convection
heat transfer by movement of air or water (through physical touch)
radiation
infrared heat emitted to enviroment / infrared rays
conduction
heat transfer by movement of air or water
wet heat exchange
evaporation
evaporation
sweat changing from liquid to vapor
primary mechanism of heat loss during exercise:
evaporation
critical temperature theory
states the brain shuts down around 40-41 degrees C
what is the body’s thermostat
prepptic : anterior hypothalamus (POAH)
3 WAYS TO INCREASE HEAT PRODUCTION IN RESPONSE TO COLD
shivering , non shivering , exercise
involuntary ways to increase heat production in response to cold
shivering
non- shivering
voluntary ways to increase heat production in response to cold
exercise ( muscular contraction)
what is the wet bulb globe thermometer
enviormental heat stress index
dry temperature
wet bulb temperature (humidity)
globe temperature ( radiation)
how helpful is it for athletic events
when is it safe to practice
hydration breaks
when to cancel events
prevent heat illness
how will dehydration affect the components of q
cardiovascular drift occurs
skin blood flow
core temperature
performance
heat & illnesses symptoms
heat cramps
heatstroke (medical emergency)
heat exhaustion
heat cramps
sodium loss + dehydration
painful spasms ( calves, abdomen)
heat exhaustion
severe dehydration
weak rapid pulse
dizziness
nausea
fainting
low plasma volume
heatstroke (medical emergency)
core temperature >40 c
hot , dry skin
confusion / altered mental state
possible unconsciousness
immediate cold-water immerssion needed
why is it more difficult to deliver o2 when we are at altitude
less oxygen loaded into blood
hypoxia in tissues
lower Vo2 max
dehydration risk increase , why ?
dry air increases respiratory water loss
cold induced diuresus
increased ventilation
blunted thirst
how to counteract dehydration
drink more water than thirst signals
warm fluids
electrolytes
avoid acohol / caffeine early on
caloric increases , why ?
higher metabolic rate
increased carbohydrate rate
shivering
harder breathing
how to counteract caloric increase
increase calorie intake
high-carb diet
snacks between meals
iron-rich food ( for rbc production)
altitude sickness
acute mountain sickness
HAPE
HACE
actue mountain sickness
headache
nausea
fatigue
dizziness
shortness of breath
timeline: begins 6-48hrs after arrival : last 2-4 days
HAPE ( pulmonary edma)
fluid in lungs
extreme shortness of breath
frothy sputum
fatigue
life- threatening
timeline: 2-4 days
HACE ( cerebral edma)
severe headache
confusion
loss of coordination
possible coma
life - threatening
timeline : 2-5 days
Acute effects on VO2 max, HR, Q, Ventilation, Hematocrit
vo2 max decrease (8-11% per 1000m above 1500m)
HR increases
Q intially increases (due to HR rise)
stroke volume decrease (plasma volume drops)
ventilation increases immediately (hyperventilation)
hematocrit increases (due to plasma volume loss , no RBC rise yet
Long term effects on hematocrit, why does it happen ?
chronic hypoxia → kidneys release epo→ increase RBC production
higher hematocrit
higher hemoglobin
more o2 - carry capacity
takes two weeks to fully occur
what is blood doping ?
artifically increasing RBC count by blood transfussion
advantages of blood doping
increase hemoglobin
increase o2 carrying capacity
vo2 max increase
delayed fatigue
Live High , Train low benefits to competition at sea level
live at high altitude → stimulate EPO→ more RBCs
train at low altitude → maintaining high- intensity workouts
benefits of live high, train low
increase vo2 max
improved endurance at sea level
enhance RBC mass
better performance 3-4 weeks after returing
coldness illness
hypothermia
frostbite
hypothermia
<35 c core temp
mild : shivering / numbness
moderate: confusion
severe: no shivering , no HR
frostbite
skin tissue freezing
pale/ white skin
numbness
most common: fingers, toes , ears , nose